week 6 Flashcards
Primary OA
No know caused may be localized or generalized DIP and first CMC joints most often involved Incidence increases with age
Secondary OA
Has identifiable cause such as trauma Any age
Defined as a gradual loss of articular cartilage due to degenerative joint disease and chemical factors
Osteoarthritis
Clinical Manifestations of OA
Joint pain Boney enlargement Stiffness Tenderness Limited motion Crepitus Malalignment Joint deformity Inflammation (edema, soft-tissue can be observed in acute exacerbations)
OA joint deformities
CMC jt commonly effected Characterized by thumb adduction and subluxation from the trapezium, MP hyperextension, IP joint flexion Pinch is painful Heberden’s nodes (DIP jts) Bouchard’s nodes (PIP jts
OT Treatment for OA
Patient Education Weight management Joint protection techniques Adaptive equipment Environmental modifications Energy conservation
Joint protection techniques
Respect pain Minimize force and load on joints Balance rest and activity Use larger, stronger joints Good body mechanics Avoid positions of deformity Decrease effort and resistance Maintain ROM
OT interventions
Modalities (PAMs)- depending on your state and facility Pain-free ROM AROM exercises Isometric strengthening General conditioning Avoid pinch strengthening Splinting Thumb – Short opponens splint Custom, prefab, neoprene
Chronic autoimmune disorder affecting 1.3 million Americans synovial membrane of a joint becomes inflamed and wears away at the bone, cartilage and soft tissues which contributes to joint deformities
RA
Clinical Manifestations of RA
Symmetric polyarticular pain Swelling Morning stiffness (1-2 hours) Joint deformities Rheumatoid nodules Fatigue Depression
Joint swelling and inflammation, no destructive changes, possible presence of osteoporosis on X-ray “flare”
Stage 1, Early phase Acute phase of RA
synovium begins to invade the soft tissues producing tenosynovitis and limiting joint movement (no joint deformities yet), adjacent muscle atrophy
Stage 2, Moderate Phase (Proliferative) of RA
synovial erosion causes irreversible changes including joint deformities, loosening of ligamentous insertions, impairment of tendon function, muscle atrophy and joint disorganization. X-ray evidence of cartilage and bone destruction and osteoporosis
Stage 3, Severe Phase (Destructive):
synovial activity “burnt out” fibrosis or bony ankylosis
Stage 4, Terminal phase (Chronic)
Extensor tendons slip to the ulnar aspect of the metacarpal head
Ulnar deviation of digits at MP joints (Ulnar drift)
PIP hyperextension with DIP extension lag, due to erosion of the PIP volar plate, rupture of lateral band or FDS rupture
swan neck deformity
PIP flexion contracture with DIP hyperextension (caused by rupture or lengthening of the central slip of the EDC)
Boutonniere deformity
OT treatment for RA
Education Prevent joint deformities Sleep and Rest Modalities (PAMs) Therapeutic exercise Adaptive equipment Splinting For rest, deformity, function Joint Protection Fatigue management Energy Conservation
aidsthe immune system inremoving and destroying waste, debris, dead blood cells, pathogens,toxins, and cancer cells. The lymphatic system removes excess fluid, and waste products from the interstitial spaces between the cells.
lymphatic system