W13 - PAEDIATRIC ORTHOPAEDIC; THE BIG 3; THE ACUTE LIMPING CHILD Flashcards

1
Q

Common Presentations of Bow Legs & Knock Knees

A
  • varus leg can progress to normal period or valgus period
  • mostly resolve by the teens
  • physiological cause commonly

> Treat rarely!!

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2
Q

Common Presentations of In Toeing & Examination

A

Negative angle on gait

  • newborn = problem in foot (commonest)
  • infant = tibia
  • school age = femur
  • Internal tibial torsion; spontaneous resolution
  • Metatarsus Adductus; also resolves

*doesnt improve w/ neuromuscular disorders

> reassurance; resolution
Never operate before 10yo!

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3
Q

Common Presentations of Flexible Flat Feet

A

Nil medial long. arch

  • flat foot normal at birth, and diminishes with age
  • if foot mobile - normal variant
  • in soles = no benefit
  • obesity, ligament laxity
  • Jacks Test
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4
Q

Common Presentations of Curly Toes

A
  • Strong family hx; tightness flexor tendons
  • mostly cosmetic issue, 1/4 improve spontaneously

> Flexor tenoromy (over 6yo) when posing functional problem

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5
Q

Common Presentations of Variable Walking Age

A

12 mos mean average, but not the normal

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6
Q

Recognize MSK symptoms that suggest need for referral (5 S)

A

S ymptoms - e.g pain causing problems
S ymmetry (lack of)
S tiffness - hindfoot = underlying bony abn.
S yndromes - associated syndromes = unlikely resolution
S ystetmic Illness

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7
Q

Significance of bone or joint pain that is worse at night is

A

infection or tumour until proven otherwise.

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8
Q

Manufacturing Defects Vs Packaging Defects

A
  • Interruption during critical time window of development can give rise to MSK disorders
    +alongside congenital defects
  • In-utero positioning giving rise to altered forces once birthed; can correct over time once birth
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9
Q

What phenomenon contributes to Bent Legs

A

dt rotational deformities

FEMORAL ANTEVERSION
40º at birth and decreases w/ age = 10º anteversion by 16yo
⇧femoral anteversion = ⇧ability for medial rotation

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10
Q

Describe the common presentation, management and complications of DDH

A

Development Dysplasia of the Hip
* congenital short bone, gait lack of confidence evident
F>M, Left hip > Right hip
* RF: first born, breech presentation, FHx, additional LL deform., Increased wt.

  • ORTOLANI’S SIGN = dislocated hip
  • BARLOW’S SIGN = dislocated hip
  • Piston Motion Sign
  • Hamstring Sign
  • Examination
  • USS for early dx (Vs late dx via XR)
  • universal USS Vs selective

> Simple splint <3mos
Closed reduction and spica cast

> Open reduction and capsule reefing 1yr+
“” w/ femoral shortening 18mos+

*6yo+ and bilateral; 10yo+ and unilateral = leave alone

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11
Q

Describe the common presentation, management and complications of Perthes’ disease

A

Typically: male, primary school age, short stature, limp, knee pain on exercise, stiff hip joint

*Avascular necrosis of hip, rpt minor trauma, familial hx

> Maintain hip motion; avoid painful activities
Analgesia
Splints, physio
?Osteotomy in 7yo+

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12
Q

Describe the common presentation, management and complications of SUFE

A

Slipped Upper Femoral Epiphysis = post-trauma, displacement through growth plate, w/ epiphysis always slipping down and back

  • Teen, overweight, left groin pain, painful wt. bearing;
  • external rot. posture and gait
  • XR = best @ lateral view + AP view

=> Line of Klein = cut epiphysis parallel to gr. trochanter
+ endocrine abn
+ bilateral progression

Chronic @ 3weeks
Angle or Proportion
Stable = able to wt. bear Vs Unstable = Prognosis

> Stable = Pinned in-situ
Unstable = open reduction but AVN high risk

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13
Q

Stages and progression of Perthe’s Disease

A

1) Initial Stage
2) Fragmentation Stage
3) Reossification Stage
4) Healed Stage

  • younger pres = better prognosis
  • with nearer the head being round, the better the outlook
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14
Q

What can delayed diagnosis of SUFE lead to

A
  • Progression of slip w/ increased risk of early OA
  • Stable lesions becoming unstable
  • AVN of femoral head
  • Impingement
  • chondrolysis
  • Limb shortening
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15
Q

Significance of chronic slips

A

New bone formation at the slipped site, making it difficult to group; hypertrophic zone nil ossification

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16
Q

Understand the investigation of the limping child

A

?foreign bodies, erythema, swelling, deform
? shoes
? scoliosis, midline dimples+hair = older children = spinal pathology

Common pres:
limp; pain; general malaise; temp; recent infection; trauma; pseudoparalysis

  • PHYSICAL EXAM.
  • TEMPERATURE
  • XR
  • USS
17
Q

Know the importance of presenting features and treating septic arthritis

A

Limp, pseudoparalysis, swollen and redness, refusal to move joint, pain, temperature

  • mainly knee and hip affected
  • multiple routes of entry: think spread and direct insult
  • inflamm investigations
  • XR, USS
  • Synovial fluid: wcc, gram stain, culture

> IV Abx, empirically for 2w; 6w in total

18
Q

Know the importance of presenting features and treating acute osteomyelitis

A

*10-15yo commonest: pain, local symptoms, fever, reduced movement

  • RF: blunt trauma, recent ifnection
  • Terminal branches/vasc loops; low pO2 = inhibited phagocytosis, trauma
  • S. aureus
  • Inflamm bloods workup
  • Followed by imaging + bone biopsy
  • Blood cultures

> IV Abx
Sx: indicated by culture/abscess = drainage, debridement; failed abx

19
Q

Understand aetiology of transient synovitis

A
Commonest cause (bar trauma) of limping child between 5-10yo (0-5 common behind variant and trauma)
* Limp, slight unwell, Hx of infection, APYREXIC
  • low CRP, normal WCC = bloods not as inflamm as OM or Septic Arth. *
20
Q

Know subtypes, and symptoms/signs suggestive of JIA

A

Juvenile Idiopathic Arthritis subtypes
=> PAUCIARTICULAR = 1 or 2 joints
=> POLYARTICULAR
=> SYSTEMIC ONSET

  • Persistent arth. of at least 6w
  • F>M
  • Post-strep infection
21
Q

Understand why children with pauciarticular JIA need eye screening.

A

Early Onset Pauciarticular JIA, commonly associated in boys, has associations with the eyes

  • Glaucoma, cataracts and permanent visual damage (including blindness) are all complications that could result from severe uveitis
22
Q

Know how to apply Kocher’s criteria

A

Points on

PYREXIA
NIL WT BEARING
WBC >12,000/mL
ESR >40mm/hr

Chance of septic arth. = increases w/ greater points

23
Q

Know the importance of presenting features and treating infective myositis

A

Presentation of infective myositis is with pain localised to one or more muscles (although in most cases it is a single muscle), with variable degrees of systemic inflammatory manifestations, depending on the pathogen

*S. aureus

> Drainage of abscess (CT guided)
Abx

!progression to compartment syndrome, infective osteomyelitis, or systemic shock (sepsis)

24
Q

Red Flags to Consider with Limping Child

A

Neoplastic concern

  • NIGHT PAIN
  • INCIDENTAL TRAUMA
  • CESSATION OF SPORT/GOING OUTSIDE
  • SWEATS, FATIGUE
  • ABN BLOODS: atypical Hb, blood film, platelets

> Paediatric/Oncology Opinion

25
Q

Common causative agent of Septic Arth.

A

S. aureus

26
Q

Pathognomic Signs of Infective Cause of Paed. Ortho Problem

A
  • Non Wt Bearing
  • Night Pain
  • Systemic Symptoms
27
Q

2 ½ year old boy, left leg pain & limp, painful but willing to be examined, normal temperature

  • Abn XR Film
  • Low reticulocytes and monocytes
A
  • No suggestive inflamm/infective cause
  • Low reticulocyte = infiltrative
  • Low reticulocyte = Bone Marrow dysfunction, liver, kidney dysfunction
  • Low Monocyte = Malignancy

(Dx of mets neuroblastoma)