04: EKG Reading Flashcards

1
Q

What order should ECGs be read?

A
  1. Calibration
  2. Heart rate
  3. Heart rhythm
  4. Intervals (PR, QRS, QT)
  5. Mean QRS axis
  6. P-Wave abnormalities
  7. QRS abnormalities
  8. ST-segment/T-wave abnormalities
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2
Q

How should an ECG be calibrated?

A

Speed = 25mm/sec

Voltage = 10 mm/mV

One large box (5x5mm) = 200ms, 0.5mV

One small box (1x1mm) = 40ms, 0.1mV

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3
Q

Identify the various components of an ECG tracing.

A
  • P-wave: depolarization of atria
  • QRS complex: depolarization of ventricular muscles/(repolarization of atria)
  • T-wave: Repolarization of ventricles
  • PR interval: P-wave to onset of QRS complex
  • QT interval: beginning of QRS to end of T-wave
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4
Q

Describe electrocardiogram deflections.

A

Depolarization current toward (+) electrode: upward deflection recorded

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5
Q

How do you calculate heart rate?

A

Count off boxes between two QRS complexes: 300-150-100-75-60-50

If irregular rhythm (e.g., AFib): Count $ of complexes during 6 seconds and multiply by 10. (ECGs have time markers spaced 3 sec apart.)

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6
Q

Describe sinus heart rhythm.

A
  • Every P wave followed by QRS
  • Every QRS preceded by P wave
  • P-wave upright in leads I, II, III
  • PR interval greater than 0.12 sec (3-5 small boxes)
  • If HR <60bpm, sinus bradycardia
  • If HR >100bpm, sinus tachycardia
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7
Q

What is the Corrected QT (QTc)?

A
  • Divide the measured QT by the square root of the R-R interval (in seconds)
  • If measured QT interval <50% in two consecutive QRS complexes, likely normal
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8
Q

For the PR interval, identify:

  1. Normal
  2. Decreased
  3. Increased
A
  1. 120-200 ms (3-5 small boxes)
  2. Pre-excitation/WPW, junctional rhythm
  3. First-degree AV block
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9
Q

For the QRS interval, identify:

  1. Normal
  2. Decreased
  3. Increased
A
  1. = 100ms (= 2.5 small boxes)
  2. Atrial flutter
  3. Bundle branch block, ventricular ectopy, drug effect (e.g., antiarrhythmics), or ↑K+
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10
Q

For the QTc interval, identify:

  1. Normal
  2. Decreased
  3. Increased
A
  1. = 450 (male) or 460 (female) ms
  2. ↑Ca, Short QTc (2/2 hypercalcemia, congenital, digoxin)
  3. ↓K, Ca, ischemia, congenital, toxic drug effect (e.g., antiarrhythmics), Long QTc
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11
Q

Describe the types of AV blocks.

A
  • 1st Degree: Large PR interval (>200ms or 1 large box).
  • 2nd Degree, Mobitz I (Wenckebach): Progressive prolongation of PR interval culminating in a non-conducted P wave.
  • 2nd Degree, Mobitz II: Intermittent non-conducted P waves *without *progressive prolongation of the PR interval.
  • 3rd Degree: Complete absence of AV conduction – none of the supraventricular impulses are conducted to the ventricles (rhythm is maintained by a junctional or ventricular escape rhythm).
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12
Q

Describe the mean QRS axis.

A
  • Mean direction of electrical forces in the frontal plane (limb leads) as measured from Lead I
  • Normal = -30° to 90° (positive in lead I, II)
  • Right axis deviation = 90° to 180° (**negative in I)
    • Increased RV mass/electrical impulse
    • DDx: RVH, pulmonary embolus, LPFB
  • Left axis deviation = -30° to -90° (negative in II)
    • Increased LV mass/electrical impulse
    • DDx: Inferior MI, LVH, LAFB
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13
Q

Describe normal and abnormal P waves.

A
  • Look in leads:
    • II: 1 (RA) & 2 (LA) both upward
      • Enlargement: >2.5sbox high, peaked (RA) or wide, bifid (LA)
    • V1: 1 up, 2 down
      • Enlargement: >1sbox
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14
Q

Describe normal and abnormal Q waves.

A
  • Under normal circumstances, Q waves are not seen in the right-sided leads (V1-3); (waves represent absence of electrical forces from infarcted cells)
  • Abnormal: width >/= 1 small box wide & depth >25% of QRS height
  • Pathological Q waves usually indicate current or prior myocardial infarction (must be in at least 2 consecutive leads; in presence of LBBB, Q waves not helpful in diagnosis of MI)
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15
Q

Describe the localization of myocardial infarctions by leads.

A
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16
Q

Describe the localization of myocardial infarctions to coronary arteries.

A
  1. Inferior (II, III, aVF): RCA
  2. Anteroseptal (V1, V2): LAD
  3. Anteroapical (V3, V4): LAD (distal)
  4. Anterolateral (V5, V6, I, aVL): CFX
  5. Posterior (V1, V2; tall R wave, not Q wave): RCA
17
Q

What are the indicators of left ventricular hypertrophy?

A
  • Larger-than-normal electrical forces generated by LV
  • Left lateral leads (V5, V6, I, aVL) have taller-than-normal R waves +/- T-inversions
  • Right lateral leads (V1, V2) have opposite– deeper-than-normal S wave
18
Q

What are the indicators of bundle branch blocks?

A
  • 2/2 ischemia or degenerative damage
  • Slow myocyte-to-myocyte spread of electrical activity: delay prolongs depolarization/widens QRS
    • QRS duration 0.10-0.12sec (2.5-3sbox) = incomplete BBB
    • QRS duration >0.12sec (>3sbox) = complete BBB
19
Q

What are the indicators of a right branch block?

A
  • 2/2 right atrium strain (pulmonary embolism, rate-related aberration)
  • R’ (secondar R wave) in V1 (‘M’ pattern)
  • Slurred S in V6 (‘W’ pattern)
20
Q

What are the indicators of left bundle branch block?

A
  • 2/2 degenerative disease, cardiomyopathy, myocardial infarction
  • Deep S waves in right precordials (V1-3)
  • Broad/notched R wave in lateral leads (I, V5-6)
21
Q

Describe the progression of ECG abnormalities in myocardial ischemia/infarction.

A
  1. Minutes-hours: ST elevation
  2. Hours: Q-waves
  3. Hours-days: T inversions
  4. Days: ST-T changes resolve +/- continued Q waves
22
Q

What are other ST/T-wave abnormalities?

A
  • Short QT: hypercalcemia, hyperkalemia
  • Long QT: hypocalcemia, genetic, drugs
  • Inverted T waves: ischemia, LVH, digoxin
  • Peaked T waves: hyperkalemia
  • U-waves (image below): hypokalemia, hypomagnesium