Week 2 Flashcards

1
Q

brief overview of central vestibular pathology symptoms talked about in case history

A

milder in nature with gradual onset

*often associated with headache, visual changes or neurological deficits

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2
Q

brief overview of peripheral vestibular pathology symptoms talked about in case history

A

tends to be sudden onset, decreases with visual fixation, often accompanied by otologic symptoms (aural fullness, fluctuation on hearing, tinnitus)
*more likely to report nausea and vomiting

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3
Q

nystagmus with peripheral lesion

A

horizontal and torsional
inhibited by fixation
doesnt change with gaze

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4
Q

nystagmus with central lesion

A

purely vertical or horizontal
nor inhibited by fixation
direction changing with gaze

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5
Q

otologic symptoms with peripheral lesion?

A

hearing loss and tinnitus common

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6
Q

otologic symptoms with central lesion?

A

uncommon

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7
Q

latency after provocation with peripheral lesion

A

longer (>15 seconds)

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8
Q

latency after provocation with central lesion

A

short

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9
Q

fatigibility of peripheral lesion

A

yes

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10
Q

fatigability of central lesion

A

no

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11
Q

duration with peripheral lesion

A

variable

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12
Q

duration with central lesion

A

long

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13
Q

neurologic symptoms with peripheral lesion

A

no

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14
Q

neurologic symptoms with central lesion

A

yes

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15
Q

loss of consciousness with peripheral lesion

A

no

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16
Q

loss of consciousness with central lesion

A

possible

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17
Q

vertigo

A

an illusionary sense of movement or rotation

*can be pt spinning or the world spinning around them

18
Q

presyncope

A

lightheadedness just prior to falling

  • often pts report feeling like they are going to faint, but don’t actually
  • *if they faint, its not a disorder of the ear
  • need to look at heart, blood pressure, carotid artery blockage and do so quickly
19
Q

orthostatic hypotension

A
  • common cause of presyncope
  • this is when someone either stands up too quickly or sits up too quickly from the supine position
  • this happens in the young and old populations but see an increase with age (inadequate vascular supply, reduced reflex compensation)
20
Q

how to diagnose orthostatic hypotension

A

blood pressure is taken laying, sitting, and standing

*if a sig changes (>20 systolic pressure) is notes then it is orthostatic hypotension

21
Q

treating orthostatic hypotension

A

medication management: what medications could be causing the issue?
*counseling: pts should be urged to move slowly to allow the change in blood pressure to equalize

22
Q

vasovagal attacks

A
  • reduction of cardiac output by decreasing blood pressure and pulse rate
  • maybe w/ sight of blood, or a frightening event, heat exposure, bowel movement
  • dizziness, pallor, sweating, tingling hands, nausea
  • pts may pass out
  • EKG and medical eval often negative
  • tilt table is important
23
Q

treating vasovagal attacks

A

reevaluation of meds
education
counseling to reduce frusteration and anxiety

24
Q

heart disorders (leading to dizziness)

A
  • arrythmias
  • heart disease acquired or congenital
  • coronary artery disease
25
psychological disequilibrium
* anxiety induced, associated with panic attacks * usually lasting 10-15 minutes * accompanied by palpitations, parethesias, sweating, shortness or breath and dizziness * hyperventilation
26
ocular disequilibrium
* visual abnormalities send incorrect messages to the brain about spacial positioning * very common after changes in corrective lenses, or eye surgery * pts will have to adjust to the change by returning to life and allowing compensation of the VOR * motion sickness falls in this category
27
motion sickness
* occurs when there is a mismatch between vestib and visual systems * this is also why reading a book in a car can make some people really sick * Dramamine reduces the input of the vestib system and reduces conflicting message
28
pathologies that have short length dizziness
* BPPV * perilymph fistula * SSCD * vascular insufficiency * Chiari malformation * Iatrogenic
29
pathologies that have a medium length dizziness
* meniere's disease * migraine * metabolic * syphilis * panic attacks * TIA * Iatrogenic
30
pathologies that have a long length dizziness
* labyrinthitis * temporal bone trauma * stroke * multiple sclerosis * Iatrogenic (caused by medical treatment) * autoimmune inner ear disease
31
static evaluation (bedside eval)
* assess for spontaneous nystagmus * have pt look straight, right, left, up, and down with and without fixation (Fenzels lenses) look at eyes fro spontaneous or gaze nystagmus * results: - --horizontal nystagmus that is conjugate and without fixation= peripheral - --vertical or torsional (rotary) and may be stronger in one eye than the other and with fixation= central
32
headshake test (bedside exam)
* a test of the VOR. have pt shake head back and forth in horizontal plane for 20-30 seconds. have stop abruptly and open eyes with frenzel's lenses or infra red goggles * Results: - --horizontal nystagmus beats away from the lesion or beats towards the healthy ear (usually) - --horizontal nystagmus beats toward the lesion after a delay or after the initial nystagmus sops
33
head thrust (bedside exam)
* a test of the VOR. the pt is asked to gaze steadily at a target in the room. move the pts head from side to side gently and then move briskly from one side to the other and hold while observing eyes position * results: - --eyes remain fixed on the target=normal - --eyes make a compensatory movement after the head is stopped to fixate on the target=abnormal * this test can indicate if the output of one or both labyrinths is depressed
34
dynamic visual acuity (bedside exam)
* a test of the VOR. have patient read a Snellen eye chart and establish visual acuity or static acuity. then rotate the pts head at 1-2 Hz horizontally and have them read the chart again * results: - --loss of one or two lines=normal - --loss of three lines indicated a VOR deficit
35
saccades (bedside exam)
* have pt rapidly shift gaze from nose to finger or wiggle fingers on each side at varying distances * results: - --grossly abnormal responses can indicate cerebellar dysfunction - --poor eyesight, unable to focus the fovea onto the target
36
smooth pursuit (tracking) (beside exam)
* have pt follow your finger as it moves slowly through their visual field * results: - --grossly abnormal responses can indicate cerebellar dysfunction - --again, interpret with caution
37
Rhomberg (bedside exam)
* have the pt stand with feet together, eyes open and arms to the side or across the chest. then have pt close their eyes * sharpened Rhomberg= have pt stand heel to toe with 1 foot in front and close eyes with arms at side or across chest, then close eyes * results: - -excessive sway (eyes closed)-peripheral lesion (usually to the side they lean) - -excessive sway (eyes pen)- proprioceptive weakness
38
gait/tandem gait (beside exam)
* have pt walk heal-to-toe in a straight line * tandem gait test- have patient walk heal-to-toe in a tight circle or figure eight * results: - --pt staggers or consistently leans to one side or the other=likely central
39
fakuda stepping (bedside exam)
* pt is asked to step (march) in place with eyes closed for 100 steps * results: - --rotation of the pt= peripheral lesion - --less than a 45 degree rotation= normal
40
fistula test (beside exam)
* manual setting of temp or pneumatic otoscopy | * nystagmus and dizziness is indicative of a fistula
41
dysduadichokinesis (bedside exam)
* rapid alternating movements such as tapping foot to floor of touching thumb to each finger rapidly * poor coordination of movements-cerebellar dysfunction
42
hyperventilating syndrome tsting
*have pt over breathe for 90 seconds and see if they can reproduce their symptoms