Wound Management Flashcards
Lipodermatosclerosis
Inverted Champagne Bottle Legs
70% of Chronic Wound is caused by?
Chronic Venous Insufficiency
Signs of Chronic Venous Insufficiency
- Pitting Oedema2. Haemosiderin Staining3. Venous ulcer - painless, irregular, copious exudate4. Atrophie Blanche5. Lipodermatosclerosis
RF of Chronic Venous Insufficiency
Obesity, DVT, Poor mobility
Tx of Chronic Venous Insufficiency
- Graduated compression - toe to knee (ankle 30mmHg)2. Address factors that delay healing3. Must exclude arterial involvement
4 Principles of Wound management
- Define Aetiology - Vascular/Mechanical/Neuropathic/Infective2. Control wound healing factors3. Select appropriate dressing4. Plan for management
Signs of lymphoedema
- Scale and keratin build-up2. Skin thickening3. Hard to pick up skin4. Non-pitting oedema
Secondary causes of Lymphoedema?
- Cancer2. Infection - Filariasis (Elephantiasis)
4 Si/Sx of Peripheral Arterial Disease (PAD)
- Claudication to rest pain2. Lower ABI - nml is 1.03. Weak pulse, poor refill4. Arterial ulcer - regular, punched out, below ankles
Tx of Arterial ulcers
- Improve flow - angioplasty/stent/bypass (essentially angina of leg)2. Amputation of digit or limb
Difference between Ischaemic or neuropathic ulcers?
Neuropathic is:1. Painless2. Bony prominence/area of pressure3. Good circulation for healingWhich are all opposite in ischaemic
RF and Mx for Pressure ulcer
- Neuropathy 2. Immobility 3. Malnutrition Mx: manage RF, risk assessment, Foam
Friction vs Shear
Friction - epidermis worn away by rubbing external surfaceShear - skin is restrained from sliding while tissue are forced to move
4 Stages of Pressure Ulcer
- Erythema remains after light pressure2. Skin loss involving epidermis/dermis3. Skin loss involving subcutaneous tissue/fascia4. Skin loss with necrosis to underlying structures (muscle/bone/joint etc)
Complication of leg ulcers
- Infection2. Gangrene3. Calcification4. Neoplastic development