week 6 Flashcards

1
Q

Primary OA

A

No know caused may be localized or generalized  DIP and first CMC joints most often involved  Incidence increases with age

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2
Q

Secondary OA

A

Has identifiable cause such as trauma  Any age

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3
Q

Defined as a gradual loss of articular cartilage due to degenerative joint disease and chemical factors

A

Osteoarthritis

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4
Q

Clinical Manifestations of OA

A

Joint pain  Boney enlargement  Stiffness  Tenderness  Limited motion  Crepitus  Malalignment  Joint deformity  Inflammation (edema, soft-tissue can be observed in acute exacerbations)

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5
Q

OA joint deformities

A

CMC jt commonly effected  Characterized by thumb adduction and subluxation from the trapezium, MP hyperextension, IP joint flexion  Pinch is painful  Heberden’s nodes (DIP jts)  Bouchard’s nodes (PIP jts

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6
Q

OT Treatment for OA

A

Patient Education  Weight management  Joint protection techniques  Adaptive equipment  Environmental modifications  Energy conservation

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7
Q

Joint protection techniques

A

Respect pain  Minimize force and load on joints  Balance rest and activity  Use larger, stronger joints  Good body mechanics  Avoid positions of deformity  Decrease effort and resistance  Maintain ROM

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8
Q

OT interventions

A

Modalities (PAMs)- depending on your state and facility  Pain-free ROM  AROM exercises  Isometric strengthening  General conditioning  Avoid pinch strengthening  Splinting  Thumb – Short opponens splint  Custom, prefab, neoprene

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9
Q

Chronic autoimmune disorder affecting 1.3 million Americans synovial membrane of a joint becomes inflamed and wears away at the bone, cartilage and soft tissues which contributes to joint deformities

A

RA

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10
Q

Clinical Manifestations of RA

A

Symmetric polyarticular pain  Swelling  Morning stiffness (1-2 hours)  Joint deformities  Rheumatoid nodules  Fatigue  Depression

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11
Q

Joint swelling and inflammation, no destructive changes, possible presence of osteoporosis on X-ray “flare” 

A

Stage 1, Early phase Acute phase of RA

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12
Q

synovium begins to invade the soft tissues producing tenosynovitis and limiting joint movement (no joint deformities yet), adjacent muscle atrophy

A

Stage 2, Moderate Phase (Proliferative) of RA

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13
Q

synovial erosion causes irreversible changes including joint deformities, loosening of ligamentous insertions, impairment of tendon function, muscle atrophy and joint disorganization. X-ray evidence of cartilage and bone destruction and osteoporosis 

A

Stage 3, Severe Phase (Destructive):

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14
Q

synovial activity “burnt out” fibrosis or bony ankylosis

A

Stage 4, Terminal phase (Chronic)

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15
Q

Extensor tendons slip to the ulnar aspect of the metacarpal head

A

Ulnar deviation of digits at MP joints (Ulnar drift)

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16
Q

PIP hyperextension with DIP extension lag, due to erosion of the PIP volar plate, rupture of lateral band or FDS rupture 

A

swan neck deformity

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17
Q

PIP flexion contracture with DIP hyperextension (caused by rupture or lengthening of the central slip of the EDC)

A

Boutonniere deformity

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18
Q

OT treatment for RA

A

Education  Prevent joint deformities  Sleep and Rest  Modalities (PAMs)  Therapeutic exercise  Adaptive equipment  Splinting  For rest, deformity, function  Joint Protection  Fatigue management  Energy Conservation

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19
Q

aidsthe immune system inremoving and destroying waste, debris, dead blood cells, pathogens,toxins, and cancer cells. The lymphatic system removes excess fluid, and waste products from the interstitial spaces between the cells.

A

lymphatic system

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20
Q

This is the fluid that is in the tissues between cells. So fluid that is not in cells but between them.

A

is interstitial fluid

21
Q

Lymph is made up of

A

protein, water, cellular components and particles, and fat. White blood cells, called lymphocytes, are one type of cellular particle in lymph. These play a roll in the immune response of the body.

22
Q

Lymphatic system development

A

The lymphatic system begins to develop during the fifth week of gestation. It develops from the mesoderm. Connective tissue, muscle, bone, urogenital and circulatory systems all develop from the mesoderm which is the middle layer of the 3 primary germ cell layers. (Embryonic development and gestation)

23
Q

difference between the lymphatic system and circulatory system

A

The lymphatic system lays parallel to the circulatory system. The lymphatic system is an open system where the circulatory system is a closed system. The circulatory system also differs from the lymphatic as it has an electric pump THE HEART! The lymphatic system relies on muscle contractions to help facilitate lymph movement, minimal muscle contractions within some lymphatic vessels and a small vacuum from lymph nodes.

24
Q

the movement of particles in a solution from an area of higher concentration to an area of lower concentration.

A

Diffusion

25
Q

movement of water molecules from a place of higher water concentration to a place of lower concentration through a permeable membrane.

A

Osmosis

26
Q

outward forces are greater than inward forces.

A

Filtration

27
Q

inward forces outweigh outward forces.

A

Reabsorption

28
Q

responsible for draining lymph from the skin and subcutaneous tissue.

A

superficial layer

29
Q

drains lymph from muscle tissue, tendon sheaths, nervous tissues, the periosteum, and joint structures.

A

deep layer

30
Q

begin the drainage system. These are located in close proximity to blood capillaries. Through open junctions the lymph capillaries are able to absorb interstitial fluid.

A

Lymph capillaries

31
Q

connect lymph capillaries and collectors. In some areas they have wall like lymph capillaries and are able to absorb lymph and have valves like lymph collectors.

A

Precollectors

32
Q

transport lymph to lymph nodes and lymphatic trunks. These vessels have valves that allow lymph to more in one direction, proximal.

A

lymph collectors

33
Q

act as protector by filtering debris from lymph, immune function with production of antibodies, and thicken lymph trough the reabsorption of water.

A

lymph nodes

34
Q

generally have a more developed muscle structure and receive lymph from superficial and deep layers. These vessels combine at the cisterna chyli and there become the largest lymph vessel called the thoracic duct.

A

lymphatic trunks

35
Q

the largest lymph vessel of the body, transports lymph to the venous angle.

A

thoracic duct

36
Q

the junction between the left internal jugular and left subclavian vein.

A

The left venous angle

37
Q

Most common insufficiency
Caused by lymphatic load exceeding capacity of the anatomical and functionally intact lymphatic system
May occur as a result of insufficient cardiac or kidney function

A

Dynamic Insufficiency

38
Q

This is when both dynamic and mechanical insufficiencies are present Both the transport capacity is reduced and the lymphatic load is increased.

A

Combined Insufficiency

39
Q

Is a reduction in transport capacity due to functional or organic causes
May occur as a result of tissue removal or damage. Tissues may be removed due to medical intervention for cancerous tissues. Damage may occur to tissues during cancer treatment such as radiation, burns to skin, or the presence of toxins such as medicines or parasites.

A

Mechanical Insufficiency

40
Q

a low-protein swelling, is a symptom rather than a disease. Several conditions may cause edema such as congestive heart failure, chronic venous insufficiency, pregnancy, immobility, constricting garments and/or jewelry.

A

EDEMA

41
Q

is a disease rather then a symptom and is a high-protein swelling. This high-protein content can result in secondary complications such as hardening of tissues, infections, and an increase in limb size.

A

lymphedema

42
Q

Edema that begins immediately as a result of trauma or infection. Generally lasts up to 5 days and then begins to subside.

A

Acute Phase

43
Q

treatment for acute phase of edema

A

Limited active motion of uninvolved areas

Balance of activity and rest for all structures to prevent inflammation or increase in edema

Elevation (ensure not contraindicated for client situation)
Loose elastic glove or stockinette
Coban
Finger bandage wraps

Observation of visible skin coloring while bulky dressings/orthoses are utilized

Light retrograde massage

44
Q

continues beyond the acute phase and becomes chronic if still present longer then 3 months.

A

subacute edema

45
Q

is hard, indurated, and resistant to pitting

A

chronic edema

46
Q

treatment for subacute edema

A

Start proximally with diaphragmatic breathing, trunk stretches, trunk exercises, easy yoga trunk stretches

Active and passive exercise
Loose elastic glove, loose stockinette, cotton finger wrap bandages and Coban
Elastic taping

47
Q

treatment for lymphedema

A

Complete decongestive therapy involves manual lymph drainage (MLD) to drain congested tissues
Short stretch compression bandaging
Education on nutrition, self massage and bandaging, skin care
Development and teaching exercise program
Measurement and ordering of compression garments to maintain limb size *requires advanced training

48
Q

precautions for treating edema

A

In the presence of congestive heart failure (CHF), kidney failure, blood clots (DVT), infection, and undiagnosed cancer DO NOT apply interventions for edema. Medical clearance needs to be secured prior to treatment.