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Finals > Vascular surgery > Flashcards

Vascular surgery Flashcards

1
Q

What is Budd-Chiari syndrome and what 2 conditions can it present as

A

Hepatic vein obstruction causing ischaemia and hepatocyte damage, presenting with liver failure or insidious cirrhosis

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2
Q

3 sx and a finding on bloods for Budd-Chiari

A

Abdominal pain
Ascites
Hepatomegaly
Raised ALT

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3
Q

Causes of Budd-Chiari

A

Primary - hypercoagulable = pregnancy, malignancy, pill, polycythaemia rubra Vera, thrombophilia
Secondary - obstructive = liver, renal, adrenal tumour causing hepatic vein thrombus

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4
Q

Investigate Budd-Chiari

A

USS
Hepatic vein Doppler
CT/MRI

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5
Q

Manage Budd-Chiari

A

Transjugular Intrahepatic Portosystemic Shunt (TIPS) or surgical shunt
Angioplasty

Anticoagulation lifelong unless varices

Consider transplant if fulminant hepatic necrosis or cirrhosis

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6
Q

5 causes of aneurysms in arteries

A

Atheroma
Trauma
Infection - mycotic aneurysm in endocarditis
Connective tissue disorders - Marfan’s, EDS
Inflammatory - Takayasu’s aortitis

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7
Q

What are true and false aneurysms?

A 
True = abnormal dilatations of arteries, involving all layers of wall
False/pseudoaneuryms = collections of blood ie after trauma, around vessel wall communicating with the lumen
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8
Q

What are the 2 types of artery aneurysms

A

Fusiform = both sides eg AAA = more common

Sac like = one side eg berry aneurysm

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9
Q

4 Common sites of artery aneurysms

A

Aorta (infrarenal)
Iliac
Femoral
Popliteal

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10
Q

5 complications of artery aneurysms

A 
Rupture
Thrombosis
Embolism
Fistulae
Pressure on other structures
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11
Q

Sx of ruptured AAA - triad, plus 4

A

Triad: 1) back pain, 2) expansile pulsatile mass, 3) hypotension/shock (haemodynamically unstable)
Intermittent or continuous abdo pain radiating to back, iliac fossa or groin
Vomiting
Syncope
Retroperitoneal haemorrhage - Cullen’s and Grey-Turner’s signs

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12
Q

Preventing AAA - 2

A

Manage BP
Quit smoking, weight loss, exercise
Statins and aspirin
Regular USS for men if >65y

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13
Q

Emergency treatment of AAA - 7

A
  1. ECG
  2. Bloods for amylase, Hb and cross match 10-40u
  3. Catheterise
  4. 2 large bore cannulas
  5. O neg blood but keep systolic bp<100
  6. Prophylactic abx - cefuroxime and metronidazole IV
  7. Surgery - clamp aorta above leak and insert graft
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14
Q

Definition of arterial and aortic aneurysms

A

> 150% dilatation of original diameter

AAA = >3cm across

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15
Q

Cause of AAA

A

Degeneration of elastic lamellar and smooth muscle loss

Genetic component

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16
Q

Sx unruptured AAA

A

Often asymptomatic, can be discovered on abdo exam incidentally
May have abdo/back pain

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17
Q

Monitoring (2 levels) and elective surgery (3 conditions) on AAA

A 
If <5.5cm, monitor by regular exam and US/CT:
- 3-4 = /year
- 4.5-5.5 = /3m
Elective surgery if 
1. >5.5 cm or
2. expanding at >1cm/y
3. or symptomatic
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18
Q

Risk factors for AAA rupture

A

Smoker
Raised BP
Female
Strong family history

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19
Q

Complication of elective surgical repair for AAA

A

Spinal or mesenteric ischaemia from dislodged thrombus debris

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20
Q

Surgery options for AAA, its pros and cons

A

If older, endovacular stent via femoral artery CT angiogram
+ = shorter hospital stay and fewer transfusions, less invasive, lower short term mortality
- = failure to totally exclude blood flow = endoleak; aneurysm may progress; higher risk of need for re-intervention
Younger patients = open surgery = clamp aorta and iliac arteries, remove and replace with prosthetic graft

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21
Q

What is thoracic aortic dissection and what can it cause at different places?

A

Blood splits aortic media with sudden tearing chest pain +- radiating to back, sequentially occluding branches of aorta

  • hemiplegia - carotid artery
  • unequal arm pulses and BP
  • acute limb ischaemia
  • paraplegia - anterior spinal artery
  • anuria - renal arteries
  • aortic incompetence and inferior MI - more proximal movement
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22
Q

2 types of thoracic aortic dissection and relevance to treatment

A

Type A = ascending aorta involved - consider for surgery

Type B = ascending aorta not involved - may be managed medically unless leaking, ruptured or compromising vital organs

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23
Q

Manage thoracic aortic dissection

A

Crossmatch 10u blood
ECG and CXR - expanded mediastinum is rare
CT/MRI or TransOesophageal Echocardiography (TOE)
Hypotensive to keep systolic BP at 100-110 = labetalol IVI

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24
Q

Early and late signs of limb ischaemia

A

Early - pain, pallor, pulseless

Late - paraesthesia, perishingly cold, paralysis

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25
Q

5 risk factors for limb ischaemia

A 
Smoking
Hypertension
Diabetes
MI
AF
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26
Q

Investigate acute limb ischaemia and when

A

CT angiogram for anatomical delineation and urgent vascular review
Within 6h or irreversible tissue damage

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27
Q

3 causes of acute limb ischaemia

A
  1. Thrombosis in situ (60%) - atheroma ruptures and thrombus forms on plaque’s cap - acute or acute on chronic
  2. Embolisation - proximal thrombus travels distally, from AF, post-MI, AAA, prosthetic heart valve
  3. Trauma - compartment syndrome
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28
Q

Investigations for acute limb ischaemia - 3

A

Serum lactate
Doppler USS
CT angiography and arteriography for pre op locating

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29
Q

Manage acute limb ischaemia - 5

A
  1. Oxygen and IV access
  2. Heparin - IVi
  3. Surgery
    - embolus = embolectomy with fogarty catheter, local intraarterial thrombolysis (tissue plasminogen activator) or bypass
    - thrombus = angioplasty, local intraarterial thrombolysis or bypass
    - amputate if mottled, non-blanching or woody muscles
  4. Look for emboli source with USS of aorta, popliteal and femoral arteries, annd echo
  5. Manage risk factors and give antiplatelet (clopidogrel or low dose aspirin)
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30
Q

Complications of limb ischaemia

A

Reperfusin injury due to sudden increase in capillary permeability - can cause compartment syndrome and substance release = hyperkalaemia, acidosis and rhabdomyolysis

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31
Q

4 levels of acute ischaemia and prognosis

A 
1 = viable, no sensory or motor deficit - only level with arterial and venous Dopplers
2a = marginal, salvageable with prompt treatment, minor sensory loss, no motor deficit - venous Doppler only
2b = immediately threatened but salvageable if immediately revascularised, sensory loss and pain at rest, mild motor deficit - venous Doppler only
3 = irreversible major tissue loss with permanent nerve damage, profound sensory and motor deficit - no dopplers
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32
Q

What is chronic limb ischaemia

A

Peripheral artery disease causing symptomatic reduction in blood supply to limbs, due to atherosclerosis

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33
Q

Staging of chronic limb ischaemia

A

Fontaine classification:

  1. Asymptomatic
  2. Intermittent claudication
  3. Ischaemic rest pain
  4. Ulceration/gangrene
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34
Q

Differentials for chronic limb ischaemia

A

Spinal stenosis - lateral radiating pain, better sitting than standing
Acute ischaemia - <14d

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35
Q

3 key features of critical limb ischaemia, plus other features

A
  1. ABPI <0.5
  2. Rest pain >2w requiring opioids, better when hanging legs off side of bed
  3. Ischaemic lesions/gangrene
    Pale and cold, weak/absent pulses, hair loss, atrophic skin, thickened nails
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36
Q

What is the condition of peripheral arterial disease affecting buttock and thigh?

A

Leriche syndrome - peripheral arterial disease at aortic bifurcation causes buttock/thigh pain and erectile dysfunction

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37
Q

Investigating chronic limb ischaemia and findings - 7

A
  1. ABPI:
    >0.9 = normal
    0.8-9 = mild
    0.5-8 = moderate
    <0.5 = severe
    [>1.2 = calcification - falsely high)
  2. Doppler USS for severity and location
  3. CT angiography
  4. CV risk assessment - BP, BM, lipids, ECG
  5. Rule out other features - arteritis (ESR/CRP), anaemia/infection (fbc), renal disease (U&Es)
  6. Thrombophilia screen and homocysteine levels if <50y and no risk factors
  7. Buerger’s - angle <20 and cap refil >15s = severe ischaemia
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38
Q

Manage chronic limb ischaemia - 2 areas

A

CVS risk factors - smoking, exercise, weight
- statins, antiplatelets, diabetes control
Surgical - percutaneous transluminal angioplasty (stent) and bypass graft if diffuse
- amputate if unsuitable/gangrene

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39
Q

Complications of chronic limb ischaemia - 4

A

Infected gangrene causing sepsis
Acute on chronic ischaemia
Amputation
Mobility and quality of life reduced

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40
Q

Progression of chronic limb ischaemia

A

Mostly stable
10-20% worse
5-10% critical
1-2% amputated

41
Q

What are young heavy smokers at risk of regarding ischaemia?

A

Buerger’s disease - thromboangiitis obliterans

42
Q

Drug to stop before angiography for limb ischaemia and why

A

Metformin - avoid metabolic acidosis

43
Q

Post op complication of amputation and manage

A

Phantom limb pain - gabapentin, can start before surgery

44
Q

What is carotid artery disease caused by

A

Atherosclerotic plaque causes either stenosis/occlusion or rupture/atheroembolism
Causes ischaemic stroke or TIA

45
Q

What makes an atherosclerotic plaque?

A

Fatty streak - lipid core - fibrous plaque

46
Q

How much of diameter is lost in mild atherosclerosis?

A

50%

47
Q

Symptoms of carotid artery disease

A

Generally asymptomatic as collaterals from contralateral ICA and vertebral arteries via circle of Willis
Carotid bruits
Stroke/TIA when rupture/occlude

48
Q

Differentials for carotid artery disease and relevant info of each - 3

A
  1. Carotid dissection - if connective tissue disease, trauma, esp <50yo
  2. Thrombotic occlusion - only differentiated on imaging
  3. Fibromuscular dysplasia = hypertrophy of vessel wall, focal neuro deficit, <50yo females
49
Q

Investigations of carotid artery disease

A

CT head
Bloods, ECG, CXR
Duplex USS/CT angiography to exclude other causes and find degree of stenosis

50
Q

Management of carotid artery disease

  • initial treatment
  • general management
  • long term medication
A

If <1.5h, IV alteplase (r-tPA) then 300mg aspirin for 14d
Screen swallowing, give o2 and keep BM 4-11mmol
Long term:
- antiplatelet clopidogrel or aspirin and dipyridamole
- statin
- Hypertension and diabetes management
- quit smoking, exercise

51
Q

Surgery for asymptomatic carotid artery disease, and ADR

A

Surgical revascularisation in <2w = carotid endarterectomy to remove atheroma and damaged intima, use temporal bypass during procedure
ADR = Stroke, damage to CN9, 10, 12, MI, bleeding, infection

52
Q

Classifications of acute mesenteric ischaemia and examples of cause - 4

A

Embolus eg from AF/murmur/valve replacement or AAA
Thrombus eg from atherosclerosis
Non-occlusive eg from hypovolaemic/cardiogenic shock
Venous eg from coagulopathy (look for DVT/PE, antiphospholipid syndrome), malignancy, inflammatory disorders

53
Q

3 risk factors for acute mesenteric ischaemic

A

Hypertension
Hyperlipidaemia
Smoking

54
Q

Sx of acute mesenteric ischaemia - 2

A

Generalised abdo pain out of proportion with clinical findings, diffuse and constant
N&V

55
Q

Bloods for acute mesenteric ischaemia

A

Lactate for acidosis
Clotting
LFT - liver ischaemia from disruption to coeliac trunk
Amylase

56
Q

5 causes of raised amylase

A 
Pancreatitis
Acute mesenteric ischaemia
DKA
Ectopic pregnancy
Bowel perforation
57
Q

2 imaging for acute mesenteric ischaemia and finding

A

CT with IV contrast for oedema, causing loss of wall enhancement and pneumatosis
AXR/CXR/CT abdo if suspect perforation

58
Q

Management of acute mesenteric ischaemia - 4

A

Resuscitation with IV access and fluids - fluid balance chart and catheterise
Broad spectrum abx
ITU if acidotic/organ failure
Surgery - excise necrotic/nonviable bowel, or revascularise with angioplasty or open embolectomy

59
Q

ADR of acute mesenteric ischaemia

A

Short bowel syndrome from excision
Necrosis or perforation
50-80% mortality

60
Q

Which 3 vessels is chronic mesenteric ischaemia in?

A

Coeliac trunk
SMA
IMA

61
Q

What age group in chronic mesenteric ischaemia in?

A

> 60yo females

62
Q

Symptoms and PMH of chronic mesenteric ischaemia - 5

A

Transient pain - when demand is increased in eating (10m-4h after) or hypovolaemia
Weight loss from anorexia and malabsorption
Loose bowels
N&V
Vascular comorbidities - stroke, MI, peripheral vascular disease

63
Q

Exam findings for chronic mesenteric ischaemia - 3

A

Cachexia
Generalised abdo tenderness
Abdo bruits

64
Q

Complications of chronic mesenteric ischaemia - 3

A

Infarct of bowel
Malabsorption
Concurrent CVD

65
Q

Investigations of chronic mesenteric ischaemia - 2

A

CT angiography

CV profile

66
Q

Treatment for chronic mesenteric ischaemia

A

Weight loss, exercise, quit smoking
Statin
Antiplatelets
Surgery if severe, progressive or debilitating symptoms - endovascular angioplasty and stent, or open endarterectomy or bypass

67
Q

What are varicose veins and where are they normally?

A

Dilated torturous segments associated with valvular incompetence
Saphenofemoral and saphenopopliteal junctions

68
Q

Causes of varicose veins and risk factors

A

Normally primary
Secondary = DVT, pelvic mass (fibroid, pregnancy) or AVM
RF: standing, obesity, pregnancy, family history

69
Q

Differentials of varicose veins - 3

A

Cellulitis
Ischaemic ulcer
DVT

70
Q

Symptoms of varicose veins

A 
Pain, ache
Swelling, particularly at the end of the day and after standing
Itching
Skin changes, ulcer, thrombophlebitis
Bleeding
71
Q

Signs on inspection of venous insufficiency - 8

A 
Examine from anterior thigh to medial calf (long saphenous vein) and back of calf (short saphenous vein):
Oedema
Varicose eczema
Thrombophlebitis - tenderness
Hard = thrombosis
Ulcers usually above medial malleolus
Haemosiderin deposits
Lipodermatosclerosis (upside down bottle)
Atrophic blanche
72
Q

Mass in groin with vascular insufficiency, investigation and treatment?

A

Saphena varix = dilatation of saphenous vein at saphenofemoral junction in groin
Sx - cough impulse and blue tinge
Duplex USS and high saphenous ligation

73
Q

Investigate varicose veins and 5 specific tests

A

Duplex USS - valve incompetence at great/short saphenous veins
Cough impulse and percussion test at SFJ
Auscultation for bruits over varicosities (AVM)
Trendelenburg’s test
Tourniquet test
Perthes disease

74
Q

What is Trendelenberg’s test for varicose veins

A

For SFJ valve competence
Lie down and raise leg (empties vein)
Put 2 fingers on SFJ (5cm below and medial to femoral pulse)
Stand up, keeping fingers in place
If varicosities are controlled, will not rapidly fill. Release fingers to confirm that they then fill
Varicosities are not controlled = incompetence at a lower level

75
Q

Tourniquet test?

A

Tourniquet tied around thigh at level of SFJ. If not controlled, move tourniquet move down leg (above then below knee) until incompetence identified

76
Q

Perthes’ test?

A

Determines if deep femoral veins are competent - tourniquet put around mid thigh while standing and patient walks for 5 mins. If saphenous veins collapse below tourniquet, deep veins are patent and communicating veins are competent
If no change, both saphenous and communicating veins are incompetent
If veins increase in prominence and pain, deep veins are occluded

77
Q

Manage varicose veins - 6

A 
Education - avoid prolonged standing, lose weight, regular walks
Compression stockings
4 layer compression bandaging for ulcers
Injection/foam sclerotherapy 
Laser coagulation 
Surgery - ligation/strip
78
Q

Complications of varicose veins and surgery

A 
Get worse with time if not treated
Haemorrhage
Thrombophlebitis from ablation and foam
DVT 
Nerve damage - saphenous and sural
79
Q

Where is the long saphenous vein system?

A

Medial thigh including saphenous opening, and posterior arch and medial perforators in calf

80
Q

Where is the short saphenous vein system?

A

Posterior thigh including saphenopopliteal junction, communication with long saphenous vein, and inconstant perforators in calf

81
Q

Features of venous ulcers on exam

A

Medial malleolus (gaiter region)
Irregular border with granulating base
Painful, especially at the end of the day
Dry, itchy, distended veins, varicose veins, oedema, varicose eczema
Atrophie blanche, haemosiderin, lipodermatosclerosis

82
Q

Features of arterial ulcers on exam

A

Small, deep, well defined with necrotic base
At sites of trauma and pressure areas
Cold, necrotic toes, hair loss, reduced or absent pulses
Sensation maintained

83
Q

Features of neuropathic ulcers on exam

A

Painless ulcers on pressure points, eg repetitive stress and injury
Burning/tingling
Punched out, variable sizes
Warm feet and good pulses
Often with vascular disease
History of peripheral neuropathy or atrophic neuropathy

84
Q

Pathophysiology of venous ulcers

A

Retrograde flow in superficial venous system causing dilatation and pooling
Reduces oxygen to skin and causes ulcer, along long and short saphenous veins

85
Q

Pathophysiology of arterial ulcers

A

Reduced arterial blood flow reducing perfusion and impairing healing

86
Q

Associated conditions with arterial ulcers

A

Intermittent claudication, peripheral artery disease

87
Q

Investigations for ulcers

A

ABPI low in arterial ulcers - show concurrent arterial disease in neuropathic ulcers
CT angiography +- MRA for arterial

Duplex shows insufficiency at junctions inn venous ulcers - show concurrent venous disease in neuropathic ulcers

Swab if infected, X-ray if suspect osteomyelitis (neuropathic more)
BM and B12 in neuropathic ulcers

88
Q

Manage venous ulcers - 5

A

Leg elevation and calf exercise
Emollient for dry skin
Antibiotics
4 layer compression bandages as long as ABPI >0.8
Radiofrequency ablation of varicose veins can improve healing

89
Q

Manage arterial ulcers

A

Conservative - improve CVD risk factors
Statin, antiplatelets, BP and BM management
Angioplasty or bypass graft +- skin reconstruction

90
Q

Manage neuropathic ulcers

A

Exercise and diet, maintain HbA1c <7%
Abx if infection +- surgical debridement
Chiropodist and foot wear

91
Q

What is a complication of neuropathy in foot?

A

Charcot’s foot - neuroarthropathy, deformity from repeated trauma
Causes swelling, distortion, pain, reduced function
- rocker bottom foot

92
Q

When does an ulcer become chronic?

A

> 4w from injury

93
Q

Complication of ulcer?

A

Marjolin’s ulcer

94
Q

What is slough in an ulcer?

A

Mix of fibrin, cell breakdown products, serous exudates, leukocytes and bacteria
Can be part of normal healing process

95
Q

Causes of ulcers

A 
Venous disease
Arterial disease
Neuropathic - diabetes
Lymphoedema
Vasculitis
Malignancy
Infection - TB, syphilis
Trauma
Pyoderma gangrenosum
Drugs
96
Q

3 types of gangrene

A 
Wet = with infection and tissue death
Dry = no infection
Gas = clostridium perfringens myositis
97
Q

Manage gangrene

A

Cultures - group A b-haemolytic strep = necrotising fasciitis or myositis
Surgical help if atypical cellulitis
Radical debridement +- amputation, + 5d benzylpenicilin

98
Q

What is gas gangrene? Sx and management

A

Clostridium perfringens myositis, risk factors = diabetes, trauma, malignancy
Early toxaemia, delirium and haemolytic jaundice
Oedema, surgical emphysema, bubbly brown pus
Remove all dead tissue, give benzylpenicillin, hyperbaric O2, clindamycin and metronidazole

99
Q

VTE prophylaxis for surgery

A

Dalteparin 5000u evening before surgery if admitted, or evening of surgery if arrived on day as long as 4h has passed since epidural
Antiembolic stockings
Intermittent pneumatic compression boots in theatre
No dalteparin or AES if neck surgery

Finals (13 decks)

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