12 Lead ECG Flashcards

1
Q

Basics: what do the following waves represent?

  1. P wave
  2. PR
  3. QRS
  4. T wave
  5. QT interval
A
  1. P wave is Atrial DEPOLARIZATION
  2. PR interval: AV conduction time (0.12-0.20 sec)
  3. QRS: Ventricular DEPOLARIZATION (0.06 - 0.10 sec)
  4. T wave: Ventricular REPOLARIZATION
  5. QT Interval: time of ventricular depolarization and repolarization (0.36 - 0.44 sec)
    QT interval is corrected bc of the effect of heart rate.
    QTc = 0.40 - 0.44 sec
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2
Q

When are Q waves considered pathologic?

A
  • Width > 30 ms (0.04 sec)
  • Depth ≥ 25% of the height of the R wave (R÷4)
  • If present in contiguous leads, indicative of myocardial necrosis. Contiguous: next or together in sequence.
    Ex: V3 and V4 or V1 and V2
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3
Q

What does one tiny box on the paper represent?

A

1 mm or
0.04 sec horizontally
0.1 mV vertically

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4
Q

Which lead placement is:

aVR?

aVL

aVF

What is the R LL’s purpose?

A

aVR is the right shoulder; stands for augmented Vector Right. It’s a positive electrode.

aVL is on the left shoulder; stands for augmented Vector Left. It’s a positive electrode.

aVF is the left lower limb lead; means augmented Vector Foot. It’s also a positive electrode.

Right LL serves as the ground; a neutral lead, like you would find in an electric plug. It is there to complete an electrical circuit and plays no role in the ECG itself.

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5
Q

Which lead is assessed to determine left vs right bundle branch block?

What is the rhyme r/t this?

A

V1

“If the QRS is wide, and the QRS is upRIGHT, it’s a RIGHT bundle branch block (BBB). If the QRS is wide and it goes LOW-er, it’s a LEFT BBB.”

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6
Q
  1. An infarct in the RCA would be seen in which leads?
  2. What are the reciprocal leads?
  3. Complications from this infarct?
A
  1. RCA = Inferior MI
    Leads: II, III, aVF
  2. Reciprocal: I, aVL
  3. AV Blocks, decreased HR (node conduction in R atria), decreased BP, Papillary muscle rupture, N/V

Right sided ECG asses V2R - V4R for possible RV MI

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7
Q
  1. An infarct in the LAD would be seen in which leads?
  2. Reciprocal leads?
  3. Complications from this infarct?
A
  1. LAD = Anterior MI
    Septal leads V1 and V2
    Anterior leads V3 and V4 (LAD/Left main)
  2. Reciprocal: II, III, aVF
  3. 2ndº Type II, BBB, 3rdº block, HF, Cardiogenic shock, VT
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8
Q
  1. An infarct in the Left Circumflex would be seen in which leads?
  2. Reciprocal leads?
  3. Complications from this infarct?
A
  1. Left Circumflex = Lateral MI
    Leads: I, aVL, V5, V6
  2. Reciprocal: II, III, aVF
  3. Ventricular aneurysm, AV blocks, PVCs, VT
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9
Q
  1. An infarct in the RCA or Circumflex (Posterior MI) would be seen in which leads?
  2. Reciprocal leads?
  3. Complications from this infarct?
A
  1. Septal leads V1, V2
  2. ST depression and/or Pathologic R waves
  3. AV Blocks, Bradycardia
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10
Q
  1. A Right Ventricular MI, supplied by the RCA, would be seen in which leads?
  2. Reciprocal leads?
  3. Complications from this infarct?
A
  1. V2R, V3R, V4R (ECG done on the RIGHT side of chest)
    If you see elevation, that’s confirmation.
  2. Reciprocal: none
  3. Complications: RV Failure, AV Blocks, N/V
    Hypotension = fluids (Caution NTG & morphine use)
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11
Q

Mnemonic Poem for First degree block:

A

“If the R is far from P, then you have a First Degree.”

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12
Q

Mnemonic poem for Wenckebach:

A

“Longer, longer, longer, drop, then you have a Wenckebach.”

Wenckebach is a 2ndº Type I block.

This poem is referring to the P to QRS relationship, P’s are further from the QRS and then a QRS is “dropped” all together.

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13
Q

Mnemonic poem for Mobitz II:

A

“If some P’s don’t get through, then you have a Mobitz II.”

OR

“If some R’s don’t get through, prepare to pace that Mobitz II!”

Mobitz II is a 2ndº Type II block.

This poem is referring to the relationship between the P’s and QRS complexes. The P’s are in consistent intervals with the QRS’s and then a QRS is blocked, or dropped.

Second-degree AV block (Type 2) is almost always a disease of the distal conduction system located in the ventricular portion of the myocardium.

Prepare to pace. This can progress to 3rd degree.

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14
Q

Mnemonic poem for 3rdº Heart Block:

A

“If P’s and Q’s don’t agree, then you have a THIRD DEGREE.”

OR

“If R’s and P’s don’t agree, prepare to PACE that 3rd degree!”

Third-degree AV block is electrocardiographically characterized by:
1. Regular P-P interval.
2. Regular R-R interval.
3. Lack of an apparent relationship between the P waves and QRS complexes.
4. More P waves are present than QRS complexes.

Third-degree heart block: The electrical signal from the atria to the ventricles is completely blocked. To make up for this, the ventricle usually starts to beat on its own acting as a substitute pacemaker but the heartbeat is slower and often irregular and not reliable.

A third degree heart block can cause a wide range of symptoms, some of which are life-threatening. This type of heart block is usually regarded as a medical emergency and may require immediate treatment with a pacemaker.

These will have bradycardic ventricle rates and atropine will NOT work. Atropine works on the SA node… and there is no communication between the atria and ventricles - so it won’t have an affect.

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15
Q

How do you pick up a posterior wall MI?

A

By seeing a reciprocal change in V1 and V2.

You will see ST depression (the reciprocal of elevation - we’d see elevation if we were able to place leads on the back). You may also see pathological R waves.

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16
Q

With INFERIOR MI’s, always look for posterior or RV infarction!

How can you interpret this?

A

POSTERIOR MI:
ST depression (reciprocal) in V1-V3

RV infarction:
ST elevation in V1-V3

17
Q

Which leads look at lateral wall?

A

V5, V6, I, aVL

I and aVL look at the high lateral wall

Lateral wall injuries often will many times have other areas affected as well; ex: Inferior in RCA dominant and/or anterior in LDA/left main.

18
Q

When there is an occlusion in this wall, you will see ST depression in V1, V2 and tall broad R waves (>0.04):

A

POSTERIOR

Can also see ST elevation in V7-V9 on their back (below their scapula)

Inferior/lateral wall d/t occlusion of RCA (90% of ppl) or Left Circumflex (10%).

19
Q

When assessing an ECG reading, which leads do you start with?

What are the reciprocal leads?

A

II, III, aVF

This reflects RCA, inferior wall.

Reciprocal: I and aVL

20
Q

Which MI (wall?) has elevation in V1-V4; reciprocal in II, III, aVF?

Which artery is occluded?

Side affects with this MI; what to watch out for?

A

ANTEROSEPTAL WALL MI

Artery: Left Anterior Descending artery (LAD)

Watch for:
HF
2nd Degree Block Type II
Complete Heart Block
Papillary muscle rupture

21
Q

Which MI (wall?) has elevation in II, III, and aVF; reciprocal in I & aVL?

Affected artery?

What to watch out for?

A

INFERIOR WALL MI.

Right Coronary Artery. If suspected RCA, monitoring lead III is preferred.

Watch for:
Bradycardia
Nausea
Vomiting
2nd Degree Type I
Right Ventricle Failure

22
Q

Which MI (wall) has elevation in V7-V9; reciprocal V1-V2?

Affected artery?

What to watch out for?

A

POSTERIOR WALL MI

RCA (90%) or Left Circumflex (10%)

Watch out for possible accompanied inferior or lateral wall MI

23
Q

Why do some pts develop pericarditis after an MI?

Description of ST segments on ECG?

A

Inflammatory response.

Slightly coved segments in V leads. May see diffuse ST changes throughout; PR depression; concave ST segment in limb leads. Looks like MI.

10-15% will develop 2-7 days post MI

Chest pain relieved by leaning forward, left radiation, deep breathing/coughing/position changes exacerbates, may have fever. NSAIDs help.

Trick: look at lead I (high lateral) and lead II (inferior)… these wouldn’t ever be elevated together in an MI - if they ARE both elevated - sign of pericarditis. Another trick is the concave nature of the ST segments in the V leads - if they look like smiley faces (you’d add the eyes) then it could be pericarditis.

24
Q

“Dead meat don’t beat”
When a muscle is infarcted, it will not conduct electricity or contract, becomes weak. What muscle degradation will lead to a heart murmur?

Treatment?

A

Papillary muscle ruptures - regurgitation

Loud systolic murmur

Treatment:
Surgical repair/replacement (valve)
Mechanical support (vent if unstable)
Hemodynamic support (Impella)

25
Q

Which complication (esp from an anterior wall MI) results in the mixing of oxygen-rich blood with venous (left to right shunt)?

Symptoms:

PA Catheter Insertion results to be expected:

A

VENTRICULAR SEPTAL RUPTURE

Symptoms: Circumoral cyanosis, S3, crackles, holosystolic murmur

Pulmonary Artery (PA) catheter will reveal (in normal pulmonary artery) 65-75% O2. If higher, 90’s% - shunting evidence.

PA cath may show increased CO (in right ventricle) d/t higher flow from left ventricle affecting reading.

Large V waves:

Treatment: OR for patch graft to septum

26
Q

Clinical signs of cardiogenic shock 2/2 to acute left ventricular HF include?

a) Hypotension, S4, pericardial friction rub
b) S3, Hypotension, systolic murmur
c) Diastolic murmur, S4, HTN
d) Crackles, S3, hypotension

How do we treat cardiogenic shock?

A

D. Crackles, S3 and hypotension

TREATMENT:
Positive inotropes (dobutamine) to help the pump
Diuretics to help decrease preload & pulm edema
Vasodilators (nitro) to help decrease afterload. As long as BP can handle it.

Impella device is a ventricular assist device that can really help these patients.

Will get an echo on these patients. ABG will probably be acidotic and will show a mix of resp/metabolic.

27
Q

Hemodynamics of cardiogenic shock:

Is the CO going to up or down?
Preload up or down?
Afterload up or down?

A

CO will be down.

Preload will be up. Fluid backing up from the pump not effectively working.

Afterload will be up. From the decreased CO stimulating the sympathetic nervous system (renin-angiotensin-aldosterone system) activating vasoconstriction

28
Q

Where’s the best place on the body to assess for mottling?

S/S of cardiogenic shock?

A

Knees.
Validated scoring tool.

S/S:
S3; Pulmonary edema
Crackles
Tachycardia
Dysrhythmias
Decreased perfusion
Decreased urine output (oliguria <0.5 mL/kg/hr)

Hemodynamics:
CO decreased (MAP <60)
Cl <2.0 L/min/m2
Elevated SVR (>1600 dynes/sec/cm^-5)
Elevated RAP/CVP (preload) RAP = right arterial pressure
PAOP > 18 mm Hg
Decreased SvO2

29
Q

Cardiogenic pulmonary edema treatment:

A
  1. Vasodilator (nitro)
  2. Loop diuretic
  3. CPAP/ BiPAP reduces preload
30
Q

Why would a patient who received a radial artery bypass be placed on nitro or diltiazem gtt and go home on dilt?

A

Radial artery bypasses are prone to spasms.

31
Q

3 Hallmark signs of Right Ventricular Infarction?

A
  1. Hypotension
  2. HR goes up (CO = HR x SV)
  3. JVD (w/clear lungs)

Tx is FLUIDS [sm boluses] (starlings law) then progresses to inotropes if needed

*** Inferior wall MI w/RV infarction are the ONLY MI pts that get fluid!!!