1533 Final Flashcards
Delirium Assessment
Confusion Assessment Model tool
CAM
Can be done without an order
Delirium can lead to
Changes in level of consciousness
Irreversible brain damage
Death
Delirium symptoms
Stupor - hypoactive Excessive activity - hyperactive Disorganized thinking Short attention Hallucinations, delusion, fear, anxiety, paranoia
Dementia symptoms are -
Where as delirium symptoms are -
Long term onset
Acute onset
Because of unknown underlying causes, delirium is considered a
Medical emergency
Delirium prevention
Reorienting Early mobilization Pain control Good sleep Enhance communication methods (hearing aids, glasses) O2 levels Hydration
Delirium treatment
Fall prevention
Discontinue none essential meds
Monitor nutrition and fluids
Familiar environment ques like fam interaction
Haldol does NOT work for
Elderly with dementia
Has opposite effect
Dementia is -
Most common dementia is -
General decline in higher brain function
AD
3 Stages of AD
Preclinical - Minor forgetfulness, small difficulties
Mild Cognitive - Noticeable mild changes that can be measured, limitations in independent living start
Severe - Significant impairment of thinking, behavior and functioning independently
Medications for Alzheimer’s
Donepezil
Rivastigmine
Galantamine
(Anticholinesterase inhibitors)
Memantine
(Preserves memory)
Risk factors for Dementia/delirium
Aging Low education Down syndrome Family history Smoking x2 to x4 fold increase Obesity Insulin resistance Dyslipidemia - high level of lipids
Dyslipidemia
High level of lipids
Med good for hyperactive delirium
Haldol
Assessment for Alzheimers
Mini COG
Can be done without orders
Assessment for cognitive performance of the elderly
Mini Mental State Examination
MMSE
In elderly, cognition may be effected by
Sensory impairment Physiologic health Environment Sleep Psychosocial influence
Delirium
Acute confused state
Alzheimer Disease
AD
6th leading cause of death
Progressive degenerative neurologic disease
AD is most common in people over
65y/o
AD Patho
Neurofibrillary tangles - nonfunctional neurons
Neurotic plaque - deposits of amyloid protein in the brain
Reality orientation with AD
Who and where a person is in a time continuum, this will only WORSER mental/emotional state of AD patient due to increased anxiety
Nursing roles in AD
Supporting cognitive function
Calm predictable routine Simple explanations Memory aids Physical and verbal stimulation Clock/calendar display
Therapies for Phobias
Cognitive Behavioral Therapy CBT
Group Therapy
Guided Mastery Therapy
Meds for phobias
Antidepressants SSRI
Paroxetine, fluoxetine
Tricyclic antidepressants imipramine
Monoamine oxidase inhibitors MOAIs
Benzodiazepines
Alprazolam
Clonazepam
SSRIs can increased suicidal thinking in patients age-
18-24
Benzos can lead to
Dependence
Anorexia Nursing interventions
Establish trust and expression Periodic weight Encourage small frequent meals PN/TPN Assess bowels Supervise patient during meal and 1 hour after
When anorexic patients eat they can develop refeeding syndrome
S/S are
Hypophosphatemia, hypokalemia, hypomagnesemia, fluid overload, edema.
Assess anorexic patients
electrolytes for refeeding syndrome
for suicidal ideations
for good sleep and energy
Weight
I&O
Selye’s General Adaptation Syndrome
GAS ARE
Alarm stage
Resistance stage
Exhaustion stage
Fetal alcohol spectrum disorder characteristics
Small head Short eye openings Flat midface (cheeks) Smooth philtrum (space under nose) Underdeveloped jaw Low nasal bridge Epicanthal folds Short nose Thin upper lip
Fetal alcohol spectrum disorder characteristics
Small head Short eye openings Flat midface (cheeks) Smooth philtrum (space under nose) Underdeveloped jaw Low nasal bridge Epicanthal folds Short nose Thin upper lip
Neonatal abstinence syndrome
NAS
occurs when mother takes
Amphetamines Barbituates Benzos Cocaine Marijuana Opiates
NAS treatment is for drug dependence
Methadone
Antabuse
Disulfiram
Creates negative reaction to alcohol
NAS baby clinical manifestations
Fussy/hard to calm Excessive sucking BUT poor feeding High pitched cry D/V Trembling or jittery
Full withdrawal symptoms - muscle tone increase, seizures, breathing problems
Peripheral neuropathy
Alcohol myopathy
GI-itis
Complications of alcoholism
Wernicke’s encephalopathy
Korsakoff’s psychosis
Alcohol cardiomyopathy
Complications of alcoholism
Cirrhosis
Blood dyscrasia
Fetal alcohol syndrome
Complications of alcoholism
Legal intoxication level
Alcohol level over 0.08
Alcohol dependence is 4 times more likely in patients with
Mental illness
SIADH
Syndrome of inappropriate ADH (antidiuretic hormone)
TOO MUCH ADH
RETAIN WATER
SIADH lab values
Hypoosmolality
Urine concentration greater than 100
Natriuresis - NA in urine greater than 30
SIADH Symptoms
Fluid in lungs
Early - Cramps, N/V
Late - Confusion, seizers, coma
Hallmark of SIADH
Hyponatremia blood serum
Hypernatremia in urine
Hypernatremia symptoms
FRIED
Fever Restlessness Increased fluids Edema Decreased urine, dry mouth
Hypernatremia can be treated with diuretics but diuretics an lead to
Hypokalemia
Potassium wasting diuretics
Furosemide
Hydrochlorothiazide
Potassium sparing diuretics
Spironolactone
is dehydration FVD
NO, dehydration is loss of water alone
INCREASE in electrolytes
in FVD unlike dehydration
Fluids AND electrolytes are lost and need replacement
Dehydration symptoms
Dry mouth Poor skin turgor Low BP High Na High HR Weight loss
Urine specific gravity
1.005-1.030
Creatinine and BUN normal
AND with dehydration
Creatinine 0.6-1.2 will be over 1.2
BUN 10-20 will be over 20
DI causes
Head injury
Pituitary tumor
Craniotomy
DI MOA
Pituitary gland shuts off ADH supply to kidneys
In DI, patients bodies cant stop peeing, as a result the symptoms will be
BP decrease Thirst Increase Hypovolemia Hypernatremia Tachycardia Urine Specific gravity low
Nursing care for DI
Monitor and replace fluids
Check neuro status, vitals, mucous membrane
DI water movement
Polydipsia
Polyuria
Nocturia
Meds for DI
Desmopressin ddAVP
Vasopressin
Because of incontinence, the elderly are reluctant to
best way to tell if a pt is dehydrated is
Drink enough water
Filling of veins in hands
COPD MOA
Chronic inflammation damages lung tissue causing scarring that narrows the airway
COPD associated complications
Chronic Bronchitis
Emphysema
COPD symptoms
Chronic cough
Sputum production
Dyspnea
Weight loss
Barrel Chest
COPD management
Smoking cessation Exacerbation management O2 therapy Pneumococcal/influenza vaccines Pulmonary rehab
Meds for COPD
Bronchodilators and Corticosteroids
Antibiotics
Mucolytics
Antitussives
Nursing management for COPD
Airway clearance
Breathing patterns
Activity tolerance
Asthma MOA
Chronic inflammation or mucous membrane and edema due to hyperresponsiveness
Asthma symptoms
Cough
Chest tightness
Wheezing
Dyspnea
Manifestations of asthma
Diaphoresis
Tachycardia
Hypoxemia
Cyanosis
Fast acting and long acting meds for asthma
Fast - fast beta 2 agonists
Anticholinergics
Long - long beta 2 agonists
Corticosteroids
Teach asthma patients to
Avoid triggers
Use inhaler
Monitor peak flow
When to seek aid
Gas exchange corticosteroid
Triamcinolone
Use bronchodilator _
Use Corticosteroid _
First
Second
Use mouth wash after
Corticosteroids
WILL cause thrush
Anticholinergics
Will decrease acetylcholine relaxing breathing
Ipratropium bromide
RSV children should be placed on what precaution
Droplet
Can be cohorted
To prevent RSV
Wash hands at day care and after exposure to individuals with cold symptoms
Hospitalize RSV child with what symptoms
Tachypnea
Retraction
Poor oral intake
Lethargy
Severe, continuous asthma that is unresponsive to meds
Status Asthmaticus
Diagnosing Asthma
Episodic symptoms of airflow obstruction
Airflow it at least partially reversible,
Other causes are excluded
Blood pH
PaCO2
HCO3
7.35-7.45
45-35
22-26
Metabolic acidosis
Bicarbonate less than 22
DKA
Kidney failure
Diarrhea
HYPERkalemia
Kusmaul breathing
Metabolic acidosis
Diamox
Decreases bicarb reabsorption
Treats metabolic alkalosis
Can cause metabolic acidosis
A/B of vomiting
Metabolic alkalosis
loss of H
Treat metabolic alkalosis with
Antiemetic
Stop suction
Stop diuretics “loop and thiazide especially”
Monitor ABGs
Give diamox
Flu symptoms
Hyperactive reflex
Confusion
A/B
Metabolic Alkalosis
Metabolic alkalosis S/S
Bradypnea Hypoventilation
under 12
HYPOkalemia
EKG changes
Tetany
Tremors
Muscle weakness
Fatigue
Metabolic acidosis s/s
Kusmaul breathing
Confusion
Weakness
Lob BP
N/V
HYPERkalemia
Respiratory acidosis S/S
Neuro drop Drowsiness, confusion, fatigue Headaches RR less than 12 Hypotension
Resp Acidosis interventions
Admin O2
Encourage cough
Deep breathing
Watch K
Respirator alkalosis s/s
Resp rate over 20
Confusion
Fatigue
Tachycardia
Tetany
EKG changes
Muscle cramps
Positive Chauvstics
Resp alkalosis interventions
Monitor lytes
Paper bag
Hold breath
Calcium gluconate for tetany
3Ps of Diabetes
Polyuria
Polydipsia
Polyphagia
Type 2 diab
Patient becomes insulin resistant due to high blood glucose over time
T2 diab risk factors
Diet
Lifestyle
Medication
A1C
Checks blood sugar for past 3-4 months
Brown thick skin on neck and armpits
Acanthosis nigricans
T2 Diab sign
Normal unfasted glucose value
Pre diabetic value
Diabetics value
70-115
NA
200+
Fasting glucose value
Fasted pre diabetic value
Fasted diabetic value
Less than 100
100-125
126+
A1C normal value
Pre diabetic value
Diabetic value
less than 5.7
5.7-6.4
Over 6.5
Sugar under 70 hypogly
Brain will die
Diabetic complications of kidneys
Nephropathy
Kidneys will DIE
creatinine over 1.2
Diabetic complications of nerves
Loss of sensation Sugar foot Slow healing Retinopathy Blindness
Diabetic complications of heart and brain
HTN
Atherosclerosis
CVA
Stroke
Oral agents only work for _ diab
T2
PO drugs for t2 diab
Toxic to
Metformin
Glipizide Glyburide
Pioglitazone
Acarbose
Toxic to liver
for diabetics, avoid FOOT
Flipflops, high heels, nylon
OTC corn removal
Overly hot baths, use thermometer
Toe injuries, daily inspection, use mirror
NOs of diabetic foot care
NO callous removal
NO heavy powder
NO rubbing with hands
NO hot baths
Diabetic diet good
High fiber
Complex carbs
BROWN beans, rice, bread, peanut butter
Whole wheat/grain/milk
Diabetic diet bad
Simple sugars Soda Candy White bread/rice Juice #1 offender
Antithyroid meds
Methimazole
Propylthiouracil
Can cause hypothyroidism
Hypothyroidism symptoms
Low and Slow
Weight gain, shot metabolism Unable to tolerate cold Goiter Tiredness Fatigue Slow HR Depression Memory loss
Hypothyroidism
Skin
GI
Hair
Dry, rough, cold
Constipation
Thin brittle
Myxedema
Swelling of face and eyes
Hypothyroidism
Hypothyroidism risk factors
Women
middle to older age
Hashimotos
Iodine deficiency
Pituitary tumor
Hypothyroidism monitor
HR BP RR
Blood glucose
Weight
Meds for hypothyroidism
Levothyroxine
Liothyroinine
Lotrix
Hypothyroidism
how it is diagnosed
Under secretion of T3 and T4
Blood test for t3 t4 and TSH
INsulin
protein hormone made in pancreas
Puts sugar and potassium INto cells
In t2 diab pt becomes insulin _
resistant
Best t2 diab treatment
Diet Lifestyle Exercise
Insulin peaks =
plates
Give food
IV only insulin
REGULAR
Right in the vein
Clear days before cloudy days
Regular Clear insulin first
NPH Cloudy insulin second
RN of insulin
Regular first
NPH iNtermediate second
Best location for insulin injection
Abdomen
rotate daily
NEVER aspirate fat tissue
NEVER massage or hot compress
Insulin on sick days
Still give
Monitor closely
LONG Acting insulin
‘Old guys”
NO peak NO mix SEPARATE syringes Duration 24+h NO risk of hypoglycemia