Visual System Disorders Flashcards

1
Q

what are the 3 “vital signs” of ophthalmology?

A

1) Visual acuity
2) Pupils
3) intraocular pressure

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2
Q

What is an easy way to determine if decreased vision is only a refractive error?

A

Have them read a chart with only one eye open… and then have them read the same chart with only one eye open looking through a “pinhole”… If it’s easier to read, then its a refractive problem

*because a small hole will only allow perpendicular light to travel back to the retina, thus it won’t require focusing

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3
Q

What are the 5 steps involved in pupillary constriction?

A

1) retinal ganglion cells
2) pretectal nuclei
3) edinger-westfall nuclei
4) Parasympathetics via CN 3
5) Ciliary Ganglion to ciliary muscle for constriction

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4
Q

Give an example of left eye relative affarent pupillary defect

A

RAPD is a optic nerve problem (afferent signal). In this case, the left eye would not constrict with direct stimulation (AND there wouldn’t be a consensual response). But if light was shined in the right eye, the left eye would constrict (be consensual response) because that is efferent, not afferent.

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5
Q

How do you determine if double vision is neurologic or non-neurologic?

A

have the pt. cover one eye, if their vision is still “double” then it is a refractive problem (aka monocular diplopia = non-neurological)

if the pt. sees double ONLY with both eyes open, then it is neurological (binocular dipoplia = neurological)

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6
Q

What does an increased cup:disc ratio indicate?

A

Glaucoma (increased intraocular pressure) tends to cause an increased cup size… thus the C:D ratio will increase (because normally the disc is larger than the cup)

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7
Q

What does a diabetic retinopathy look like?

A

numerous microaneurisms in vessels of retina

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8
Q

what should you suspect in a person with blurry vision, visual field loss, and loss of color vision?

A

optic nerve probs

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9
Q

clinical characteristics of optic neuritis (3 of em)

A

1) 92% have ocular pain
2) rapid onset of central vision loss (5ish days)
3) unilateral presentation!
* 77%female, mean age 32

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10
Q

What is best treatment for optic neuritis?

A

IV steroids

*oral prednisone actually increased the risk of recurrence (bad!)

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11
Q

what are the outcomes of optic neuritis?

A

with IV steroids, they will recover most (if not all 70% back to 20/20) visual acuity and stays with them for a long time

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12
Q

what is the risk of developing MS with optic neuritis?

A

Depends on # of other white matter lesions at the time of optic neuritis…

  • 0 other lesions = 22% chance of MS @ 10 years
  • 1 or more white matter lesions = 56% chance of MS @ 10 years
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13
Q

Giant Cell arteritis characteristics (3 of em)

A

1) Rapid (hours to days) visual acuity loss
2) Jaw pain is very sensitive (headache, wt. loss also common)
3) extremely high erythrocyte sedimentation rate (measure of inflammation in body)

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14
Q

Diagnosis and treatment of Giant cell arteritis

A

temporal artery biopsy = gold standard

9-12 mo. of steroids!

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15
Q

visual field loss with pituitary tumor

A

optic chiasm compression (aka only the axons that cross) resulting in a bitemporal hemianopsia

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16
Q

visual field loss with left cerebrovascular event

A

right homonymous hemianopsia (aka can’t see right field of each eye)