Week 1 Flashcards

1
Q

What is Cognitive Neuropsychology?

A
Examine variety problems associated with 
brain injury 
•  Study patients 

understand mind 

understand patients 

rehabilitation
•  Explain symptoms brain injured patients 
relative to normal cognitive function
– eg. perception, memory, language, movement
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2
Q

Optic Aphasia

A

inability to name
visually presented objects with intact
visual object recognition processes
(patient can mime the use of objects)

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3
Q

Utilization Behaviour

A
actions for use 
of objects intact but patient uses 
objects in an inappropriate context.
– Occurs because of poor task based 
control and actions are triggered by 
strong “use” of environmental cues
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4
Q

What is Cognitive Neuropsychology? (how does it build on other realms of psych)

A

Cognitive Psychology: study mental processes
– eg. face and object recognit
ion, speech, write, planning
• Neuropsychology: study how particular brain
structures and processes mediate behaviour
• Cognitive Neuropsychology: combination of both
emphasis on understanding the mind
• Developmental Cognitive Neuropsychology: study
the development of normal cognitive function
• Cognitive Neuroscience: branch of neuroscience,
study of the neural systems (brain) that carry out
cognitive function
• Cognitive Neuropsychiatry

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5
Q

Aims of Cognitive Neuropsychology

A

Explain patterns of impaired and intact
cognitive function in brain injured patients
• Link these patterns of damage to components
of theory/models normal cognitive function
• Information from brain injured patients
(impaired and intact processes)

conclusions (theories and models) normal
cognitive function

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6
Q

How do we achieve these two aims?

A
Dissociations
•  Dissociations between tasks allow inferences 
to be made about the working of the intact 
mind
Task 1 (word)
Task 2 (face)
•  Patient A
Good
Poor
•  Tasks must tap separate Cognitive Processes
•  BUT what if 1 easier than 2?
Double Dissociation
•  2 patients with opposite performance 
patterns
Task 1(word)
Task 2 (face)
•  Patient A
Good
Poor
•  Patient B
Poor
Good

Associations
• Look for pattern of
impairments across tasks
• Association of symptoms
– patient KM impaired on three
tasks - face, word and object
recognition
– Then they must all use the same
processor?
Association deficits – all 3 tasks require
common cognitive processes
• No overlap between processes and discrete
sets of cognitive processes are mediated by
adjacent brain areas
• e.g., Gerstmann’s syndrome (damage to left
parietal cortex) – acalculia, finger agnosia,
right-left disorientation and dysgraphia
• Association is ok but Dissociation or Double
Dissociation is better

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7
Q

Dissociations

A

Dissociations between tasks allow inferences
to be made about the working of the intact
mind
Task 1 (word)
Task 2 (face)
• Patient A
Good
Poor
• Tasks must tap separate Cognitive Processes
• BUT what if 1 easier than 2?

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8
Q

Double Dissociation

A
Double Dissociation
•  2 patients with opposite performance 
patterns
Task 1(word)
Task 2 (face)
•  Patient A
Good
Poor
•  Patient B
Poor
Good
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9
Q

Associations

A
Look for pattern of 
impairments across tasks
•  Association of symptoms
– patient KM impaired on three 
tasks - face, word and object 
recognition
– Then they must all use the same 
processor?

Association deficits – all 3 tasks require
common cognitive processes
• No overlap between processes and discrete
sets of cognitive processes are mediated by
adjacent brain areas

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10
Q

Gerstmann’s syndrome

A
An example of an association:
(damage to the left parietal cortex)
-acalculia
-finger agnosia
-right-left disorientation
-dysgraphia
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11
Q

acalculia

A

Acalculia is an acquired impairment in which patients have difficulty performing simple mathematical tasks, such as adding, subtracting, multiplying and even simply stating which of two numbers is larger.

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12
Q

finger agnosia

A

s the loss in the ability “to distinguish, name, or recognize the fingers”, not only the patient’s own fingers, but also the fingers of others, and drawings and other representations of fingers.

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13
Q

right-left disorientation

A

Left-right disorientation describes confusion of the right and left limbs and suggests a lesion in the dominant parietal lobe. It can be tested by asking the patient to show you their right and then their left hand, and then asking them to touch their left ear with their right hand and vice-versa.

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14
Q

dysgraphia

A

Dysgraphia is a deficiency in the ability to write, primarily handwriting, but also coherence.[1] Dysgraphia is a transcription disability, meaning that it is a writing disorder associated with impaired handwriting, orthographic coding (orthography, the storing process of written words and processing the letters in those words), and finger sequencing (the movement of muscles required to write)

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15
Q

Studies of Groups

A

Traditional Neuropsych:
group patients based on common co-occurrence of symptoms (study “syndromes” eg gerstmanns)
aim of neuropsych is to determine location of brain regions
not concerned with specific cognitive processes

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16
Q

Studies of individuals

A

this is the approach taken by cognitive neuropsych
syndromes and categories are too coarse
options - subdivide syndromes based on assocation of disorders:
– e.g. acquired dyslexia
deep dyslexia, neglect
dyslexia, attentional dyslexia
Each patient single case requiring unique
explanation
– Case studies used in Co
gnitive Neuropsychology
• Neuropsychology: patient group vs.. normal
group
>
loss info

17
Q

Cognitive Neuropsychology aims

To have generalisability

A
Theory/model cognitive function must 
account for all cases disorder
•  Theory/model must also explain normal 
cognitive function 
•  Based on experimental data
•  1) cognitive psychology
•  2) cognitive neuropsychology patient data 
and 
•  3) computer modelling
18
Q

Assumptions of Cognitive Neuropsychology

A

Neurological specificity (isomorphism) – some
correspondence between mind and brain
(monism)
• Universality assumption – all cognitive
architectures the same initially
• Transparency – analysis of pattern of intact and
impaired performance and pattern of errors by
patients

nature and function of impaired
processes and components (which component
or module was disrupted)
• Converging operations – data multiple sources

19
Q

What is the Modularity Hypothesis (MH)?

A

mental life is made possible by activity of multiple cognitive processors or modules
-e.g. reading, face recognition, speech, -higher order eg. decision making etc

modules independent but inter-communicate
modules > diagrams of cognitive processes

fodor came up with the MH.
in terms of our cognitive processes we have modules (eg - object recognition a distinct module to face recognition which is a distinct module from word recognition)

data from double dissocation cases shows that we don’t have 1 module that does all your cognitive processes

  • each process/system impaired separately
  • semi-independent
20
Q

Problems with use of diagrams

A

• Diagrams limited e.g. single word vs. syntax
• Lack specification internal workings modules
• Basis patient data

post-hoc theories
(falsifiable)
• Diagrams superimposed on outline left
hemisphere

21
Q

17
Problems with use of diagrams
• Diagrams limited e.g. single word vs. syntax
• Lack specification internal workings modules
• Basis patient data

post-hoc theories
(falsifiable)
• Diagrams superimposed on outline left
hemisphere
Cognitive Neuropsychology today

A

diagrams used
• Careful study mind-brain
• Used to explain the overall workings of an
information processing system – pathways of
information flow and the specific components
of the system

22
Q

Modularity of Mind -revisited

A

Information encapsulated: processing within
module isolated from rest of system
• Domain specific: 1 type of input e.g. visual,
spoken word
• Mandatory module operation: automatic e.g.
recognition familiar persons
• Innate
• Fast operation
• Neural specificity
• Higher level processes not modular
– e.g. reasoning, decision making

23
Q

Functional modularity

A

mental processes are functionally independent but intercommunicate
eg. object reco vs face reco

24
Q

Anatomical modularity

A

specific brain region

25
Q

neurochemical modularity

A

specific neurotransmitter

26
Q

Knowledge modules

A

independent knowledge modules, independent of other bodies of knowledge

27
Q

Processing modules

A

independent processor of

information