Week 3 Recognition of the Sick Child Flashcards
1
Q
What’s the anatomical difference between children and adult?
A
Airway
- smaller, ↑resistance (more rapid reaction to impact)
- soft laryngeal cartilage (different way in position head for CPR)
Respiratory
- less lung capacity
Circulatory
- less blood but faster circulation (loss of blood has greater impact)
- sick children general display tachycardia
Neurologic
- proportionally large heavy head
- thin but flexible skull (injury is hard to detect if underlying)
Musculoskeletal
- flexible and plastic bones
- soft rib cartilage (risk of underlying injury)
Immunity
- immature immunity (vaccination)
Metabolic & Thermoregulation
- higher metabolic rates (SpO2 coming down when unwell)
- less subcut fat (variable baseline to body temperature)
2
Q
What are the common causes and clinical features of children cardiac arrest?
A
Respiratory obstruction ↓ Respiratory failure → ↑ Respiratory depression
CARDIAC FAILURE Fluid loss ↓ Circulatory failure → ↑ Fluid maldistribution
3
Q
What is the Paediatric Assessment Triangle?
A
Work of breathing: ?exertion
Appearance: ?cyanosis, rashes
Circulation: ?tachycardia
4
Q
What is the structured assessment for children?
A
Airway:
- Inspiratory noises being abnormal: bubbly, snoring, stridor
- Changes in vital signs (ie. ↑RR, ↓SpO2)
- Croup (upper airway inflammation)
Breathing:
- Effort: how hard is the baby/child working → ?RR
- Efficacy: how effective they are in gas exchange → ?cyanosis ?HR
Circulation:
- Direct signs: ?HR ?BP ?Capillary Refill Time
- Secondary signs: level of consciousness ?confusion ?agitation
- Perfusion: ?Colour (pale; mottled) ?Cap refill<3sec
Disability: A - alert ?drowsy V - respond to voice P - respond to pain U - unresponsive
Modified GCS (paediatric version)
Fluids:
- Intake & Output
- Hydration status ?sunken eye ?dry mucous membranes
- Gastroenteritis: virus-induced, damage to gut mucosa and result in absorbing disturbance*
- Antiemetic is not preferred due to AEs*
Temperature:
- 36.5~37.5
- Antipyretics given as a comfort to distress (ie. Paracetamol)
5
Q
Croup Management
A
Croup: sternal recession, trachea tug
- Positioning
- Frequent Obs
- PRN O2/Intubation
- administer medications:
• dexamethasone
• Nebulised adrenaline
6
Q
Bronchiolitis
A
Etiology:
- Most common severe LRTI
- RSV* (Respiratory Syncytial Virus)
Pathophysiology: - mucosal inflammation/edema - bronchioles constrict during expiration causing air trapping in alveoli and hyperinflation of lungs - impaired gas exchange
S/S:
- rhinorrhea, cough, fever
- tachypnea, tachycardia
Initial management:
- frequent VS
- humidified O2
- nasopharyngeal suctioning
- infection control
7
Q
Asthma
A
Etiology:
- genetic defects
- allergens
Examination:
- Prolonged expiration
- Generalised wheeze
- Be aware of Slient Chest*
Management:
- Oxygen (high flow)
- Beta-Agonists (eg. Salbutamol)
- Reduce inflammation & mucus in airways
- Anticholinergics (Ipratropium bromide)
- Works on nerves that tighten airways (works in 30 minutes)
- Oral Corticosteroids (Prednisolone)
- Reduce inflammation & mucus in airways