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Neuromuscular practice- Adutls > Week 3 > Flashcards

Week 3 Flashcards

1
Q

What is postural control?

A

Involves controlling the body’s

position in space for stability and orientation

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2
Q

What is postural orientation?

A

Ability to maintain appropriate

relationship between body segments and between body and environment for a task

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3
Q

What is postural stability?

A

Ability to control the center of mass (COM) in relationship to the base of support (BOS)

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4
Q

What is Center of Mass (COM)?

A

Point at center of total body mass, determined by finding weighted average of the COM of each body segment

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5
Q

What is Center of Gravity (COG)?

A

Vertical projection of COM

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6
Q

What is Base of Support (BOS)?

A

Area of body that is in contact with support surface

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7
Q

What is center of Pressure?

A

Center of the distribution of the total force applied to the supporting surface. COP moves continuously around the COM to keep COM within support base

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8
Q

All tasks require __

A

All tasks require postural control

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9
Q

Every task has an ___ component and a ___

component

A

Every task has an orientation component and a stability

component, and each varies with the task and the environment

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10
Q

The relationship between orientation and stability is ____

A

Inverse

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11
Q

What does postural control emerge from?

A

An interaction of the individual, the task with its inherent postural demands, and the environmental constraints postural actions

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12
Q

What are the components of balance?

A
  • Postural tasks
  • Individual
  • Environment
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13
Q

What are the motor components of posture and the individual?

A

Musculoskeletal, muscle synergies

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14
Q

What are the sensory components of posture and the individual?

A

Sensory systems, sensory organization

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15
Q

What are the cognitive components of posture and the individual?

A

Cognitive strategies, cognitive resources (cognitive doesn’t necessarily mean conscious control)

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16
Q

What is included in the cognitive components of posture and the individual?

A

Attention, motivation, intent; also ability to adapt in response to changing demands, anticipatory aspects of postural control based on previous experience and learning

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17
Q

What is the relationship between task being handled at once and attention available for posturing and balance?

A

Inverse, the more task being being handled at once, the less attention available for posturing and balance

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18
Q

What are the support surfaces components of posture and the environment?

A

Changing support surfaces affects organization of muscles and forces needed to balance

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19
Q

What are the sensory context components of posture and the environment?

A

Differences in visual and surface conditions affect way sensory info used for balance

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20
Q

What are the cognitive load components of posture and the environment?

A

Multi tasking affects the way things like attention are used for balance

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21
Q

What may the attention required for postural task lead to?

A

May reduce performance of second task

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22
Q

What is the definition of postural task: steady state?

A

Ability to control COM relative to BOS in fairly predictable, stable and non-changing conditions

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23
Q

What is the definition of postural task: proactive?

A

Ability to activate muscles in legs and trunk for balance control in advance of potentially destabilizing voluntary movements. Relies on feed forward condition (anticipatory posture for destabilization)

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24
Q

What is the definition of postural task: reactive?

A

Ability to recover a stable position following an unexpected perturbation. Relies on feed back mechanism

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25
Q

What are the contributions to postural control?

A
  • Body alignment
  • Muscle tone
  • Postural tone
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26
Q

How does body alignment contribute to postural control?

A

Minimizes effect of gravitational forces

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27
Q

How does muscle tone contribute to postural control?

A

Keeps body from collapsing in response to pull of gravity

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28
Q

How does postural tone contribute to postural control?

A

Keeps body from collapsing in

response to pull of gravity. The activation of the anti-gravity muscle

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29
Q

Where is fixed support recovery of balance?

A
  • Ankle

* Hip

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30
Q

When is ankle strategy used?

A

In small perturbations. Requires good ROM of the ankle

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31
Q

When is hip strategy used?

A

When the perturbation is larger and or faster, and when the BoS is concurrent or smaller than the feet

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32
Q

When is the change in support recovery of balance used?

A

When there is a change in BoS

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33
Q

What is the stepping strategy?

A

Re- aligning the BoS under a falling CoM. Always preceded by a medio-lateral anticipatory postural adjustment. Before you can take a quick step, there has to be a shift in CoM in one direction or the other

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34
Q

Who are those that will find the stepping strategy very difficult to accomplish?

A

Patients with decreased ability to weight shift over their BoS. (stroke or weakness due to injury. will do for reach-to-grasp instead)

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35
Q

How does the reach-to-grasp strategy work?

A

By extending the BoS by using the arm

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36
Q

____ makes it possible to ensure that the forces generated from one point doesn’t produce instability in the other parts of the body

A

CNS makes it possible to ensure that the forces generated from one point doesn’t produce instability in the other parts of the body

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37
Q

What do postural muscles activated before voluntary

movements do?

A

Minimizes disturbance to balance

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38
Q

What do visual, somatosensory, and vestibular systems that provide info about body position and movement in space with respect to gravity and environment do?

A

Each provide different frame of reference for postural control

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39
Q

What are the spinal contributions to controlling postural orientation and stability?

A

Tonically active extensor muscles for antigravity support in postural orientation; somatosensory contributions to postural control

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40
Q

What are the brainstem contributions to controlling postural orientation and stability?

A

Level of postural tone, automatic postural synergies that occur during poor posture, vestibular contributions to postural control

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41
Q

What are the basal ganglia contributions to controlling postural orientation and stability?

A

Postural Set (ability to quickly change muscle patterns)

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42
Q

What are the cerebellum contributions to controlling postural orientation and stability?

A

Control of adaptation (ability to modify postural muscle amplitude)

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43
Q

What are the consequences of impaired stability?

A
  • Loss of functional independence
  • Reduced or restricted participation
  • Reduced confidence
  • Increased fall risk
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44
Q

What are the characteristics of alignment as a problem in motor systems?

A

Often changed in persons with neuromuscular dysfunction

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45
Q

What are the characteristics of postural sway as a problem in motor systems?

A

Increased postural sway area and velocity; asymmetrical sway area. Sway areas will be biased towards the active area

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46
Q

What are the characteristics of functional stability limits as a problem in motor systems?

A

Reduced limits of stability

(reduced COP excursion). Inability to move CoP over the BoS, causes instability

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47
Q

What are the characteristics of steady-state balance in sitting as a problem in motor systems?

A

All about control of trunk

segmentally; good prognostic indictor of outcome following stroke and TBI.

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48
Q

What are the components of impaired reactive balance?

A
  • Sequencing Problems
  • Coactivation
  • Delayed onset of postural responses
  • Problems modifying postural strategies
  • Impaired central set
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49
Q

What are sequencing problems?

A

The inability to sequence firing muscle. Simply messes with the sequence with which the needed muscles are activated

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50
Q

What is central set?

A

The ability to adapt quickly to changes in task and or the environment. Gets impaired due to neurological injuries

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51
Q

What are the components of impaired change in support strategies?

A
  • Lack of anticipatory lateral weight shift
  • Delayed onset of support strategy
  • Take many steps to recover
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52
Q

When is anticipatory lateral weight shift critical?

A

Before any stepping strategy can occur.

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53
Q

What are the components of impaired anticipatory postural control?

A

• Heavily dependent on previous experience and
learning
• Lack of anticipatory activation in “affected side” in stroke, brain injury
• Lack of anticipatory trunk activation
• Seen with lesions in many areas of nervous system
- Circuitry for anticipatory postural control involves
supplementary motor cortex, basal ganglia, and cerebellum

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54
Q

What are the characteristics of sensory problems that can affect reactive balance?

A
  • Triggers centrally organized postural synergies
  • Providing direct sensory feedback to scale magnitude of automatic postural responses. This ensures that the sensory response given is proportionate to the instability/imbalance felt
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55
Q

What are the characteristics of sensory problems that can affect anticipatory balance?

A
  • Profound changes in motor adaption

* Earlier activation of anticipatory postural adjustments (compensation)

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56
Q

What are the characteristics of perceptual problems that can affect postural control?

A

• Perceptions of verticality: often impaired in stroke,

correlates with poor balance

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57
Q

What are the characteristics of balance and falls self-efficacy as a problem in cognitive systems?

A
  • Confidence in ability to perform activities without losing balance or falling
  • Lack of self-efficacy is common in persons with neuromuscular dysfunctions
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58
Q

What are the characteristics of impaired postural stability and dual-task interference as a problem in cognitive systems?

A
  • Amount of attention required depends on difficulty
  • Impaired postural control increases attentional demands of balance
  • Less reserve for other tasks
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59
Q

What is agnosia?

A

An inability to recognize or perceive familiar sensory information even though the sensory system is without deficit.

Disturbance of one of the sensory modes of visual,
tactile, proprioceptive, and auditory, or may involve
problems in body scheme

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60
Q

What is visual agnosia?

A 
• Perception without meaning,
inability to recognize visual stimuli despite adequate visual
functioning
• Failure to recognize familiar
colors, objects or name them
using vision
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61
Q

What is prosopagnosia?

A

Failure to recognize people or faces. Highly uncommon

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62
Q

How is the evaluation of agnosia done?

A

• Rule out sensory deficit and
anomia
• Object recognition
• Visual Identification of Objects

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63
Q

How do we manage visual agnosia?

A
  • Use real objects
  • Physical guidance
  • Practice discrimination between objects
  • Compensate with other sensory modalities
64
Q

What is auditory agnosia?

A

Failure to identify meanings of sounds, cannot discriminate between speech and non speech sounds

65
Q

How do we manage auditory agnosia?

A

Drilling or practice compensate with other sensory modalities (usually done by speech
pathologist)

66
Q

What is tactile agnosia?

A

Astereognosis- inability to recognize objects by handling them even though the tactile sensations are without deficit

67
Q

How is tactile agnosia evaluated?

A

• Place objects in hand, have pt hold it, then remove it
and place on a tray; have pt show you which object he
had in his hand
• Ayres’ Manual for Perception: 10 plastic geometric forms placed in clients hand and they are to identify the object by finding it on a chart

68
Q

How do we manage tactile agnosia?

A

Practice or compensation(visual is the most common)

69
Q

What is apraxia?

A

An inability to perform voluntary purposeful movements even though the sensory, motor, and cerebellar systems are without deficit. It is
a motor programming disorder. Misuse of objects or using them in an incorrect manner

70
Q

What is ideomotor apraxia?

A

The inability to produce gestures or perform a purposeful motor task on command even though the person understands the concept of the task. Occurs when there is a failure to transmit the plan of action and convert it to a motor movement in the frontal lobes.

71
Q

What is ideational apraxia?

A

Patient is unable to formulate a plan of action. They cannot develop the sequence or create the motor patterns necessary to execute the task. They cannot describe the task or items that would be used to
complete the task

72
Q

What are the differences between ideomotor and ideational apraxia?

A

• Pt with ideomotor apraxia will not be able to do an activity
to command, but will be able to do it automatically at the
appropriate time
• Pt with ideational apraxia could not do the task at all even though they have the motor capacity to do it (or complete it incorrectly)

73
Q

What are the general guidelines for the evaluation of apraxia?

A

• Perform a detailed sensory and physical functioning
assessment
• Evaluate visual agnosia to rule out object recognition
problems
• Evaluate both UE/LE
• Does the pt seem bothered by the errors they have
made or can they recognize the errors

74
Q

What are the characteristics of different assessments for apraxia?

A

• Dynamic Assessment: task range from simple to complex; peel a banana, butter bread and cut in half, pour soda into a glass and drink with a straw, fit letter into an envelope and put a stamp on it, make a bowl
of cereal
• Multi-Level Action Test: 3 tasks; making a slice of toast
with butter and jam, wrapping a present, and packing
a lunchbox.

75
Q

What are the management methods for apraxia?

A

• Keep verbal commands to a minimum, put task at
subcortical(automatic) level, physically guide, visualize the task.
• Performance can improve with imitation and
repetition
• Use simple or isolated tasks
• The more familiar and typical the surroundings the
better (good argument for home health)
• Use cue (picture) cards taped to objects required for everyday function, stating what the object is and how to use it

76
Q

What is constructional apraxia?

A

Impairment in producing two or three dimensional designs, whether copying, drawing or constructing. The impairment cannot be attributed to ideomotor apraxia or a primary motor impairment. Unable to understand the relationship of the parts of a task or object to the whole.

77
Q

What causes constructional apraxia?

A

Lesion of the right cerebral hemisphere

78
Q

What are the characteristics of constructional apraxia?

A

• May be related to body scheme problems
• People who did poorly on block design tests and
object assembly tests did not acquire dressing and
grooming skills
• Management: practice simple construction of tasks
and progress to more complex, use physical guidance, backward chaining

79
Q

What is backward chaining?

A

Start with a completed project and work in reverse

80
Q

How is constructional apraxia evaluated?

A
  • Copying designs-House Test, Clock Test
  • Copy Shapes
  • Block Design
  • Peg Board Design
81
Q

How is constructional apraxia managed?

A
  • Practice simple construction of tasks and progress to more complex
  • Physical guidance
  • Backward chaining
82
Q

What is unilateral neglect?

A

Inability to perceive and integrate stimuli and perceptions from one side of the body, even though the sensory system is normal and visual system intact

83
Q

What side does unilateral neglect usually involve?

A

The left side (right parietal cortex damage)

84
Q

What does a left hemisphere/ parietal cortex damage lead to?

A

Minimal right neglect

85
Q

What does a bilateral parietal cortex lesion lead to?

A

Severe right neglect

86
Q

How do we manage neglect?

A
  • Increase stimulation to side of brain being neglected
  • Bilateral movements with weight bearing
  • Forced use
  • Compensate with visual scanning
  • Verbal cueing
  • Adapt the environment
87
Q

What is somatagnosia?

A

• Lack of awareness of body structure and failure to
recognize how parts relate to each other and the whole body
• Inability to identify any part of one’s own or another’s body

88
Q

What is the clinical presentation of somatagnosia?

A

Patient might complain of their arms being heavy, with a lot of difficulties in dressing

89
Q

How do we manage somatagnosia?

A

Provide tactile input, identification of body parts, bilateral activities. It is critical to keep instructions very simple

90
Q

What is right and left disorientation?

A

Complex neuropsychologic process that uses several

higher functions, including visuospatial processing, memory, language, and integration of sensory information

91
Q

What causes right and left disorientation?

A

Damage to parietal lobe at angular gyrus

92
Q

What is the presentation of right and left disorientation?

A

Inability to distinguish right and left

93
Q

Right and left disorientation is one component of Gerstmann’s syndrome, what are the others?

A
  • Acalculia and discalculia: Inability to solve small maths problems
  • Agraphia and disgraphia:
94
Q

What is anosognosia?

A

Inability to recognize and a refusal to accept the presence or severity of one’s own paralysis (Denial). Typically transient and may be associated with decreased mental and intellectual functioning.

95
Q

How do we manage anosognosia?

A

Very difficult, they may refuse any compensations or activities

96
Q

What are spatial relations disorders?

A
  • Inability to appropriately discriminate the body in relation to two or more objects or directions
  • Inability to detect relationship of objects or self to objects in space
97
Q

What is figure ground discrimination?

A

Inability to visually distinguish an object from the background in which it exists or locate an object that is not prominent in the visual array

98
Q

What is the clinical presentation of figure ground discrimination?

A

Cannot find things in a cluttered drawer, white shirt on white sheet, locate shirt
buttons

99
Q

How do we manage figure ground discrimination?

A

Compensation with visual scanning, amplify stimulus, simplify/adapt the environment, and repeated practice

100
Q

What is topographical disorientation?

A

Inability to perceive the concepts of up, down, in , out, in front of, etc. Difficulty identifying certain landmarks

101
Q

What is the clinical presentation of topographical disorientation?

A

Difficulty getting from one place to another

102
Q

What are the management options of topographical disorientation?

A

Practice, mark route with colored dots on a neighborhood map

103
Q

What is depth and distance perception?

A

Inability to perceive and judge direction, distance, or depth. Difficulty in identifying change in surfaces

104
Q

What are the management options for depth and distance perception?

A

Can use specialized glasses, practice, adapt environment

105
Q

What is vertical disorientation?

A

Distorted concept of what is vertical

106
Q

___ highly associated with pushing after Right hemisphere damage

A

Neglect highly associated with pushing after Right hemisphere damage

107
Q

____ highly associated with pushing after Left hemisphere

damage

A

Aphasia highly associated with pushing after Left hemisphere damage

108
Q

What is the anatomy associated with pusher syndrome?

A

Left or Right Posteriolateral thalamus

109
Q

What is the posterolateral thalamus’s role in vision?

A

Provides info about movement and cues for judging upright posture

110
Q

What is the posterolateral thalamus’s role in vestibular?

A

Informs person about head position relative to gravity and about head movement

111
Q

What is the posterolateral thalamus’s role in somatosensation?

A

Provides information about

weight bearing and relative position of body parts

112
Q

What is graviception?

A

Perception of body position, equilibrium, and direction of gravitational forces

113
Q

Pusher syndrome is the distortion of ____

A

Subjective postural

vertical

114
Q

Patients with pusher syndrome experience a mismatch between ___ and ____

A

Patients with pusher syndrome experience a mismatch between visual and postural vertical, Visual vertical based on vestibular AND visual inputs

115
Q

What are the prognosis of pusher syndrome?

A

• Does not affect functional outcomes
• Rarely still evident at 6 months
• BUT.. It does slow process significantly
• Need 3.6 weeks longer to reach same functional
outcome as those without pusher syndrome
- Goal should be to shorten rehab time
• Pushing can be fairly well compensated for by brain,
as compared with aphasia or neglect
• Those with R CVA and pusher syndrome significantly worse

116
Q

What are the test conditions of the Modified SCP (M-SCP)?

A
  • Static sitting at bedside, feet on floor
  • Static standing with a full erect posture
  • Transferring from bed to chair or wheelchair with squat pivot
  • Transferring from bed to chair or wheelchair using stand pivot
117
Q

What is the recommended measure for pusher syndrome?

A

BLS recommended for identifying contraversive
lateropulsion
• Across several functional tasks (rolling to walking)
• Only scale originally written in English
• Best reliability and responsiveness

118
Q

What are some of the current evidence of treating pusher syndrome?

A

• Treat in earth vertical positions (sitting, standing,
walking)
• Allow pushing to occur so pt experiences falling
• Make pts visually aware of tilted position
• Assist with active correction
• Reaching vertical position actively
• Trained to use visual orientation
- Are you upright?

119
Q

What are the characteristics of visual feedback treatment for pusher syndrome?

A

• Realize the disturbed perception of erect body
position
• Visually explore the surroundings and the body’s
relation to the surroundings
• Learn the movements necessary to reach a vertical
body position
• Maintain the vertical body position while performing
other activities.

120
Q

What are some activities for the pusher?

A 
• Patient needs to see that they are or are not oriented
upright
  - Use of vertical structures
• Active reaching (goal directed) to temporarily extinguish pushing
• Managing transfers
  - Forward weight shift is key
  - Which side do we go to?
• Blocking the unwanted movement
121
Q

What are the pathways of postural control?

A
  • Reticulospinal Tract
  • Vestibulospinal Tract
  • Medial Corticospinal Tract
122
Q

What are the effects of a SCI on posture?

A
  • Varied Muscle Activation
  • Delayed Responses
  • Tone and Spasticity
  • Bracing
123
Q

What’s the biggest difference in postural control in the SCI

versus able-bodied population?

A
  • Sporadic Muscle Activation

* Varied Trunk Control

124
Q

If you put a vertical line through the side of a person with normal posture, what structures does it bisect?

A

Ear, acromion, greater trochanter, L3-4, and lateral malleolus

125
Q

What are some postural control techniques?

A 
• Obtain normal alignment 
• Upright in sitting
  - Easy first position
  - Can use UE for support
• Progressing to standing if able
• You may need more than 2 hands
126
Q

What are some ways to challenge our patients once they have achieved normal posture?

A
  • Alter BOS
  • Alternating UE support
  • Alternating support surfaces
  • Altering sensory inputs
127
Q

What are some ways to challenge our patients with dynamic tasks once they have achieved normal posture?

A
  • Ball Toss
  • Reaching Activities
  • Unsupported Exercises
  • Sports and Games
128
Q

What are the biggest risk factors in the SCI population?

A
  • Autonomic Dysreflexia (T6 or above)

* Orthostatic Hypotension

129
Q

What is autonomic dysreflexia?

A

A rapid increase in BP due to some noxious stimuli. Gradually move them into a standing position and try to figure out what the problem is

130
Q

What is the greatest risk factor for exercise in SCI?

A

Therapist

131
Q

What is scoliosis secondary to?

A

Poor posture (sitting position) and abnormal muscle activation (iSCI or spasticity)

132
Q

How do we manage scoliosis?

A
  • Stretch tight muscles/ mobilize structures
  • Position into proper vertebral alignment
  • Stabilize in proper alignment and perform core stability exercises in the alignment
  • For SCI, begin in sitting and work your way up
133
Q

What does instability look like?

A 
Problems with:
• Static posture
• Dynamic mobility
• Joint stability
• Maintaining a weightbearing position
• Closed chain and open chain actitivities
134
Q

What are the basics in determining the best treatment for our patients?

A
  • Identify the limitations in function
  • Determine the specific underlying impairments
  • Prioritize the problems in an effective plan of care
135
Q

What are the questions that we have to think of during our examination of a patient with instability?

A
  • How is instability impacting function?
  • What is impact of this instability on specific movement components? On the rest of the body? Above and below instability?
  • What is the likely cause versus the consequence?
  • What other systems are involved?
  • Tests and measures?
  • Impairment measures?
136
Q

What are the questions that we have to think of during our evaluation of a patient with instability?

A

• Is instability direct cause of functional limitation?
• What are contributing factors? What are
consequences of instability?

137
Q

What are the things to think about when deciding on the intervention for instability?

A
  • First determine remediation versus compensation or both

* Address barriers: joint mobility, muscle length, strength, sensory awareness

138
Q

What are some specific interventions to address stability?

A
  • Resistance exercise
  • Stance weightbearing exercise
  • Postural exercise
  • Biofeedback/Estim
  • Taping/strapping
  • Splinting/bracing
  • Pressure garments
  • Body weight supported treadmill training
139
Q

What are the main approaches to visual field loss?

A
  • Adaptation/compensatory – more favorable option
  • Substitution
  • Restitution: actual restoration of visual loss
140
Q

What are the components of compensation as an intervention for visual field loss?

A

Scanning and search training

141
Q

What are the components of restoration as an intervention for visual field loss?

A

Light stimuli to border area of visual field loss to increase visual field

142
Q

What are the components of substitution as an intervention for visual field loss?

A

Peli prism: high strength prism placed above or below the pupil and they will reflects the images from the impaired visual field to the intact one

143
Q

What is strabismus?

A

Misalignment of the 2 eyes (both eyes don’t point to same target)

144
Q

What causes ocular abnormalities?

A

Cranial nerve problems

145
Q

What are some interventions for ocular motility deficits?

A

• Pharmacological - Botox, used as an assessment prior to surgery
• Compensatory – occlusion to eradicate 2nd image; adaptive head postures and increase in lighting
• Substitutive – Prisms to reduce diplopia
• Restorative – vergence exercises or in more chronic
cases with ocular muscle surgery

146
Q

What is reduced central vision?

A

Reduced visual acuity and contrast sensitivity

147
Q

What are the treatment options for reduced central vision?

A

Retest vision after BI, adjust glasses; low vision aids like magnifiers; modification of light and environment

148
Q

What are visual perceptual deficits?

A

Visual inattention/neglect

149
Q

What are visual perceptual deficits treatment methods?

A

• Substitutive: perceptual retraining and visual scanning
• Compensatory: scanning therapy, ADL training and
provision of aids and modifications; smooth pursuit
training

150
Q

What are the different ways to screen for visual deficits?

A 
• Visual acuity
• Contrast sensitivity
  - Pelli-Robson Contrast Sensitivity Test
• Visual Fixation/Attention Testing
  - See during visual tracking
  - Warren Fixation Test  
• Ocular Alignment/Range of Motion Testing
  - Double H
  - Convergence
• Depth Perception
  - Straw and stirrer test
• Visual Field:
   - Peripheral testing
   - Confrontation testing (test one eye at a time)
   - Tests for neglect
151
Q

What is the role of the PT in visual deficits?

A

• Detection of a problem
• Consultation and referral
- Neuro optometry
- Neuro othalmology

152
Q

What are the characteristics of the Postural Assessment Scale for Stroke (PASS)?

A

• 12 item performance based measure of postural
control
• Specifically sensitive in first 3 months
• Measures ability to maintain stable postures and
equilibrium during positional changes related to sitting, laying down and vice versa
• 10 minutes to administer
• No special equipment

153
Q

What are the characteristics of the Function in Sitting Test

(FIST)?

A
  • 14 item test
  • Developed and validated for adults with acute stroke
  • Easy to administer
  • Performed at bedside
154
Q

What are the characteristics of the Static Standing Test?

A 
• 5 standing positions, no UE support, goal is to
maintain at least 30 seconds
• Feet apart (10 cm)
• Feet together
• Stride stance (10 cm)
• Tandem stance
• Single leg stance

No cut offs

155
Q

What are the characteristics of the Trunk Impairment Scale?

A
  • Static sitting balance
  • Dynamic sitting balance
  • Coordination
  • All done from pt sitting on edge of bed or mat

Neuromuscular practice- Adutls (8 decks)

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