Week 2 Flashcards

Study Guide

1
Q

S & Symptoms of Respiratory Distress Syndrome

A

-Expiratory Grunting
-Hypothermia
-Hypotonic Muscle Tone
-Central Cyanosis
-Lethargic
-Hypoglycemia
-Tachypnea

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2
Q

S & S of Neonatal Sepsis

A

-Expiratory Grunting
-Hypothermia
-Hypotonic Muscle Tone
-Lethargy
Tachypnea

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3
Q

S & S of Neonatal Abstinence Syndrome

A

-Hypoglycemia, although strong sucking, will not feed
-Tachypnea
-Tremors

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4
Q

What is a preemie at risk for if respiratory distress syndrome id left untreated?

A

-Increased Atelectasis due to lack of surfactant

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5
Q

The treatment plan for a respiratory exacerbation and failure to thrive for Cystic Fibrosis

A

*airway clearance therapy (chest physiotherapy and postural drainage)
*Nebulized bronchodilators and mucolytics
*High-Calorie diet with pancreatic enzymes (before the meal)
*IV antibiotic therapy
*Regular Pulmonary function tests
*Psychosocial support for the family

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6
Q

Should you give high levels of oxygen to a client with cystic fibrosis?

A

No, due their chronic respiratory condition, their body relies on CO2 levels not o2 levels, like someone with COPD

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7
Q

Place clearance techniques in order for optimal mucous clearance:
1. Administer Bronchodilator (Albuterol) via nebulizer
2. Perform chest physiotherapy and postural drainage
3. Encourage coughing and suction if needed
4. Administer mucolytic agent (Dornase Alfa)

A

1) Administer Bronchodilator (Albuterol) via nebulizer
2) Administer mucolytic agent (Dornase Alfa)
3) Encourage coughing and suction if needed
4) Perform chest physiotherapy and postural drainage

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8
Q

What is cluster care?

A

A nursing technique that involves performing multiple tasks for a patient at the same time, instead of doing them individually-use in order to provide rest/ recovery

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9
Q

Biological Growth and Physical Development-external proportions

A

Infant-growth from trunk
head is the largest part, proportions will alternate with growth

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10
Q

Growth and Development-Skeletal and maturation, most accurate measurement?

A

*epiphyseal plate, site of longitudinal growth of the long bones

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11
Q

Metabolism-Physiological Changes

A

With each degree of fever increase, basal metabolism increases 10%, corresponding increase in fluid requirement

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12
Q

Spends more time in sleep…

A

infants

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13
Q

The most important influence on growth

A

Nutrition

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14
Q

Universal medium of play

A

*children learn what no one else can teach them

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15
Q

What are genes?

A

Segments of DNA that contain genetic information to control certain physiological functions/ characteristics

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16
Q

Congenital anomalies/ birth defects

A

2-4%, classified as deformity, disruption, dysplasia, or malformation

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17
Q

Congenital anomalies (structure) or birth defects are seen…

A

immediately after birth

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18
Q

Genetic Disorders can be caused by…

A

chromosomal abnormalities

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19
Q

Examples of genetic disorders…

A

*sickle cell anemia
*down syndrome
*Turner syndrome
*Muscular dystrophy

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20
Q

Genetic disorders are not…

A

always seen after birth, but growing/ environmental factors

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21
Q

Role of the Nurse in genetics

A

Perform and full assessment (no history), I.D. then refer through testing, educate parents on discovery and treatment

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22
Q

Health problems-Newborns, how does head trauma occur

A

Falls, birth, shaking…ICP can cause brain bleeding
…paralysis (Cranial Nerve 7)-most common

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23
Q

Newborn Head trauma causes intraventricular bleeding which increases ICP effecting…

A

Neural development
-watch for S &S of hemorrhage and neural system decline

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24
Q

The most common newborn fracture is clavicle caused by….

A

birth positioning, LGA…immobilize the arm with good alignment w/ the body, tell parents to be gentle

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25
Q

Structural Defects-Cleft lip

A

-formed during embryonic
-development from incomplete fusion of the oral cavity
-Surgery should be performed first 2-3 months of life

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26
Q

Structural defect-Cleft Palate

A

Forms during embryonic development, incomplete fusion of the palate
-surgical repair at 6-12 months

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27
Q

Newborns/ infants with a cleft palate are at risk for.

A

Aspiration, infection, conduction hearing loss and malnutrition

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28
Q

Pre-Op consideration for CL: Educate about proper feeding and care, assess the ability to feed

A

*encourage breast feeding
*wide based nipples- unidirectional (firm to palate, supine)
*squeeze cheeks to decrease gap
*more burping
*upright position, cradle head
*syringe feedings
*consider aspiration

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29
Q

Educate about proper feeding and care, assess the ability to feed, CP-Pre-Op

A

-Upright position and cradle head with feeds
-Special bottle one way valve and special nipple, vacuum seal
-Frequent burps
-Syringe feeds

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30
Q

Post-op CL and CP repair

A

*pain management
*elbow restraints (release hourly)
*no pacifiers (hard)
*Cl repair: supine/ side-lying
*CP repair: prone (drainage), NPO then slow progression>soft diet)

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31
Q

Post-op CP & CL Education/ complications

A

*diet/ feeding techniques
*complications:
-ear infections (mid ear(
-difficulty eating
-altered structure/ recurrent infections
-speech/ language impairment (CP)
-dental-abnormal eruptions

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32
Q

Before Feeding: CP/ CL

A

check gag reflex, GI sounds (increase w/ tolerance), swallow reflex

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33
Q

High-risk Newborn (preemie-1st 28-days)…why?

A

Hyperbilirubinemia
Respiratory Destress
neonatal seizure
newborn sepsis
NEC
Drug-exposure
Phenylketonuria

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34
Q

Hyperbilirubinemia-characterized by:

A

*jaundice, yellow skin/ sclera/ nails

35
Q

Hyperbilirubinemia-Causes
*ask if previous hx. w/ other children

A

-Physiological Jaundice-inability to excrete bili initially
-breastfeeding-hormone increase bili, not enough milk (dehydration), C-section
-Hemolysis (ABO incompatible/ hemolytic disease)
-inadequate liver function
-cephalic hematoma
-umbilical cord (broken RBC)

36
Q

Hyperbilirubinemia- Diagnosis

A

Blood test, heel-stick

37
Q

bilirubin encephalopathy

A

-deposits of unconjugated bilirubin into brain cells

38
Q

Kernicterus

A

yellow staining of brain cell from bilirubin ecepalopathy

39
Q

Bilirubinemia-Treatment

A

-prevent increase in bili, minimize treatment S/E
-phototherapy
-exchange transfusion
-hydration/ breast milk-remove for only 15 min. pump after for bottle

40
Q

Phototherapy-insoluble bili to soluble

A

*IV fluids-fluid loss-sweat
*moves to stool (yellow)-effective
-recheck labs for decrease

41
Q

Exchange transfusion-aggressive

A

*through central line-umbilical cord, * (3) nurse documents VS-every 5 minutes @ bedside
*after-phototherapy

42
Q

Respiratory Distress Syndrome, high risk-preemies

A

*developmental delay in lung maturation, not enough surfactant, alveoli collapse expiration (atelectasis), unequal alveoli inflation (inhalation)
*unable to inflate lungs

43
Q

RDS- clinical manifestation

A

-tachypnea
-dyspnea
-intercostal/ substernal retractions
-fine inspiratory crackles
-nasal flaring
-pallor
-cyanosis
-grunting

44
Q

RDS-management (preemie anticipation-dexamethasone)

A

*Intubation
*Provide liquid surfactant (inhalation)
*half-dose one side, middle, other side rest of dose (push ambu after dosing)
-prevent hypotension, acid-base balance

45
Q

RDS- dessating before surfactant

A

1) suction first-cannot 1-2 hours after med admin
2) give oxygen
3)then fluids (electrolyte imbalances)
-order ABGs/ BMP
-keep warm

46
Q

Neonatal Sepsis- causes

A

-bacterial infection in blood stream
-across placenta (bloodstream, birth)
-ingestion/ aspiration infected amniotic

47
Q

PKU expected findings

A

-FTT
-vomiting
-irritable
-musty urine odor
-microcephaly
-heart defects

48
Q

Neonatal Sepsis-early (< 3 days after birth), late (1-3 weeks), diagnosis (clinical S/S), manifestations?

A

-hypothermia
-drainage
-weak suck/ decreased intake
-V/ Diarrhea
-poor weight gain
-large residual (gavage)
-abd. distension
-respiratory distress
-hypoactive/ tonic
-pale

49
Q

Prevent Neonatal Sepsis

A

Screen mom-GBS
good hygiene
isolation
breastfeeding

50
Q

Manage/ Care Neonatal Sepsis

A

-supportive (O2), fluids, transfusion, electrolyte acid-base balance
-Pan-Culture (blood, stool, urine), 3 days, 1 week
-give broad spectrum, them specified antibiotic
-Perform BMP/ ABG

51
Q

Neonatal Sepsis- closely assess/ monitor…complications

A

-meningitis and septic shock

52
Q

NEC-life-threatening, preemies high-risk

A

*poor blood supply causes infection

53
Q

Why NEC?

A

Intestinal ischemia and inflammation
Bacterial/viral infection
Enteral feeding
Immature GI mucosa
Medical NEC**

54
Q

What to do…NEC?

A

Serial X-ray and abd girths (4-6 hr)
Monitor for blood emesis and stools
Watch labs, cultures for changes and growth
NPO for bowel rest (5-7 days)
begin antibiotics, NGT suctioning (low-intermittent)
CVC and TPN/ IL nutrtion

55
Q

Drug Exposed Infant (Alteration in breathing to death), how to test?

A

Newborn urine, hair, or meconium sampling
Before feeding and at least 2 hours after birth, then every time before feeding NAS score b/c putting on medication can be discharged after 24-hours of low NAS score and mother is provided stability

56
Q

PKU-test

A

heel-stick to assess at least 24-hours after birth b/c they have to eat first retest in 7-10 days to ensure no false negatives

57
Q

PKU?

A

inborn error of metabolism inherited as an autosomal recessive trait (the PAH gene is located on chromosome 12q24), is caused by a deficiency or absence of the enzyme needed to metabolize the essential amino acid phenylalanine

58
Q

PKU confirmation

A

Places infant at risk for convulsion, diagnose 24 hours after fed, if the test is confirmed for a second time provide a low protein formula

59
Q

SIDS

A

Third leading cause of infant mortality under 1 year of age
Education aboutBack to SleepCampaign
Pacifier use linked to decreasedincidences

60
Q

Poisoning-Emergency Treatment

A

First Priority: Stabilize the patient, then determine what was consumed.
Treatment:
Remove from exposure.
Chelation therapy (lead poisoning antidote) may be needed.
Screening:
Screen for lead poisoning at 1 year old.
Health Effects:
Brain/nervous system damage.
Learning/behavioral issues.
Slowed growth, hearing/speech problems, lower IQ.
Decreased attention span and school performance.
Gasoline Poisoning: Be aware of risks; follow similar treatment guidelines.
Always call poison control first for advice.

61
Q

Testicular Torsion

A

Testicular Torsion, testes rotate on spermatic cord, cutting off blood supply, must fix within 6 hours to save, can be caused by trauma or birth defect

62
Q

Sexually transmitted infections

A

*best is prevention, educate!!!
*any case of STI must report to the health department

63
Q

Airway Diameter

A

If infection of inflammation they can block to airway

64
Q

Pulse Oximeter: Respiratory

A

Always connect to pulse oximeter.
Position patient to facilitate oxygen intake.
Be cautious: the light from the pulse oximeter can burn the baby’s skin.
Check/change the pulse oximeter every few hours, especially in infants or preemies.
Readings may not be accurate if not positioned properly.

65
Q

Inhaler Use

A

With metered-dose inhalers or dry powder inhalers, ask children to rinse their mouth after use.
This helps avoid thrush or candida infections from steroid medication.

66
Q

Do NOT perform CPT on patients with

A

Blood clots
Pulmonary embolism
Broken ribs
Spinal injuries
High intracranial pressure (ICP

67
Q

Early signs of hypoxia

A

Tachycardia (increased heart rate)
Tachypnea (increased breathing rate)
Restlessness
Pale skin

68
Q

late signs: hypoxia

A

Bradycardia (decreased heart rate)
Bradypnea (decreased breathing rate)
Lethargy
Confusion
Cyanosis (bluish skin color)

69
Q

100% O2

A

Oxygen saturation of 100% is not ideal; it can lead to oxygen toxicity, which can affect hearing.

70
Q

Suctioning

A

Hold suction for less than 5 seconds for infants.
Hold suction for 10 seconds for toddlers and older children.
Oxygen levels will drop during suctioning but should return to normal once suctioning stops.

71
Q

VAP

A

Ventilator associated pneumonia

72
Q

Artificial airways complication

A

Artificial Airways
Suctioning
Oral care (VAP!)
Complications
Decannulation, occlusion

73
Q

Otitis Externa

A

swimmers ear
not drying, main cause inflammation
-S & S-itching, drainage
Treat w/ steroid ear drops, Tylenol for pain/ itching, irrigate ears

74
Q

otitis media

A

Child often tugs on affected ear, fussiness, fever
Short eustachian tubes
Recurrent if smokers in family
Recurrent can lead to tonsillitis

75
Q

Tonsillitis

A

Enlarged tonsils can interfere with breathing, nasal and sinus drainage, sleeping, swallowing, and speaking.

76
Q

Enlarged tonsils also can disrupt the function of the eustachian tube, which can impede hearing.

A

Risk for recurrent otitis media!

77
Q

Acute tonsillitis

A

Tonsils become inflamed and reddened
Small patches of yellowish pus also may become visible.
Acute tonsillitis may become chronic

78
Q

More serious in pediatric airway due to smaller airway

A

Tonsillitis

79
Q

Positive culture for Group A beta hemolytic strep (GABHS)

A

tonsilitis

80
Q

Epiglottitis

A

Not as threatening since Hib vaccine
Look for inspiratory stridor, drooling

81
Q

Laryngotracheobronchitis

A

Barking cough

82
Q

croup syndromes

A

life-threatening condition; partial or full occlusion, horse/ froggy/ stridor, chest retractions, dyspneic, must have intubation ready at bedside
fever

83
Q

lateral neck X-ray to confirm diagnose, never culture the throat-will make it worse

A

croup syndrome