093014 EKG Flashcards

1
Q

P wave represents

A

atrial depolarization (first right, then left, but both are combined into the P wave)

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2
Q

if you prolong AP duration, what happens on the EKG?

A

prolongation of QT interval (QT interval changes can be arrhythmia-genic)

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3
Q

in the heart the sequence of repolarization is opposite to that of

A

depolarization, b/c AP duration is shorter near the outer epicardium (the last cells to depolarize)

so you get a positive T wave

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4
Q

precordial leads

A

in transverse plane (V1-V6)

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5
Q

limb leads

A

front plane leads

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6
Q

bipolar leads are leads

A

I, II, III

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7
Q

aVL’s degree deflection

A

-30 degrees

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8
Q

lead II’s degree deflection

A

60 deg

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9
Q

if you have RBBB, what will happen on EKG?

A

prolongation of total time to depolarize the two ventricles (the left will depolarize first and then the right)

this prolongation widens out the QRS

also, get deflection of electrical activity to the right

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10
Q

septal depolarization is from

A

left to right

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11
Q

as lateral wall of LV is depolarized, why is depolarization directed leftward as opposed to rightward?

A

bc electrical forces of thick LV outweigh those of RV

electrical forces of thick LV-depolarization is directed leftward and posteriorly toward V6

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12
Q

interpreting EKG-what to look for?

A
check voltage calibration
check heart rhythm
HR
measure intervals (PR, QRS, QT)
calculate mean QRS axis
evaluate P wave 
evaluate QRS (hypertrophy, bundle branch block, infarction)
evaluate ST/T wave
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13
Q

in standard cases along vertical axis 1 mm represents?

A

0.1 mV

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14
Q

time in standard horizontal axis on EKG-with recording speed of 25 mm/sec, 1mm = ?

A

0.04 seconds

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15
Q

PR interval should be btwn

A

120-200 ms

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16
Q

normal sinus rhythm criteria

A

every P followed by QRS
upright P in I, II, III
PR interval btwn 120 and 200 ms
HR btwn 60-100 bpm

17
Q

how to calculate HR from EKG

A

1500/ number of small boxes between two beats
or
count off 300-150-100-75-60-50

if rhythm is irregular, can count number of QRS during 6 seconds and multiply by 10

18
Q

corrected QT interval

A

measured QT/square of RR interval (seconds)

you do this because QT can vary with heart rate

other thing you can do–QT interval should normally be less of half of RR

19
Q

mean QRS axis is defined as

A

average of instantaneous lectrical ofrces generated during ventricular depolarization in the frontal plane

20
Q

normal mean QRS axis is

A

-30 to 90 deg

21
Q

how do you roughly estimate the QRS mean axis?

A

look at leads I and II- if net deflection is positive in both of these leads, the mean QRS axis is within normal range of -30 to +90 degrees.

22
Q

how to use geometric method to find mean QRS axis

A

pick any two frontal leads

draw perpendiculars from them and where they intersect is the mean axis

23
Q

how to use inspection method to find mean QRS axis

A

look for isoelectric frontal lead, then draw a perpendicular line to this lead. this perpendicular line is the mean QRS axis, but to determine which direction it’s pointing, you need to look at another lead to determine which direction

24
Q

if you have R atrial enlargement what would you see on P wave in leads II and V1?

A

in lead II-taller P wave

in lead V1-taller right atrium wave

25
Q

L ventricle hypertrophy-what would you see in lead V1?

A

deeper S wave

26
Q

L ventricle hypertrophy-what would you see in lead V6?

A

bigger QRS

27
Q

if you have L atrial enlargement what would you see on P wave in leads II and V1?

A

in lead II-a second left atrium peak

in lead V1-a deeper left atrium depression

28
Q

if you have R ventricular hypertrophy, what would you see on EKG?

A

in V1: increased R-R is greater than S

in V6: deep S

29
Q

with fascicular blocks, QRS interval is

A

reasonably normal, as opposed to bundle branch block

30
Q

with left anterior fascicle block, what is the direction of activation?

A

initial inferior followed by dominant superior direction of activation

31
Q

with left posterior fascicle block, what is direction of activation?

A

initial superior followed by dominant inferior direction of activation

32
Q

in bundle branch block, why is activation of the blocked part slow?

A

because relies on activation from cell to cell rather than rapidly conducting HIs purkinje system

33
Q

in left bundle branch block, why does the Q wave disappear in V6?

A

because you don’t have the normal left to right ventricular septum depolarization

34
Q

criteria for RBBB

A

QRS complex
RsR’ (M shaped ) QRS complex in lead I
widened or “slurred” S wave in leads I and V6

35
Q

when you have ST segment elevation, can you have reciprocal ST depression in some leads for the case of myocardial infarction?

A

yes