Week 7 Flashcards

1
Q

Musculoskeletal development in infants

A
  • Matrix for skeleton laid down in first month of conception
  • Joints and muscles formed by 3 months gestation and ready
    for further development
  • Features at birth
    -Kyphosis from c-spine to sacrum
    -Hip flexion/lateral rotation
    -Medial rotation of tibia
    -Equinus position of feet
    -Varying limitation of hip, knee, elbow extension
  • Response physical demands
  • Muscle tissue growth
    -Tension leads to longitudinal growth
    -Rapid foetal growth passively stretches muscles and stimulates it
    to grow longer at the same rate as the bone.
  • Bone growth
    -After birth, long bones grow in length at epiphyseal plate
    -Cartilaginous plate (Proliferates, Chondrocytes, Converted to bone, Ossification)
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2
Q

Changes in lower limb alignment

A

Newborn:
- Moderate genu varum
6 months:
- Minimal genu-varum
1 to 2 years:
- Legs straight
2 to 4 years:
-Physiologic genu valgus
16 year old females:
- Slight genu valgum
16 year old males
- Slight genu varum

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3
Q

Conditions

A
  • Fractures
  • Clubfoot (congenital talipes equino-varus)
  • Postural foot/hand deformities
  • Developmental dysplasia of the hip
  • Torticollis/deformational plagiocephaly
    -Obstetrical nerve palsies
    -Osteogenesis imperfecta
    -Arthrogryposis
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4
Q

Developmental dysplasia of the hip (DDH)

A
  • Abnormal development of hip joint

Aetiology:
- Intrauterine positioning resulting in a stretched posterior capsule
- Neurological condition
- The hip presents as dislocated, dislocatable or subluxed

Management:
- All babies screen at birth
- Irremovable splint fitted on day 1 (von rosen splint)

Physio:
- Need to understand DDH
- May suspect the condition if:
* limb length discrepancy
* asymmetry of movement
* limited range of hip abduction
* asymmetrical thigh folds
* limping or waddling gait (toddler)
* may be associated with torticollis
- Immediate referral for orthopaedic
review

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5
Q

Congenital muscular torticollis (CMT)

A
  • Involves shortening of the sternocleidomastoid (SCM)
    muscle + tumour
  • Tilt their head…
  • toward the side of the affected muscle
  • rotate toward the contralateral side.
  • Upper trapezius muscle can also be involved and contribute to the neck deformity
  • Unknown cause
  • If torticollis persists, craniofacial deformities or plagiocephaly can occur
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6
Q

Torticollis

A
  • Secondary to
  • Skeletal
  • Neurological
  • Muscular (Sternomastoid tumour, Muscular tightness, Postural)
  • Visual/vestibular disorder
  • Aim for active correction
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7
Q

Deformational plagiocephaly

A
  • Postural condition
  • Oblique, misshapen, parallelogram head
  • Repeated external pressure to the head as a result of being in the one position for extended lengths of time (sleeping)
  • Flattening of the occipital bone,
  • More prominent forehead and cheek on that side
  • The ear pushed forward compared with the other

Prevention/management:
- Infant positioning is associated with early head orientation and plagiocephaly development. Early parent-reported asymmetry during awake and sleep time is an important indicator for the need for
professional assessment and advice.
-From birth – alternate the head position when sleeping
-Encourage active head control strategies (prone, sideways lift etc)
-If no improvement after 2 sessions, ensure a medical review to rule out pathology (craniosynostosis, cervical rib)
-Do not refer for helmet unless specialist review has occurred

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8
Q

Brachial plexus palsy

A

Causes:
- Prenatal (position in utero)
- Trauma during labour
- Delivery (shoulder dystonia)
- Klumpke (C7-8, T1 nerve roots)
- Erb (C5-6 nerve roots)

Assessment:
- Background information
- Medical review
- CNS + PNS examination (gentle) and documentation
- X-ray – fractures, hemidiaphragm(phrenic nerve injury)
- Serology e.g. exclude septic join

Objective:
- Observe – supine and prone (including alertness, shoulder muscle
wasting, marked deformities)
- Spontaneous movement whole baby
- Sucking – looking for oromotor involvement
- Active movement – shoulder abduction (Moro), elbow flexion (recoil or stroking of biceps belly), wrist extension (placing reaction)
- Passive movement – including shoulder ER in neutral
- Sensation
- Other problems: # clavicle or humerus, postural foot deformities,
torticollis, DDH

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8
Q

Brachial plexus palsy

A

Causes:
- Prenatal (position in utero)
- Trauma during labour
- Delivery (shoulder dystonia)
- Klumpke (C7-8, T1 nerve roots)
- Erb (C5-6 nerve roots)

Assessment:
- Background information
- Medical review
- CNS + PNS examination (gentle) and documentation
- X-ray – fractures, hemidiaphragm(phrenic nerve injury)
- Serology e.g. exclude septic join

Objective:
- Observe – supine and prone (including alertness, shoulder muscle
wasting, marked deformities)
- Spontaneous movement whole baby
- Sucking – looking for oromotor involvement
- Active movement – shoulder abduction (Moro), elbow flexion (recoil or stroking of biceps belly), wrist extension (placing reaction)
- Passive movement – including shoulder ER in neutral
- Sensation
- Other problems: # clavicle or humerus, postural foot deformities,
torticollis, DDH

Management:
- Pain relief
- Parental reassurance (90% recover within 3 months)
- Minimal handling – do not passively move/range arm
- Gentle massage and stroking to facilitate active movement
- Swaddling with hands to mouth
- Swaddled bathing
- Positioning - affected side up for feeding and sleeping
- Education parents/staff re supporting arm with handling
- Peanut pillow

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9
Q

Palsies

A

Sciatic
- presents with foot drop
- Managed with splinting, passive movements and stimulation of affected muscle groups

Facial:
- Presents with feeding difficulties and asymmetrical facial movements
- managed with oral stimulation, including jaw and check support for feeding
- Watch for other asymmetries (torticollis)

Radial
- Presents with wrist drop
- managed with splinting, stimulation of affected groups

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10
Q

Osteogenesis imperfecta

A
  • Brittle Bone Disease
  • Failure of bone matrix formation
  • Congenital osteoporosis
  • Blue sclera
  • Early hearing loss
  • Developmental approach
  • Equipment – bean bags, seating etc.
  • # identification and immobilisation
  • Parental support
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11
Q

Arthrogryposis multiplex congenital

A
  • Non progressive neuromuscular syndrome
  • Joint contractures
  • Muscles (poorly developed, replaced by fibrous tissue)
  • Early treatment of deformities (clubfoot, hip and knee contractures)
  • Casting/Splinting
  • Handling and positioning
  • Developmental car
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