Week 8- Pressure ulcers/ dressing Flashcards
Who experiences a loss of skin integrity?
Patients confined to bed for long periods, patients with motor or sensory dysfunction, and patients who experience muscular atrophy and reduction of padding between the overlying skin and the underlying bone are prone to pressure ulcers.
What is a pressure ulcer (related to cap closure pressure)?
Who is at risk for this?
What is the initial sign of pressure?
localized areas of infarcted soft tissue that occur when pressure applied to the skin over time is greater than normal capillary closure pressure (25 to 32 mm Hg)
Critically ill patients= lower capillary closure pressure and are at greater risk for pressure ulcers.
The initial sign of pressure is erythema caused by reactive hyperemia, which normally resolves in less than 1 hour.
What contributes to the development of pressure ulcers (list)?
Immobility, impaired sensory perception or cognition, decreased tissue perfusion, decreased nutritional status, friction and shear forces, increased moisture, and age-related skin changes all contribute to the development of pressure ulcers.
Of the 7 contributing factors to pressure ulcers, how does Immobility contribute to development? Where are the most susceptible areas?
Immobility
Weight-bearing bony prominences are most susceptible to pressure ulcer development because they are covered only by skin and small amounts of subcutaneous tissue.
Susceptible areas: sacrum and coccygeal areas ischial tuberosities greater trochanter heel knee malleolus medial condyle of the tibia fibular head Scapula elbow
Of the 7 contributing factors to pressure ulcers, how does Sensory Perception or Cognition contribute to development?
Patients with sensory loss, impaired level of consciousness, or paralysis may not be aware of the discomfort associated with prolonged pressure on the skin and therefore may not change their position themselves to relieve the pressure.
Also… I THINK they may not be able to say when they’re wet and may have less control of bladder
How does Decreased Tissue Perfusion contribute to the development of pressure ulcers? What three types of patients are at higher risk and why?
Any condition that reduces the circulation and nourishment of the skin and subcutaneous tissue increases the risk of pressure ulcer development.
Patients with diabetes mellitus experience an alteration in microcirculation.
Similarly, patients with edema have impaired circulation and poor nourishment of the skin tissue.
Patients who are obese have large amounts of poorly vascularized adipose tissue.
How does Altered Nutritional Status contribute to the development of pressure ulcers? Which vitamin and other nutritional elements are especially important?
Nutritional deficiencies, anemias, and metabolic disorders.
Anemia decreases the blood’s oxygen-carrying ability and predisposes a patient to pressure ulcer formation.
Patients who have low protein levels or who are in a negative nitrogen balance experience tissue wasting and inhibited tissue repair.
Serum albumin is a sensitive indicator of protein deficiency; lowered serum albumin levels are associated with hypoalbuminemic tissue edema and increased risk of pressure ulcers.
Vitamin C and trace minerals, are needed for tissue maintenance and repair
How does friction and shear contribute to development of pressure ulcers? What are f and s? and what area is the most susceptible to this type of skin damage?
Friction is the resistance to movement that occurs when two surfaces are moved across each other.
Shear is created by the interplay of gravitational forces and friction.
When shear occurs, tissue layers slide over one another, blood vessels stretch and twist, and the microcirculation of the skin and subcutaneous tissue is disrupted.
The sacrum and heels are most susceptible to the effects of shear.
How does increased moisture contribute to development of pressure ulcers? What is a complication?
Prolonged contact with moisture from perspiration, urine, feces, or drainage produces maceration (softening) of the skin.
The lesion may continue to enlarge and extend deep into the fascia, muscle, and bone, with multiple sinus tracts radiating from the pressure ulcer.
May result in sepsis.
What are Gerontological Considerations of development of pressure ulcers?
Older adults: reduced skin elasticity, decreased collagen, and muscle/tissue atrophy.
Polypharmacy and concomitant medical conditions may affect wound healing.
Decreased inflammatory response, little subcutaneous padding over bony prominences and decreased nutritional intake
How does the nurse assess a patient’s risk for pressure ulcers?
The nurse assesses the patient’s mobility, sensory perception, cognitive abilities, tissue perfusion, nutritional status, friction and shear forces, sources of moisture on the skin, and age.
The nurse performs the following:
• Assesses the total skin condition at least twice a day
• Inspects each pressure site for erythema
• Assesses areas of erythema for a blanching response
• Inspects for dry skin, moist skin, and breaks in skin
• Determines the presence of incontinence
• Notes any drainage and odour
• Evaluates the level of mobility
• Notes restrictive devices (e.g., restraints, splints)
Evaluates circulatory status (e.g., peripheral pulses, edema)
• Assesses the neurovascular status
• Evaluates the nutritional and hydration status
• Reviews the patient’s record for laboratory studies, including hematocrit, hemoglobin, electrolytes, albumin, transferrin, and creatinine
• Notes any present health problems
• Reviews current medications
Spinal Cord Injury Pressure Ulcer Scale (SCIPUS): is used to measure the risk for pressure ulcer development for individuals with a spinal cord injury who are in a rehabilitation centre
How can the nurse help a patient improve Peripheral Arterial Circulation ?
Arterial blood supply to a body part can be enhanced by positioning the part below the level of the heart.
For the lower extremities: elevating the head of the patient’s bed or by having the patient use a reclining chair or sit with the feet resting on the floor.
The nurse instructs the patient to walk to the point of pain, rest until the pain subsides, and then resume walking
The onset of pain indicates that the tissues are not receiving adequate oxygen, signalling the patient to rest before continuing activity.
Not all patients with peripheral vascular disease should exercise.
Conditions that worsen with exercise include leg ulcers, cellulitis, gangrene, or acute thrombotic occlusions.
Which patients are at risk for vascular compression?
If the arteries are severely sclerosed, inelastic, or damaged, dilation is not possible.
What are some nursing interventions for vascular compression? What is some teaching the nurse can do?
Nursing interventions may involve applications of warmth to promote arterial flow and instructions to the patient to avoid exposure to cold temperatures, which causes vasoconstriction.
A hot water bottle or heating pad may be applied to the patient’s abdomen, causing vasodilation throughout the lower extremities.
Nicotine from tobacco products causes vasospasm, reduces circulation to the extremities.
Emotional upsets stimulate the sympathetic nervous system, resulting in peripheral vasoconstriction.
Crossing the legs for more than 15 minutes at a time should be discouraged because it compresses vessels in the legs.
How can the nurse help to relieve pain associated with reduced perfusion, vascular compression, ulcers?
Analgesic agents such as oxycodone (OxyContin) plus acetylsalicylic acid (Aspirin), or oxycodone plus acetaminophen (Tylenol) may be helpful in reducing pain so that the patient can participate in therapies that can increase circulation and ultimately relieve pain more effectively.
What teaching can the nurse do to help the patient maintain tissue integrity?
Poorly perfused tissues= susceptible to damage and infection. When lesions develop, healing may be delayed or inhibited because of the poor blood supply to the area.
Advising the patient to wear sturdy, well-fitting shoes or slippers to prevent foot injury and blisters may be helpful, and recommending neutral soaps and body lotions may prevent drying and cracking of skin.
Instruct the patient not to apply lotion between the toes because the increased moisture can lead to maceration of tissue.
Feet should be patted dry. Stockings should be clean and dry.
Fingernails and toenails should be carefully trimmed straight across and sharp corners filed to follow the contour of the nail.
Good nutrition promotes healing and prevents tissue breakdown (adequate protein and vitamins is necessary for patients with arterial insufficiency)
Vitamin C is essential for collagen synthesis and capillary development.
Vitamin A enhances epithelialization.
Zinc is necessary for cell mitosis and cell proliferation.
Weight reduction: Obesity strains the heart, increases venous congestion, and reduces circulation
What causes venous insufficiency? What are the clinical manifestations?
Venous insufficiency results from obstruction of the venous valves in the legs or a reflux of blood through the valves.
Clinical Manifestations
Postthrombotic syndrome: by chronic venous stasis, resulting in edema, altered pigmentation, pain, and stasis dermatitis.
symptoms less in the morning and more in the evening.
Stasis ulcers develop as a result of the rupture of small skin veins and subsequent ulcerations.
When these vessels rupture, red blood cells escape into surrounding tissues and then degenerate, leaving a brownish discolouration
The pigmentation and ulcerations usually occur in the lower part of the extremity(medial malleolus of the ankle).
What are Postthrombotic syndrome? Stasis ulcers?
Manifestations of venous stasis
Postthrombotic syndrome: by chronic venous stasis, resulting in edema, altered pigmentation, pain, and stasis dermatitis.
symptoms less in the morning and more in the evening.
Stasis ulcers develop as a result of the rupture of small skin veins and subsequent ulcerations.
When these vessels rupture, red blood cells escape into surrounding tissues and then degenerate, leaving a brownish discolouration
Where do ulcers and pigmentation usually occur in a venous ulcer?
The pigmentation and ulcerations usually occur in the lower part of the extremity(medial malleolus of the ankle)
what are the complications of venous stasis?
Venous ulceration is the most serious complication
Cellulitis or dermatitis
what are the interventions of venous stasis?
Elevating the leg, and compression of superficial veins with graduated compression stockings.
The legs should be elevated frequently throughout the day (at least 15 to 30 minutes every 2 hours).
At night, the patient should sleep with the foot of the bed elevated about 15 cm.
Compression of the legs with graduated compression stockings reduces the pooling of venous blood, enhances venous return to the heart, and is recommended for people with venous insufficiency.
It is recommended that stockings with 30 to 40 mm Hg pressure be used during the first year post-DVT
Each stocking should fit so that pressure is greater at the foot and ankle and then gradually declines to a lesser pressure at the knee or groin.
Stockings should be applied after the legs have been elevated for a period, when the amount of blood in the leg veins is at its lowest.
skin is kept clean, dry, and soft.
What is a leg ulcer and what are the manifestations?
Leg Ulcers
A leg ulcer is an excavation of the skin surface that occurs when inflamed necrotic tissue sloughs off.
Clinical Manifestations
The symptoms depend on arterial or venous in origin
The severity of the symptoms depends on the extent and duration of the vascular insufficiency. The ulcer itself appears as an open, inflamed sore. The area may be draining or covered by eschar (dark, hard crust).
What are arterial ulcers?
Arterial Ulcers
Chronic arterial disease is characterized by intermittent claudication (a condition in which cramping pain in the leg is induced by exercise, typically caused by obstruction of the arteries.)
If the onset of arterial occlusion is acute, ischemic pain is unrelenting and rarely relieved even with opioids.
Arterial ulcers are small, circular, deep ulcerations on the tips of toes or in the web spaces between the toes.
Ulcers often occur on the medial side of the hallux or lateral fifth toe and may be caused by a combination of ischemia and pressure
Arterial insufficiency may result in gangrene of the toe (digital gangrene), which usually is caused by trauma.
Débridement is contraindicated in these instances.
Managing dry gangrene is preferable to débriding the toe and causing an open wound that will not heal because of insufficient circulation.
Dry gangrene of the toe in an elderly person with poor circulation is usually left undisturbed.
Where do arterial ulcers usually occur?
Arterial ulcers are small, circular, deep ulcerations on the tips of toes or in the web spaces between the toes.
Ulcers often occur on the medial side of the hallux or lateral fifth toe and may be caused by a combination of ischemia and pressure
What are venous ulcers?
Chronic venous insufficiency is characterized by pain described as aching or heavy.
The foot and ankle may be edematous.
Ulcerations are in the area of the medial or lateral malleolus (gaiter area)
Typically large, superficial, and highly exudative.
Patients with neuropathy frequently have ulcerations on the side of the foot over the metatarsal
Heads (painless).
What type of pain is associated with arterial ulcers?
Chronic arterial disease is characterized by intermittent claudication (a condition in which cramping pain in the leg is induced by exercise, typically caused by obstruction of the arteries.)
If the onset of arterial occlusion is acute, ischemic pain is unrelenting and rarely relieved even with opioids.
What type of pain is associated with venous ulcers?
Chronic venous insufficiency is characterized by pain described as aching or heavy.
Where do venous ulcers usually appear and what is their general appearance?
Ulcerations are in the area of the medial or lateral malleolus (gaiter area)
Typically large, superficial, and highly exudative.