Vision loss Flashcards

1
Q

what are the types of ARMD

A

dry and wet

dry - develops very slow

wet - sudden onset requires urgent referral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

signs of ARMD

A

Foveal reflex is absent - pinpoint-sized “sparkle” of light that may be observed when light from an ophthalmoscope illuminates the interior of the eye

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

treatment for dry ARMD

A

No specific treatment

stop smoking
counselling/education
vitamins
visual rehabilitation and aids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

investigations in someone with loss of vision

A

VA
Visual fields
fundoscopy - check cornea is clear, look for red reflex and examine the funds

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

investigations in someone with loss of vision

A

VA
Visual fields
fundoscopy - check cornea is clear, look for red reflex and examine the fundus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

symptoms of wet ARMD

A

sudden loss of vision - days to weeks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

risk factors for dry ARMD

A

female
HTN
smoking
previous cataract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

prognosis of ARMD both dry and wet

A

WONT GO BLIND

peripheral vision sparred

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

diagnosis of both ARMD

A

made based on appearance of retina

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

management of wet ARMD

A

fluid and/or blood develops in the retina
intra-vitreal, anti VEG-F injections

needs urgent referral to ophthalmologist

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

causes of central vision loss

A

ARMD - wet and dry

diabetic retinopathy cataract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

sudden painful loss of vision in a non inflamed eye

A

GCA
migraine
optic neuritis
benign intracranial hypertension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

sudden painless loss of vision

A

retinal detachment - floaters and flashes
vitreous haemorrhage
central retinal artery occlusion - check BP, pulse, carotids, heart look for murmur, bruits and afibrillation refer to cardiologist
central retinal vein occlusion - check intra-ocular pressure, BP, viscosity
wet ARMD
trauma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

gradual loss of vision

A
cataract
ARMD - dry and wet 
diabetic retinopathy
primary open angle glaucoma 
tumour
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

anatomy of the aqueous production and IOP

A

IOP is due to the production of aqueous humour by the ciliary body, it flows over the anterior surface of the lens through the pupil and drains through the trabecular meshwork into Schlemm’s canal
alternative uveoscleral pathway drains 10% of aqueous

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

normal range of IOP

A

10-21 mmHg

17
Q

What does glaucoma mean

A

multifactorial optic neuropathy, with acquired loss of optic nerve fibres

thinning of neuroretinal rim of the optic disc results in the characteristic optic cupping and a corresponding loss of the visual field

can occur with or without increased IOP

18
Q

classification of glaucoma

A

1) primary or secondary (absence or presence of causative factors)
2) open angle or closed angle
3) acute or chronic
4) age of onset: congenital, juvenile, adult
5) level of IOP: normal tension glaucoma, or ocular hypertension

19
Q

most common type of glaucoma

A

primary open acute glaucoma - POAG

20
Q

signs and symptoms of POAG

A

usually asymptomatic until disease is advanced
painless loss of vision, can cause blindness if not treated
optic disc cupping, thinning of optic disc rim thinning and notching
raised IOP

peripheral vision affected - patients can’t drive

21
Q

treatment of POAG

A
can cause blindness if not treated 
aim to control IOP 
topical drops - carbonic anhydrase inhibitors, beta blocks, prostaglandin analogues, pilocarpine all lower IOP
laser and surgical options 
regular monitoring
22
Q

RF for POAG

A
DM
FH - relatives should be tested regularly 
race - afro Caribbean 
old age
raised IOP 
corneal thickness 
being short sited - myopia 
vascular features - CVD, vasospasm, systemic HTN