W06 - PAEDS: Approach to assessing children; Children are not small adults; Adolescent Health; SUDI Flashcards

1
Q

Physiological differences and risks to children

A

dt diff SA:vol, water content, metabolic activity
more at risk of
- hypothermia
- dehydration
- hypoglyc.

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2
Q

Wt and food amounts conversion

A

0.45lb = 1kg

1 ounce = 29.6 ml

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3
Q

Developmental Red Flags

A

Loss of developmental skills

Referral elsewhere

Hearing loss (simultaneous referral for audiology/ ENT)

Persistent low muscle tone/ floppiness

No speech by 18 months, esp if no other communication (simultaneous referral for urgent hearing test)

Asymmetry of movements/ increased muscle tone

Not walking by 18m/ Persistent toe walking

OFC > 99.6th / < 0.4th / crossed two centiles/ disproportionate to parental OFC

Clinician uncertain/ thinks that development may be disordered

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4
Q

Areas of development

A

Fine motor skills

Gross motor

Social & self-help

Language & Comms

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5
Q

Child Health Screening

A

Primary care w/ midwives. Recorded in red book.

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6
Q

SUDI: triple risk model

A

a

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7
Q

SUDI

A

The sudden and unexpected death of a child under 1 for which no adequate case is found after a thorough post mortem and case report

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8
Q

Significance of Back to Sleep Campaign

A

Since the 1980s onwards, campaign highlighting awareness of certain practices which saw a decrease in cot deaths.

  • potentially preventing rebreathing when baby is left to lie on front

NOW:
- chaotic, lower socioeconomic, high parity, inappt. sleeping conditions, illicit drugs, alcohol, LATER NEONATAL AGE than before

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9
Q

SUDI vs SIDS

A

SIDS subset of SUDI, where no suspicious circumstances or untoward features are found.
* SIDS incidence is decreasing

whereas SUDII = leaves room for potential explanation for death to appear later. In practice, more commonly used etc.
SUDI better than ‘unascertained’ for optics, and leaves case ‘open’.

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10
Q

Significance of increasing complexity of investigating SUDI

A

positive findings will arise the more tests are ran d/t the nature of variability in testing results, however minor they may be.

*consider impacts of continuous testing and results

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11
Q

3 typical cases of SUDI

A

OBVIOUS HOMICIDE
* suspicious circumstances:
* evidence of fractures
* genetics contribute to PREDISPOSITION not CAUSATION

NATURAL CAUSE IDENTIFIED
*congenital
* SEPSIS

UNKNOWN CAUSE
* most common
*

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12
Q

Methadone withdrawal in infants

A
  • excessive feeding, highly irritable, mouthing a lot prior to death.
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13
Q

Milestones

A
  • social smile ~2mos onwards
  • sitting ~3mos onwards, ~6mos +/- hand support, 8mos: w/o support
  • walking ~9mos pull to stand, ~12mos walking development
  • first words: ~9mos sounds, then ~12mos simple single words

*refer if not walking by 18mos

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14
Q

Developmental Assessment

A

a

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15
Q

6-8w Review (GP & HV)

A

ID, feeding, parental concerns, development, measurement, examination, sleeping position

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16
Q

27-30mos Review

A

carried out by HV
- ID data
- development
- physical measurements

17
Q

Growth Monitoring

A

Physical measurements: wt., length, head circumference (OFC)

18
Q

Wt. and Length and OFC

A

Birth:
3.3kg, 50cm, 35cm (OFC)

4mos:
6.6, 60cm,

12mos:
10kg, 75cm, 45cm (OFC)

3y:
15kg, 95cm

19
Q

Causes of failure to thrive in early life

A

DEFICIENCIES: lactation, feeds, prematurity, palatal abn., neuromusc. disease

INCREASED METABOLIC DEMANDS: disease of organ, infection, anemia, CF, malignancy

EXCESSIVE NUTRIENT LOSS: reflux, pyloric stenosis

20
Q

Failure to Thrive Algorithm

A

HX => PC, dietary assessment, birth/family/social

PHYS. EXAM => dysmorphic features, anthropometry, systems, development

TRIAL OF FEEDING IN HOSPITAL => observing feeding, handling, SALT assessment, developmental assessment

Good intake => non-rganic or organic

Poor intake => Organic cause; Feeding disorder; Non-organic or mixed

20
Q

Failure to Thrive Algorithm

A

HX => PC, dietary assessment, birth/family/social

PHYS. EXAM => dysmorphic features, anthropometry, systems, development

TRIAL OF FEEDING IN HOSPITAL => observing feeding, handling, SALT assessment, developmental assessment

Good intake => non-rganic or organic

Poor intake => Organic cause; Feeding disorder; Non-organic or mixed

21
Q

Understand key aspects of adolescent brain development

A
  • bio, psycho, social development between 11-25yrs
  • developing prefrontal cortex
  • concrete thinking dev => abstract thinking
  • self-id, self-consciousness, independence
  • risk taking behaviours and exp.

support development by:
- normalise challenges
- suport goal setting and problem solving
- negotiating support
- reminders
- opportunities to recognise success

22
Q

Discuss how young people may present within healthcare settings including health outcomes

A
  • over half of MH problems are established by age 14 and 75% by age 24
  • ?rising prevalence
23
Q

Be familiar with the impact of adolescence on chronic conditions such as diabetes

A
  • highest graft failure rates; kidney loss/rejection
  • HbA1c deterioration
24
Q

Consider important communication adaptations

A
  • offer seeing adolescent on their own: standard, chaparone, followed by bringing parents

*set agenda, aim for conversation, explore priorities/concerns

  • discuss adherence
  • discuss understanding of disease and treatments, and assess motivation
25
Q

Adolescent Social Hx

A

HEADSS

Home
Education/Employment
Activities
Drugs/Alcohol
Sexuality
Suicide/Self-Harm

26
Q

Transition from paeds to adult services

A

At 16yrs
- begin process of promoting independence etc early!
- ensure good udnerstanding & eduation on condition and medication
- INITIAL JOINT APPT. W/ ADULT AND PAED TEAMS
- checklists to improve