Vascular PP1b Flashcards
CEA: considerations
GA vs Regional: pros cons
what to ready
GA: patient stays still. can manipulate gases, RR, Co2. Cons: BP swings
Regional: pt awake. gold standard for neuromonitoring. no BP swings
con: have to stay still, no orthopnea, back pain, anxiety, OA
ready to convert to general if needed
CEA: induction and maintainance
discuss plan
Also: ssep, herparin dosing
quick induction. LTA lidocaine. smooth no bucking no HTN or BP swings. propofol ok. ROC x1, but dont redose. opioids and benzo, small doses what good neuro exam on wake up
a line, 2 IV, all standard monitors
maintaince: normothermic, euvolemic, VG depth depends on SSEP or MEP
watch BP closely when clamped baroreceptos (hypotension, bradycardia). give heparin before clamp AVT 200-240
CEA emergence considerations
BP changes
neuro exam
coughing
PONV
when do most strokes occur
rapid and quick. HTN and tachycardia common. blunt w LTA, lidocaine, small doses or precedex
want a good neuro exam at wake up
prevent coughing and bucking
zofran, decadron, pherigan to prevent PONV
most stokes occur postop from thrombus
CEA and regional placement
how, where
efficecy
best neruo monitoring
considerations if using this method of sedation
regional for CEA
superficial cervical plexus block: C2-C4. SCM, superficial and inferior cervical plexus, scalene muscle
not shown to be more effective than general
awake patient
OA, anxiety, back pain, orthopnea
give mild benzos and opioids, avoid resp depression. want to try and have patient awake, be ready to convert to general
what do EEG, SSEP, temporal window look at
EEG looking at corticol structures
SSEP deeper brain structures
transcranial doppler: temporal window (decrease in blood flow and thrombos)
EEG changes
normal, ischemia, infarction
latency and amplitude
normal 50mls/100g brain tissue
ischemia <18
infarction <12
decrease in amplitude by 50% and increase in latency by 10%
cerebral pulse oxemitry
where specifically in the brain is it looking
like spo2 but for brain
keep within 20% of baseline
tell if there is disruption in blood flow to brain. getting oxygen to the grey matter
SSEP
most common nerve monitored
how sensitive
latency and amplitude changes that are important
anesthetic dosing with SSEP or MEP
median
80%, MEPS are more sensitive
50% decrease in amplitude and 10% increase in latency
SSEP less than 1 mac. MEP less than .5 mac
transcranial doppler
what does it tell you
where is it looking
looking at the temporal window, reflecting blood flow in MCA
tell you if decrease blood flow and thrombus formation
carotid stump pressures
discuss
this is looking at flow during clamping to the contralateral side
clamp on, measure pressure distal to clamp. inserted at common CA
40-50 mmhg is normal
angioplasty and stenting
where does this procedure take place
what compensatory function may be disrupted
heparin and ACT goal
IR
baroceptors may be disfunctional. may see hypotension and bradycardia. have pressors ready, glyco ready
Heparin and ACT >300
discuss CEA postoperative Considerations A.B.C.C.T.
AIRWAY: RLN and SLN injury. may have respiratory depression, airway compression from bleeding
BLEEDING: most common postop 1-5, leading to aitway compromise
CAROTID BODY DENERVATION: leading to hypertension. rule out bladder distention, pain, hypercapnia,hypoemia (esmolol, NTG, labetolol)
CHS: cerebral hyperperfusion syndrome: HA, seizures, cerebral edema
THROMBUS: formation MI and stroke