Week 5 - external environment & one's own representations [2] Flashcards
Evidence for dbl diss: episodic /// semantic memory
CASE KC - Tulving et al, 1988 –> patient demonstrated inability to recollect personally experienced events from birth, but pre-trauma knowledge about maths, geography & general knowledge was preserved
(Green and Hodges, 1996) BRAIN –> patient experiences progressive loss of knowledge surrounding public figures (‘semantic dementia’) but showed no detectable increase in retrograde amnesia for personal experiences.
Anatomy of memory - which regions for which purpose?
Hippocampus - episodic memory (events)
Parahippocampal region - semantic memory (facts)
(Graham et al., 2000) Observations of semantic dementia? What does this suggest?
SEMANTIC DEMENTIA- temporal lobe atrophy
- no recognition of famous faces, buildings or sort animals into the correct category
- could mimic actions of objects through gestures but could’t say what they were
EARLY ALZHEIMERS - hippocampal atrophy
CASE MS - outline
29 year old, surgery of the RHS occiptal lobe due to epilepsy. Hemaniopic.
Test subject on implicit memory task (priming task) against healthy and amnesic controls
CASE MS - findings
- no sig diff in conceptual priming
- less visual priming effect is seen for MS on word completion task. Impaired explicit memory.
CASE MS - implications
supports idea that there is a double dissociation between implicit memory /// explicit memory
- explicit memory has a distinct processing system from implicit memory
- perceptual priming is damaged in MS
What is learning?
Examples of implicit and explicit learning
An enduring change in behaviour that results from experience
Implicit – riding a bike, driving a car
Explicit - conscious recall of info eg for an exam
Example of implicit memory task
Cognitive task, such as filling in the blanks of a word (word completion task).
Repetition priming gives the participant a question along with a previously presented cue that could evoke words that may be related to this one cue
What areas of the memory are spared and impaired in amnesia ?
Spared: STM and non-declarative memory
Impaired: episodic memory definitely, semantic typically.
Organ of representation – what is meant by this?
The brain re-constructs events to plan for the future.
The brain doesn’t ‘replay’ events, it re-represents them, therefore is prone to error.
Organ of representation –> what areas are involved in autobiographical retrieval and semantic planning?
- Medial prefrontal cortex
- Posterior mediolateral cortex
- Laterotemporal cortex
Bartlett, 1932 study ‘ the war of the ghosts ‘ – what did this study involve and demonstrate?
Wanted to investigate how the recall of a story is impacted by existing knowledge/cognitive schemas (due to influences such as cultural background or unfamiliarity)
Hypothesised that memory is stored and retrieved according to cultural schemas
What happens when we forget, and why do we forget?
Forgetting is functional to remembering –> we are not making economical use of cognitive resources if we retain detailed info within the brain indefinitely. It is an adaptive feature that facilitates updating
As retrieval decreases, accessibility decreases
Why do amnesiacs forget?
Memories fade as a function of time. Lack of glutamate is argued to be a causative factor in amnesia –> facilitate biochemical changes required for memory formation.
Primacy and recency effect (Capitani, Della Sala, Logie, Spinnler, 1992) - what is it?
Reflects typical functioning of the STM and LTM. The serial position curve is a demonstration of this.
Recency effect is a property of all types of memory systems, including verbal STM
Experiments show that when participants are presented with a list of words, they tend to remember the first few and last few words and are more likely to forget those in the middle of the list.