Ventricular Arrythmias Flashcards
Causes of ventricular arrthymias
Drugs, electrolytes, orthostatic hypotension, HEART DISEASE, genetics
What is the difference between monomorphic and polymorphic PVCs
Mono: One ectopic location
Poly: Multiple ectopic locations
VT rates
> 3 beats
HR: >100
Describe NSVT
Terminates within 30 seconds
Where does v-tach originate from when it produces an S wave or LBBB
RV or septum
Where does vtach originate from if it produces a RBBB
LV
Which types of VT occur without structural heart changes
PVCs and VT
What electrolyte disturbance causes an S wave
Hyperkalemis
What do q waves indicate
Prior MI
S/s of vtach
Hypotension, syncope, palpitations, dizziness, exercise intolerance, lightheadedness
Vtach treatment plan
Remove offending factor
Antiarrythmic drugs
Catheter ablation
ICD implementation
What is the first line antiarrythmic drug for VT and its side effects
What are other options?
BB
Bradycardia, hypotension, negative ionotropic
Second line: Non-dihydropyridine CBB and produce negative ionotropic and vasodilators effects (cardizem and verapamil)
Sodium channel blockers: flecanide avoid with long term use in structural heart disease, can prolong QT
Potassium Channel blocking agents: Sotalol, Dofetilide, avoid with torsades/ qt prolong/ hypokalemia, and bradycardia
What is the antiarrhythmic of choice for heart disease patients that are not a candidate for ICD
Amiodarone
Adverse effects of amino
Bradycardia, thyroid, pneumonia, pulmonary fibrosis,
Describe an ICD
Endocardium leads to the right ventricle that control pulsations. Terminates vtach with cardioversion.
