Weel 4: Respiratory A Flashcards

1
Q

Define is asthma

A

by chronic airway inflammation

defined by the history of respiratory symptoms such as wheeze, shortness of breath, chest tightness and cough that vary over time and in intensity, together with variable expiratory airflow limitation”

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2
Q

Explain the pathophysiology of asthma

A

When exposed to a trigger smooth muscles of the air ways contract and narrow the air layer.
and
trigger causes mucosal lining to become swollen and secrete more mucus.
= block the airways and increase difficulty f breathing

  • these make exhalation and inhalation harder therefore causing excess of air in lungs= hyperinflation

Asthma patients have chronically inflamed air ways that make they hypertensive to triggers.

Mucosa and smooth musclee
Affects smaller airways

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3
Q

Link the key pathological processes of asthma to the clinical manifestations/symtoms

A

Smooth muscle contrcation= feels like chest is tightening

Increased inflammation + secretion= coughing

Constriction and narrowing of airways= wheezing

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4
Q

Explain hyperinflation and what it causes.

A

Hyperinflation= the trapping of air inside the lungs which causes body to work harder to move air in and out of the lungs.

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5
Q

What are some key clinical manifestations of asthma?

A
  • wheeze
  • SOB
  • chest tightening
  • coughing
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6
Q

During a sever/life threatening asthma exacerbation, what are some key clinical manifestations?

A
  • Prominent inspiratory and expiratory wheezing
  • Respiratory Rate > 30 breaths per minute
  • Pulse Rate > 120 beats per minute
  • Use of accessory muscles
  • Agitation
  • Perspiration
  • Inability to speak e.g. single word responses, unable to speak a sentence
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7
Q

Explain the key com[ponents of an asthma patient assessment.

A

Gain a brief health history

  • subjective data (have they been hospitalised for asthma before? asthma management plan? prescribed medications?)
  • Objective info (ntegumentary: have they got diaphoresis, cyanosis? Respiratory: nasal discharge? polyps? increased WOB? are they positioned for maximal respiratory effort? Cardiovascular: tachycardia? Important diagnostic findings: chest x-ray, arterial blood gases during an asthma attack)

Primary assessment
- DRSABCD

Secondary

  • do they look well? tripoding?
  • Head to toe assessment
  • Full set of vitals
  • pain assessment

Focoused resp assessment

  • inspect
  • auscultate
  • percussion
  • palpate
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8
Q

Explain each component of a focused resp assessment

A

Inspect
Observe the overall appearance (does the patient appear well/unwell?)

Observe the patient’s colour; both centrally and peripherally

Observe the patient’s respiratory rate, rhythm and depth & respiratory effort (are they using accessory muscles?)

Inspect the symmetry and shape of chest

Inspect the tracheal position

Can you hear any audible sounds?

Auscultation
Listen for breath sounds

Auscultate the lung fields - comparing both sides, inspiration and expiration

Percussion
Percuss the thorax
Palpation
Bilateral symmetry of chest expansion

Palpate the skin feeling for skin temperature, turgor and moisture

Assess capillary refill both centrally and peripherally

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9
Q

How do you manage an episode of acute asthma?

A
  1. correct significant hypoxemia
  2. reverse airflow obstruction quickly
  3. plan to prevent further events
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10
Q

How can you correct significant hypoxemia?

A

Oxygen therapy via a Simple face mask / Hudson Mask

Reverse Airflow Obstruction Quickly with

  • Reliever medication: immediate symptomatic relief (Example: Salbutamol)
  • Preventer medication: inhaled corticosteroids (ICS) or single preventer inhalers for longer term treatment (Example: Budesonide)

In a sever situation:

  • DRSABCDE
  • Provide oxygen therapy as required
  • Provide medications as required and/or prescribed
  • Escalate to medical treating team and assess need to initiate MET call
  • Escalate to Code Blue if needed
  • Position the patient to maximise chest expansion
  • Stay with the patient
  • Provide reassurance to reduce patient anxiety
  • Encourage pursed lip-breathing
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11
Q

What are the nursing consideration of someone with asthma?

A

Education

  • triggers
  • medication use
  • when to call 000
  • reducing acute events
  • knowledge of early signs of events

Intercollabroative approach
- check in with GP

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12
Q

Explain some interprofessional collaboration that may be needed to treat and manage asthma.

A

Community nurse: Completion of an asthma management plan and provide focused education and review

Physiotherapist: Providing chest physiotherapy

Exercise physiologist: Tailored assessment and exercise

General Practitioner (GP): Ongoing review/assessment and the prescription of required medications

Respiratory Physician: For patients with uncontrolled asthma

Asthma is often self-managed by patients via an asthma-management plan; however, collaborative support encourages the patient to engage with their management plan and achieve positive overall health outcomes.

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13
Q

What are the nursing consideration of someone with asthma?

A

Education

  • triggers
  • medication use
  • when to call 000
  • reducing acute events
  • knowledge of early signs of events

Intercollabroative approach
- check in with GP

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14
Q

Explain some interprofessional collaboration that may be needed to treat and manage asthma.

A

Community nurse: Completion of an asthma management plan and provide focused education and review

Physiotherapist: Providing chest physiotherapy

Exercise physiologist: Tailored assessment and exercise

General Practitioner (GP): Ongoing review/assessment and the prescription of required medications

Respiratory Physician: For patients with uncontrolled asthma

Asthma is often self-managed by patients via an asthma-management plan; however, collaborative support encourages the patient to engage with their management plan and achieve positive overall health outcomes.

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15
Q

Define chronic obstructive pulmonary disease

A

progressive, chronic lung disease, characterised by irreversible obstruction of the airways

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16
Q

What are some key characteristics of COPD?

A
  • preventable
  • treatable
  • airfow limitation is not always fully reversible
  • slow progressive obstruction of the airways with periods otic exacerbations and increased periods of dyspnea and sputum production.
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17
Q

What comes under the umbrella term of COPD?

A
  • chronic bronchitis
  • emphysema
  • asthma?
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18
Q

Explain chronic bronchitis

A

It is the hyper secretion of mucus and a produce cough that lasts longer than 3months and has occurred for at least 2 consecutive years.

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19
Q

What causes chronic bronchitis?

A
  • Patient inhales harmful irritants (cigarette smoke, pollutants)
  • Airway becomes inflammed and infiltrated with neutrophils, macrophages and lymphocytes into bronchial wall
  • Bronchial inflammation causes bronchial oedema
  • Goblet cells increase in size and quantity within the airway and epithelium
  • Thick mucus is then produced which cannot be cleared due to damaged ciliary function
  • Over time the airway becomes narrowed and airway obstruction can occur
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20
Q

Explain emphysema

A

the abnormal and permanent enlargement of gas-exchange airways accompanied by the destruction of alveolar walls.

Obstruction results from changes in the lung tissues, rather than mucus production and inflammation in chronic bronchitis. The major mechanism of airflow limitation is the loss of elastic recoil.

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21
Q

What causes emphysema?

A

The cause of emphysema is cigarette smoking however air pollution and childhood respiratory infections are contributing factors.

22
Q

What is the pathophysiological process of emphysema?

A
  • Destruction of alveolar space (which destroys portions of the pulmonary capillary bed and increases the volume of air in the alveoli
  • Alveolar destruction –> large air spaces in lung tissue and air spaces
  • The air spaces are then not able to participate effectively in gas exchange
  • Expiration is challenged due to a loss of elastic recoil
  • This reduces the volume of air that is expired and air becomes trapped in lungs
  • Air trapping –> increases chest expansion
  • Causing reduced gas exchange, increased work of breathing, hypoventilation and hypercapnia
23
Q

What are the clinical manifestations of COPD?

A
  • SOB
  • Persistent cough
  • Dyspnoea
  • recurrent or sever pulmonary infection
  • Barrel chest
  • Digital clubbing
  • fatigue

Worsened clinical manifestations that can occur over time include;

  • haemoptysis (coughing up blood)
  • pneumonia (acute infection of the lungs)
24
Q

What is involved in the assessment of a patient with COPD?

A

Primary
Secondary
Focused
- Inspect
- Observe the overall appearance (does the patient appear well/unwell?)
- Observe the patients colour both centrally and peripherally
- Observe the patient’s respiratory rate, rhythm and depth & respiratory effort (are they using accessory muscles?)
- Inspect the symmetry and shape of chest
- Inspect the tracheal position
- Can you hear any audible sounds?

  • Auscultation
    - Listen for breath sounds
    - Auscultate the lung fields
  • Percussion
    - Percuss the thorax
  • Palpation
    - Bilateral symmetry of chest expansion
    - Palpate the skin feeling for skin temperature, turgor and moisture
    - Assess capillary refill both centrally and peripherally
25
Q

Explain what is entailed in each step of a non specific resp assessment

A

Primary assessment
If not already performed = airway, breathing, circulation, disability (mental status assessment), exposure

Secondary Assessment
Comprehensive, systematic body systems assessment (or head-to-toe assessment)

General appearance → looks well / unwell, discomfort, skin colour → pallor or flushed face

Full set of vital signs

Pain assessment: PQRST, OLDCARTS, is the patient experiencing chest pain?

Respiratory Focused Assessment

  • inspect
  • auscultate
  • percuss
  • palpate
26
Q

What are the nursing managements for someone with COPD?

A
  • O2 therapy (to treat hypoxemia)
  • patient education
  • patient reassurance
  • airway clearance
  • breathing retraining (pursed lip breathing)
27
Q

What should be the aim of patient education surrounding COPD?

A
  • minimise symptoms
  • maximise quality of life
  • empower patients to independently manage their COPD at home.

Further goals include;
- Improving gas exchange;
- improving oxygenation and reducing carbon dioxide
Improving airway clearance (reduce mucous production and increase exporation)
- Improving breathing rate and pattern
- Promoting independence with ADL’s and self-care activities.
- Improving nutritional intake
- Preventing and treating infection

28
Q

What other health care professionals can nurses collaborate with to increase patient outcome and care of someone with COPD?

A

interprofessional collaboration to ensure the patient is provided with holistic and comprehensive support.
Some examples include;
- Dietician: nutritional therapy can support a side effect of weight loss and ensure the patient is achieving nutritional requirements
- Surgical therapy: some patients may require a lung reduction, bullectomy or a lung transplant.
- Physiotherapy: pulmonary rehabilitation and chest physiotherapy
- Exercise physiology: promotion of safe exercise and activity
- Mental health support: patients with COPD have often had to consider major life adjustments to manage their health therefor mental health support can assist with this process

29
Q

Explain pneumonia

A

an acute infection of the lung parenchyma.
(The lung parenchyma comprises a large number of thin-walled alveoli, forming an enormous surface area, which serves to maintain proper gas exchange.)

Pneumonia can be caused by bacteria, viruses, fungi, protozoa or parasites.

30
Q

What are the four types of pneumonia?

A
  • community acquired
  • aspiration pneumonia
  • medical care associated pneumonia (eg whilst in hospital, from being intubated and ventilated etc)
  • opportunistic (secondary) pneumonia
31
Q

What can cause pneumonia?

A
  1. aspiration of normal flora from the nasopharynx or oropharynx
  2. inhalation of microbes present in the air
  3. Haematogenous spread from infection elsewhere in the body.
32
Q

Explain the pathophysiology of pneumonia

A

A microorganism must pass the following;

  • Upper airway (nasopharynx and oropharynx)
  • Compromised systemic defence mechanisms (humoral and complement-mediated immunity
  • Impaired mucociliary clearance
  • Impaired cough reflex
  • Alveolar macrophages
  • Accumulation of secretions

Once passes these;

  • An acute inflammatory response ensues including inflammatory mediators, cellular infiltration and immune activation.
  • This damages the bronchial mucous membranes and alveolar-capillary membranes causing an accumulation of thickened fluid that includes white blood cells, neutrophils, plasma proteins and cellular debris (pus) in the alveoli and terminal bronchioles.
  • This impairs gas exchange and can lead to dyspnoea, hypoxia and hypercapnia.
33
Q

What are some clinical manifestations of pneumonia?

A
  • increased resp rate
  • increased work of breathing/SOB
  • accessory muscle use
  • decreased SpO2
  • Productive cough
  • Cyanosis (late sign)
  • Chest tightening/pain (ensure it isnt cardiac pain)
  • Fatigue, general malaise, headache and decreased appetite
  • Fever/chills
34
Q

Explain a nursing assessment for a patient presenting with Pneumonia:

A
  • Obtain a brief history
  • obtain subjective and objective information
  • Documentation of all clinical findings
  • Review all medical notes regarding clinical presentation and patient’s treatment plan

Primary assessment

  • If not already performed = airway, breathing, circulation, disability (mental status assessment), exposure

Secondary Assessment

  • Comprehensive, systematic body systems assessment (or head-to-toe assessment)
  • General appearance → looks well / unwell, discomfort, skin colour → pallor or flushed face
  • Full set of vital signs
  • Pain assessment: PQRST, OLDCARTS, is the patient experiencing chest pain?

Respiratory Focused Assessment

  • Inspect
    - Observe the overall appearance (does the patient appear well/unwell?)
    - Observe the patients colour both centrally and peripherally
    - Observe the patient’s respiratory rate, rhythm and depth & respiratory effort (are they using accessory muscles?)
    - Inspect the symmetry and shape of chest
    - Inspect the tracheal position
    - Can you hear any audible sounds?
  • Auscultation
    - Listen for breath sounds
    - Auscultate the lung fields
  • Percussion
    - Percuss the thorax
  • Palpation
    - Bilateral symmetry of chest expansion
    - Palpate the skin feeling for skin temperature, turgor and moisture
    - Assess capillary refill both centrally and peripherally
35
Q

What nursing managements should take place to treat pneumonia?

A
  • Correct hypoxia, oxygen therapy and/or high flow
    Maintain administration of ordered medications (timely administration of antibiotics etc)
  • Regular pain assessment and pain relief
  • Encourage deep breathing and coughing exercises
36
Q

What nursing management should be carried out in a life-threatening episode of pneumonia

A
  • DRSABCDE
  • Provide oxygen therapy
  • Provide medications as required and/or prescribed
  • Escalate to medical treating team and assess need to initiate MET call
  • Escalate to Code Blue if needed
  • Position the patient to maximise chest expansion
  • Stay with the patient
  • Provide reassurance to reduce patient anxiety
37
Q

What are some key points that nurses must be aware of surrounding pneumonia?

A
  • must be mindful not to carse health care associated infections
    0 ensure patient has adequate education
  • support person with ADL’s as they are likely fatigued
38
Q

Explain some interprofessional collaborative health care measures measures.

A

Medical team: diagnose, order diagnostic tests, prescribe medication

Physiotherapy: chest physiotherapy

Pharmacist: support and education of medication regime

39
Q

Explain tuberculosis

A

Mainly affects the lungs, TB can infect any organ in the body

40
Q

Explain TBs latent activities

A

TB often stays latent and capitalises on the body when its immune system is compromised by another infection or when the person ages. It then becomes active and this is when symptoms present.

41
Q

How is TB transmitted?

A

via inhalation

42
Q

What can TB do to over come the body immune system and infect the lungs?

A
  • over come the turbulent air intake and mucus then travel further into the air ways.
  • TB inhibits lysosomal activity that occurs when the Tb is phagocytosed by macrophages. - it proliferates
  • at this point this is considered primary TB*
  • they are likley asymptomatic

At around 3 weeks cell mediated immunity= a granuloma forms around the TB infection
- caseous necrosis occurs (death of inside tissue)

TB also infected Hilar lymphomas of and classifies= this is what is seen on X-rays

TB= an aerobe= profers areas of greater oxygenation

43
Q

What can systemic miliary infect?

A
  • kidneys
  • Meninges of the brain (meningitis)
  • lumbar vertebrae (Potts disease)
  • Adrenal glands (Addisons disease)
  • Liver (hepatitis)
  • Cervial lymph nodes
44
Q

How can you test for TB/how is it diagnosed?

A

Positive tuberculin skin test (The tuberculin skin test however has been found to be associated with a high number of false positive results. For this reason the interferon gamma release assay (Quantiferon Gold), has been developed)

Sputum culture

Chest x-ray

  • purified protein derivative (PPD) intradermal skin test
    Tuberculin (component of the bacteria)- if the immune system has previously been exposed to TB the skin will react with induration and not just redness.
  • Interferon gamma release assay (IGRA)
    looks for evidence in the blood for previous TB infections

Spetum testin= looks for TB microbes

Chest X-ray= looks for calcification of active disease

45
Q

What is the treatment of TB

A

use of single drug for a long drug such as 9 months

46
Q

Explain the pathophysiology of TB

A

TB is spread by respiratory droplets (saliva, mucus) and transmitted via inhalation (airbourne).

Macrophages are released by the body to fight the tubercule bacilli (TB). If the body successfully fights the TB and the person does not become sick and the TB is latent. Although it can become active after a period of time, sometimes months or years. If the TB is not destroyed, they can grow and destroy the macrophages.

When the bacteria settles in the lung it multiplies causing inflammation. When the body’s defense mechanisms become overwhlemed, the TB continues to multiply and the person becomes unwell. The bacteria travels to the lymphatic system where the lymphocytes initiate the immune system response.

The inflammtory process in the lungs activates the alveolar macrophages and neutrophils to try to overwhlem the TB bacteria. They engulf the bacteria forming lesions called tubercles. This infected tissue dies forming material that is necrotic with scar tissue surrounding this in an attempt to isolate the TB.

47
Q

What are some clinical manifestations of TB?

A

Active infection

  • Chronic cough
  • sputum production
  • loss of appetite
  • weight loss
  • fever
  • night sweats
  • fatigue
  • chest pain
  • haemoptysis

Latent infection - Usually asymptomatic

48
Q

What is the nursing assessment of someone with TB?

A

Nursing assessment for a patient presenting with TB:

  • Obtain a brief history
  • obtain subjective and objective information
  • Documentation of all clinical findings
  • Review all medical notes regarding clinical presentation and patient’s treatment plan

Primary assessment

Secondary Assessment

Focused resp assessment
(inspection, auscultation, percussion, palpation)

49
Q

How can TB be treated?

A
  • consists of antibiotic therapy for a minimum of 6 months. This acts to control active or latent TB infection and prevent its transmission.
  • never involves single drug therapy as drug resistance appears quickly.
  • Medication regimen for tuberculosis are centred around medications in which the Mycobacterium tuberculosis is sensitive to. These medications include: isoniazid, rifampin, pyrazinamide and ethambutol.
  • those with TB isolate at home
50
Q

What are some nursing care considerations

A
  • Nurses must be mindful of maintaining droplet precautions; refer to infection control guidelines and always consult the treating team for guidance on required PPE
  • Ensure the patient has been provided with adequate education
  • Support the patient with ADL’s if they are fatigued and require support
51
Q

What is an interprofessional approach to TB?

A

Infection control: to assess and ensure appropriate PPE requirements

Pharmacist: medication education and dispensing

Physiotherapist: Providing chest physiotherapy

General Practitioner (GP): Ongoing review/assessment and the prescription of required medications