Wk 5: mat emergencies 1 Flashcards

1
Q

Define shoulder dystocia

A

“vaginal cephalic delivery that requires additional manoeuvres to deliver the fetus after the head has delivered and gentle traction has failed” (RCOG 2012)

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2
Q

What is the cause of a shoulder dystocia?

A

= bony impaction of the fetal anterior shoulder (usually) behind the maternal symphysis pubis. Occasionally the fetal posterior shoulder impacts over the maternal sacral promontory.

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3
Q

What documentation needs to be recorded in a shoulder dystocia?

A
  • Time of birth of the head
  • The time and sequence of manoeuvres used
  • Which was the anterior shoulder
  • The time of birth of the body
  • The condition of the baby
  • Umbilical cord lactates/gases
  • Estimated blood loss
  • Assessment of perineum and vagina
  • Staff members present during emergency.
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4
Q

What are some risk factors for shoulder dystocial?

A
  • infant of a diabetic mother
  • maternal obesity
  • gestational age (risk increases with age)
  • prolonged first stage
  • prolonged second stage
  • IOL (except for maternal diabetes and suspected macrosomia where evidence supports it reduces the risk of shoulder dystocia in this group of women)
  • Labour augmentation
  • Instrumental/assisted vaginal birth
    *fetal size is not a good predictor alone as the majority of new borns >4500g do not develop shoulder dystocia.
    ** in majority of shoulder dystocia cases there are no risk factors
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4
Q

What complications are associated with shoulder dystocia?

A

Maternal
- increased risk of PPH
- 3rd and 4th degree tear
- uterine rupture
- future obstetric issues
- psychological side effects

Neonatal
- increased risk of need for resus
- brachial plexus injury
- a network of nreves that conduct signals from the spinal cord to the arm and hand. Can result in erb’s palsy= flacid upper arm (recovers in 12 months usually)
- Klumpe’s Palsy is less common and is characterized by a limp hand and no movement of the fingers.
- fractured humerus/clavicle
- can occur unintentionally or intentionally. Quick to heal
- hypoxia
- death

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5
Q

Explain the steps of management for shoulder dystocia.

A
  • call for help
  • McRobers’ manoeuvre
  • suprapubic pressure + routein axial traction
  • ?episiotomy
  • delivery of posterior arm
    or
  • internal rotation maneuvers

*Repeat from top or try all fours

If still no release consider;
- cleidotomy
- zavanelli manoeuvre
- symphysiotomy

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6
Q

Recall some signs of shoulder dystocia?

A
  • There is difficulty with the birth of the face and chin
  • The head is born but remains tightly applied to the vulva
  • The chin retracts into the perineum (the turtle sign)
  • The anterior shoulder does not birth with normal downward traction
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7
Q

What is the goals of the manouvers?

A
  1. Increase the functional size of the bony pelvis
  2. Decrease the bisacromial diameter of the fetus
  3. Change the relationship of the bisacromial diameter within the bony pelvis by rotating the fetus into the wider oblique diameter
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8
Q

What are some key practice points in the management of a shoulder dystocia?

A
  • shoulders must be rotated using pressure on the scapula or clavicle. Never rotate the head
  • AVOID EXCESSIVE TRACTION AT ALL TIMES= anything but gentle axial pressure has been shown to cause increased harm with no benefit.
  • avoid fundal pressure= increased risk of brachial plexus injury, uterine rupture and haemorrhage from
    potential detachment of fundal placenta.
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9
Q

Explain McRobers manoeuvre

A
  • Remove or lower the bottom of the bed and manipulate her buttocks to the extreme edge.
  • With the aid of an assistant either side
  • legs are straightened and then thighs are abducted and hyperflexed onto the abdomen
    (McRobert’s position).
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10
Q

Explain Suprapubic pressure (also known as Rubin 1)

A
  • The accoucheur applies gentle downwards traction to the baby’s head.
  • Simultaneously the assistant adopts a CPR-hand position over the anterior shoulder.
  • The initial pressure applied is continuous.
  • If delivery is unsuccessful, a rocking motion may be applied.
    *aim is to reduce the diameter of the fetal shoulders and rotate the anterior shoulder into oblique diameter to slip the shoulder under symphysis pubis.
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11
Q

Explain the use of episiotomy in a shoulder dystocia

A
  • may be used to assist with the effectiveness of internal maneuvers
  • consider completing it during rubins 1 if predicting that baby will not come.
  • assistant may be required to elevate the baby’s head to improve the view of the perineum (thereby reducing potential trauma to the baby’s face).
  • This allows the accoucheur to use both hands to cut (or extend) the episiotomy.
  • The accoucheur applies gentle downwards traction to the baby’s head.
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12
Q

WHat is the correct hand position and insertion point for internal maneuvers during a shoulder dystocia?

A
  • sacral hollow as it is the most spacious part
  • hand position has been described ‘as if putting on a tight bracelet’ where the fingers are compressed and the thumb tucked in to the palm.
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13
Q

Describe rubins 2

A
  • accoucheur’s hand is inserted into the vagina and digital pressure is applied to the posterior aspect of the anterior shoulder pushing it towards the fetal chest. This rotates the shoulders forward into the more favourable oblique diameter.
  • Completion of the birth is then attempted using normal downward traction.
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14
Q

Explain the combined Rubins 2 and wood screw

A
  • While maintaining the McRobert’s position
  • the accoucheur introduces their second hand and locates the anterior aspect of the posterior shoulder.
  • Pressure is applied to rotate the posterior shoulder.
  • Completion of the birth should be attempted once the shoulders move into the oblique diameter.
  • If this movement is unsuccessful continue rotation through 180°
    and attempt delivery
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15
Q

Explain reverse wood screw

A
  • Pressure is applied to the posterior aspect of the posterior shoulder attempting to rotate it through 180° in the opposite direction to that described in the Wood’s screw manoeuvre.
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16
Q

Explain ‘removal of the posterior arm’ in a shoulder dystocia

A
  • The accoucheur passes their hand into the vagina over the chest of the fetus to identify the posterior arm and elbow.
  • Apply pressure to the antecubital fossa to flex the elbow in front of the body, and / or grasp the posterior hand to sweep the arm across the chest and deliver the arm.
  • This is followed by rotation of the fetus into the oblique diameter of the pelvis, or through 180°, bringing the anterior shoulder under the symphysis pubis.
  • The fetus is usually in an attitude of flexion with the arms flexed over the chest.
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17
Q

Explain rotation onto all fours in the context of shoulder dystocia

A
  • may facilitate delivery by increasing the pelvic diameters and allowing better access to the posterior shoulder.
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18
Q

Explain posterior axilla sling traction (PAST) tachnique

A
  • uses a sling that is placed around the posterior axilla.
  • is typically a suction catheter or an in-out urinary catheter which is folded into a loop and threaded through the baby’s posterior axilla.
  • Downward traction can then be applied to the sling to deliver the posterior shoulder.
  • If the posterior arm does not follow, it can be swept out more easily as room has been created by removing the posterior shoulder.
  • The sling can also be used to rotate the shoulders if traction on the shoulder fails.
  • In order to rotate the shoulders, traction should be applied laterally towards the baby’s back then anteriorly while digital pressure is applied behind the anterior shoulder to assist rotation.
  • double sling may be used
  • The ends of the sling can be clamped to allow greater tration
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19
Q

Recall the definition of a PPH

A

= as a blood loss of 500 ml or more from the genital tract, within 24 hours after birth.

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20
Q

Recall the definition of a severe/major PPH

A

= a blood loss of 1000ml or more after birth.

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21
Q

Recall the definition of a secondary PPH

A

is a blood loss of 500ml or more occurring from 24 hours postpartum until 6 weeks postpartum

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22
Q

Recall the most common cause of a PPH

A

Uterine atony is the most common cause of PPH- 70-90%

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23
Q

Explain why we must assess each women blood loss in relation to her and not just these numerical values.

A
  • normal adult blood volume is approximately 70ml/kg however in pregnancy this increases to approximately 100ml/kg.
    = This is a physiological process to assist in protecting women against haemorrhage.
  • Young, healthy women often compensate extremely well during haemorrhage and often signs of clinical deterioration are not as clear cut and negatively reassure us as healthcare professionals that the women is not clinically compromised.
  • This masking of clinical deterioration suggests that any sign/s of hypovolaemia should be taken seriously and resuscitation commence promptly.
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24
Q

What are the four T causes of a PPH?

A

TONE: (70%) - uterine atony, distended uterus, uterine muscle exhaustion

TISSUE: (10%) - retained products of conception, invasive placenta e.g. placenta percreta/accreta

TRAUMA: (19%) - cervical, vaginal or perineum, pelvic haematoma, uterus

THROMBIN: (1%) - blood clotting disorders, inherited or acquired including Disseminated intravascular coagulation (DIC)

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25
Q
A
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26
Q

Explain the initial non-pharmacological management of a PPH?

A
  • Lie flat
  • Administer O2
  • Keep warm
  • Insert IDC
  • Assess blood loss
  • Massage uterus and expel clots
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27
Q

Explain the initial pharmacological management of a PPH?

A

Oxytocin 10 units IM/IV
Syntometrine 1ml IM
or
Ergometrine 250microg IM and 250microg IV
**(Hypertension -Syntometrine &
Ergometrine contraindicated)

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28
Q

What obs are necessary and at what frequency in the initial management of a PPH?

A

BP, HR, RR and SaO2 5/60
Temp 15/60
Blood loss
1/24 urine output

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29
Q

What other actions may need to occur in the initial management of a PPH?

A
  • Intravenous access 2 x 14 or 16g
  • Collect blood samples (FBE, clotting,
    x-match, fibrinogen)
  • Rapid fluid replacement
    -Give 2L initially
  • Commence fluid balance chart
  • Avoid excessive crystalloid use.
  • ?blood products (O-ve RBC)
  • Use rapid infuser/warmer FFP, Platelets, cryoprecipitate
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30
Q

What are the management options for tone related PPH?

A
  • Fundal massage
  • Expel clots
  • Drugs:
    - oxytocin/Syntometrine/ergometrine if not already
    - Tranexamic acid 1g 100ml 0.9% NaCI IV
    - Carboprost 250microg/1ml IM
    - Oxytocin infusion 40 units/1L/4 hours
    - Misoprostol 600microg PR or buccal
  • Bi-manual compression
  • Bakri balloon
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31
Q

What are the management options for trauma related PPH?

A
  • inspection for tear (vaginal and vervix)
  • repair lacerations
  • ?packing uterus/vagina
  • consider presence of vaginal haematoma
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32
Q

What are the management options for tissue related PPH?

A
  • Deliver the placenta
  • Expel clots from uterus and vagina
  • Inspect placenta and membranes
  • ?need for surgical removal of retained products
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33
Q

What are the management options for thrombin related PPH?

A
  • check blood picture
  • consider: Severe preeclampsia,
    placental abruption, sepsis,
    FDIU, amniotic fluid embolism,
    hereditary bleeding disorders e.g. von
    willebrand’s
34
Q

When a PPH is escalating what are some options to gain better support?

A
  • activation of a massive transfusion protocol
  • advice from haemotologist via pathology
35
Q

When should transfer to theatre be considered?

A
  • Exploration of the uterine cavity
  • Consider uterine tamponade with Bakri balloon
  • Consider uterine/vaginal packing
  • Repair perineal/vaginal/cervical tears
  • Angiography and embolisation
  • Manual removal of placenta
  • B-Lynch suture (compression suture around whole uterus)
  • Ligation of bleeding vessels
  • Consider aorto-caval compression
  • Hysterectomy
36
Q

What condition is commonly associated with secondary PPH?

A
  • endometriosis
37
Q

What assessments should be carried out on a women with suspected secondary PPH?

A

Thorough history:
– Parity
– Intrapartum details and complications
– Obstetric risk factors
– Any relevant medical/family history

Clinical assessment:
– Vital signs
– Vaginal loss
– Uterine size
– Pain

38
Q

What investigations/management points would you want if you have a pt with suspected secondary PPH?

A
  • Insert 16g IV x 2
  • FBC, CRP, G&H [or cross matching depending on history], coagulation studies, serum hCG
  • Vaginal swabs
  • If signs of infection: MSU
  • If febrile T >38º C: blood cultures
39
Q

What is the key management principles of a secondary PPH?

A
  • is the woman stable
  • assessment
  • investigations
  • imaging
  • subsequent management for the unstable
  • IV antibiotic regimine
39
Q

What imaging might you want if you have a pt with suspected secondary PPH?

A
  • pelvic u/s and doppler if retained tissue suspected
  • other imaging as needed
40
Q

What subsequent management might you want if you have a pt with confirmed secondary PPH?

A
  • Treatment (resuscitation, uterotonics, bed rest, antibiotics, surgical evacuation & specialised options).
  • Admit all women with secondary PPH who have had a caesarean birth.
  • discuss w/ Senior Registrar/ Consultant, to ?d/c w/ antibiotics.
  • debrief the woman once she is haemodynamically stable
41
Q

What is the IV antibiotic regimen if a women is febrile/septic post secondary PPH.

A

IV for 48hrs then switch to oral, if no longer febrile/septic
- Ampicillin 2g STAT, then 1g 6/24
- Metronidazole 500mg 12/24
- +/- Gentamicin 5mg/kg daily

*if a penicillin allergy
- Clindamycin or Lincomycin 900mg 8/24
- +/- Gentamicin 5mg/kg daily

Oral Antibiotic Regimen – if not febrile
- Amox/clav 875/125 12/24, for 7 days
- Metronidazole 400mg 8/24, for 7 days

**Penicillin allergy
- Ciprofloxacin 500mg 12/24, for 7 days
- Metronidazole 400mg 12/24, for 7 days

42
Q

For TXA, what is the
- Dose/route
- max
- administration considerations

A

Dose/route: 1g IV over 10 mins

Max: 2nd dose may be given if bleeding persists >30mins or stops and restarts w/in 24hrs of first dose.

Consider: rapid administration may cause hypotension and dizziness
- use pump/infusion device

43
Q

For oxytocin, what is the
- Dose/route
- max
- administration considerations

A

Dose:
- 5IU IM
- 5IU IV over 1-2mins

Max: may repeat after 5 mins to max total dose of 10IU

and

Dose: 5-10 IU per hr IV via infusion pump
Reconstitution: oxytocin 30 IU in 500ml CSL or NS, infuse at 83-167ml

Administration considerations: use instead of ergo if BP is elevated.
- ensure placenta is expelled

44
Q

For ergometrine, what is the
- Dose/route
- max
- administration considerations

A

Dose/route:
- 250 mcg IV over 1-2mins
- 250mcg IM

Reconstitution: **only for IV dilute 250mcg up to 5ml w/ NS, (to make 50mcg/ml)

Maxi:
- for IV= may repeat 2-3mins to max total dose of 250mcg - 1mg
- for IM= may repeat after 5mins to max total dose of 500mcg - 1g

Administration
- give with antiemetic
- contraindicated with retained placenta, pre-eclampsia
- may cause severe hypertension

45
Q

For misorpostol, what is the
- Dose/route
- max
- administration considerations

A

Dose: 800-100 mcg PR

Max: repeat dose not recommended

Considerations: use when oxytocin and ergo are not successful
- due to slow onset of action, consider early administration

46
Q

For carboprost, what is the
- Dose/route
- max
- administration considerations

A

Dose:
- 250mcg IM
- 500mcg intramyometrial

Max:
- for IM= may repeat after 125mins to max dose of 2mg (= 8 doses)
- for intrautrerine= not recommended

Considerations:
- manufacturers don’t recommend intrauterine
- commence cardiac monitoring and O2 administration prior

47
Q

For fibrogen concenrate, what is the
- Dose/route
- max
- administration considerations

A

Dose:
- in response to fibrinogen levels.
- if levels unknown, then 5-70mg/kg of body weight IV at rate not exceeding 5ml/min

Reconstitution: w/ 50ml of sterile water and swirl gently to ensure fully dissolved
- do not shake

48
Q

For cryoprecipitate, what is the
- Dose/route
- max
- administration considerations

A

Dose:
- dependaent on fibrogen levels
- one adult standard dose= to 10 whole blood units

Considerations: stored frozen so defrost 30mins before administration

49
Q

What are some risk factors for a PPH?

A
  • Previous retained placenta or PPH
  • Previous caesarean section
  • Placenta praevia, percreta and accreta
  • Antepartum haemorrhage especially from placental abruption
  • Pre-eclampsia/ Hypertensive disorders
  • Body Mass Index above 35
  • Maternal weight below 60kg (due to smaller circulating volume)
  • Increased maternal age
  • existing uterine abnormalities e.g. fibroids
  • Maternal haemoglobin < 90 g?L at the start of labour
  • Spurious labour - prolonged latent phase of labour
  • Induction of labour - use of oxytocin or misoprostol in labour
  • Prolonged labour - 1st, 2nd or 3rd stage
  • Precipitate labour or incoordinate labour
  • Operative vaginal birth - forceps/vacuum
  • Caesarean section particularly if in 2nd stage
  • Placental abruption
  • Pyrexia in labour
  • Genital tract trauma - vaginal of cervical lacerations
  • Uterine rupture
  • Retained placental tissue
  • Maternal bleeding disorders
  • Grandmultiparity
  • Multiple gestation
  • Maternal Anaemia/ Maternal Iron deficiency
  • Polyhydramnios
  • Previous or current macrosomia >4500g
  • Shoulder dystocia
50
Q

What are the 5 basic/overarching steps to managing a PPH?

A
  1. escalation
    - involve senior clinician promptly, CALL FOR HELP immediately
  2. Communication
    - staff assistance, handover, activate MASSIVE TRANSFUSION protocol as per organisational policy, call PIPER if support/advice required
  3. monitoring
    - ongoing assessment and weight measuring of blood loss (weight 1 ml=1 gram) is essential, collect blood for group and hold and cross match, prevent hypothermia, frequent (5 minutely) vital signs including conscious state, heart rate, respiratory rate, blood pressure, O2 saturation, uterine tone and blood loss. Urine output
  4. resus
    - DRSABCD approach, signs and symptoms of hypovolaemia, speed of blood loss, the woman’s haemoglobin level pre birth, Intravenous access - wide bore cannula, pathology collection, Intravenous fluid administration (3 litres of crystalloid solution to 1 litre of blood loss, ?colloid fluid from 3rd litre), O NEGATIVE blood to be available for use.
  5. treatment of cause
    - check if the uterus is well contracted, check completeness of placenta/membranes, examine the cervix/vagina/perineum for tears, observe wounds and cannula sites for any ooze that may suggest signs of clotting disorders.

**stop the bleeding

51
Q

What is non-pharacological intervention you can perform to stop PV bleeding?

A
  • Bimanual compression of the uterus
  • w/ sterile gloves, insert pringle hand into wrist then form fist
  • place first into anterior fornix and apply pressure against anterior wall of uterus
  • w/ other hand, press deeply into abdomen behind uterus, applying pressure against posterior wall of uterus
  • maintain compression for 20-30 mins or until bleeding is controlled and the uterus contracts.
52
Q

In a setting where multiple uterotinics are available which is recommended?

A

oxytocin (10 IU, IM/IV) is the recommended uterotonic agent for the prevention of PPH for all births.

53
Q

What is TXA and when should it be used?

A

= Tranexamic acid
= a competitive inhibitor of plasminogen activation, and it can reduce bleeding.
- management for anticoagulant
- used IV within 3hrs of birth + standard drugs recommended for those with PPH or c/s.
- used in standard PPH management

54
Q

What are some non-surgical and non-pharmacological means to manage PPH?

A
  • bimanual compression
  • intrauterine balloon tamponade
  • nonpneumatic anti-shock garment
  • aortic compression
55
Q

Define uterine rupture

A

= a rare occurrence and is a result of all 3 layers of the uterus rupturing creating changes to the wellbeing of the woman and/or baby (complete rupture).
Incomplete rupture= all layers except the peritoneum rupture. [Note: the definition of complete or incomplete rupture is variable in the literature]

56
Q

What are some risk factors for uterine rupture?

A
  • previous uterine surgery includes myomectomy, c/s etc. (ensure u have a full surgical history for these patients)
  • fetal macrosomia
  • TOLAC
  • IOL
  • Grand multi
  • overdistended uterus e.g. multiple pregnancy
  • malpresentation/position
  • use of oxytocics including prostaglandins for IOL
  • obstructed labour
  • uterine/abdominal trauma
  • uterine scar, perforation or uterine anomaly
    *previous c/s scar rupture risk is least with spont labour
57
Q

What are some indications of uterine rupture?

A
  • persistent, prolonged, profound fetal brady
  • Abnormal FHR pattern suggesting fetal compromise
  • Abdominal pain: acute onset of scar tenderness
  • Any atypical pain:
    - chest pain
    - shoulder tip pain
    - pain previously controlled by analgesia
    - pain between contractions
  • Cessation of previously efficient uterine activity
  • Loss of station of presenting part
  • Abnormal labour progress
    Vaginal bleeding
  • Maternal tachycardia, hypotension or shock
58
Q

Define acute uterine inversion

A

= The fundus of the uterus inverts and collapses down into the uterus as it protrudes downwards.
- potentially life threatening complication of the 3rd stage of labour.

59
Q

What is the most common cause of uterine inversion?

A

= almost always is a result of applying cord traction before the uterus has firmly contracted and the placenta separated from the uterus.

60
Q

What are the degrees of uterine inversion?

A

First degree
- the uterus is partially turned out, fundus of uterus reaches the internal os.

Second degree
- the fundus has passed through the cervix but not outside the vagina.

Third degree
- the fundus is prolapsed outside the vagina.

Fourth degree
- the uterus, cervix and vagina are completely turned inside out are are visible.

61
Q

What are the types of uterine inversion?

A

Acute - occurs within the first 24 hours
Subacute - occurs within the first 24 hours- 30 days
Chronic - occurs after 30 days and is rare.

62
Q

What are risk factors for uterine inversion?

A
  • excessive traction on the umbilical cord to manage the birth of the placenta
  • inappropriate fundal pressure- combining fundal pressure and cord traction to expel the placenta
  • short umbilical cord
  • use of fundal pressure to expel the placenta while the uterus is atonic
  • multiparity
  • abnormally adherent placenta
  • vaginal birth after caesarean (VBAC)
  • abnormalities of the uterus e.g. unicornuate uterus
  • previous uterine inversion
  • fetal macrosomia
  • precipitate labour
  • Connective tissue disorders e.g. Marfan syndrome
  • spontaneous occurrence
  • precipitate labour
  • placenta acreta
  • use of muscle relaxants e.g. uterotonics
63
Q

What are some signs and symptoms of uterine inversion?

A
  • suddenly appears shocked and collapses *** this level of shock and collapse does not correlate with blood loss.
64
Q

Why does uterine inversion result in shock?

A

= due to the vasovagal (neurogenic) shock with bradycardia and hypotension. The best way to resolve neurogenic shock is to replace the uterus.
- Should the woman then have a PPH following the uterine version (after the uterus has been replaced and placenta birthed), she will be tachycardic and hypotensive due to hypovolaemic shock.

65
Q

What is the relationship between uterine inversion and PPH?

A

PPH risk is high following a uterine inversion due to uterine atony in up to 90% of cases.

66
Q

What is the management of a uterine inversion?

A
  • replace uterus (priority as the longer the uterus remains prolapsed the more oedematous the uterus becomes and over time a constriction ring may develop resulting in uterine replacement becoming more difficult)
  • call for help
  • maternal resus
  • IV cannulation x2
  • IV fluids
  • path tests taken
  • effective communication w/ women and team
67
Q

What are some immediate management actions for managing an acute uterine inversion?

A
  • lie flat
  • give facial oxygen 10L/min
  • inform women/partner clearly and communicate
  • alert theatre
  • RR, HR, BP, O2
68
Q

What resus measures should be completed in the case of a urine inversion?

A
  • x2 large IV cannule (14 or 16 guage)
  • send bloods; FBC, clotting, cross match 4 units of blood
  • commence 2L crystalloid IV
69
Q

What is involved in replacing the uterus in the management of a uterine inversion?

A
  • attempt immediate manual replacement of the uterus
  • ?transfer to theatre for analgesia if woman is stable and no pain relief.
  • inform consultant OB
  • alert anesthetics, OT and appropriate staff
70
Q

What is the management of a uterine inversion if immediate replacement of the uterus fails?

A
  • transfer to OT
  • consider uterine relaxants e.g. GTN, SC terbutaline, general anesthetic
  • attempt replacement either manually or by hydrostatic method

If still unsuccessful
- prepare for laparotomy w/ consultant OBGYN

71
Q

What the management of a uterine inversion once successfully replaced?

A
  • manage PPH (as 90% of cases are followed by PPH)
  • do not remove placenta until in theatre
  • to prevent haemorrhage
    - oxytocin bolus (IM syntometrine or synt)
    - commence oxytocin infusion
  • TXA if does PPH
  • consider further uterotoncis as required
72
Q

What is the best prevent for uterine inversion?

A
  • wait for all 4 signs of placental separation before CCT
  • guarding uterus
73
Q

Define an amniotic fluid embolism (AFE)

A

= occurs when amniotic fluid, fetal cells, hair and other debris enters the maternal pulmonary circulation and causes an allergic-like reaction and cardiovascular collapse

74
Q

What can cause an AFE?

A

= Although poorly understood, it is thought to be the result of an abnormal and exaggerated, allergic-like reaction to the amniotic fluid that enters the mother’s bloodstream, a normal part of the birth process.

75
Q

What are complications of AFE?

A
  • maternal collapse
  • often cardiac arrest
76
Q

When is an AFE most likely to occur?

A
  • in labour
  • shortly after
77
Q

What are the signs and symptoms of a AFE?

A

**Women are conscious at the onset of symptoms

Acute symptoms of-
- appear to have an anaphylactoid reaction due to immune response
- anxiety
- agitation
- coughing
- shortness of breath followed by
- adult respiratory distress syndrome (ARDS)
- cardiovascular collapse ( hypotension, tachycardia and possible arrhythmias)
- cardiac arrest
- DIC can quickly develop leading to massive maternal haemorrhage
- Pulmonary hypertension and acute right ventricular failure is not uncommon
- Encephalopathy
- Seizures
- Maternal/infant mortality

78
Q

What is the treatment of an AFE?

A
  • Support the cardiovascular and respiratory systems
  • Early correction of clotting abnormalities with blood products
  • Liaising with Intensive Care Unit and Haematology staff
79
Q

What is a perimortem caesarean section

A

= caesarean section/laparotomy without general anaesthetic, that is initiated after Cardiopulmonary resuscitation has commenced following extensive trauma/maternal collapse e.g. motor vehicle accident, domestic violence, cerebral haemorrhage.

80
Q

When is a peri-mortem caesarean section recommended?

A

= for women who look visibly pregnant or 20 weeks gestation or more (uterine fundus at the level of the umbilicus = 20 weeks gestation)
- with in 3-5 mins of cardiac arrest

81
Q

What is the benefit of performing a peri-mortem c/s

A

= to enhance adequate circulating blood volume for the woman due to the fetal birth and removal of placental circulation, resulting in a reduction in aorto-caval compression/obstruction and increased cardiac output and improve venous return, improve chest compressions and improve respiratory mechanics.

82
Q

What are some key practice points of peri-mortem resus/c/s

A
  • CPR must continue thought peri-mortum c/s
  • L) side lower
  • Irreversible brain damage can occur within 4-6 minutes for a pregnant woman due to aorto-caval compression from the pregnant uterus as venous return is impaired and cardiac output reduced.
  • Everyone must be mindful that once cardiac output in the woman is restored, her risk of bleeding are extreme.
  • Have a neonatal team ready to receive and resuscitate the baby once it is born.
  • intimate partner violence/domestic violence increases during pregnancy especially in the 3rd trimester and women are most commonly struck in the abdomen by a blunt or penetrating object.
    ** this may cause womans collapse
  • do consider placenta abruption