Wound Care, Abx & Immunisations Flashcards

1
Q

Wounds types & healing: 6 & 4

A

Traumatic:
- abrasion; epithelialisation & healing under scab
- cut; primary intention
- stab; primary intention
- impalement; primary intention followed by surg intervention
- laceration; secondary intention OR primary after debribement

Iatrogenic:
- incision; epithelialisation
- puncture; primary intention
- acid/alkali burns; primary intention
- split skin removal; primary after surg intervention

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Healing types & closures

A

Epithelialisation; epithelium growths over a denuded surface

Primary: edges can be approximated & wound actively closed using techniques

Secondary: edges cannot be approximated & wound needs to heal from bottom thru granulation; left open & wound closes naturally

Tertiary: delayed primary closure due to infection risk, wound closed w techniques after time

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

HEIDI

A

H- Hx; wound, Pt, environment
E- Examination; Pt’s localised skin, circulation, etc
I- investigation; consider pathology, radiology, haematology or sonography
D- Dx; aetiology/pathophysiology
I- Implement; immed & long term

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

TIME

A

T- Tissue; non-viable tissue (slough/necrosis), foreign bodies

I- Inflammation; & infection

M- Moisture; imbalances, oedema, temp, pH

E- Edges; rolled, raised, undermined, calloused

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Wound Exam general

A
  • location
  • type
  • dimensions
  • tissue types/wound bed
  • exudate type & amount
  • odour
  • wound edges
  • condition of surrounding skin
  • pain
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Tissue types & wound bed

A

Epithelialisation: pink, often irregular, at wound edges

Granulation: Red, bumpy, associated w healing wound (reducing size)

Agranular: Red/pink/pale; smooth; not healing (not reducing)

Slough: white-yellow; smooth; soft, sloppy & stringy to tenacious & adherent

Gelatinous slough: firm, gelatinous yellowish coating over the wound

Necrosis: black/tan; hard & dry OR moist & leathery

Hypergranulation: friable; spongy; exceeds over the wound edges

Fat: white-yellow globular

Tendon: white-yellow striated

Bone: white-yellow hard

Others; sutures, mesh, tumour

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Clinical Pathway Wounds

A

1- Irrigation & wound cleaning
2- x-ray for foreign bodies PRN
3- primary or delayed primary closure
4- dressing PRN
5- analgesia PRN
6- ADT PRN
7- referral & Pt info

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Suture/Staple location & days

A

Face - 3-5
Neck - 3-4
Upper extremity - 7-10
Hand - 10-14
Chest - 7-10
Back - 10-14
Buttocks - 10-14
Legs - 8-10
Foot - 10-14

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Pressure injury classifications

A

Stage I- intact skin w unblanchable redness, usually over bony prominence; pigmented skin may be of diff colour than surrounding
Stage II- partial thickness loss of dermis; shallow ulcer w a red-pink bed but no slough; can present as intact/ruptured serum filled blister
Stage III- full thickness tissue loss; subcut fat may be visible, but no bone, muscle or tendon; slough, if present, does not obscure the depth of tissue loss; ulcer edge may be undermined
Stage IV- full-thickness tissue loss w exposed bone/muscle/tendon; may have slough, eschar, undermining, or tunnelling
Unstageable - full-thickness tissue loss & base of ulcer is covered by slough or eschar

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Venous Ulcers

A
  • often painful: elevation may help, often worsened by dependency of limb
  • poss Hx of varicose veins, oedema that is worse at the end of the day, haemosiderin staining
  • typically in the gaiter area (lower 1/3 of medial/lateral leg above the malleoli); edge is usually sloping & may be irregular; base maybe be granulating & sloughy; may be heavy exudate
  • surrounding skin: pale scarring, venous dermatitis, or eczema
  • unless comorbid arterial comprise, pulses should be normal

Rx:
Compression therapy: graduated comp therapy improves healing rate; reduces recurrence; must assess ankle brachial pressure index (ABPI) prior to use!
Ulcer dressing: local wound conditions should be optimised for healing; lock away fluid dressing, intact/active for 1 wk, low profile & addresses local wound cond to promote moist healing
Pain management: mod to sever pain can impact ability to tolerate compression; manage pain!
Leg elevation: can aid venous return, reduce pain, reduce oedema; encourage during inactive periods
Exercise: reg gentle exercise (walking, dorsiflexing foot) to maintain calf pump function
Skincare: maintain skin integrity of lower limbs, prevent dryness w bland & simple moisturisers
Pt Ed: about disease process & how manage it

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Arterial ulcers

A
  • all Pts must be assessed for PAD (peripheral arterial Dx)
  • often painful, aggrav+ by elevation, but relieved by dependency
  • other features: vasc+ Dx, signs of ischaemia, pallor on elevation, redness on dependency, abnormal ABPI
  • usually on toes, bony prominences, sides of feet or heel; edge may appear punched out; base may be sloughy or covered by eschar; usually minimal exudate

Rx
Ulcer dressing: no specific perfect dressing, select to suit wound
Pain management: very common & may req pain relief prior to dressing changes
Pressure area management: ? Referral to podiatrist to assess feet & footwear; advise Pt to wear well-fitting, closed footwear

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Sterile technique

A

Free from microorganisms
Meticulous hand washing, use of sterile field, sterile gloves & dressing & instruments
Sterile to sterile rule!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Clean technique

A

Free of dirt, marks or stains
Meticulous hand washing, maintain a clean environment, using clean gloves & sterile instruments, preventing direct contamination of materials/supplies
NO sterile to sterile rule applies
Appropriate for long-term care, home care, & some clinical settings; for Pt’s not high risk for infection; for Pt’s receiving routine dressings for chronic wounds

Eg; removing staples/sutures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Aseptic technique

A

Free from pathogenic microorganisms
Purposeful prevention of the transfer of organisms from one person to another by keeping the microbe count as low as poss

Egs; wound care, wound closure, insertion of catheters or PEG tubes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

No touch technique

A

Method of changing dressings with directly touching the wound or an surface that might come in contact w the wound
Clean gloves w sterile solution/supplies/dressings

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

LACERATED

A

L-Look: consider time of injury, depth, potential for retained FB & note any functional loss
A-Anaesthetise: choice of drug, max safe vol, ability to explore wound thoroughly
C-Clean: consider best wound decontamination method
E-Explore: satisfy yrself that the wound is suitable for closure
R-Repair: consider best approach to closure & Pt positioning
A-Apply: apply an appropriate dressing
T-Tetanus: check Pt’s status & consider ADT in relation to wound
E-Educate: Pt about ongoing wound care, complications, & removal of sutures
D-Documentation: wound assessment, repair & advice

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Everting sutures

A

Simple continuous running: to close multiple layers with one suture; not cut till the end

Continuous locking (blanket): single suture is passed in & out of tissue layers & looped thru free end before passing thru the next - holding the prev one in place

Simple interrupted: each individual stitch is placed, tied & cut in succession

Horizontal mattress: stitches are placed parallel to the wound edges

Vertical mattress: uses deep & superficial bites, each crossing the wound at right angles. Works effectively for the approximation of edges of deep wounds

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Skin closure tapes: Adv, Ind & technique

A

Advantages:
- rapid application, little/No Pt discomfort, low cost, no needle injury risk, Allows skin to breathe without moisture collecting under strip, less risk of irritation, no injection req

Indications:
- Minor injuries; lacerations/incisions
- Provide additional tension for wounds closer with Intracutaneous sutures
- Where early replacement of staples/sutures is req to improve cosmetics results
- suitable for thin, fragile or sensitive skin

Technique:
- prepare skin w tincture if benzoic compound to aid adhesion
- place strips with sufficient space between each to allow drainage of fluid
- tell Pt to keep area dry for 72 hrs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Cyanoacrylate glue: Adv, Ind, Technique

A

Adv:
- easy, fast, no pain, no removal/injected anaesthesia req

Ind:
Clean, short <5cm lacs under low tension & w good approximation

Technique:
- hold wound edges in opposition w fingers of non-dominant hand/forceps
- apply thin layer of adhesive across the entire wound, including a margin of at least 5-10mm on either side of wound
- hold for at least 30 secs before removing pressure/tension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Staples: Adv, Ind & Technique

A

Adv:
- ease of use, rapidity, cost-effective, minimal damage to host, low infection risk, off strong closure

Ind:
- scalp lacs that do not req extensive haemostasis
- linear non-facial lacs caused by shear forces (sharp objects)
- can also be used high on trunk

Technique:
- before inserting, it is important to line up wound edges w the centreline indicator on stapler head
- typically use forceps to evert & precisely line up for each staple

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Bites & Clenched fist Injs: high risk x5

A
  • wounds w delayed presentation (>8hrs)
  • puncture wounds that cannot be debribed properly
  • wounds; hands, feet, face
  • wounds involving; bones, joints, tendons
  • immunocompromised Pts
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Bites & Clenched fist Injs: Abx Mild [common: CC : or MD/T

A

Amoxicillin + Clavulanate
875 + 125mg PO 12 hrly

Pts sensitive to penicillins:
Ciprofloxacin 500mg PO 12 hrly,
PLUS:
Clindamycin 450mg PO 8 hrly
OR:

Metronidazole 400mg PO 12 hrly
PLUS:
Doxycycline 200mg PO 1st dose & 100mg PO daily
OR:
Trimethoprim + Sulfamethoxazole 160 + 800mg PO 12 hrly

Duration: 5/7

23
Q

Bites & Clenched fist Injs: Abx Mod-Sev [M PO + C/C IV]

A

Metronidazole 400mg PO 12 hrly
PLUS:
Ceftriaxone 1gm IV daily
OR:
Cefotaxime 1gm IV 8 hrly

For severe/penetrating wounds, duration:
14/7 & reps combo IV & PO

24
Q

Boils & Carbuncles: Rx & Abx options

A

Usually stand-alone incision & drainage is sufficient
Spreading cellulitis/systemic symptoms:
- incision, draining & Abx
- wound swab prior to Abx Rx

Abx:
Di/flucloxacillin 500mg PO 6 hrly
OR (Pen sens):
Cephalexin

Duration: 5/7

Modify post lab results

25
Q

Cellulitis: risk factors x 9, 3 x notes

A
  • lymphoedema
  • skin breakdown/site of entry
  • venous insufficiency
  • leg oedema
  • obesity
  • neutropenia
  • immunocompromised
  • chronic renal Dx
  • cirrhosis

Approx 80% cases gram +ve (MRSA, streptococcus progenies/other streptococci)

  • purulent cellulitis, an abscess may or may not form
  • systemic signs & findings of leukocytosis & bacteraemia more typical in immunocompromised Pts
26
Q

Cellulitis: man* x 3, Abx; mild, mod, severe

A

Management:
- rest & elevate affected area
- incision & drainage of any abscess found
- use non-adherent dressings

Mild early cellulitis:
di/flucloxacillin 500mg PO 6 hrly
Pen Sens:
Cephalexin 500mg PO 6 hrly

Duration 5-10/7

Purulent cell*:
Mild - nil
Mod - trimethoprim OR doxycycline

Severe or not improving after 48hrs:
IV flucloxacillin (cephazolin for Pen Sens)

27
Q

Impetigo types: x 2, Aff Vs Remote

A

Most often in children

Crusted or non-bulbous impetigo - yellow crusts & erosions that are irritating/itchy but not painful

Bulbous impetigo - irritating blisters that erode rapidly, usually caused by Staph Aureus

Affluent comm:
1- staph A
2- streptococcus pyogenes

Remote comm:
Streptococcus pyogenes is main culprit

28
Q

Impetigo man*: non-remote/remote/recurring

A

Non-remote Comm:
Localised skin sores - mupirocin 2% ointment topically to crusted areas 8hrly 7/7
Multiple skin sores/recurrent - di/flucloxacillin 500mg PO 6hrly up to 10/7 (can be ceased upon resolution)
Pen Sens: Cephalexin

Remote communities (central & Nth Aus)
Benzathine penicillin 900mg IM single dose
OR;
Trimethoprim + sulfamethoxazole 160 + 800mg PO 12hrly 5/7
OR;
Trimethoprim + sulfamethoxazole 320 + 1600mg PO daily 5/7

Recurrent impetigo/resistant organism:
Trimethoprim + sulfamethoxazole 160 + 800mg PO 12hrly 5/7
OR;
Trimethoprim + sulfamethoxazole 320 + 1600mg PO daily 5/7
- if not responding, modify therapy according to culture results

29
Q

Diabetic foot inf* info: acute Vs chronic, active - assess & R?

A

Acute infections: staph A or streptococci - if not on recent antimicrobials

Chronic infections: polymicrobial; involving gram +ve & -ve, aerobic & anaerobic organisms

Active infection: wound cultured recommended to guide therapy

Assess vascular supply to affected foot

Refer for surg assess/debribement if wound needing investigation

30
Q

Diabetic foot infection: man* ABX; mild-mod Vs severe

A

Refer for surg assess/debribement

Mild - Mod (no osteomyelitis/septic arthritis):
Amoxicillin + clavulanate 875 + 125mg PO 12hrly 5/7
OR;
Cephalexin 500mg PO 6hrly PLUS Metronidazole 400mg PO 12hrly 5/7

Pen Sens:
Ciprofloxacin 500mg PO 12hrly 5/7
PLUS Clindamycin 450mg PO 8hrly 5/7

Severe/systemic toxicity:
- septic shock, bacteraemia, necrosis/gangrene, deep ulceration, severe cellulitis, osteomyelitis/septic arthritis
Aggressive IV Abx req!!

31
Q

Wound infections: surg site; man* & Abx; mild-mod Vs Severe

A

Collect swab for gram stain & cultures
Minor: often drainage & irrigation w NaCl will resolve issue

Mild- Mod w assoc cellulitis:
DI/flucloxacillin 500mg PO 6hrly 5/7 (pen Sens: Cephalexin)

Gram -ve suspected:
Amoxicillin + clavulanate 875 + 125mg PO 12 hrly 5/7

Severe (w systemic features):
Flucloxacillin 2gm IV 6hrly
Pen Sens: cephazolin IV 8hrly
+ also Gram -ve: add gentamicin

32
Q

Wound infections: post-traumatics: x 4, Abx; mild Vs severe & prophylactic

A
  • Ensure tetanus up to date
  • Clean & debribe carefully
  • Immobilise & elevate
  • Abx Not routinely req, but prophylactic for contaminated wounds

Likely pathogens:
Staph A, strep pyogenes, clostridium perfringens, & aerobic gram -ve
Penetrating foot injury: pseudomonas aeruginosa is common

Management:
Abx if >8hrs old or difficult debribement

Mild:
Amoxicillin + clavulanate 875 + 125mg PO 12hrly 5/7
Pen Sens: Cephalexin 500mg PO 6hrly
PLUS metronidazole 400mg PO 12 hrly 5/7

Severe:
Cephazolin 2g IV
PLUS; metronidazole 500mg IV 12hrly
OR; alternatives in ATG

33
Q

Wound infections: non-contaminated

A

Abx gen not req
Yes if; > 8hrs or debridement difficult

34
Q

Wound infections: contaminated/infected Abx

A

Mild:
Amoxicillin + clavulanate 875 + 125mg PO 12hrly
Pen Sens: Cephalexin 500mg PO 6hrly PLUS metronidazole 400mg PO 12hrly

Severe:
Cephalexin 2g IV 8hrly
PLUS
Metronidazole 500mg IV 12hrly
OR ATG alternatives

Switch to PO ASAP & totally IV + PO 5-7/7

35
Q

ENT: acute pharyngitis &/or tonsillitis; high risk Pts x 3, & other causes x 2

A

ATG high risk Pts for Abx
- 2-25 yo w sore throat in communities w high incidence of acute rheumatic fever (indig central & Nth Aus, Māori & Pacific Islander peops)
- Pts w existing rheumatic heart Dx
- Pts w scarlet fever

Other causes of acute pharyngitis:
Epstein-Barr virus (no Abx req),
Peritonsillar abscess, & rarely, diphtheria (req Hosp!)

36
Q

ENT: Acute epiglottis

A

Relatively rare in children in Aus

Adults presenting = urgent management & hosp

37
Q

ENT: otitis externa ; Rx & other x 1

A

AKA: swimmers ear
Most commonly: pseudomonas aeruginous & S Aureus
Rx: keep ear dry, refer to GP for management & ear drops

Boil: can cause localised otitis externa, Abx can be prescribed

38
Q

ENT: otitis media

A

Dx difficult as inflammation of middle ear often assoc+ w viral URTIs

Rx: referral to GP is wise for assess/manage

39
Q

ENT: pertussis; S&S, Req*, Rx

A

Caused by bordetella pertussis
Typically present w; cough &/or paroxysms of coughing (inspiratory whoop or post-tissue vomiting)
Infants <6 months are at greatest risk of morbidity/mortality
Notifiable disease in Aus

Rx: azithromycin OR trimethoprim + sulfamethoxazole OR clarythromycin
Infected individuals req 5/7 Abx before they’re allowed to mingle w others

40
Q

ENT: acute rhinosinusitis: Def x3, Rx x 1;5, ?Abx 3;1

A

Rapid onset inflam+ of nose & paranasal sinuses w 2 or more additional symptoms:
- nasal blockage
- nasal discharge
- facial pain/pressure
- reduction/loss of sense of smell

Most common form is Viral, w acute bacterial Inf* a complication

Rx:
Viral usually resolves within 7-10/7 without Rx
OR poss Rx;
- adequate & regular oral analgesia (paracetamol, NSAIDs)
- saline nasal sprays
- topical nasal corticosteroids
- topical decongestant for no more than 3 consecutive days
- topical ipratropium bromide may lessen rhinorrhoea

?Abx:
- Pt’s w symptoms lasting >7/7; w purulent discharge, sinus tenderness, or maxillary toothache
OR; Pt has severe symptoms & high fever @ onset lasting >3/7
OR; worsening of symptoms after initial improvement

Abx:
Amoxicillin + clavulanate
OR see ATG

41
Q

Resp: Viruses

A

SARS - coronavirus from 2002

Avian influenza - influenza A (H5N1) re-emerged in Vietnam 2003

All standard precautions: airborne & droplet & contact

COVID 19 - follow current advice

Influenza - A, B, or C (A & B more common) w changes in their surface antigens involving H (cell attachment) & N (viral shedding) glycoproteins

Assessment:
- Hx - age, immunisation status, chronic health cond, immunocompromised, pregnancy, smoking
- Onset - incubation period of 1-3 days
- S&S - malaise, feverishness, chills, headache, anorexia, nasal discharge, sneezing

Severe cases: IV fluids & resp support or vasopressors
- those w pneumonia will req Abx

42
Q

Resp: TB: def; 1 & 2*, Dx, Rx

A

Aerobic bacteria, usually gram neutral
Incubation: from infection to primary lesions 4-12/52
- degree of transmission depends upon No of bacilli in droplets, virulence of bacilli, bacilli exposure to sun or UV, & opportunities for aerosolisation

Usually affects lungs, but can affect any organ.
Primary Pulmonary TB: initial infection that forms primary lesion & usually resolves spontaneously (10% chance of developing active TB during lifetime)
Secondary Pulmonary TB: endogenous reactivation of latent infection, usually localised to upper lobes

Dx: usually by S&S, chest X-ray & skin tests

Rx: may be protracted w first line & second line therapeutic agents (see ATG)

43
Q

Resp: CAP: Ass x 4, Red Flags x 6, Abx: mild-mod Vs severe

A

Most commonly Strep pneumoniae, rarer mycoplasma pneumoniae, chlamydia pneumoniae & legionella
Haemophilus influenza <5% of CAP, often pre-existing COPD

Ass:
- Hx & exam
- refer for chest X-ray,
- O2 sats/blood gases
- investigate pathogen: ? Sputum sample, blood cultures, or nose/throat swab

Red flags:
- RR >30 bpm
- BP <90 mmhg
- O2 sats <92 %
- HR >100 bpm
- acute onset confusion
- multi-lobar involvement on X-ray

Abx:
Mild - mod: usually penicillin based
Severe - broader spectrum empirical Abx
Flow chart on Aus Abx therapeutic guidelines

44
Q

Sepsis: S&S x 4, severe x 3, Man* x 4

A

1* causes: resp, GU, infra-abdominal, skin & 1* bacteraemia

Systemic signs: (2 or more!!)
- abnormal temp - >38 or <36
- tachycardia >90 bpm
- tachypnoea >20 bpm or PaCO2 <32 mmHg
- abnormal WCC - >12000/microL or <4000/microL or >10% immature (band) cells

Severe sepsis:
- plus organ dysfunction: SBP <90 mmHg or MAP <70 mmHg, elevated lactate, or oliguria

Septic shock:
- persisting hypotension despite Rx w fluids

Management:
Haemodynamic resus:
- adequate fluid resus before vasopressors or inotropes
- norad, adrenaline or vasopressin (?Clin guidelines)
- maintain MAP >65 mmHg
Supportive measure for perfusion:
- ETT & mechanical vent w approp sedation & paralysis
Early Abx:
- collect approp cultures & commence within 1hr of presentation
- choose approp Abx (source, Pt factors, renal function, immunocompromise)
- initial course: Broad enough to cover likely pathogens
Source control:
- control/contain: drainage, debribement, repair

45
Q

Foodborne illness: V Vs B, S&S x 2, Man* x 2 + 1 x 7, Rx x 3

A

Viral causes: norovirus, rotavirus, astrovirus
Bacterial: salmonella, C Diff, C perfringens, campylobacter, yersinia, cryptosporidium, Giardia, E Coli

Norovirus: seasonal, winter, usually 24-48 hrs; viral shedding occurs for 24hrs post cessation of symptoms

Presentation:
- nausea, Vomiting, diarrhoea, abdo cramping
- systemic: fever, dehydration & malaise

Management:
- Dx: comprehensive Hx - most self limiting, though could also be sign of more complex prob
- electrolytes & FBC - Pt’s w prolonged symptoms
Stool samples for:
- watery diarrhoea & signs of hypoVol
- bloody diarrhoea
- fever >38.5*C
- elderly >70 yo
- immunocompromised
- pregnant women
- pt’s w comorbidities (eg; IBS)

Rx:
- appropriate PPE!!
- supportive care: antiemetic or antimotility (loperamide)
- maintain hydration: glucose containing solution (eg; gastrolyte) or parenteral dehydration (severe dehydration or continued V or unable to tolerate oral fluids)

See AATG for management of specific pathogens

46
Q

Gastroenteritis in Aged Care: S&S x 3, Man* x 3

A

S&S:
- >/= 3 loose bowel motions above baseline in 24hrs
- >/= 2 episodes of vomiting over 24hrs
- nausea, V, D, abdo pain/tenderness in Pt who has stool based pathogen detected

Management
- early recognition
- infection control practices
- Rx & contain at NH

47
Q

UTIs: uncomp Vs comp, S&S x 4 conds, invest* x 2, Man* x 4 cond

A

Uncomp: healthy person w normal urinary tract
Comp: anatomical abnormality, urinary obs or incomplete bladder emptying.

Ass:
- Hx: risk factors of comp!!
- S&S:
Lower tract inf (cystitis); dysuria, freq, suprapubic discomfort, macroscopic heamaturia, usually no fever (ex; males w prostatitis)
Pyelonephritis; loin pain, fever >38*C, chills & urinary symptoms
Ureteric calculus; sever pain
IDC Pts: usually no lower symptoms but may have loin pain/fever

Investigation:
- MSU (midstream urine); reagent strip for leuks, nitrites, haematuria, proteinuria
- Urine for culture/sensitivity; Pt’s w recurring Inf, catheterised Pts w S&S, poss pyelonephritis, poss Comp UTI, males, elderly, or Inf* not clinically evident.

Management
Acute simple cystitis:
- trimethoprim 300mg daily for 3/7 (alt: Cephalexin or amoxicillin/clavulanate)

Acute Uncomp pyelonephritis: amoxicillin/clavulanate 875/125mg 12 hrly 10/7 (alt: Cephalexin or trimethoprim)

Comp UTI - structural: guided by culture & sensitivity

Comp UTI - catheter: change catheter if req & Rx if symptomatic as per Comp UTI

48
Q

STIs

A
  • often working alone so assessment limited to Hx & referring

Follow local guidelines & refer to STI clinic for follow up

Notifiable:
- chlamydia, gonorrhoea & syphilis (there are more)

49
Q

HIV

A

Human immunodeficiency virus: makes peops immunocompromised

AIDS: acq imm deficiency syndrome; sever life threatening consequence of HIV - often result of damage to immune Sys

Refer** if concerned, GPs have broad guidelines around this.

50
Q

HSV Type 1 & 2: Ass x 4, man* x 2

A

Viruses that cause oral & genital infections
- treatable w antivirals

Ass:
- most are subclinical, so difficult
- symptomatic HSV 1 - pro-labial lesions
- symptomatic HSV 2 - genital herpes
- HSV encephalitis: acute onset fever & neuro symptoms; hemiparesis, cranial nerve abnormalities, ataxia, focal seizures & altered GCS/behaviour
- comprehensive pathology & radiology req for formal Dx

Management:
- healthy Pts w 1* inf - oral acyclovir (or similar) 7-10/7
- HSV encephalitis- IV acyclovir

51
Q

Varicella: Ass x 4, Comp x 3 & Manage x 3

A

Ass:
- febrile w vesicular rash
- assoc non-specific symptoms; headache, malaise, loss of appetite
- most inf* self limiting
- complications; extreme age/immunocompromised

Complications:
- bacterial superinfections of skin lesions leading to Nec Fasc
- some children may develop visceral involvement & CNS complications
- pneumonitis may develop, esp pregnant women

Management:
- supportive care only for most healthy
- acyclovir (& similar) decrease lesions & shorten course of started within 24hrs of rash onset (also considered for higher risk Pts)
- secondary skin inf* likely caused by group A Strep - Rx w first-gen cephalosporin

52
Q

Herpes Zoster: Ass x 4, man* x 5

A

Herpes Zoster Ass:
- prodrome of malaise, headache & photophobia
- S&S; pain, itching & paraesthesias in one or more dermatome
- development of macropapular rash that becomes vesicular; eruption does not cross the midline
- HZ opthalmicus can cause blindness!!

Management:
- antivirals may hasten resolution of many S&S, but Not post-herpatic neuralgia
- commence AVs within 72hrs of rash onset: any time for immunocompromised Pts
- more severe disease: ? IV AVs
- consider opioids for pain
- consider corticosteroids for pain

53
Q

Tetanus: contraind x 2, precautions x 2, & side effects x 2, dose

A

Tetanus:
- caused by clostridium tetanus - spores found in soil & can enter wounds
- acute, often fatal, disease - neurotoxin that acts on CNS & causes muscle rigidity & spasms
- usually an incubation period of 3-21 days
- most deaths in >70yo, esp women, & may be assoc w minor injury

Immunisation:
- DTP vax only in Aus
- antitoxin produced dose not prevent future infection but protects body from toxin produced by inf*

Indications:
Tetanus prone wounds

Contraindications:
- known, severe allergic reaction/hypersensitivity
- Pts <10yo req reduced dose

Precautions:
- current acute illness
- pregnancy

Side effects:
- pain at injection sight
- less common; headache, lethargy, malaise, myalgia, fever, bacterial neuritis (spec; brachial neuritis - not common)

Preg Cat: A

Admin:
Adults: 0.5mL IMI single dose only

Paeds (>/= 10yo): as above

54
Q

Influenza: contraind x 4, precautions x 1, side effects x 4, & Clin prac points x 5, dose

A

Influenza:
- virus types A & B affect humans
- surface antigens: haemagglutinin (H) & neuraminidase (N)
- aerosolised or direct contact w secretions

Severe disease:
- advanced age
- lack of prev exposure
- greater virulence of strain
- chronic Dx; heart/lung Dx, renal Dx, diabetes
- pregnancy, immunocompromised, smoking

Vax:
Usually contain 2 x A & 1 x B strains

Contraindications:
Severe allergic reaction or hypersensitivity to;
- seasonal influenza vax
- gentamicin, neomycin, or polymycin Abx
- polysorbate 80, octoxinol 9 or formaldehyde (components of vax)
- chicken products (eggs, feathers)
Current immunosuppressant Thx
Pregnancy/lactation
Current episode of febrile illness - >38.5 temp
Hx G-B syndrome

Precautions:
- current warfarin, theophylline (asthma& COPD), phenytoin or phenobarbitone (anticonvulsants) Thx

Side effects:
- allergic reaction
- discomfort, redness, swelling at injection site
- influenza-like illness
- headache

Clinic Prac Points:
- immunisation usually takes 2 wks post-admin to occur
- must be administered yearly to maintain immunity
- storage - cold chain must be maintained
- be prepared for adverse events
- observe for min 15mins post admin

Preg Cat: B2

Admin:
Adults - 0.5mL IMI single dose only (dependant on vax)