Week 3 Flashcards

1
Q

Integumentary System

A
  1. Epidermis
  2. Dermis
  3. Subcutaneous tissue (hypodermics)
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2
Q

Epidermis

A
  • protective barrier
  • 0.05-0.1mm thick
  • regenerate every 28 days
  • contains melanocytes and keratinocytes
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3
Q

Dermis

A
  • connective tissue, highly vascular
  • contains collagen (strength)
  • upper (thin) layer, lower (thicker) reticular layer
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4
Q

Subcutaneous tissue (hypodermis)

A
  • attaches skin to muscle and bone
  • contains fat and loose connective tissues
  • regulates temperature
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5
Q

Integumentary system function

A
  • protects underlying tissues from the environment
  • protection against bacteria, viruses, and excessive water loss
  • fat within the subcutaneous layer insulates and protects from trauma
  • interpret sensory information physiologic response
  • temperature regulation
  • secretion of sebum and sweat
  • helps synthesize vitamin D
  • melanin screens and absorbs ultraviolet light
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6
Q

Assessment data for integumentary assessment

A

hx of trauma, surgery, skin disease
health hx can explain alterations in skin colour:
- jaundice (liver disease)
- old wounds (diabetes mellitus)
- cyanosis (COPD)
- pallor (anemia)

Medications for acne, pruritus or rashes
Hx of skin biopsy and results
Exposure to sun
Allergy to pets? Are there pets in the home
Any new cleaning agents being used in the home

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7
Q

What does skin indicate about heath status

A
  • heart disease
  • colour
  • temperature
  • liver disease
  • hydration
  • potential signs of cancer (moles, growths)
  • tell about hygiene practices
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8
Q

Hair, Skin and Nails Physical exam summary

A
  1. Inspect the skin
  2. Palpate the skin
  3. Note any lesions
  4. Inspect and palpate hair and nails
  5. Teach self examination
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9
Q

Mole assessment

A
  1. Asymmetry
  2. Border
  3. Colour
  4. Diameter
  5. Evolution
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10
Q

Age related considerations for integumentary system

A
  • decreased skin turgor
  • dryness
  • benign neoplasms (barnacles as seborrhoea keratoses)
  • vascular lesions
  • increased skin fragility, skin shearing
  • fewer melanocytes (grey and white hair)
  • less volume in the dermis and subcutaneous layer, wrinkling
  • nails become brittle and prone to splitting and yellowing
  • hypothermia
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11
Q

Cultural and social considerations for integumentary

A

Recognition of unique clinical manifestations of disease
- related to variation in skin pigmentation and hair texture
- genetic advantage of dark pigmentation and lower incidence of skin cancer
- variation in sexual skin areas affected by hormones
- environmental adaptations affecting sweat glands

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12
Q

Biopsy

A
  • specimen collection Typically used with suspended malignancy
  • consent, pre-op, preparation, and post- op care
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13
Q

Microscopic tests

A
  • used to identify causative substance
    Ex. Culture and sensitivity
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14
Q

Woods lamp

A
  • uses black light
  • certain substances floresce (e.x head lice, scabies, fungal infections)
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15
Q

First line of defence

A

Epithelial cells
- skin, GI, GU, respiratory tract

Pathogens sloughed off with dead skin cells

Coughed, sneezed, expelled (vomit)

Flushed out by urine

Removed via stool (diarrhea)

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16
Q

Second line of defence

A

Inflammation (occurs rapidly)

A response to
- infection
- trauma
- immune response to allergens

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17
Q

Wound healing phases

A
  1. Inflammatory phase
  2. Proliferative phase
  3. Maturation/ remodeling
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18
Q

Inflammatory phase

A

-Hemostasis
- acute inflammation

Mast cells
- activate inflammatory response
- located in the skin and GI tract

Histamines
- vasodilation
- smooth muscle constriction
- antihistamines are important here to decrease inflammatory response

Phagocytic system
- eliminate pathogens and foreign debris
- living cells that recognize, attach, engulf and destroy

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19
Q

Local manifestations of inflammation

A

Redness
- hyperaemia from vasodilation

Heat
-increased metabolism at the inflammatory site

Pain
- nerve stimulation by chemicals, pressure from fluid exudate

Swelling
- fluid shift to interstitial space; fluid exudate accumulation

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20
Q

Patch testing

A

Evaluating response to different allergens

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21
Q

Exudate (fluid)

A

Typically serous (watery) in mild inflammation
- ex. Abrasions, rashes, blisters

Typically, thicker in severe inflammation
- ex. Pneumonia
- can contain pus (purulent)
-hemorrhagic exudate can be excreted from tissues with serious injury (burn)

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22
Q

Primary intention

A

Wound is closed, with surgical intervention. Edges are brought together. Best choice for clean, fresh wounds in sufficiently vascularized areas

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23
Q

Second intention

A
  • Would is left open and allowed to heal spontaneously.
  • Good for contaminated/ infected wounds.
  • Increased scarring
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24
Q

Tertiary intention

A
  • delayed primary closure
  • good for wounds which are contaminated/ infected initially
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25
Q

Complications during wound healing

A
  • adhesions
  • contractures
  • dehiscence
  • evisceration
  • fistula formation
  • infection
  • hemorrhage
  • excess granulation
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26
Q

Nursing management of inflammation and healing

A
  1. Prevention
  2. Nutrition
  3. Early recognition
  4. Acute intervention
    - fever management
    - consider hot and cold application
    - compression and immobilization
    - wound management
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27
Q

Causes of pressure injuries

A
  • pressure
  • friction
  • shearing force
  • moisture
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28
Q

Risk Factors for Pressure injuries

A
  • advanced age
  • nutrition
  • comorbidities
  • duration
  • incontinence
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29
Q

Nursing management of pressure injuries

A
  • impaired skin integrity
  • repositioning
    -devices
  • identify stage, type, extent
  • document and assessment
  • wound care
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30
Q

Nursing assessment and documentation

A
  • on admission
  • every shift
  • ongoing basis
  • Braden scale
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31
Q

Wound management

A

Purpose
A) cleaning and debriding the wound to remove debris and dead tissue from the wound bed
B) controlling inflammation and treating infection to prepare the wound for healing
C) providing moisture balance for healable wounds and moisture reduction for non-healable and maintenance wounds

  • negative pressure wound therapy
  • hyperbaric oxygen therapy
  • electrical stimulation
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32
Q

Vitamin A (retinol)

A

Derived from animal products and green and yellow/orange fruits/veggies

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33
Q

B- complex vitamins

A

Deficient in malabsorption, poor nutrition, ETOH abuse and prolonged fever/dehydration

34
Q

Vitamin C (ascorbic acid)

A

Deficiency resulting from poor nutrition (scurvy)

35
Q

Vitamin D

A

Regulates the absorption and utilization of calcium and phosphorus

36
Q

Right drug for the right bug

A

Selection of anti microbial is based upon assessment, pharmacological and clinical judgement, and microbiological identification

37
Q

Antibiotic classifications

A
  • classified into broad categories based upon chemical structure and MOA
  • common categories are sulfanomides, penicillins, cephalosporins, carbapenems, macrolides, quinolones, aminoglycosides and tetracyclines
38
Q

Empiric therapy

A

Treatment of an infection before specific culture information has been reported or obtained

39
Q

Targeted or definitive therapy

A

Antibiotic therapy tailored to treat organisms identified with cultures

40
Q

Prophylactic therapy

A

Treatment with antibiotics to prevent an infection, as in intro abdominal surgery or after trauma

41
Q

Antibiotics

A
  • are medications used to treat bacterial infections
  • ideally before beginning antibiotic therapy of suspected areas of infection should be cultured to identify the causative organism and potential antibiotic susceptibilities. However, risk vs benefit is always considered
42
Q

Classes of antibiotics

A
  • sulfonamides
  • penicillins
  • cephalosporins
    -Macrolides
  • quinolones
  • aminoglycosides
  • tetracyclines
43
Q

Penicillins G is..

A

Injectable

44
Q

Penicillins V is

45
Q

Penicillins G and Penicillin V

A
  • inhibits cell was synthesis
  • binds to the active site of penicillin-binding proteins (PBP) that halts peptidoglycan synthesis
  • as a result, bacterial cells die from cell lysis
  • bactericidal
46
Q

Pen G and Pen V prevention and treatment of infections caused by susceptible bacteria such as

A
  • many gram positive bacteria, included streptococcus spp, enterococcus ssp., and staphylococcus spp
  • litttle gram negative coverage (greater coverage with extended spectrum penicillins)
47
Q

B- lactation antibiotics

A
  • penicillins
  • cephalosporins
  • carbapenems
  • monobactams
48
Q

Impetigo

A
  • linked to seasons, hygiene
  • contagious
  • commonly perioral lesions, honey “crust”, prurtis, tenderness
  • strep or staph
  • local or systemic antibiotics (depending on the severity)
49
Q

Folliculitis

A
  • related to shaving, heat, oils, friction, moisture, DM
  • commonly staph
  • small pustules, tenderness
  • local or systemic antibiotics (depending on the severity)
  • warm compress, hygiene
50
Q

Cellulitis

A
  • subcutaneous tissue involvement
  • commonly Steph or strep
  • erythema, tenderness, Edelman, fever, malaise (commonly in lower legs)
  • rest elevation, mosit heat
  • systemic antibiotics
51
Q

Scabies

A

Caused by sarcopetes scabies
Transmitted by direct contact

Clinical features
- severe itching, especially at night
- presence of burrowing tracks
- crusting

Treatment
- topical 5% permethrin
- cohabitants and sexual partners
- bleach for clothing and hard surfaces
- consider secondary infection
- treatment of puritus

52
Q

Bed Bugs

A

Treatment considerations:
Lesions: No treatment needed
Pruritis: antihistamines, corticosteroids

53
Q

Melanocytes

A

Contain melanin, giving pigment colour to skin and hair and protects the body from damaging ultraviolet light

More melanin = darker skin colour

54
Q

Keratinocytes

A

Produce keratinocytes fibrous protein, vital to the protective barrier function of skin

55
Q

Wheal

A

Skin leasion
- firm
- edematous (insect bite, urticaria)

56
Q

Plaque

A

Skin lesion
- elevated, superficial, solid
- ex. Psoriasis

57
Q

Macule

A

Skin lesion
- flat, change in skin colour
- ex. Freckle, flat mole

58
Q

Pustule

A

Skin lesion
- elevated, superficial, furulent fluid
- ex. Acne

59
Q

Papule

A

Skin lesion
- elevated, solid
- ex. Wart, elevated moles

60
Q

Vesicle

A

Skin lesion
- superficial collection of fluid
- ex. Varicella (chicken pox), herpes zoster (shingles) and second degree burns

61
Q

Sublethal injury

A

Alters function without causing cell death

62
Q

Lethal injury

A

An irreversible process that causes cell death

63
Q

Causes of cell injury

A
  • heat
  • cold
  • radiation
  • electro thermal
  • mechanical trauma
  • chemical trauma
  • virus
  • bacteria
  • fungus
  • neoplastic growth
64
Q

Common topical antibacterial

A
  • polysporin, neosporin, bacitracin (most commonly treats skin infections from staph and strep)
  • mupirocin (bactroban) *impetigo
  • silver sulfadiazine (flamazine) *burns
65
Q

Common topical antifungals

A
  • candidiasis, tinea ringworm: clotrimazole
  • clotrimazole
  • nystatin
  • terbinafine hydroclordide
66
Q

Less common antiviral ointments

A

Acyclovir: herpes, HPV

67
Q

Common topical anesthetic *pain

A
  • lidocaine (with or without prilocaine)
68
Q

Common topical anti-inflammatories or antipruritic

A

Corticosteroids: used for inflammation or pruritus, the “sones”
Variable % of concentration, variable potency
Ex. Hydrocortisone, betamethasone, ect

69
Q

Common oral antihistamines

A

May treat urticaria, angiogram, pruritus, contact dermatitis and allergic cutaneous reactions
- different generations of antihistamines, first generation often causes drowsiness as a side effect

Diphenhydramine (Benadryl)
Fexofenadine (Allegra)
Cetrizine (reactine)
Lortadine (Claritin)

70
Q

Structures of the eye

A

External structures
- eyebrows, eyelids, eyelashes, lacrimal system, conjunctiva, cornea, sclera, extraocular muscles

Internal structure
- iris, lens, ciliary body, choroid, retina

The eyeball or globe is composed of three layers
1. Outer layer is sclera and transparent cornea
2. Uveal tract (iris, choroid and collard body)
3. Innermost layer is the retina

71
Q

Refractive media

A

For the light to reach the retina, it must pass through several structures
Cornea-> aqueous humour-> lens-> vitreous humour

All these structures must remain clear for light to reach the retina and stimiulate the photoreceptors cells. The cornea, which is normally transparent, is the first structure through which light passes, it is responsible for most of the light refraction nexessary for clear vision

72
Q

Myopia

A

Nearsigntedness

73
Q

Hyperopia

A

Farsightedness

74
Q

Astigmatism

A

Caused by corneal unevenness resulting in vision distortion

75
Q

Presbyopia

A

Type of hyperopia due to aging, usually beginning around age 40

76
Q

Diagnostics: Snellen chart

A
  • determines visual acuity (distance vision)
  • normal vision is 20/20
77
Q

Diagnostics: ophthalmoscopy

A
  • using light and magnification to examine the back of your eye (including the retina and optic nerve)
78
Q

Diagnostics: Tonometry

A
  • used to measure the pressure inside your eyes (IOP)
  • screens for glaucoma and monitors treatment for glaucoma
  • non-contact tonometry= eye puff test
79
Q

Nursing interventions (non-pharm) *eyes

A
  • knowing the pts vision needs (reading glasses, etc)
  • mellow lighting
  • warm/ cool eye compress
  • gentle cleaning eye lubricant
  • sunglasses
  • reduce exposure to irritants
80
Q

Eye health promotion

A
  • regular visual exams (every 3-5 years until the age of 40) (2-4 years until 65) (2 yearly after 65)
    (Yearly eye exams if family hx glaucoma or if from African American descent)
  • a balanced diet and fluid intakes
  • reduce risky lighting (blue screen exposure)
  • sunglasses
  • reduce exposure to irritants with protective eyewear