VP7+8 Flashcards

1
Q

Hyperaemia

A

The active process of increasing the blood flow to a tissue during eg. inflammation.

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2
Q

Congestion

A

A passive process due to decreased outflow from a tissue, eg. secondary to a local obstruction (something tight around a limb)

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3
Q

Haemorrhage

A

Escape of blood from vessels

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4
Q

Petechial haemorrhage

A

Small haemorrage, 1-2mm diameter either due to a capillary rupture or clotting problem

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5
Q

Ecchymoses haemorrhage

A

A heamorrhar 1-3cm diameter, your regular bruise.

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6
Q

Suffusive haemorrhage

A

A heamorrhage that is covering a large area

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7
Q

Rhectic haemorrhage

A

A rupture causing a haemorrhage, eg a fracture-related haemorrhage

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8
Q

Haemorrhagic diathesis

A

Increased tendency for haemorrhages after minor injuries, eg due to clotting problems.

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9
Q

Haemostasis

A

Blood clotting, arrests bleeding from ruptured or damaged blood vessels. Either primary, secondary or tertiary.

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10
Q

Primary haemostasis

A

The platelet response (turning dendritic) to exposed collagen fibers and von Willerbands factor.

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11
Q

Secondary haemostasis

A

Activation of the coagulation cascade by activated platelets (releasing factor 12 to activate factor 11 = thrombin).

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12
Q

Tertiary haemostasis

A

Fibrinolysis, the breakdown of fibrin into plasmin to balance the reaction.

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13
Q

Thrombus

A

A formation of thrombocytes, fibrin and other blood elements in blood vessels. Will have a firm attachment to the blood vessel wall.

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14
Q

PM clot

A

Coagulation of the blood as a normal post mortem change, esp found in the ventricles of the heart. Smooth surface and easily removed.

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15
Q

Embolism

A

The breaking off of a tissue/aggregation that will travel in the blood stream to a distant part.

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16
Q

Ischaemia

A

Lack of blood flow

17
Q

Infarction

A

Segmental or localised area of ischaemia (failure/reduction in blood flow).

18
Q

Disseminated intravascular coagulation (DIC)

A

Widespread intravascular activation of the coagulation cascade by either (1) tissue factor release (2) thrombin activation or (3) extensive endothelial damage, leading to thrombosis, ischaemia and micro-infarctions.