Vestibular System Flashcards
What 2 places does information from the vestibular nuclei go to?
- Ocular Muscles (VOR)
2. Spinal cord (VSR)
What 3 sections is the vestibular section divided into?
- Peripheral (sensory input, ss canals and otoliths)
- Central Processor (vest. nuclei and vest cerebellum)
- Mechanism for Motor Output (VSR, VOR)
True or False. The Bony Labyrinth is filled with PERILYMPH and the Membranous Labyrinth is filled with ENDOLYMPH.
True
What direction does the urtricle lay/hang? What direction does the saccule lay/hang? Motions that each detect?
utricle: horizonally - horizontal plane motions (front-to-back, left-to-right)
saccule: vertically - saggital plane motions (front-to-back, up-and-down).
What two types of cilia does the hair cells contain?
- Stereocilia (50-70)
2. Kinocilium (1)
True or False. Movement TOWARD the kinocilium DECREASES the rate of nerve firing.
False. movement toward it increases the rate and movement away decreases the rate.
What is the crista ampullaris. What is the maculae?
crista ampullaris - sensory area of the ampullae composed of a tuft of blood vessels and nerve fibers.
Maculae - the sensory areas containing the saccule and utricle.
Define:
- cupula
2. otoconia
- gelatinous membrane that covers hair cells to completely seal the semicircular canals
- calcium carbonate crystals that cover the gelatinous materal covering the hair cells in the otolith organs. Lags behind when movement, causing hair cells to bend.
What are the 2 advantages of the spatial arrangement of the semicircular canals?
- Sensory redundancy (in case one side is lost)
2. Allows brain to ignore changes that occur to both sides at once (changes in body temp or chemistry).
Damage to the vestibular cortex can result in what 2 conditions?
- loss of awareness of “upright” position
2. unilateral pusher syndrome
Explain where the following pathways from the vestibular nuclei lead to and what they do:
- Medial longitudinal fasciculus
- Vestibulospinal tracts
- Vestibulocollic pathways
- Vestibulothalamocortical
- Vestibuloautonomic
- Vestibulocerebellar
- extraocular nuclei & superior colliculus - eye movements (VOR)
- LMN’s - posture influence
- XI nuclei - influences head position
- cortex - conscious awareness of head position/movement
- reticular formation - vomiting/nausea
- vestibulocerebellum - eye movements and postural muscle activity via VOR and VSR respectively.
What symptoms does BPPV produce and what does it not produce?
BPPV produces short-lived (30s-2min) vertigo with rapid changes in head position (ext with head turned). It does not produce loss of hearing, “fullness” in the ear, or tinnitus.
- Is BPPV more common in men or women?
- Is it usually unilateral or bilateral?
- How long does it usually last?
- What is the name of the test used to dx it and positive sign of it?
- women
- unilateral
- 6-12 mo with certain cases on/off for years
- Dix-Hallpike positioning test. + is vertigo and nystagmus.
Explain the 2 pathophysiological theories behind BPPV.
- Cupulolithiasis - deposition of otoconial material onto the cupula
- Canalithiasis - debris of higher density than endolymph is free-floating in the canal.
What are the 4 signs/symptoms of vestibular neuritis?
- Acute onset of prolonged, severe rotational vertigo
- Imbalance
- Nausea
- Nystagmus toward the good ear (not while fixating).