Who AM I?

Question 1

I penetrate the cell wall of susceptible bacteria. I bind irreversibly to 30S and 50S ribosomal subunits, intracellular structures that synthesize proteins. As a result, the bacteria cell membrane becomes defective and cannot synthesize the proteins necessary for its growth and replication.

Answer 1

Gentamicin (Aminoglycoside)

Question 2

I am a bacteriacidal agent that causes cell death. I interfere with the enzymes that are required for synthesis of bacterial DNA and therefore are necessary for growth and replication.
You can’t give me to children <18, or with dairy, or with pregnant women.

Answer 2

Ciproflaxicin

Question 3

I enter the microbial cell and reversibly bind to the 50S subunits of ribosomes, thereby inhibiting microbial protein synthesis and leading to cell death. I have both bacteriacidal and bacteriostatic activity against susceptible bacteria.

Answer 3

Erythromycin

Question 4

I treat nausea and vomiting. I block dopamine from receptor sites in the brain and in the chemotherapy trigger zone. I antagonize D2 receptors in the area postrema of the midbrain, thereby decreasing effects of dopamine in the brain. I also possess M1 Muscarinic and H1 Histamine blocking effects.

Answer 4

Promethazine

Question 5

I relieve nausea and vomiting by blocking the action of acetylcholine in the brain.

Answer 5

Hydroxyzine.

Question 6

I antagonize serotonin receptors, preventing their activation by the effects of emetogenic drugs

Answer 6

Ondansteron

Question 7

I work by slowing perstalsis of the smooth muscle in the intestine.

Answer 7

Diphenoxylate with atropine.

Question 8

I inhibit bacterial cell wall synthesis by binding to one or more penicillin binding proteins. I am more effective at treating gram positive bacteria, but I am occasionally used for gram negative. I also treat infective endocarditis.

Answer 8

Ampicillin

Question 9

I inhibit the third and last step of bacterial cell wall synthesis by binding to one or more penicillin binding proteins. I am often used as a prophylaxis involving surgical procedures

Answer 9

Cefazolin

Question 10

I inhibit bacterial cell wall synthesis by binding with penicillin binding proteins. I am given IM/IV, at a max dose of 4 grams per day. I am considered a broad spectrum antibiotic.

Answer 10

Impenem-Cilastin

Question 11

I inhibit bacterial cell wall synthesis by binding with penicillin binding proteins. I can be given to patients with a known penicillin allergy because of my structure.

Answer 11

Aztreonam

Question 12

I act as an antimetabolite of PABA which micro-organisms need to produce folic acid. Folic acid is necessary for the production of bacterial intracellular proteins. I enter into the reaction instead of PABA, and compete for the enzyme involved and cause formation of nonfunctional derivatives of folic acid. I halt multiplication of new bacteria, but have no effect on exisiting ones.

Answer 12

TMP-SMZ

Question 13

I penetrate microbial cells by using passive diffusion and an active transport system. I bind to the 30S ribosomes and I inhibit microbial protein synthesis.

Answer 13

Tetracycline.

Question 14

I am given to patients with acne. I supress the growth of propionbacterium acnes with sebaceous follicles by reducing the free fatty acid content in the sebum

Answer 14

Tetracycline.

Question 15

I inhibit sodium and chloride reabsorption in the ascending loop of henle, where absorption of most filtered drugs takes place.

Answer 15

Furosemide

Question 16

I decrease absorption of water, sodium chloride, and bicarbonate in the distal convulted tubule.

Answer 16

HTCZ

Question 17

I inhibit mineral corticosteroid receptors and block the effects of aldosterone, in the renal tubules. I block the sodium retaining effects of aldosterone.

Answer 17

Spirinolactone

Question 18

I kill bacteria by entering into their cells and damaging genetic material

Answer 18

Nitrofurantoin

Question 19

I consist of soluble fibers that are largely unabsorbed by the small intestine. When H20 is added, these substances swell and become gel like.

Answer 19

Psyllium Preparations

Question 20

I cause the stool to absorb water and swell stimulating peristalsis and defecations

Answer 20

Psyllium prepartions

Question 21

I allow H20 to penetrate the stool.

Answer 21

Docusate sodium

Question 22

I facilitate admixing of fat and water in the stool. Stools are softer and easier to expel, little if any laxative effect.

Answer 22

Docusate sodium

Question 23

I am the strongest and most abused laxative. I irritate the GI mucosa, pull water in the colon and stimulate peristalsis. I produce a watery stool and may lead to fluid, electrolyte and acid base imbalances.

Answer 23

Bisacodyl

Question 24

I increase the osmotic pressure in the intestinal lumen resulting in the retention of water, which distends the bowel and stimulates peristalsis. I produce a semi fluid stool and may lead to fluid and electrolyte imbalances.

Answer 24

Polyethylene glycol. (Peg)