week 1 Flashcards

1
Q

impairment vs diagnosis

A

Per the Guide to Physical therapy
Disease – The intrinsic pathogen or active pathology (i.e. cancer, IDDM, arthritis)

Impairment – Loss of normal anatomical, physiological, psychological status of an organism. (i.e.- decreased MMT, ROM, sensory changes.

Functional Limitation – limitation of performance of an organism as a whole. (i.e.- inability to ambulate, inability to feed oneself)

Disability – Limitation or disadvantage to perform socially defined roles within a culture, as it relates to age and gender. (i.e. quadriplegic patient)

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2
Q

Upper extremity evaluation- student notes 2024

A
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3
Q

Examination and evaluation for PT visit

EXAMINATION
The process of obtaining a history,
performing a systems review, and
selecting and administering tests and
measures to gather data about the
patient/client. The initial examination is
a comprehensive screening and specific
testing process that leads to a
diagnostic classification. The
examination process also may identify
possible problems that require
consultation with or referral to another
provider.

A
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4
Q

Evaluation for PT visit

EVALUATION
A dynamic process in which the
physical therapist makes clinical
judgments based on data gathered
during the examination. This process
also may identify possible problems that
require consultation with or referral to
another provider.

A
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5
Q

What kind of screen is the upper quadrant screen?

What is another name for it?

A

It is a neuro screen and it is know as the neuro screen

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6
Q

When is upper quadrant screen performed

A

When there are any symptoms distal to the acromion. or any suspicion of neurological or unusual involvement

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7
Q

In the upper quadrant screen there in UMN Tests. These consist of what?

Also called upper quarter screen

Also called Neuro screen or neurological screen

A

UMN tests” refers to tests for Upper Motor Neuron (UMN) lesions. These tests are used to identify signs of damage to the upper motor neurons, which are part of the central nervous system. Examples of UMN tests include:

Babinski Reflex: Stroking the sole of the foot to see if the big toe extends upwards (a positive sign in adults suggests an UMN lesion).
Clonus: Rapidly dorsiflexing the foot and observing for rhythmic contractions (more than three beats are considered abnormal).
Hoffmann’s Reflex: Flicking the nail of the middle or ring finger and observing for thumb flexion (a positive sign suggests an UMN lesion).
Increased Deep Tendon Reflexes (DTRs): Hyperactive reflexes such as exaggerated knee jerk or biceps reflex.
Spasticity: Increased muscle tone that results in stiff and awkward movements.

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8
Q

When do you do over pressure when using Active ROM

A

When there is no pain in normal ROM

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9
Q

What is end feel

A

sensation of feel at the end of a joints ROM

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10
Q

PROM measures what?

A

PROM

. Testing for inert tissue involvement
. Remember this is osteokinematic movement, not accessory
. Overpressure to check end feel
· End feel
. Sensation of PT’s hands’ “feel” at the end of a joint’s ROM
. Gentle overpressure at end range

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11
Q

What does PROM not measure?

A

Arthrokinematic motions, or acessory motion

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12
Q

What are joint surface levels or accessory motions

These are also known as joint play/ Arthrokinematic Assessment

A

Joint surface level” or “accessory” refers to accessory joint movements, which are the small movements that occur between the joint surfaces during normal motion. These movements are essential for full, pain-free range of motion and proper joint function. They are not under voluntary control and are necessary for the larger, voluntary movements of the joint. Accessory movements include:

Glide (Slide): One joint surface moves parallel to the plane of the adjoining joint surface.
Roll: One joint surface rolls over another like a tire rolling on a road.
Spin: One joint surface rotates around a stationary axis.
These movements are assessed and sometimes mobilized by physical therapists to improve joint function, reduce pain, and restore normal movement patterns.

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13
Q

Are arthrokinematic assessments done passively or actively

A

Passively

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14
Q

Passive vs. active rom

PROM vs AROM

A

Passive vs. active rom

. If active and passive motions are limited/painful in the same direction, the lesion is an inert tissue
. If active and passive motions are limited/painful in the opposite direction, the lesion is in the contractile tissue

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15
Q

Inert tissue

A

Inert Tissue
Inert tissues are those that do not contract or produce movement. They provide structural support and stability. Examples include:

Ligaments: Connect bone to bone and stabilize joints.

Joint Capsules: Enclose the joint and contain synovial fluid for lubrication.

Cartilage: Provides cushioning and smooth movement at the joints.

Bursae: Small fluid-filled sacs that reduce friction between moving parts.

Inert tissue lesions: If both active and passive motions are limited or painful in the same direction, it suggests that the problem is with inert tissues. For example, pain in both active and passive shoulder flexion might indicate issues with the shoulder joint capsule or ligaments.

Example:
Inert Tissue Lesion: Limited and painful active and passive shoulder abduction could indicate an issue with the shoulder joint capsule or a ligament sprain.

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16
Q

Contractile tissue

A

Contractile Tissue
Contractile tissues are those that can contract and produce movement. They include:

Muscles: Generate force and movement by contracting.

Tendons: Connect muscles to bones and transmit the force produced by muscles to move the bones.

Contractile tissue lesions: If active and passive motions are limited or painful in opposite directions, it suggests that the problem is with contractile tissues. For instance, pain during active elbow flexion and passive elbow extension may indicate an issue with the muscles or tendons involved in flexion.

Example

Contractile Tissue Lesion: Painful active wrist extension and painful passive wrist flexion might suggest an issue with the extensor muscles or tendons in the forearm.

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17
Q

More examples of short term goals

A

Short Term Goals:

. By 3 weeks patient will have:

. Patient will have increased Upper Extremity
Functional Scale by 9 points

. Patient will have increased shoulder external
rotation strength as measured by 1/2 MMT
grade

. Patient will have increased shoulder
abduction AROM by 15 degrees

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18
Q

More examples of long term goals

A

. Long Term Goals:

. By 6 weeks patient will have:
. Increased Upper Extremity Functional Scale by 18 points
. Patient will be able to drive without
compensatory motion

. Patient will be able to engage in all work
activities without compensatory motions

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19
Q

Start of Clinical Anatomy and Bio Mechanics of the Shoulder_Student Notes 2024

A
20
Q

joints of the shoulder

A

. Glenohumeral Joint

. Acromioclavicular Joint

. Sternoclavicular Joint

. Scapulothoracic Joint

21
Q

Sterno clavicular joint

A

· Plane synovial
. Saddle shaped surfaces
· Disc gives joint 3df
. Disc separates joint into 2
compartments
. Little inherent stability structurally
but strong ligs
. CP- clavicle maximal ER when
shoulder maximally elevated

22
Q

Sterno clavicular joint motions

A

Protraction

Retraction

· Elevation

Depression

· Rotation

23
Q

Ac joint

Acromioclavicular joint

A

· Strong ligaments, weak capsule
. Coracoclavicular- vertical joint
stability
· Acromioclavicular- prevents joint
separation

. CP position: arm in maximal
elevation

24
Q

Glenohumeral Joint

A

Glenohumeral Joint

. Ball and socket joint
. 3 degrees of freedom
. Arthokinematic glide- convex on concave -
roll/glide in opposite direction
. Increased mobility versus stability
· Stabilizers: passive: ligaments (Glenohumeral:
superior, middle, inferior; coracohumeral
ligament; labrum
. Stabilizers: active: muscles (rotator cuff
muscles: supraspinatus, infraspinatus, teres
minor and subscapularis)
. Mobility provided by rotator cuff muscles and
deltoid

25
Q

glenohumeral joint

A

ball and socket (spheroidal- 3 df

Glenoid fossa faces superior and anterior

Convex humerus on concave glenoid fossa of the scapula

Roll and glide in the opposite directions

closed pack position is abduction and external rotation

26
Q

Glenohumeral motions

A

GH: Motions

· Flexion/extension
. Abduction/adduction
· ER/IR

. Combined motions:
circumduction, horizontal
ab/adduction

. Scapular elevation (scaption)
elevation in scapular plane-
functional plane

27
Q

Start of Shoulder Soft Tissue Lesions Student Notes 2024

A
28
Q

Musculotendinous meaning

A

muscle and tendon

29
Q

Stage approach for shoulder disorders

A

1st stage screen for red and yellow flags

2nd stage pathoanatomical diagnosis

3rd stage rehabilitation

30
Q

Red flag conditions

A

tumor

History of cancer
Symptoms and signs of cancer, including unexplained weight loss, pain
not correlated with mechanical stress, and unexplained fatigue
Unexplained mass, swelling, or deformity

Infection

Red skin
Fever
Systemically unwell

Fracture or unreduced dislocation

Significant trauma
Seizure
Acute disabling pain
Acute loss of motion
Deformity or loss of normal contour

Neurologic lesion

Unexplained sensory or motor deficit

Visceral pathology

Pain not reproduced with shoulder mechanical stress
Pain or symptoms with physical exertion or respiratory stress
Pain associated with gastrointestinal symptoms
Scapular pain associated with ingestion of fatty foods

31
Q

Sub acromial impingement syndrome

Also known as SIS

Also known as impingement syndrome

A

Is a cluster of signs and syndroms

Shld Pain with overhead upper extemity motion

“Painful ark sign”

shoulder pain at night which is usually because of direct compression. (sleeping on affected side

Shoulder pain during repetitive motion, ex: overhead work activities

32
Q

Neer Classification of Impingement

A

Neer Classification of Impingement

Three stages of impingement according to age and pathological changes (Neer 1983).

STAGE I: <25 years, reversible lesion, edema and hemorrhage.

STAGE II: 25-40 years, tendinitis and fibrosis (bursa may become
fibrotic and thickened).

STAGE III: >40 years, tendon degeneration, bony spurs, rotator cuff
tears, long head of biceps rupture.

Not all of these symptoms have to be there. Sometimes its just one or two

33
Q

Signs and Symptoms of impingement syndrome

A

. AROM may display decreased motion or a painful arc
. PROM may be painfree (contractile lesion)
. Resistive may be painful in the primary motion of the involved structure
. Mobility testing normal unless there are long term capsular changes

34
Q

Etiology

A

Etiology
- Seen in over head
repetitive activities or
trauma

  • Exacerbating factors -
    laxity and
    inflammation,
    postural
    malalignments
    (kyphotic posture,
    rounded shoulders)
35
Q

Intrinsic causes- Physiological causes

A

Intrinsic Causes- ‘Physiological Causes’

. Degenerative tendinopathy

· Muscle weakness and/or tissue stress

. Decreased collagen integrity

. Inflammation

· Vascular causes- ‘avascular zone’ RC tendons

. Intrinsic causes can cause extrinsic causes and vice-versa - physiological
causes can affect bio-mechanical causes and vice-versa

36
Q

Extrinsic causes- Bio mechanical origin

A

. Mechanical Cause

Extrinsic Causes- ‘Bio-Mechanical
Origin’

. Tendons affected by humeral migration or decreased space

. Compression of tendons

. Many potential causes: postural impairments, motion impairments

· Altered scapulohumeral dynamics

37
Q

Three Types of Extrinsic
Impingement

A

Three Types of Extrinsic
Impingement

. Primary Impingement: A mechanical,
space limiting condition in the
suprahumeral region

Secondary Impingement: Abnormal
biomechanics (hypermobility or posture)

Internal Impingement: Posterior RC gets
“pinched” intra-articularly when arm is in
90 degrees of Abduction and ER secondary to anterior laxity

· It is possible to see a patient who presents
with a combination of primary and
secondary impingement (swimmer)

38
Q

Primary impingement

A

Primary Impingement

· Decreased space
- Spurs, OA, outlet stenosis, or
thickened CA ligament, hooked
acromion
. Acromion types: Type I straight,Type II
curved
Type IlI hooked

39
Q

Secondary impingement

A

Secondary Impingement

. Occurs when the biomechanics of the shoulder are altered by:
- GH instability
- Faulty movement patterns
- Neurological pathology
- Adhesive capsulitis

40
Q

Internal impingement

A

Internal Impingement

. Impingement of the cuff
(undersurface and midsubstance) on
the posterosuperior glenoid labrum

· Usually posterior RC gets “pinched”
when arm is in 90 degrees of
Abduction and ER secondary to
anterior laxity

· Who gets it?
- Overhead throwing athletes
(pitchers, volleyball players, etc)

41
Q

Internal impingement is linked to?

A

-linked to micro instability
- Scapular dyskinesis

42
Q

differntial diagnosis that you would need to rule out before making a diagnosis of impingement

A

· Glenohumeral instability
. Cervical radiculopathy
· Calcific tendinitis
· Adhesive capsulitis
. Glenohumeral arthritis
. Acromioclavicular arthritis
. Rotator cuff tendonosis (correlate with impingement), result of
impingement in some cases

43
Q
A

Diagnosis of impingement syndrome

· Resistive:

  • May present with weak and/or painful supra and/or infraspinatus,
    deltoid and biceps.(long head of biceps may be compromised)
  • Decreased strength of ER compared to IR
  • Weak scapular stabilizers (trap, serratus, etc)

. Joint mobility: tight posterior capsule, hypo-mobile upper T-spine (regional interdependence), limited scap mobility

· Special Tests:
- Impingement sign (Neer) and test
- Hawkins’ sign
- Infraspinatus Muscle Test (External Rotation Resisted Test)
- The Painful arc sign test

44
Q

Infraspinatus muscle strength test

Also known as infraspinatus muscle test

also known as External rotation resisted test

A
45
Q

If all 3 tests are positive it would have ratio of 10.56

A

two tests would be 5.03

46
Q
A