Week 2 ECG Flashcards

1
Q

What is sinus tachycardia? Describe the ECG

A

Regular fast HR
Same distance between each part of the wave. >100bpm.

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2
Q

What is sinus bradycardia? Describe the ECG

A

Regular slow HR <60bpm.
Same distance between each wave just not as many per second.

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3
Q

Describe sinus arrhythmia? What can cause it?

A

2 fast, one slow heartbeat = regular, not normal.
Can be due to breathing, breathing in increases HR, breathing out decreases HR.
Regularly irregular pattern.

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4
Q

What are premature ventricular contractions (PVCs)?

A

Extra or missed beats caused by a group of cells in the ventricles that cause an early beat.
They don’t normally require investigation. May cause palpitations and a sense of the heart “skipping a beat”.

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5
Q

What is a unifocal PVC?

A

When the extra beats both looks the same.
The heart is generating 2 extra beats from the same place - not to worry about.
Can be caused by too much caffeine.

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6
Q

what are Multifocal PVCs?

A

Multiple extra beats that look different.
More concerning.
Coming from different places to each other.

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7
Q

What is bigeminy?

A

Every other beat is an extra one.

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8
Q

What is trigeminy?

A

Every third beat is an extra one.

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9
Q

What does more than 3 PVCs in a run indicate?
What does the ECG look like?

A

Ventricular tachycardia (non-sustained) = VT.
Normal ECG beats followed by larger QRS complex - ventricular contractions.

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10
Q

What is the issue with sustained VT?

A

It is ventricular fibrillation (VF).
Does not allow the ventricles to fill so can’t maintain cardiac output.

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11
Q

What is atrial fibrillation? How can you identify it?

A

Rapid and chaotic depolarisation within the atria.
No P wave on the ECG.
Increases risk of thrombosis/embolism (blood clot) - causes 20% of all strokes.

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12
Q

How to identify wolf parkinson white syndrome? What is it?

A

Very short PR interval (0.08s) - speeds everything up.
The electrical impulse bypasses the AV node.
Pre-excitation syndrome.

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13
Q

How to identify atrioventricular blocks
How are they sometimes treated?

A

Longer PR intervals - slows everything down.
if 2nd or 3rd degree block may be fitted with a pacemaker.

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14
Q

Right bundle branch block
How to identify?
Issues?
What happens

A

M shape in the QRS of V1 and W in V6 - MARROW
Fairly common in athletes, doesn’t really cause any major issues or symptoms.
Signal affected goes to the right ventricles from the bundle of His

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15
Q

Left bundle branch block
How to identify?
Issues?
What happens?

A

Tall R waves in V5-V6, deep S waves in V1-3.
W in V1, M in V6 QRS - WILLIAM
Do not exercise if you have this, if blockage is in left ventricle oxygen won’t be getting to the body - danger for exercise.
RV depolarises normally and first via RBB, delayed activation of LV as must be depolarised by the RBB with depolarisation from R to L.

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16
Q

Left ventricular hypertrophy
When to expect?
Issues?
What does the ECG look like?

A

Athletes, thin chest walls (not much space between heart and electrode makes voltage seem higher than it is). Uncontrolled, long lasting hypertension and aortic stenosis (narrowing of heart valve) - left ventricle has to thicken to pump blood as at higher pressure.
Often considered normal in athletes.
Thickened walls leads to prolonged depolarisation (R) and delayed repolarisation (ST and T in lateral leads).

17
Q

How to identify myocardial ischemia?
What suggests it is a blockage of the left main coronary artery?

A

ST depression >1mm. Leads II, III and V4
T wave inversion (flattening) can be seen but not always.

Widespread ST depression across multiple leads.

18
Q

How to identify myocardial infarction?

A

ST elevation (STEMI = ST elevated MI).

19
Q

When is medical clearance needed?

A

Known disease but no symptoms.
Any signs or symptoms suggestive of disease.

If cleared they can progress from light to moderate exercise.

20
Q

Who should be supervising different patients.

A

Low risk - no-one, safe to exercise alone.
Medium risk - a HCP with exercise testing experience
High risk - a HCP with exercise testing experience, doctor must be immediately available (ILS trained for administration of drugs if CV event).

21
Q

What are some contraindications to exercise?

A

Unstable anything e.g. angina, diabetes, arrhythmia, tachycardia.
Resting SBP> 180/200
Resting DBP > 100/110
Symptomatic Hypotension
Acute infections
Heart disease

22
Q

What is phase 0?

A

Rapid depolarisation
Na+ channels open for rapid influx of Na.

23
Q

Phase 1

A

Rapid depolarisation
Na channels close/become inactivated - no Na influx.
K+ channels open for brief K+ influx then close rapidly.

24
Q

Phase 2

A

Plateau (longest phase)
Influx of Ca2+ - released from sarcoplasmic reticulum to initiate contraction
K+ efflux (causes plateau)

25
Q

Phase 3

A

Rapid repolarisation
Ca2+ channels closed (no more contraction)
K+ channels remain open until restores the membrane potential to resting value

26
Q

Phase 4

A

Resting potential
Stable at -90mV in normal working myocardial cells
Na closed
Ca closed
K+ stay open until the next action potential arrives and Phase 0 starts again

27
Q

Suggest some abnormal ECG findings that would require further investigation

A

T wave inversion
ST segment depression
Pathologic Q waves
Complete LBBB
QRS>140ms duration
Ventricular pre-excitation
Prolonged QT interval
Sinus bradycardia <30bpm.
Atrial tachycardia
Ventricular arrhythmias
Atrial enlargements
Complete RBBB
Axis deviations

28
Q

What are some causes of sudden cardiac death that can be identified using ECG?

A

Hypertrophic cardiomyopathy
Wolf-Parkinson-White syndrome
Right ventricular outflow tachycardia
Marfan syndrom
Congenital long QT syndrome
Electrolyte disturbances (hypokalemia or hyperkalemia)

29
Q

Normal ECG findings that do not require any further investigation

A

Increased QRS voltage for LVH or RVH
Incomplete RBBB
T wave inversion < 16 years
Sinus bradycardia or arrhythmias
ST elevation and T wave inversion in black athletes.