11 - ENT Flashcards

1
Q

What bacteria most commonly cause otitis media and what are some risk factors for developing this?

A

Usually preceded by viral URTI

Bacteria: Haemophilus Influenza, Strep Pneumonia, Moraxhella Catarrhalis

Risks: passive smoking, formula fed, craniofacial abnormalities, attend nursery

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2
Q

How does acute otitis media present in children?

A

Children will often have history of URTI

  • Infant: fever, vomiting, irritability, lethargy, poor feeding
  • Child: ear pain, reduced hearing, fever, cough, coryza symptoms
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3
Q

How does AOM present on examination?

A

Always check the ears and throat of unwell children

  • Bulging red TM
  • May be yellow or cloudy
  • Loss of light reflex
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4
Q

How is uncomplicated AOM managed in children?

A

Most will self-resolve within 3 days

  • Ibuprofen/Paracetamol: For fever and pain relief. Most cases will self-resolve within 3 days without antibiotics
  • Antibiotics: If indicated give Amoxicillin for 5-7 days or Erythromycin. Give delayed until 3 days of symptoms
  • Safety Net: advice to parents on when to seek further medical attention
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5
Q

What is the criteria for antibiotics in AOM?

A
  • Systemically unwell
  • Immunocompromised
  • Perforated eardrum or discharge
  • <2 years old and bilateral
  • Present for ≥4 days
  • <3 months old
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6
Q

Which patients need hospital admission for AOM?

A
  • Severe systemic infection
  • Suspected complications e.g meningitis, mastoiditis, intracranial abscess, sinus thrombosis, or facial nerve paralysis
  • Children younger than 3 months of age with a temperature >38°C
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7
Q

What are some complications of AOM?

A
  • Hearing loss (usually temporary)
  • Otitis media with effusion
  • Perforated eardrum
  • Recurrent infection
  • Mastoiditis
  • Sinus thrombosis
  • Meningitis
  • Intracranial abscess
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8
Q

What is Glue ear and some risk factors for this?

A

Otitis Media with Effusion

  • Parental smoking
  • Recurrent AOM
  • Cleft palate
  • Down’s syndrome
  • Cystic fibrosis
  • Allergic rhinitis
  • Reflux
  • Enlarged adenoids

Those with Downs and Cleft should be regularly checked for OME by ENT

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9
Q

How does Glue ear present and what do you see on otoscopy?

A

Presentation

  • Reduction in conductive hearing
  • Speech and language developmental issues

Examination

  • Dull tympanic membrane
  • Fluid level or air bubbles
  • Retracted TM
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10
Q

How is OME diagnosed?

A
  • History of hearing loss
  • Otoscopy findings
  • Audiometry
  • Tympanometry
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11
Q

How is OME managed?

A
  • Active observation for 3 months: often self resolves, if not by this time send for audiometry and ENT review
  • Myringotomy and insertion of grommets: will fall out after a year, faster referral if significant hearing loss
  • Hearing aids
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12
Q

How is chronic supportive otitis media managed in children?

A

Perforation - complication of AOM

  • Refer to ENT
  • Antibiotics, topical corticosteroids and intensive aural cleaning
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13
Q

What bacteria cause OE and how is otitis externa in children managed?

A

Always need to rule out ottorhea from an otitis media causing the otitis externa

  • Clean ear: If cannot visualise TM. Use micro suction, dry swabbing
  • Topical antibacterial: Acetic acid 2% with or without steroid for 1 week
  • Paracetamol/Ibuprofen: for pain
  • Safety net: avoid getting wet, come back if not resolving
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14
Q

Why are topical antibacterials only used for a short period of time in OE?

A

Can lead to chronic OE caused by a fungal infection

Use clotrimazole

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15
Q

What are common causes of hearing loss in children?

A

Congenital or Acquired

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16
Q

Children with deafness often present on the NHSP. Other children may present with speech and language or behaviour difficulties. How is hearing loss in children managed?

A

MDT

  • Speech and language therapy
  • Educational psychology
  • ENT specialist
  • Hearing aids for children who retain some hearing
  • Sign language
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17
Q

What is the pathophysiology of mastoiditis and some risk factors for this?

A

Complication of AOM

  • Mastoid air cells in temporal bone and communicate with middle ear via small canal
  • Infection can spread from the middle ear into the mastoid air cells causing infection of the bone of the mastoid air cells, causing necrosis and subperiosteal abscess
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18
Q

How is mastoiditis diagnosed and how does it present?

A

Diagnose with CT WITH CONTRAST

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19
Q

How is mastoiditis managed?

A
  • IV antibiotics: usually co-amoxiclav or ceftriaxone
  • Surgical intervention: mastoidectomy
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20
Q

What are the complications of mastoiditis?

A
  • Facial nerve palsy
  • Hearing loss
  • Meningitis
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21
Q

What is the epidemiology and pathophysiology of periorbital cellulitis?

A

Infection of tissues anterior to orbital septum

  • More common in those under 10 and in winter
  • Commonly from ethmoidal sinus as young children not yet formed their frontal sinuses until age 7 and thin lamina papyracea
  • Other causes: dental infection, endophthalmitis, trauma, foreign bodies, insect bites, skin infections (impetigo), eyelid lesions (chalazia, hordeola)
  • Organisms: Staph aureus, Strep pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, group A streptococcus
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22
Q

What are some signs and symptoms of pre-septal cellulitis?

A
  • Acute onset
  • Absence of orbital signs (e.g painful eye movements, proptosis)
  • Eyelid oedema
  • Erythema
  • Fever
  • History of URTI or sinusitis
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23
Q

What are some differentials for pre-septal cellulitis?

A
  • Orbital cellulitis
  • Nephrotic syndrome
  • Allergic reaction
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24
Q

What are some investigations done for diagnosis of pre-septal cellulitis?

A

Thorough history (previous episodes, recent URTI) and clinical examination

  • Vital signs
  • Dentition
  • Anterior rhinoscopy with swab
  • Comprehensive ophthalmic examination
  • Neurological examination
  • Bloods
  • Contrast CT
  • ABG and lactate if considering sepsis
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25
Q

How is pre-septal cellulitis managed?

A
  • Refer for assessment
  • If well give PO antibiotics and treat outpatient
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26
Q

How does orbital cellulitis present?

A

Infection of ocular muscles and fat behind the anterior septum

  • Redness and swelling around the eye
  • Severe ocular pain
  • Visual disturbance
  • Proptosis
  • Ophthalmoplegia/pain with eye movements
  • Eyelid oedema and ptosis
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27
Q

How can you tell the difference between pre-septal and orbital cellulitis?

A

Orbital has:

  • Reduced visual acuity
  • Proptosis
  • Ophthalmoplegia/pain with eye movements
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28
Q

What classification is used for peri/orbital cellulitis?

A

Chandler Classification

  1. Pre septal
  2. Post septal
  3. Subperisoteal abscess
  4. Intaorbital abscess
  5. Cavernous sinus thrombosis
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29
Q

How is orbital cellulitis investigated and managed?

A

Ix

  • FBC: WBC elevated, raised inflammatory markers.
  • Clinical examination involving complete ophthalmological assessment
  • CT with contrast
  • Blood culture and microbiological swab to determine the organism

Management

  • Admit to hospital due to risk of intracranial spread and cavernous sinus thrombosis
  • Refer to ENT and Ophthalmology
  • IV antibiotics inc Cef/Met
  • Surgical drainage if not improving
  • CT if red flags of orbital cellulitis
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30
Q

What are the complications with orbital cellulitis?

A
  • Sight threatening
  • Life threatening: cavernous sinus thrombosis, intracranial abscess, meningitis, sepsis
  • Impaired ocular motility
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31
Q

What are the complications of rhinosinusitis in children?

A
32
Q

How may rhino sinusitis present in children?

A

Usually caused by viral infection and resolves in 2-3 weeks

  • Nasal blockage or congestion
  • Mouth breathing
  • Discoloured nasal discharge
  • Cough during the day or night due to post nasal drip
33
Q

How is acute sinusitis managed in children?

A

<10 days: paracetamol or ibuprofen and may self resolve, safety net

>10 days: high dose nasal corticosteroid e.g mometasone

Systemically unwell: PO phenoxymethylpenicillin, if complications like orbital cellulitis admit to hospital

34
Q

How is allergic rhino sinusitis managed in children?

A
  • All: Saline irrigation and allergen avoidance
  • Mild: topical antihistamines
  • Severe: topic corticosteroids, sodium cromoglicate, montelukast
35
Q

When is epistaxis abnormal in children?

A

Under the age of 2 - refer to ENT

36
Q

What are some risk factors for epistaxis in children?

A
  1. Activities with risk of nasal trauma or straining/raising ICP e.g. rugby, gymnastics
  2. Coagulopathies
  3. Hayfever or regular URTIs
  4. Medication use
37
Q

What do you need to rule out when a child has epistaxis?

A
  • Foreign body
  • Septal haematoma
38
Q

If a child has recurrent epistaxis from one nostril what do you need to consider?

A

Juvenile nasopharyngeal angiofibroma

Especially if male aged 12-20, refer to ENT

39
Q

How is epistaxis in children managed?

A

First aid

  • Lean child forwards over a bowl and encourage them to spit any blood out
  • Pinch the soft part of the nose and hold for at least 15 minutes
  • Put ice pack on neck

Primary care/ A&E management

  • If any bleeding points identified, obtain verbal consent from parent for nasal cautery with silver nitrate. Do not cauterise both sides of the septum in the same episode.
  • If bleeding continues, call ENT who will consider placing an anterior or a posterior nasal pack
  • In recurrent cases, a full blood count and clotting profile should be checked

DISCHARGE WITH NASEPTIN OINTMENT BD FOR 2 WEEKS

40
Q

What advice can be given to parents after a child has epistaxis to prevent recurrence?

A

Avoid

  • Strenuous physical activity
  • Bending forwards e.g. to tie shoelaces
  • Hot drinks/food/showers
  • Blowing nose
  • Picking nose
  • Spicy food
41
Q

What are the causes of tonsillitis?

A
  • Most common: Viral such as EBV or Adenovirus
  • S.Pyogenes
  • S.Pneumoniae
  • S.Aureus
  • Haemophilus influenzae
  • Morazella catarrhalis
42
Q

How may tonsillitis present in children?

A

Usually aged 5 to 10 or 15 to 20

  • Fever
  • Sore throat
  • Painful swallowing/poor oral intake
  • Vomiting
  • Abdominal pain
  • Headache

Examination: red, inflamed and enlarged tonsils, with or without exudates

Always examine the ears to visualise TM and check for lymphadenopathy

43
Q

What is the FeverPAIN score and how do you interpret the results?

A

Work out likelihood of infection being due to Streptococcus

Score of 4 or more warrants antibiotics

44
Q

What is the Centor Score and how do you interpret it?

A

Estimate the probability that tonsillitis is due to a bacteria infection, and will benefit from antibiotics

Score of 3 or more give antibiotics

Tender lymphadenopathy

45
Q

How is tonsillar size graded?

A

Based on how much space they take up in oropharynx

Enlarged tonsils could be due to recurrent tonsillitis leading to scarring, doesn’t mean it is tonsillitis

46
Q

What are some differentials for tonsillitis in children?

A

Based on how much space they take up in oropharynx

Enlarged tonsils could be due to recurrent tonsillitis leading to scarring, doesn’t mean it is tonsillitis

47
Q

How is tonsillitis in children managed?

A
  • Exclude other serious pathology, e.g meningitis, epiglottitis and quinsy
  • Calculate FeverPAIN and Centor score
  • Educate parents with likely viral tonsillitis, and give safety net advice to return if the pain has not settled after 3 days or the fever rises above 38.3ºC. May consider delayed prescription
  • If Centor ≥ 3 or the FeverPAIN score is ≥ 4. or immunocompromised or history of rheumatic fever then prescribe antibiotics
48
Q

When do children with tonsillitis need admission to hospital?

A
  • Immunocompromised
  • Systemically unwell, dehydrated
  • Stridor
  • Respiratory distress
  • Peritonsillar abscess
49
Q

When do children qualify for a tonsillectomy according to SIGN guidelines?

A
  • 2 episodes of Quinsy
  • Recurrent febrile convulsions secondary to episodes of tonsillitis
  • OSA stridor or dysphagia secondary to enlarged tonsils
50
Q

What are some complications of tonsillitis?

A
  • Chronic tonsillitis
  • Peritonsillar
  • Otitis media
  • Scarlet fever
  • Rheumatic fever
  • Post-streptococcal glomerulonephritis
  • Post-streptococcal reactive arthritis
51
Q

How is post tonsillectomy bleeding managed in children?

(important)

A

Management:

  • Call ENT registrar
  • Get IV access
  • Bloods inc FBC, clotting screen, G+S and crossmatch
  • Keep the child calm and give adequate analgesia
  • Sit them up and encourage them to spit the blood
  • Make child NBM incase an anaesthetic and operation is required
  • IV fluids for maintenance and resuscitation as required
  • Definitive: hydrogen peroxide gargle, adrenaline soaked swab, back to theatre

If there is severe bleeding or airway compromise, call an anaesthetist

52
Q

How does quinsy present in children?

A
  • Tonsillitis symptoms: Sore throat, painful swallowing, fever, referred ear pain, swollen tender lymph nodes
  • Trismus
  • Change in voice “hot potato voice”
  • Deviated uvula
  • Drooling
53
Q

What are some differentials for quinsy (peritonsillar abscess)?

A
  • Tonsillitis
  • Peritonsillar cellulitis
  • Retropharyngeal abscess
  • Epiglottitiis
54
Q

How is quinsy managed?

A
  • Refer to ENT
  • IV fluids (if dehydrated)
  • Broad spectrum IV antibiotic (Benzylpenicillin plus Metronidazole)
  • PO Dexamethasone
  • I+D under GA
  • Tonsillectomy within 6 weeks
55
Q

What is an important investigation to do with Quinsy?

A

Glandular fever screen

Giving amoxicillin with glandular fever can result in permanent skin rash

Give benzylpenicillin instead of co-amoxiclav

56
Q

What is epiglottis caused by and how does it present?

A

HiB and Strep pneumoniae, less so HiB since vaccines

  • Acute onset
  • High grade fever
  • Sore throat
  • Stridor (late sign)
  • Tripod position
  • Toxic looking child
  • No cough
57
Q

What are some differentials for epiglottis?

A
  • Laryngotracheobronchitis (Croup)
  • Inhaled foreign body
  • Retropharyngeal abscess
  • Tonsillitis
  • Peritonsillar Abscess
  • Diphtheria
58
Q

How is epiglottis diagnosed and investigated?

A

ALWAYS SECURE AIRWAY FIRST

  • Keep calm with parent and intubation equipment should always be nearby
  • Throat Swabs: Both bacterial and viral swabs taken on intubation
  • Blood Tests: FBC, cultures and CRP (once airway secured)
  • Lateral Neck X-Ray
    1) Thumb-Print Sign (swollen epiglottis) (5)
    2) Thickened aryepiglottic folds
    3) Increased opacity of the larynx and vocal cords

X-Rays can help rule out epiglottitis if a normal epiglottis is visualised, but should not waste time in a critical situation

59
Q

How is epiglottis managed?

A
  1. Secure the Airway: escalate to senior ENT and anaesthetist
  2. Oxygen: Parent can hold mask near child’s face
  3. Nebulised Adrenaline
  4. IV Antibiotics cover for haemophilus influenza (e.g. cefotaxime/ceftriaxone
  5. IV Steroids
  6. IVI: NBM until resolved
60
Q

What are some complications with epiglottis?

A
  • Airway compromise
  • Mediastinitis: If infection spreads to the retropharyngeal space
  • Retropharyngeal or Parapharyngeal cellulitis/abscess
  • Pneumonia: Especially following intubation
  • Meningitis
  • Sepsis
61
Q

What is laryngomalacia and the epidemiology of this?

A

Congenital abnormality of the larynx making it floppy

Most common cause of stridor in infants

Usually presents at 4 weeks and resolves by 2 years

62
Q

What are some risk factors for laryngomalacia?

A

Neuromuscular disease

63
Q

How does laryngomalacia present?

A
  • High-pitched inspiratory stridor worse on lying flat or on exertion
  • Normal cry
64
Q

What are some differentials for laryngomalacia?

A
  • Vocal Cord Paralysis
  • Subglottic stenosis
  • Laryngeal Atresia
  • Croup (Laryngotracheobronchitis)
  • Supraglottitis / Epiglottitis
  • Trauma (laryngeal fractures caused by direct trauma or strangulation
65
Q

What investigations are done for laryngomalacia?

A

Flexible endoscopy (laryngoscopy) via the nose or mouth to view the larynx

Rigid endoscopy if below level of larynx

66
Q

How is laryngomalacia managed?

A

99% of cases resolve over time

Mild: parental reassurance that will resolve by 12-16 months, may worsen at 6 months or during URTI

Severe: e.g feeding difficulties, faltering growth, respiratory distress and apnoeas. Endoscopic aryepiglottoplasty aka supraglottoplasty

Life-Threatening: Respiratory distress usually exacerbated by URTI, SEE IMAGE

67
Q

What is glandular fever and the epidemiology of this?

A

Infectious mononucleosis (IM) caused by Epstein-Barr virus

Triad: sore throat, fever, lymphadenopathy

Usually age 1-6 or 18-22

68
Q

How does glandular fever present in history and examination?

A

History

  • Sore throat
  • Swollen neck
  • Fever
  • Headaches
  • Nausea & vomiting
  • Generally tired all the time, despite adequate sleep
  • Generalised aches and pains in the muscles and joints

The odynophagia typically lasts 7-10 days but generalized malaise, aches and myalgia and cervical lymphadenopathy may well persist for several weeks

Examination

  • Enlarged inflamed tonsils (kissing tonsils)
  • Significant cervical lymphadenopathy
  • Abdominal tenderness & splenomegaly
  • Hepatomegaly
  • Palatal petechiae
69
Q

What investigations are done to diagnose glandular fever?

A

-FBC: elevated lymphocytes

-LFTs: elevated in week 2 or 3 until week five

-Monospot test: may not show for a few weeks

FBC and monospot test in 2nd week for diagnosis

70
Q

What antibiotic should not be given in glandular fever and why?

A

Ampicillin/Amoxicillin as a maculopapular, pruritic rash develops

71
Q

How is glandular fever managed?

A
  • Rest
  • Fluids
  • Avoid alcohol
  • Avoid kissing
  • Simple analgesia for any aches or pains
  • Avoid contact sports for 4 weeks to reduce risk of splenic rupture
72
Q

What are some complications of glandular fever?

A
  • Post viral fatigue: may last up to a year
  • Malignancy: Burkitt and Hodkgin’s lymphoma, nasopharyngeal cancer
  • Splenic rupture
  • Encephalitis
73
Q

What are red flags for foreign bodies?

A
74
Q

Where do inhaled foreign bodies tend to go?

A

Right main bronchus

75
Q

What investigations are done for a ingested/inhaled foreign body?

A
  • Plain film chest radiograph (assess for the foreign body, if radio-opaque, the presence of surgical emphysema, any widening of retropharyngeal tissue or loss of cervical lordosis)
76
Q

How are ingested/inhaled foreign bodies removed?

A
  • If the FB is visualised in the oropharynx: Magill forceps
  • Otherwise, endoscopy under GA