Week 2 - Acute Muscle Cramps & Doms Flashcards

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1
Q

What is EAMC

A

Exercise associated muscle cramps

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2
Q

What are the symptoms of EAMC?

A
  • painful, spasmodic, involuntary contractions
  • 1-3 minute duration
  • late in game or post exercise
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3
Q

Common sites for EAMC?

A
  • calf (gastrocnemius)
  • foot ( intrinsic muscles)
  • thigh (quads and Hamstrings)
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4
Q

What are the causes for EAMC?

A
  • no underlying metabolic, neurological, or endocrine disease
  • no benefit of “specific” pre-exercise fluid & electrolyte intake
  • no significant association between history of stretching and EAMC
  • some individuals more predisposed
  • doesn’t seem to be dehydration or electrolyte imbalance
  • may be altered neuromuscular control (alpha motor neurons have increased excitation and decreased inhibition from golgi tendon organs)
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5
Q

What is the best EAMC management?

A
  • passive stretching (may increase tension in golgi tendon organ leads is reflex inhibition to increase afferent reflex inhibition to alpha motor neuron
  • symptomatic relief within 10-20 seconds - maintain stretch until fasciculation ceases
  • pickle juice - due to activation of oralphyrangeal reflex - reduces alpha motor neuron activity
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6
Q

What are DOMS?

A
  • delayed onset muscle soreness
  • soreness following vigorous, unaccustomed exercises
  • 24-48 hours after exercise, particularly eccentric actions
  • downhill running & skiing common cause in untrained individuals
  • complex process - microtrauma of muscle cells and connective tissue followed by local inflammatory process within extra cellular space which sensitises nerve endings
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7
Q

Signs and symptoms of DOMS?

A
  • pain
  • local muscle swelling/muscle stiffness
  • muscle strength deficits
  • elevated plasma creatine kinase (marker of muscle breakdown)
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8
Q

How to deal with DOMS?

A
  • during symptomatic period (up to 10 days) modify exercise regime
  • massage, cryotherapy, stretching, active recovery may provide relief
  • curcumin (turmeric) may reduce inflammation and pain by modulating inflammation and cytokine flux by influencing COX-2 signalling
  • protective effects conferred by repeated (preparatory/sub maximal) eccentric bouts
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