Visual Fields Flashcards

1
Q

! Rule out VF defect
! Document VF defect
! Localize pathology in visual pathway ! Monitor disease process over time

A

Reasons for performing perimetry

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2
Q

superior 60
inferior 75
temporal 100
nasal 60

A

normal limits of vf

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3
Q
!  Corresponds to the optic nerve 
!  15 ̊ temporal to point of fixation
!  1.5 ̊ below horizontal meridian
!  Diameter:
      !  5 ̊ horizontal 
      !  7 ̊ vertical

A

physiologic blind spot

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4
Q

VF examination strategies

A

kinetic perimetry

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5
Q

! Tests differential light sensitivities of specific retinal locations
on a fixed grid pattern
! Spacing between points varies on type of examination area

A

static perimetry

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6
Q

! Grey scale
! Decibel sensitivity plot
! Total deviation numerical & probability plot
! Numerical plot in decibels
! Compares sensitivity at each point to normal population of similar age
! Pattern deviation numerical & probability plot
! Adjusts for generalized depression or elevation of VF

A

automated static perimetric plots

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7
Q

! Fixation losses
! False positives
! False negatives

A

reliability indices

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8
Q
!  30-2
!  24-2
!  10-2
!  Macular
!  Nasal step: additional 12 locations up to 50 degrees nasal
A

humphrey vfa

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9
Q

! Generalized reduction in retinal sensitivity

A

vf defect depression

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10
Q

! Focal area of reduced sensitivity surrounded by an area of
normal sensitivity
! Absolute : defect persists when maximum stimulus is used (ie. blind spot)
! Relative: defect present to weaker stimulus, but disappears with brighter stimulus

A

scotoma vf defect

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11
Q

where are the axons in the optic nerve

A

behind the orbit and

near the chiasm

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12
Q

! Containscrossed(nasal)anduncrossed (temporal) fibers
! Typically situated directly above sella turcica
! Superior: hypothalamus and floor of third ventricle
! Inferior:pituitarygland
! Lateral: IC A and cavernous sinus

A

chiasmal anatomy

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13
Q

! Pituitarytumorinvolves

chiasm first

A

central: directly over sella

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14
Q

! Pituitarytumorinvolves

chiasm first

A

prefixed: anterior to sella

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15
Q

! Pituitary tumor damages optic nerve(s) first

A

post fixed: posterior to sella

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16
Q

! Extend from chiasm to LGB
! Fibers from temporal half of ipsilateral eye and nasal half
of contralateral eye
! Visual fields will produce homonymous hemianopia ! Typically incongruous

A

optic tracts

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17
Q
!  Relaynucleus
!  Positionedalonglateralaspectof
midbrain
!  Further organization of fibers
!  VF lesions will be hemianopic (congruous or incongruous)
A

lateral geniculate body

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18
Q

! SuperiorVF
! MeyersLoop:temporallobe
! Temporal lobe lesions: “pie in thesky”

A

inferior fibers (optic radiations)

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19
Q

! InferiorVF
! Loopposteriorlythrough parietal lobe
! Parietallobelesions:“pieonthe floor”

A

superior fibers

20
Q

! Medial aspect of occipital lobe ! Significant input from macula
! Central 30 degrees occupy 83% of the striate cortex

A

visual cortex

21
Q

blood supply of optic chiasm

A

circle of willis

22
Q

blood supply of visual cortex

A

! Posterior cerebral artery

! Middle cerebral artery

23
Q
!  Unilateral
!  Invades vertical hemianopic line
!  May respect horizontal line d/t separation of fiber bundles !  Complete/partial vision loss
!  Color vision defect
!  RAPD
A

lesions of optic nerve clinical findings

24
Q
!  Optic neuritis
!  Trauma
!  Space occupying lesion (adenoma, glioma, meningioma) !  Ischemic optic atrophy (NAION, AION)
!  Papilledema
!  Nutritional/toxic insult
!  Glaucoma
A

causes of lesions of optic nerve

25
Q
!  Vision loss
!  Typically painless
!  Progressive
!  Bilateral asymmetric
!  Headache possible
!  Stretching of diaphragma sella
A

chiasmal disease

26
Q
!  Signs
!  +/- VA loss
!  VF defect
!  Optic atrophy
!  APD
!  Dyschromatopsia
!  +/- endocrine dysfunction
!  Diplopia: if adjacent cavernous sinus is involved
A

chiasmal disease signs

27
Q
!  Pituitary adenoma
!  Suprasellar meningioma !  ICA aneurysm
!  Optic nerve glioma
!  Uncommon: !  Trauma
!  Inflammation
A

chiasmal disease: etiology

28
Q
!  Causes: compressive disease
!  Aka junctional scotoma due to
compression of Von Willebrand’s Knee
!  Due to post-fixed chiasm
!  Clinical findings
!  Ipsilateral RAPD
!  +/-diplopia
!  Ipsilateralcentralscotoma
!  Contralateralsuperiortemporaldefec
A

anterior chiasmal syndrome

29
Q

! Pre-fixed chiasm
! Macular fibers cross posteriorly in chiasm
! Tumor impinges on posterior chiasm and optic tracts
! Clinical findings
! Central bitemporal hemianopic defects (nasal macular fibers) ! Homonymous hemianopia due to optic tract compression

A

posterior chiasmal syndrome

30
Q

! Causes
! Suprasellar aneurysm
! Pituitary gland tumors
! Suprasellar meningioma and glioma
! Third ventricular dilation due to obstructive hydrocephalus
! Clinical presentation ! Bitemporal hemianopia ! Bilateral optic atrophy

A

middle chiasmal syndrome

31
Q

! Causes
! Distention of 3rd ventricle causing pressure on each side of
chiasma
! ICA aneurysm
! Clinical findings
! Binasal hemianopia ! Partial optic atrophy

A

lateral chiasmal lesions

32
Q

! Acromegaly: prominent brows/nose/chins
! Cushing Syndrome: moon face, truncal obesity, buffalo hump ! Galactorrhea
! Amenorrhea
! Decreased libido, infertility

A

endocrine dysfunction

33
Q

! Band pallor: common
! Diffuse pallor
! APD
! Dyschromatopsia

A

optic atrophy

34
Q

! MRI with contrast
! Neurosurgical referral ! Endocrine workup
! Monitor visual fields
! Every 4-6 months until stabilization, then annually

A

chiasmal disease manaement

35
Q

! Homonymous: nasal VF of one eye and temporal VF of fellow eye
! Congruous: nasal and temporal defects closely resemble each other
! Fibers posterior to chiasm become more segregated in terms of what part of the VF they represent
! Decreased VA not common unless lesion also involves optic nerve or occipital lobes
! Usually due to cardiovascular disease

A

post chiasmal disease

36
Q
!  Causes:
!  Tumors
!  Ischemic stroke
!  Aneurysms of superior cerebellar or PCA
!  Clinical findings:
!  +/- congruous hemianopia
A

optic tract lesions

37
Q
!  Causes:
!  Vascular occlusions
!  Primary & secondary tumors 
!  Trauma
!  Clinical findings:
!  Incomplete incongruous quandranopsias 
!  Neurologic deficits predominate

A

lesions of optic radiations

38
Q
!  Incongruous “pie in the sky”
!  Affects Meyer’s Loop: inferior retinal fibers
!  Neurologic symptoms !  Seizures
!  Hemiparesis
!  Hemisensory loss
!  Aphasia
A

temporal lobe lesions

39
Q
!  Incongruous “pie on the floor”
!  Affects superior retinal fibers
!  Neurologic symptoms !  Hemiplegia
!  Hemisensory loss
!  Visual neglect
!  Aphasia
A

parietal lobe lesion

40
Q

! Congruous homonymous hemianopia with macular sparing

A

middle cerebral artery infarct

41
Q

! Congruous homonymous macular defect

A

posterior cerebral artery infarct

42
Q

! Incongruous homonymous hemianopia
! Fibers segregated into R & L side of visual pathway
! Contralateral APD because temporal VF is 40% larger than nasal VF
! Band optic atrophy

A

optic tracts

43
Q

! Congruous

! Very small lesion and very difficult to isolate

A

LGN post chiasmal vf defects

44
Q

! Incongruous
! Pie-shaped superior homonymous hemianopia ! Seizures and hallucinations common
! Neurologic symptoms predominate

A

temporal lobe post chiasmal vf defect

45
Q

! Congruous, complete or incomplete

! Neurologic symptoms predominate

A

parietal lobe post chiasmal vf defect

46
Q

! Absolute congruity
! Superior and inferior fibers completely separated ! Extreme respect for vertical and horizontal midline
! Homonymous central or macula-sparing defects ! Absence of neurologic symptoms

A

occipital lobe post chiasmal vf defect