Wrist and hand pathology/management Flashcards

1
Q

Instability Etiologies:

A

Microtrauma

Macrotrauma

Other

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Microtrauma

A

Gymnasts: Repetitive stress on the joints from intense training and high-impact activities can lead to instability.

Weight lifters: Heavy lifting can strain and potentially damage stabilizing structures around joints.

Manual therapists: Repeated manipulation and physical demands can cause joint stress over time.

Secondary instability
*injury from macro trauma and 
instability from micro trauma

Capsule gets more and 
more stretched - repetitive motion leads to instability

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Macrotrauma

A

FOOSH (Fall On Outstretched Hand): A common cause of joint instability due to sudden, forceful impact. Any significant trauma can potentially cause joint instability.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Other Factors

A

Neurological: Conditions affecting neuromuscular control can lead to joint instability due to impaired proprioception or motor control.

Rheumatoid arthritis: Chronic inflammation can weaken and damage the stabilizing structures around joints.

Hypermobility syndromes

Lack of tone - bigger problem in 
shoulder = less bony stability more soft tissue from muscle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Hypermobility syndromes:

A

Ehlers-Danlos Syndrome: A connective tissue disorder that often leads to joint hypermobility and instability.

Marfan’s Syndrome: A genetic disorder that affects connective tissue and can result in joint laxity.

We should know about it if they have it
*affects soft tissue extensibility
*we can’t “fix” it
*they don’t have stability from CT -> joint issues over time

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Beighton score items:

A

Passive Extension of the Little Finger: The examiner checks if the little finger can be extended backward beyond 90 degrees. (1 point for each hand)

Passive Extension of the Thumb: The examiner checks if the thumb can be placed against the forearm. (1 point for each hand)

Elbow Hyperextension: The examiner checks if the elbows can extend backward beyond 10 degrees. (1 point for each elbow)

Knee Hyperextension: The examiner checks if the knees can extend backward beyond 10 degrees. (1 point for each knee)

Forward Flexion of the Trunk: The patient is asked to bend forward with straight knees and try to touch the floor with their palms. (1 point if they can touch the floor)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Beighton Score Interpretation:

A

The total score is out of 9 points.

A higher score indicates greater joint hypermobility.

Score of 4 or more: Suggests significant joint hypermobility and may indicate a hypermobility syndrome, such as Ehlers-Danlos Syndrome.

Score of 6 or more: Often used as a threshold for diagnosing hypermobility syndromes in some guidelines.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

3 Pillars of stability

A

Bony, neuromuscular, capsuloligamentous

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Bony Stability:

A

contribution of the bone structure and its alignment to joint stability

Proper anatomical fit and congruence of the joint surfaces help in maintaining stability

If bones are not in right place - they won’t fit nicely in puzzle = NO arch

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Neuromuscular Stability:

A

role of muscles, tendons, and the nervous system in providing joint stability

ability of the muscles to contract and support the joint as well as the role of proprioception and neuromuscular control

Extrinsic m. = for the wrist = provide compression where 
bones fit together = maintain arch
*line of pull is mostly compression

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Capsuloligamentous Stability:

A

contribution of the joint capsule and ligaments to joint stability

provide passive support and restrict excessive movements

*proximal row ligaments = usually most devastating to wrist stability** scapholunate (more commonly injury)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Stability of the wrist:

A

Shape of carpal bones -> transverse carpal arch

Intrinsic muscle (interossei and lumbricals) activity to maintain the arch

Compressive loading from extrinsic muscles

Ligamentous constraints: the most important factor for wrist stability
> palmar radiocarpal ligaments
> dorsal radiocarpal ligaments
> intercarpal ligaments

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Loose 
Instability: Wrist sprain

A

MOI: FOOSH, trauma, taking weight through the hand, very common!

May have visible swelling
Pain at extremes of wrist motion
Pain with RI in most directions
Tender to palpation at joint line or over carpals

Differ. Dx- r/o fx, especially the scaphoid and lunate

Wrist is small - a little bit of swelling = can impact all motions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

fracture vs sprain tenderness:

A

*Tend to palpation = over ligament uncomfortable (kind of hurts), pinpoint over bone for fracture (pass out/throw up)

*Wrist sprain = hurts, can move don’t want to / fracture = natural thing to not move = something is wrong

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Loose 
Distal radio-ulnar joint instability (DRUJ)

A

Pain with pronation/supination

h/o fall, or impact sports, racquet sports

Increased laxity between the distal radius and ulna

Identified with (+) piano key test or radioulnar ballottement

Piano key – ulnar meniscal triquetral instability

Ballottement (def) – a palpatory technique for detecting or examining a “floating” object in the body

MOI
*FOOSH, impact sports
Think football someone running into you and you put your hands up
Racquet sports - violent supination/pronation or rotational force

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Loose 
triangular fibrocartilage complex (TFCC) disruption

A

Fall on supinated outstretched hand or due to chronic repetitive rotational loading

c/o medial wrist pain just distal to ulna = Does not travel, but can refer upward the length of the ulna

Pain increases with end range pronation/supination, and forceful gripping
*stability of wrist, extrinsic m. contribute to compression
*co-contract = compression of joint & ulnar deviation (position of power) - TFCC is loaded a little bit more

May have painful clicking w/wrist motion = *fibrocartilage area

Tenderness localized to dorsal anatomic depression distal to ulnar head

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

(TFCC) disruption
Acute Management:

A

Long-arm cast or splint fitted with the elbow in 90 degrees of flexion, and the forearm and wrist in ulnar deviation and extension for 6 weeks, if the TFCC is unstable

*stabilize up = rotation is occurring = pronation and supination needs to be limited so things heal - If just wrist is splinted - rotation will still occur at elbow

UE treatment proximal kinematic chain

After cast removal
> A/AAROM first emphasis on flexion/extension initially (straight plane), followed by pronation/supination, radial/ulnar deviation

> After 2 weeks can start strengthening if asymptomatic

If identified after the initial healing phase – bracing, strengthening, surgery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Loose Carpal Instability
Multi-link structure – Zigzag effect


A

Mobile proximal row “intercalated” between 2 rigid structures

Can get rotational collapse

Lunate most frequently dislocated- can dislocate dorsally (DISI scapholunate instability) or volarly (VISI-lunotriquetral instability)
**named for when lunate ends up

When you flex
*scaphoid flexed
*triquetirum extends
*Lunate stays put
= keeps them together

If scapho-lunate ligament ruptures
= no counterbalance = lunate goes with triquetrium into extension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Loose 
Scapholunate Instability (DISI)

A

lunate migrates dorsally

**Named based on what happens in flexion

FOOSH, with forces transmitted through the wrist in extension and ulnar deviation

Weakness with grasping objects

chronic vague wrist pain

+ tenderness over scaphoid and/or lunate
+ laxity between the scaphoid and lunate
+ scaphoid shear test
+ Scaphoid (Watsons) shift test

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Loose 
Lunate triquetral instability (VISI)

A

Second most common instability (VISI- lunate tilts ventrally)

Similar signs and symptoms to scapho-lunate instability, except for location

+ Ballotment test (lunotriquetral instability) Reagan’s Test

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Loose 
Mid carpal instability

A

Insufficiency of the intercarpal ligaments, may not be symptomatic, and pt may be able to sublux and reduce the jt at will

Clunk can be felt as the distal row of carpals jumps back into place at the end of ulnar deviation

Between proximal and distal row
*shearing them on each other

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Loose 
Acute management principles

A

Protect injury
Control pain and inflammation
Control /eliminate edema

Restore full painfree ROM in entire kinematic chain

Prevent ms atrophy

Minimize detrimental effects of immobilization

Scar management if needed
Maintain fitness

*some grade III’s can heal depending on where it is

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Loose 
Acute management swelling:

A

Swelling follows acute sprain, with degree of swelling corresponding to severity of injury
*swelling is a secondary problem

Pulling fluid out = more worried about chronic
*not in first 48 hours
Don’t drain joints early on - do if it impacts ROM and function

***Fluid is not the problem, what causes the fluid is the problem

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Loose 
Acute management splinting:

A

Immobilize the joint- custom or off the shelf splint that allows fingers to move, holds wrist in 10 degrees of extension

Mild sprain splint for 3-5 days, longer for moderate to severe sprain ice throughout the day,

After splint d/c : AROM, taping to provide support, decrease pain, strengthening

Times where you can move joint farther but swelling is the limiting factor
*not the ligament itself

Don’t give NSAIDs early on - don’t knock down inflammatory process in first 48 hours
*want the natural healing process to occur

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Loose 
Hypermobility/Instability

Exercise considerations

A

We work in diagonal 3D planes not straight planes

Functional oblique motion from radial ext (RD and ext) to ulnar flexion (UD and flex)

Dart-throwing motion – less scaphoid and lunate motion than during pure flex-ext

Stress reduced in the scapholunate interval

End range supination stresses the TFCC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Secondary consequence of instability

A

Transient neuro symptoms due to compromise of the carpal tunnel
> Loss of the transverse carpal arch

compression or irritation of the median nerve within the carpal tunnel

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

Symptoms of Carpal Tunnel Syndrome

A

Numbness: Especially in the thumb, index finger, middle finger, and part of the ring finger.

Tingling: Often described as a “pins and needles” sensation in the affected fingers.

Pain: May radiate up the arm or occur at night.

Weakness: Difficulty with fine motor tasks or grip strength.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Loose 
UCL sprain

A

Skier’s thumb, gamekeeper’s thumb, break-dancer’s thumb

UCL sprain- apply force to stress UCL; tested at 0- and 30-degrees flexion

+ test is pain and extreme laxity

Angulation 15 degrees greater than uninvolved side =30-35 degrees of motion- need for surgery

Risk of Stener lesion (torn UCL gets entrapped beneath adductor pollicis aponeurosis

complete rupture of UCL = possible of ends to get stuck under
*surgery is recommended for this reason
*if not in right some = won’t heal right = chronic instability

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

Loose 
UCL sprain management

A

Protect injury
Control pain and inflammation
Control /eliminate edema
Restore full painfree ROM in entire kinematic chain
Prevent ms atrophy
Minimize detrimental effects of immobilization

With thumb = you can wear just thumb splint
*not up to elbow

Limit valgus essentially = prevent it from going 
backwards

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Loose 
CMC OA

A

Distinguishing feature = loss of retroposition

Destruction of joint to the point where use stability from static stabilizers and bone

Splinting, stretch adductor pollicis, avoid adduction positions

Try to get it back to more functional position
*stretching
*avoid adducted position

31
Q

Splints (OTC)

A

Don’t cross MCP joint = can’t bend fingers = lose function
*immobilization would impact other joints

32
Q

Rheumatoid diseases / family

A

Chronic, progressive (flare-remitting), systemic, autoimmune

Affects the entire body; bilateral, symmetrical

Acute stage: pain, swelling, warmth, limited motion (commonly: MCP, PIP, wrist joint)

Jt inflammation ➔ soft tissue damage ➔ muscle weakness and imbalance ➔ pain, stiffness, joint damage, instability, deformity
> Ulnar deviation of the MCP joints
> Boutonnière deformity
S> wan-neck deformities of the digits.

33
Q

Ulnar drift

A

Ulnar drift happens in the MCP first then wrist = More distal to proximal - fingers first then ulnar deviation of wrist

Ulnar drift and palmar subluxation (pronation) due damage to collateral ligaments and extensor mechanisms = Alignment of extensors thrown off = bow like effect

Ulnar deviation of hand due to weakening of capsuligamentous structures of the MCP and “bowstring effect” of extensor communis tendon

34
Q

Ulnar drift Causes:

A

\synovitis, weakening of radial collateral ligament, ulnar displacement of extensor tendons, contracture of ulnar side intrinsics, dysfunction of radial side intrinsics, ulnar displacement of flexor tendons

35
Q

Boutonniere (“buttonhole”) deformity

A

Extended MCP and DIP, Flexed PIP

Rupture of central tendinous slip of the extensor hood

Common extensor tendon that inserts on the base of the middle phalanx is damaged (can also happen in sports: severe flexion of PIP or trauma to dorsal PIP ➔ damage to common extensor tendon)

36
Q

Boutonniere (“buttonhole”) deformity
Intervention (due to sports trauma):

A

4 weeks in a splint that holds the PIP joint in full extension, while allowing the DIP joint to flex.

Gentle AROM exercises can begin for flexion and extension of the PIP joint at 4–8 weeks, with the splint being reapplied between exercises.

General strengthening usually begins at 10–12 weeks. For a return to competition, an additional 2 months is required.

Don’t pull
on tendon
until it heals
slowly 
load 
tendon to 
help with 
remodeling and restore function

Tendons healing on bones = slow process

Hope extensor heals back downward = get function back

37
Q

Swan neck (recurvatum) deformity

A

Flexed MCP and DIP, Extended PIP

Least functional of all deformities

Contracture of intrinsics or tearing of volar plate

Destruction of the oblique retinacular ligament of the extensor mechanism leads to posterior (dorsal) displacement of the lateral bands of the extensor mechanism.

38
Q

Swan neck (recurvatum) deformity leads to:

A

an increased extensor force across the PIP joint with a resulting hyperextension of the PIP joint and chronic injury to the volar plate ➔ The extended position of the PIP joint stretches the FDS and FDP tendons. ➔ The pull on the FDP tendon causes a passive flexion of the DIP joint.

Over time volar plate gets more damaged as hyperextension increases

39
Q

Swan neck (recurvatum) deformity Intervention:

A

Application of a small figure-of-eight splint that prevents the PIP joint from fully extending, while still allowing full flexion range.

Splint that lets you flex but not extend

40
Q

Rheumatoid arthritis: Interventions

A

Stabilize the wrist!
Control inflammation

Focus on joint systems not isolated joints

Patients with stiff joints because of scarring do poorly after soft-tissue surgery. Patients in this group require aggressive and sustained therapy, often for 3–4 months.

Patients with joint laxity require careful intervention and control of the ROM and the direction of motion by the use of splints for many months after surgery.

41
Q

Stiff 
Joint Hypomobility

Related Pathologies and Etiology of Symptoms

A

RA /JRA, DJD, trauma, dislocation, fracture, surgery

Contracture and adhesion post-immobilization

42
Q

Stiff 
Post-traumatic/Post-fracture: 
Nonoperative Management

Management: Protection Phase

A

Educate the patient (e.g. length of symptoms, joint protection, activity pacing and modification; low but frequent repetition)

Reduce effects of inflammation or synovial effusion and protect the area (e.g. frequent controlled movement to reduce contracture; Grade I-II mobilization)

Maintain soft tissue and joint mobility (PROM, AAROM, flex/ext; pron/supin; multi-angle muscle setting)

Maintain integrity and function of related areas (shoulder, elbow exercise/activities; elevate if with swelling/edema)

Control pain and protect joints

Maintain joint and tendon mobility and muscle integrity

43
Q

Stiff 
Post-traumatic/Post-fracture: 
Nonoperative Management

healing potential:

A

Closer it heals to thumb = better healing potential

Closer to ulna = worse potential to heal

44
Q

Stiff 
Post-traumatic/Post-fracture: 
Nonoperative Management

Management: Controlled Motion Phase

A

Increase soft tissue and joint mobility
> Joint mobilization (non-thrust – grade I-IV)
> Manual stretching & self-stretching with weight; low intensity, long duration; dynamic splint
> Home instructions

Improve muscle performance and functional abilities
> Low load exercises in open and closed chain
> Control and coordination
> Muscle endurance + strength; involve other joints proximal to the elbow; progress to functional activities

45
Q

Stiff 
Post-traumatic/Post-fracture: 
Nonoperative Management

Management: Return to Function Phase

A

Improve muscle performance
> Progress strengthening exercises; progress to simulate daily activities e.g. pushing, pulling, lifting, carrying, gripping

Restore functional mobility of joints and soft tissues (manual or mechanical stretching; joint mobilization)

Promote joint protection
> E.g. chronic RA  modify high load activities

46
Q

Stiff 
Wrist Arthroplasty
Indications for Surgery:

A

Severe pain

Deformity causing imbalance of soft tissues

Subluxed/dislocated radiocarpal joint

Low demand UE functional needs

Bilateral wrist involvement and arthrodesis will not improve function

Anytime you replace a joint
*stability over mobility
Will NEVER get full ROM after a replacement
*too much motion is worse than too little motion

47
Q

Stiff 
Wrist Arthroplasty

Postoperative Management

A

Immobilization
Exercise
*** Stability of the wrist takes priority over mobility (only 35 dg flex is needed)

Outcomes
> Pain relief
> Wrist and forearm ROM, strength, and function

48
Q

Total joint arthroplasty – principles

A

Total joint arthroplasty is considered when all other management has been exhausted

Primarily for pain reduction

Full restoration of ROM is NOT expected

Quality of life measure

49
Q

Stiff wrist/hand management principles

A

Restore ROM
Restore accessory motions, joint mobility
Restore strength
Consider that strength within the available range will be greater than strength available within the newly gained range

Strengthen through the full arc of motion!

Restore upper limb function
Task specific, work specific, sports specific

50
Q

Painful (just a few examples)**

A

CTS (median n), AIN, pronator syndrome
Ulnar n entrapment (cubital tunnel or guyon’s tunnel)
DeQuervain’s tenosynovitis
RA

51
Q

Tendinopathy – etiology and classification

Reactive vs degenerative

A

Inflammation presence:
Reactive Tendinopathy: Inflammation is present and contributes to the acute response. This inflammation can be addressed with rest, ice, and anti-inflammatory medications.

Degenerative Tendinopathy: Inflammation is less prominent. The focus is more on addressing structural changes and improving tendon health through rehabilitation and strengthening exercises.

Onset and duration
> Tendinitis resolves more quickly
> Tendinosis can become more reticent to change

52
Q

Reactive Tendinopathy

A

result of acute overload or excessive mechanical stress on the tendon

acute tendon swelling and increased cellular activity

increased vascularity and a reactionary inflammatory response without significant structural changes in the tendon

Symptoms usually resolve relatively quickly with appropriate rest and reduction in stress

53
Q

Degenerative Tendinopathy (Tendinosis)

A

Chronic overload or repetitive stress over time, often associated with aging or long-term use of the tendon

influenced by poor biomechanics or inadequate recovery

Characterized by a breakdown of collagen fibers, disorganized tendon structure, and an increase in non-inflammatory cells and matrix

Persistent pain, stiffness, and decreased functional capacity of the tendon

54
Q

Tendinopathy – etiology and classification

Load

A

Too much load or too little load-bearing capacity

Rest-activity cycle - Tendon undergoing micro damage
metabolism undergoing to tendinoses
*lose mechanical properties

Decreased collagen integrity and vascular supply with increased age

55
Q

Micro-Damage and Repair

A

Micro-Damage: Repetitive or excessive loading of the tendon leads to micro-damage in the collagen fibers. This damage is usually minor and is part of the natural cycle of tendon use and repair.

Repair Mechanism: Tendons have a limited capacity for self-repair. When subjected to normal, controlled stress, the tendon undergoes a repair process where damaged fibers are replaced and collagen synthesis occurs.

During rest, the metabolic processes focus on repairing the micro-damage and restoring tendon structure

56
Q

Principles of tendon loading

A

Progressive loading of tendon facilitates collagen synthesis = High quality collagen synthesis - need progressive
loading to get it

Tensile loading can be imparted with both active muscle contraction or passive stretching

Volitional activation can be isometric, concentric or eccentric

Follow the soreness rules

Tendons only work through tensile forces
*w/in tendon itself compression is not created
*only works by pulling

57
Q

soreness during warm-up that continues:

A

2 days off, drop down 1 level

58
Q

soreness during warm-up that goes away:

A

stay at level that led to soreness

59
Q

soreness during warm-up that goes away but redevelops during session:

A

2 days off, drop down 1 level

60
Q

soreness the day after lifting (not muscle soreness):

A

1 day off, do not advance program to the next level

61
Q

no soreness:

A

advance 1 level per week or as instructed by healthcare professional

62
Q

Painful
Carpal tunnel syndrome (CTS)

A

Most common compression neuropathy

Carpal tunnel pressure on median nerve, may result from decreased tunnel space due to fluid retention (common during pregnancy, or infection, or renal dysfunction), gout, pseudo gout, can also decrease tunnel size, RA, collagen disorders, c-spine pathology, diabetes, hypothyroidism

Symptoms: Numbness, tingling, and pain in the thumb, index finger, middle finger, and part of the ring finger. Symptoms are often worse at night and can include weakness or clumsiness in the hand.

Can have transient symptoms from compression

Fluid going through the tendon as well
*helps with lubrication and protection
*general amount - not much space in wrist - lots of swelling = no ability to expand

63
Q

Half of CTS cases related to:

A

repetitive and cumulative trauma in the workplace; extreme positioning of fingers and wrist, forceful and repetitive loading, especially of finger flexors can provoke CTS

Wrist in more opposition ——> chronic compression

There is a fat pad
*but not a lot of protection here
*nerve can become easily compressed

64
Q

Painful 
Carpal tunnel syndrome (CTS)
symptoms:

A

Numbness pattern:
Volar thumb, index, long, radial ½ of ring

Pain :
Nocturnal pain/numbness

Motor weakness pattern:
Thenar muscles, esp APB, OP, FPB (superficial head) lumbricals 1 & 2
in late stages more severe, visible atrophy

OTHER:
Dropping objects
*Lose grip strength - tell tale sign!!

65
Q

CTS (Examination)

A

Use Semmes-Weinstein monofilament testing

Use Phalen, Tinel, and carpal compression test

Clinicians should assess and document patient age (older than 45 years), whether shaking their hands relieves their symptoms, sensory loss in the thumb, the wrist ratio index (> 0.67), and scores from the Boston Carpal Tunnel Questionnaire- symptom severity scale (CTQ-SSS) (> 1.9).

The presence of more than 3 of these clinical findings has shown acceptable diagnostic accuracy.

Use Purdue Pegboard Test or the Delon-modified Moberg pick-up test

Do not use lateral pinch strength testing as outcome measure (surgical or non-surgical)

Do not use grip strength to assess <3 mo. change post surgery

May assess grip strength and 3-point or tip pinch strength to compare with norms

66
Q

CTS (Intervention)

A

Recommend a neutral-positioned wrist orthosis worn at night for short-term symptom relief and functional improvement for individuals with CTS seeking nonsurgical management.
**Avoid sustained flexed wrist posturing

Do not use low-level laser therapy

Do not use thermal ultrasound (mild-moderate CTS)

Do not use iontophoresis (mild-moderate CTS)

Do not use magnets

May perform manual therapy for the cervical spine and UE (mild to moderate CTS in the short term.)

FROM THE CPG - night splint to wear at night
*keeps you from flexed position so you don’t compress the median nerve even more

There is conflicting evidence on the use of neurodynamic mobilizations for mild to moderate CTS.

May use a combined orthotic/stretching program in individuals with mild to moderate CTS who do not have thenar atrophy and have normal 2-point discrimination. Clinicians should monitor those undergoing treatment for clinically significant improvement.

67
Q

Double Crush Syndrome Overview

A

peripheral nerve is compressed or irritated at more than one location along its path, such as at both the cervical spine and the carpal tunnel

68
Q

Painful 
DeQuervain’s tenosynovitis

A

50+ usually, can be younger

Progressive stenosing tenosynovitis affecting the tendon sheaths of the first dorsal compartment of the wrist

Entrapment of the tendons due to thickening of the sheaths, Sheath around tendon, gets thick = tendon can’t slide nicely through
*over time gets stuck
*sheath gets so thick = tendon can’t go through
*tendon shortens over time

Compression increases with ulnar deviation

Severe pain with wrist ulnar deviation and thumb flexion and adduction, and with thumb extension and abduction

Crepitus may be present

69
Q

Painful 
DeQuervain’s tenosynovitis positive tests

A

+ Finkelstein’s test
+ Eichoff’s test
+ WHAT test

70
Q

Painful 
DeQuervain’s tenosynovitis positive tests
s/sx

A

APL and EPB tendons

Overuse, repetitive tasks, overexertion of the thumb or radial/ulnar deviation of the wrist predisposing factors

c/o gradual onset of dull pain in the region of the radial styloid and wrist which can radiate proximally into the forearm and distally into the thumb

Pain and swelling at the base of the thumb, which can worsen with thumb movements or gripping. There may be tenderness in the first dorsal compartment

71
Q

Pronator Syndrome

A

Affected Nerve: Median nerve.

Symptoms: Similar to CTS but can also include pain in the forearm. Pain and symptoms are often exacerbated by pronation of the forearm and can also involve sensory and motor dysfunction.

Cause: Compression of the median nerve by the pronator teres muscle or other structures in the forearm.

72
Q

Anterior Interosseous Nerve (AIN) Syndrome

A

Affected Nerve: Anterior interosseous nerve, a branch of the median nerve.

Symptoms: Motor symptoms only, including weakness or paralysis of the flexor pollicis longus, flexor digitorum profundus (to the index and middle fingers), and pronator quadratus. There is typically no sensory loss.

73
Q

Principles of tenosynovitis management

A

Principles of severity and irritability

Protection and relative rest if severe and highly irritable

Progression based on response to loading

Tendon glides within its sheath

Synovial fluid

Promote the mobility of the tendon within its container

Promote bathing the tendon in the synovial fluid

Tendon gliding -> Gentle AROM

74
Q

Splints (OTC)

A

Thumb splints
*designed to prevent flexion from occurring
*strains EPB
*minimize and utilize from contractile standpoint