Week 10 - physical exam of the hip Flashcards

1
Q

what are the clinical entities of groin pain

A

adductor related groin pain
iliopsoas related
inguinal related
pubic related
hip related
hip joint OA

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2
Q

adductor related groin pain (ACUTE)

A

 Is common is sports involving sudden change in direction.
 Presentation – acute onset, adductor longus most commonly injured muscle.
 Symptoms – groin tenderness/pain on muscle palpation, stretch or contraction.
 Clinical signs:
o Palpation – pain on muscle belly or musculotendinous junction near origin, on inferior pubic ramus.
o Adductor tenderness AND pain on resisted adduction.
o Positive tests on – resisted outer range adduction, positive squeeze test with hip neutral in long level. Passive adductor stretch
o If all three tests are positive – provide approx. 80% probability of a positive MRI in adductors.
 Can be one recurrent or chronic if not properly managed.

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3
Q

adductor related groin pain (TENDINOPATHY)

A

 Presentation – graduation onset, chronic or overuse.
 Symptoms – proximal groin pain, initially pain with increased activity, may improve once warmed up, possible loss of speed and or kicking distance, if untreated, pain tends to persist during activity.
 Clinical signs:
o Pain on palpation of adductor origin
o Pain with passive hip abduction (stretch)
o Pain with resisted hip adduction (contraction)
o Positive squeeze test @ 0 degrees and 45 degrees.

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4
Q

iliopsoas related groin pain - acute onset

A

 Rectus femoris acute injury with kicking and sprinting.
 Iliopsoas acute injury with change of direction in sports.
 Clinical signs:
o Iliopsoas or rectus femoris palpation
 Negative palpation = 100% probability of negative MRI.
 Positive palpation = poor probability of positive MRI.

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5
Q

iliopsoas related groin pain - long standing or chronic

A

 Often related to repetitive activities and or accumulated overload in activities with excessive hip flexion (e.g., kicking, dancing).
o Iliopsoas tendinopathy
o Snapping iliopsoas tendon over iliopectineal eminence/femoral head.
o Iliopsoas bursitis.
 Presentation – groin pain, deep ache, usually unilateral.
o Pain on palpation
o Pain on resisted hip flexion and or pain on hip flexion stretching.

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6
Q

snapping hip syndrome (anterior) - iliopsoas

A

Snapping hip syndrome (Anterior) – iliopsoas tendon
 Iliopsoas ‘snaps’ over iliopectineal eminence causing audible ‘clunk’.
 Presentation – ‘clunk’ anterior hip, usually no MOI or history of trauma.
o Common in young women.
 Symptoms:
o Audible ‘clunk’ – often asymptomatic – is snap felt internal or external to hip joint.
o Can be bilateral.
o Can develop pain with clunk – if painful snapping hip they may have concomitant.
o May be associated with iliopsoas bursitis.
 Clinical signs:
o Start with hip in flexion and abduction, ask patient t eccentrically lower leg into extension with guidance – see if there is a reproduction of snap.
o Or ask the patient to simply reproduce the snap.
o Palpate snap – try and change it with applied pressure over the tendon.

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7
Q

inguinal related groin pain - abdominal wall

A

Inguinal related groin pain – abdominal wall
 Rectus abdominus insertion:
o Pain in the inguinal canal region AND tenderness of the inguinal canal at insertion of the rectus abdominus on pubic rami.
o No palpable inguinal hernia is present.
o Due to acute strain while lifting or overuse injury caused by excessive abdominal contractions.
o Pain aggravated by resistance testing of the abdominal muscles or on Valsalva/cough/sneeze.

 Inguinal hernia – posterior inguinal wall weakness.
o Gradual onset, poorly localise groin pain increasing with activity e.g., kicking and coughing/sneezing.
o Pain could be felt in the groin, genital area, it could be abdominal pain and they may experience urinary symptoms or bowel symptoms.
o Need to refer for medical review immediately if suspecting an inguinal hernia.
o May also involve tears in transverse Abd or external oblique fascia/aponeurosis.

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8
Q

pubic related groin pain (osteitis pubis)

A

 Presentation:
o Sometimes referred to as “sportsman groin.”
o May be secondary to repetitive microtrauma – training overload. Insidious gradual onset.
o Unsure of true pathophysiology.
o Involves pubic bone and pubic symphysis.
o In adolescents, pubic or adductor related groin pain could be due to apophysitis.
o Could include multiple pathologies.
 Pubic bone edema/stress reaction/periostitis pubis/ enthesopathy/adductor insertion tendinopathy.
 Consider age, type or sport and loading of the athletes with presenting symptoms.
 symptoms – symphysis pain on palpation, aggravated by exercises (twisting, turning, kicking), eases with rest, symptoms may be present during and after activity, may have over 6 weeks of longstanding pain – time of onset of symptoms is important.

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9
Q

pubis related groin pain - pubic bone stress

A

 clinical signs:
o Local tenderness of pubic symphysis and on immediately adjacent bone, positive squeeze test.
 Diagnostic imaging
o Bone changes seen on X-ray, MRI, and CT.
o Bone change may be evident in early stages and degenerative changes in chronic stages.

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10
Q

differentiation of groin pain

A

palpation

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11
Q

hip related groin pain

A
  1. Femoral acetabular impingement (FAI) syndrome
  2. acetabular dysplasia and or hip instability
  3. Other conditions without distinct osseous morphology (labral tear/ligamentum teres tears, chondral pathology).
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12
Q

femoral acetabular impingement

A
  1. Femoral acetabular impingement (FAI) syndrome – “a motion related clinical disorder of the hip with a triad of symptoms, clinical signs, and imaging findings. It represents symptomatic premature contact between the proximal femur and the acetabulum.”

 Types of morphology:
o Cam – increased bone formation at anterior surface of femur – decreased femoral head-neck offset. Associated with intra-articular head-neck offset.
o Pincer – bony growth at acetabular, generally anterior.
o Mixed – both cam and pincer present.

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13
Q

cam vs pincer morphology

A

for femoral acetabular impingement

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14
Q

cam vs pincer morphology

A

for femoral acetabular impingement

Cam/pincer morphology
 Can be present radiographically but be asymptomatic (10-25% of people)
 Commonly seen in young athletes – football/soccer, ballet, hockey, martial arts – hip loading in adolescents may be a risk factor for cam morphology.

Cam morphology development:
 Mainly when the proximal femoral growth plate is open.
 Rare after closure of the proximal femoral growth plate.

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15
Q

clinical presentation of femoral acetabular impingement

A

Clinical presentation

 Diagnosis – clinical and imaging positive findings.
 Symptoms:
o Motion or position related hip/groin pain and/or pain in the back, buttock, or thigh – pain increased with walking, running, swatting, performing lateral/cutting movements.
o May also have clicking, catching, locking, stiffness, restricted ROM or giving way.
 Clinical signs:
o Hip impingement tests (FADIR) reproduce their pain.
o Limited hip ROM -> IR in flexion.
 Investigations:
o X ray – AP view of pelvis and Dunn view for the lateral femoral neck.
o MRI – 3D hip morphology, cartilage, ligamentum teres and labral lesions – cross sectional view.
o May be associated with previous slipped femoral epiphysis or congenital hip dysplasia.

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16
Q

acetabular dysplasia and or hip instability

A

 Anatomy – fibrocartilage ring and dense CT attached to the bone acetabulum rim.
o Acetabulum labrum depends on acetabulum for stability and increased contact area of acetabulum to distribute load.
o Has a blood and nerve supply, therefore is pain sensitive.

 Presentation – most common intra-articular hip injury in athletes – 22% of athletes with hip pain.

o Anterior-superior tears are most common:

 Grade I – detachment from hyaline cartilage at acetabular rim

 Grade II – cleavage tears within substance of labrum.

o Patients with a labral tear have a 40% change they may also have chondropathy.

17
Q

acetabular labral tear

A

Acetabular labral tear
 Causes:
o Trauma – single or repetitive trauma – rotation and flexion, repetitive pivoting, hip flexion, twisting or direct trauma (fall/MVA).
o Congenital – due to acetabular or hip dysplasia.
o Capsular laxity – due to systemic connective tissue disorder or repetitive activities.
o Idiopathic – may be associated with FAI (cam or pincer morphology) syndrome.
 Symptoms – anterior groin pain, gradual onset of symptoms or related to trauma/injury, pain increases with activities.
o Ask patient is pain is intermittent, a sharp catching pain, clicking, locking, catching, giving way.
o Ask patient if they have referred pain in other areas such as buttock and/or anterior thigh.
 Diagnostic imaging
o X-ray – WB AP view and lateral hip view
o MRI and MRA most sensitive, CT scan, arthroscopy gold standard.

18
Q

ligamentum teres tears

A

intraarticular ligament with proprioception and stabilization role in hip joint.
- is taut in flexion, adduction and ER.
- tears common in athletes (type 1 is a partial tear, type 2 is a complete rupture, and type 3 is degenerate ligament)

MOI - forced flexion and adduction with ER or IR or twisting motions.

diagnostic imaging - MR athrography and CT arthrography.

19
Q

 Capsuloligamentous laxity (+/- hip instability)

A

 Capsuloligamentous laxity (+/- hip instability)
o Traumatic laxity +/- instability:
* Hip dislocation – fall on flexed knee and hip or form dashboard (MVA).
* Repetitive rotation movements with axial loading
* Trauma – stretching of capsule, possible labral damage/tears of ligamentum teres.
* Recurrent dislocations.
o Atraumatic instability
* In hypermobility congenital conditions, generalised ligamentous laxity, acetabular dysplasia or in athletes (gold, football).
o Symptoms
* Feeling of instability
* Pain in hip/groin region which may increase with activity.
o Clinical signs
* Decrease hip ROM +/- pain.
* Anterior hip pain with passive E + ER in prone.
* Passive accessory glides of the hip – hypermobile.

20
Q

hip related groin pain - osteoarthritis (OA)

A

 Synovial joint’s age-related response to abnormal loading from a traumatic incident or repetitive microtrauma
 OQ can affect whole joint organ – including cartilage, subchondral bone, inflammation of the synovium and synovial lining thickening.
 Leads to pain and stiffness, muscle changes and functional limitations.

 Kallgren and Lawrence scores 1-4 based on:
o Subchondral bone sclerosis
o Joint space narrowing
o Osteophytes – acetabular and femoral
o Subchondral cysts

 Femoral head superior migration, secondary to articular cartilage loss.

 MRI – the scoring hip osteoarthritis with MRI method
o Early microscopic cartilage changes
o Osteophytes
o Subchondral cysts
o Inflammation
o Muscle atrophy
o Other joint changes
o High sensitivity (95.7%) and specificity (84.8%) in detecting cartilage lesions.

21
Q

hip OA predisposing factors

A
22
Q

anterior pain - common conditions - Hip OA

A

 Age – increased risk in over 55-year-olds, with further increased risk in those over 65.
 Gender - women increased risk.
 Genetic predisposition.
 Ethnicity or race – lower incidence in Asian cultures than white Caucasian.
 Morphological abnormalities/developmental disorders.
 Leg length difference
 History of lower limb or hip trauma.
 Occupational factors – manual labor/physical stress work and sporting activities.
 Increased weight or obesity (BMI)

23
Q

clinical diagnosis criteria for hip OA

A

set A :
- over 50 years
- hip pain
- hip IR is greater than or equal to 15 degrees.
- pain with IR
- morning stiffness for 60min or more

Set B:
- over 50 years.
- hip pain.
- hip IR less than 15 degrees.

24
Q

OA symptoms

A

 Hip joint pain and pain anterior or posterior or lateral hip region +/- referral to thigh or knee.
 Deep “ache” or discomfort with sharp stabbing pain.
 Hip joint pain and stiffness restricted mobility.
 Stiffness in early morning eases less than 60 mins.
 Stiffness after rest, decreases with movement.
 Pain usually related with activity e.g., walking or stairs.
 Difficulty putting socks and shoes on.

25
Q

physical exam for hip OA

A

 Hip accessory glides performed anterior/posterior, lateral/medial, cephalad/caudad.
o In 20 hip flexion (because this position reduced capsule tension).
o In hip extension to assess effect of capsule tension.
o In painful hip range.
 This identifies stiffness in joint and medial/up and out migratory presentation.

26
Q

OA diagnosis - clinical prediction rule

A

Hip OA diagnosis – clinical prediction rule.
 5 main predictor variables:
1. Self-reported squatting an aggravating factor.
2. Active hip flexion causing lateral hip pain.
3. Scour test + adductor causing lateral hip or groin pain.
4. Passive IR – less than 25 degrees.
5. Active hip extension causing pain.

 If patient has 3/5 predictor variables – likelihood of having hip OA is 68% (positive likelihood ratio = 5.2)
 If patient has 4/5 predictor variables – likelihood of having hip OA is 91% (positive likelihood ratio = 5.3).

27
Q

hip related groin pains (other)

A
  • stress fracture of neck of femur or acetabulum
  • stress fracture of pelvis or pubic ramus
  • nerve entrapment - Obturator nerve 0 entrapped in the fascia as entering adductor longus, brevis and pectineus compartment OR Ilioinguinal nerve – entrapped under ilioinguinal ligament – groin pain radiating to genitalia region.
  • other conditions causing groin pain not to be missed: o Orthopaedic, neurological rheumatological, urological, gastrointestinal, dermatological, oncological and surgical.
    o Lumbar spine referral.
28
Q

GTPS (1)

A

 Umbrella term for conditions causing lateral hip pain:
o Glut med/min tendinopathies or tears.
o Trochanteric bursitis -tendinopathies of the gluteal tendons and bursa.
 Prevalence – 10-25% of the population in 40–60-year-old.
o More common in females – a lower neck shaft angle is a risk factor for, and adiposity is associated with GTPS in women.
 Pathology
o Compressive loading of the gluteal tendons and bursa between greater trochanter and ITB with repetitive activities.
o Compression loads – tendon and bursa thickening.
o Thickened tendons develop reduced capacity for tensile loading, leading got tears under less loading.

29
Q

GTPS - clinical signs and symptoms

A

 Symptoms:
o Lateral hip pain, ache, intermittent, persistent or unrelating.
o Pain lying on affected site, sitting with legs crossed, prolonged standing on one leg, decreased physical exercise.

 Clinical signs
o Lateral hip tender on palpation, over the greater trochanter
o Plus- pain with at least one of the following:
 Pain EOR hip adb/add or hip IR/ER
 Positive FABER
 Active resisted abduction in adducted position
 Pain with sustained single leg stance
 Positive resisted external de-rotation
 Positive Ober’s test.

30
Q

GTPS - gluteal tendinopathy

A

GTPS: Gluteal tendinopathy
 Presentation:
o Females more than males.
o Insidious onset pain usually.
o Pain may be associated with training loads or physical activity sudden changes.
o May be acute onset, after an incident such as a fall.

 Symptoms:
o Pain and tenderness over greater trochanter
o Pain may radiate down lateral thigh.
o Pain may be worse at night.
o Pain to sleep on affected side.
o Pain with prolonged single leg stance or stairs.

 Clinical signs:
o Pain provocative tests
o *Tender on palpation over greater trochanter
o *Sustained single leg stance

 Patient reported outcome measure.
o VISA – G questionnaire

 Investigations:
o MRI – gold standard
o US.

31
Q

GTPS - snapping hip syndrome (lateral)

A

 Common in younger, active people.
 Pathology – gluteus max anterior fibres and ITB and move over the greater trochanter from posterior to anterior as the hip moved from extension to flexion causing a snapping.
o May be associated with pain and inflammation.
 Symptoms:
o Audible snap during repetitive flexion, extension, and abduction +/- pain.
o Snap increases with repetitive hip F/E.
 Clinical signs:
o Patient can usually reproduce their symptoms in:
 In side-lying with the ITB on stretch – ask patient to actively flex and extend their hip.
 Manual compression to the band proximal to greater trochanter or external rotation of hip may relieve snap.
 Trochanteric bursitis could also be present.

32
Q

posterior pain - buttock pain

A

 Common in younger, active people.
 Pathology – gluteus max anterior fibres and ITB and move over the greater trochanter from posterior to anterior as the hip moved from extension to flexion causing a snapping.
o May be associated with pain and inflammation.

 Symptoms:
o Audible snap during repetitive flexion, extension, and abduction +/- pain.
o Snap increases with repetitive hip F/E.

 Clinical signs:
o Patient can usually reproduce their symptoms in:
 In side-lying with the ITB on stretch – ask patient to actively flex and extend their hip.
 Manual compression to the band proximal to greater trochanter or external rotation of hip may relieve snap.
 Trochanteric bursitis could also be present.

Piriformis
 Piriformis syndrome and sciatic nerve entrapment
o In short posterior rotators.
 Ischiogluteal bursa
o Between hamstring tendon and origin on the ischial tuberosity.
o Usually affected in conjunction with proximal hamstring tendinopathy.