Week 3 GI Flashcards

1
Q

What causes cleft lip/cleft palate

A

Failure of the maxillary processes to fuse between 5 to 12 weeks gestation. Failure of the tongue to move down at the correct time prevents the palatine processes from fusing

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2
Q

Risk factors that increase likelihood of cleft palate & RX

A

Advanced maternal age
Poor maternal care (vitamins, nutrition, drugs taken by mother, genetically linked, linked to poor income → lack of access to care
Rx → FOLIC ACID

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3
Q

Nursing management to facilitate feeding in cleft lip/palate

A

Cannot create suction; now newborns and infants feed
Use palate shield and bottles with nozzle-type attachment
OG / NG tubes

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4
Q

Diagnosis of esophageal atresia and tracheoesphageal fistula

A

Sx: THREE Cs – cyanosis, choking, coughing à if you have secretions ending in a blind pouch, it will either come back up or go into the lungs
Soon after birth, placing NG / OG tube – continuous suctioning
Might see a distending abdomen - air trapping

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5
Q

Gastroschisis VS omphalocele

A

Gastroschisis – ABD is open, contents exposed

Omphalocele – muscle has not formed but membrane is containing contents but they are visible

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6
Q

RX for Gastroschisis

A

create silo à meshy prosthetic sack that covers ABD. Top of sack is a cinch string that is tightened over time to put contents back under cover.

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7
Q

RX for Gastroschisis, omphalocele

A

folic acid

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8
Q

Anal stenosis and anal atresia

A

Narrowing or complete covering of rectum

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9
Q

Anal stenosis

A

wall is thickened or constricted
Decreased normal stool passage
Reports of ribbon like stool

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10
Q

Umbilical hernia

A

a weakness in umbilical ring – stomach contents protruding into abd. Can resolve on its own, older children may need surgery.

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11
Q

Meckel’s diverticulum

A

Omphalomesenteric duct fails to atrophy. One of the most common congenital anomalies. Happens a lot in utero but don’t see sx until much much older (may have been plagued with GI sx for a while though)

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12
Q

S/S of Meckel’s Diverticulum

A

Inflammation
Rectal bleeding
Intestinal obstruction

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13
Q

Hirschsprung disease

A

adequate motility that causes mechanical obstruction – congenital link
Congenital absence of ganglion cells in the rectum and colon
Can cause mega colin

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14
Q

S/S in newborn

A

want to see that meconium
over time back in doctor’s office with really infrequent stooling
abd distension
bile stained emesis → bowel contents backing up
can get ostomies
going to see failure to thrive, dev. Delays, severe constipation/ diarrhea
chronic care over time → diet, stool modifications forever
prevent dehydration
rectal stimulation

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15
Q

Pyloric stenosis presentation

A

Child presents with massive vomiting (exorcist) within 20 minutes it’s coming back up; then they are hungry again
Increased peristalsis prior to vomiting
Hyperactive bowel sounds
Olive shaped mass
In pyloric stenosis, the hypertrophied pyloric muscle (feels like a small olive) causes symptoms of projectile vomiting and visible peristalsis.
Picked up on ultrasound based on clinical presentation
At SEVERE risk for dehydration and F/E imbalance – needs to be corrected prior to OR.
Quick OR visit

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16
Q

What is Intussesception

A

part of the intestine folds into the section next to it. It typically involves the small bowel and less commonly the large bowel. Symptoms include abdominal pain which may come and go, vomiting, abdominal bloating, and bloody stool.

17
Q

Three mechanisms allow reflux to occur

A

Lower esophageal relaxations
Incompetent lower esophageal sphincter
Anatomic disruption of esophagogastric junction

18
Q

Those at risk for GERD

A

Preemies

Those w/TEF repairs

19
Q

Appendicitis is cause by

A

Caused by an obstruction of the appendiceal lumen, causing edema

20
Q

Presentation of appendicitis

A
⅔ the distance on right side along umbilicus to superior iliac crest→ highly acute pain
Rebound tenderness (pain upon release)
21
Q

Three hepatic disorder

A

Biliary atresia
Viral hepatitis
Cirrhosis

22
Q

Signs of hepatic disorder

A
Jaundice
Unconjugated bilirubin floating around system → can become toxic if not corrected
Progressive yellowing of skin that moves upwards
Phototherapy
Helps body reabsorb quicker
Easy bruising, intense itching
White or clay-colored stools 
Tea-colored urine
23
Q

Potential Signs of Gastrointestinal Emergencies in Infants

A

Excessive salivation with cyanosis, coughing, and choking in newborn
Esophageal atresia and tracheoesophageal fistula
Abdominal viscera outside the abdominal cavity when born
Gastroschisis and omphalocele
Anorectal malformations