Vascular Disorder Flashcards
PAD (Peripheral Arterial Disorder)
What is it?
Reduced arterial blood flow to the extremities
Why?
Atherosclerosis
Where?
Arterial system extending from aorta to tibial artery
PAD Symptoms
Symptoms:
Intermittent Claudication:
- begins during exercise
and ends with rest
- Not constant, resolve
within 10 min
- Paresthesia (Numbness or
tingling) - Dependent rubour
- Skin change
- Skin cool to touch,
pallor, increase cap
refill, loss of hair, taut
and thin skin - Decreased circulation
- Weak pulse
PAD complication
- Continuous pain at rest
- Gangrene
- Limb threatening disease
PAD Diagnostic Test
- Anke Brachial Pressure Index
(ABPI)
- Ankle SBP/Brachial SBP
- Normal if 1 - 1.4 - Doppler Ultrasound
- Magnetic Resonance
angiography
Interventions Surgery
Surgery:
- Femoral popliteal bypass
- Percutaneous transluminal angioplasty (PTA) of the femoral
arteries - Endarterectomy
- Amputation as last resort
Interventions Drug Therapy
Drug Therapy:
- Antiplatelet (e.g. aspirin)
- Statins
- ACE inhibitors
- Meds to treat intermittent claudication
- Pentoxifylline/Trental - NSAID
Other Interventions
Nutritional Therapy
Exercise therapy
Risk Factor Modification
- Control BP, weight control, smoking cessation, blood glucose control
Intervention Surgery
- Femoral popliteal bypass
- Percutaneous transluminal
angioplasty (PTA) of the
femoral arteries - Endarterectomy
- Amputation as last resort
Acute Arterial Ischemic Disorder
- Occur suddenly, without
warning - Caused by embolism,
thrombus, or trauma
Clinical manifestations (“6 Ps”):
- Pain, pallor, pulselessness,
paresthesia, paralysis, and
perishingly cold
- Early treatment essential to
keep limb viable - Anticoagulant therapy, tPA,
surgery, amputation
Clinical Manifestation of Actue Arterial Ischemic Disorder: 6 Ps
Clinical manifestations (“6 Ps”):
- Pain,
- pallor,
-pulselessness,
-paresthesia,
- paralysis, and
- perishingly cold
Case Study: Leo Ducharme – a 76 year old man who was admitted to the hospital with rest pain in both legs and a non-healing ulcer on the big toe of his right foot.
PMHX: MI, stroke, hypertension, arthritis and type 1 Diabetes
Underwent left fem-pop bypass 5 years ago
45 year hx of tobacco use
Been using insulin for 30 years
Complains of sudden, intense increase in right foot pain for past 2 hours
Current Meds
Furosemide – 40mg/day PO
Enalapril 5mg/day PO
Insulin R with meals
Lantus insulin 50 units/day subQ
Diltiazem 240mg/day PO
ASA 325 mg/Day PO
Fish oil (self prescribed)
Objective Data
BP 148/92, irregular HR, 90 beats/min, RR – 22/min, temp 36.6 oral
Alert and oriented but anxious, no mental of physical deficits from previous stroke
Decreased right fem pulse, popliteal pulse detected with Doppler only, posterior tibial pulse with Doppler only, absent dorsalis pedis pulse
Left leg pulses weakly palpable
2cm necrotic ulcer on tip of right big toe
Thickened toenails, shiny taught skin on both legs, hair absent on both lower legs
Right foot pale, very cool, mottled in colour and decreased sensation
No peripheral edema
BS glucose 16mmol/L
What are Mr. Ducharme’s risk factors for PAD?
Does Mr. Ducharme have chronic PAD or acute arterial ischemia? What’s the difference?
What could be the cause of the sudden, intense increasing pain in his right foot?
What are Mr. Ducharme’s risk factors for PAD?
Does Mr. Ducharme have chronic PAD or acute arterial ischemia? What’s the difference?
What could be the cause of the sudden, intense increasing pain in his right foot?
What are Mr. Ducharme’s risk factors for PAD?
Hypertension, Smoking, Diabetes, Age
Does Mr. Ducharme have chronic PAD or acute arterial ischemia? What’s the difference?
Acute arterial ischemia
What could be the cause of the sudden, intense increasing pain in his right foot?
Caused by an emoblism because of his history of heart conditions
Nursing Diagnosis for PAD what are some interventions.
- Ineffective Tissue Perfusion.
- Impaired Skin integrity.
- Acute Pain
- Activity Intolerance
- Ineffective Therapeutic Regimen Mgmt.
- Ineffective Tissue Perfusion.
Antiplatelets
walking
- Impaired Skin Integrity
Protect feet, Should not wear tight shoes, keep warm, pt have loss of feeling on feet
- Acute Pain
NSAIDS, anything that increase circulation can help, elevating head of bed
- Activity Intolerance
Encourage gradual increase in activity levels as tolerated.
Plan rest periods to prevent fatigue.
Educate the patient on energy conservation techniques.
- Ineffective Therapeutic Regimen Management
Educate the patient and family about the importance of medication adherence.
Provide information on lifestyle changes, such as smoking cessation and healthy diet.
Ensure the patient understands their treatment plan and follow-up care.
Peripheral Venous Disorder: Venous Thrombus
Venous Thrombus:
- Formation of blood clot
Why blood clot forms?
- Virchow’s triad
- Stasis
- Hypercoagulability
- Endothelial damage
As thrombus can be enlarged-> detached -> embolus
Peripheral Venous Disorder: Superficial vein thrombosis
Where and Manifestation
- Where?:
Upper extremities (ie. IV therapy)
Lower extremities (ie. trauma to varicose vein) - Manifestation:
palpable, firm, subcutaneous cordlike vein with the surrounding area tender, reddened and warm
Peripheral Venous Disorder: Superficial vein thrombosis
Intervention
- Remove IV
- Elevation of the affected limb
- Warm & moist heat ( warm compress)
- Compression stocking
- Analgesics
- NSAIDs
Peripheral Venous Disorder—Deep Vein Thrombosis (DVT)
AKA Venous Thrombo-Embolism (VTE)
Where?
Blood clot form in deep vein
DVT (Deep Vein Thrombosis) is a condition where a blood clot forms in a deep vein, usually in the legs, causing swelling, pain, and warmth. If the clot travels to the lungs, it can cause a potentially life-threatening pulmonary embolism. Prompt treatment is essential.