Valvular Heart Disease 2 Flashcards
aortic stenosis
- what is the most common etiology?
- which valves are most affected in pts 65?
- What is likely etiology if pt is <30?
- What is another common cause of aortic stenosis?
-Three etiologies: calcific degeneration (most common) -bicuspid aortic in 65 congenital-if pt is <30 rheumatic heart disease
Physiologically what is happening in aortic stenosis
aortic valve is much narrower, and blood cannot pass through as well… leading to a huge increase in LV systolic pressure (200s) and much less aortic pressure
5 basic diagnostic tools for valvular disease?
- PEx
- CXR
- EKG
- Echocardiogram
- cardiac cath
Aortic Stenosis
- physical exam findings?
- where can you hear the abnl, where does it radiate?
- What kind of murmur?
- pulse abnormalities?
- any split abnormalities?
- systained LV impulse; little LV displacement
- Pulsus Parvus et Tardus (AKA weak carotid pulses)
- absent A2 or paradoxical A2 split
- murmur is systolic with crescendo and decresendo
- heart at base of heart (R upper sternal border) and radiates to carotid
CXR findings of aortic stenosis
- slight LVH
- post stenotic dilation
- calcification of aortic valve.
EKG findings of aortic stenosis
Echo findings
Cath findings
- due to LVH, the leads pointing to LV will be much more in amplitude
- Thickened LV
- increased LV pressure as compared to the aortic pressure
Criteria for severe stenosis? (4)
Jet velocity >4
mean gradient >40
valve area <0.6
Aortic stenosis. When should you intervene?
-Options for intervention?
When pt has sx.
w/o intervention a pt with angina will survive 5 years, syncope 3 years, and CHF 2 years
-surgical replacement; percutaneous replacement
Class I requirements for aortic stenosis indicating the need to replace AV.
- Severe AS
- Severe AS pt undergoing CABG
- Severe AS pt undergoing other valvular surgery
- severe AS pt with significant L ventricular dysfunction
Aortic regurgitation
-2 general causes; 7 specific causes
- leaflet problems
- congenital problems
- endocarditis
- rheumatic heart disease - arotic root problems
- aneurysm
- dissection
- annuloaortic ectasia
- syphilis
Pathophysiology of aortic regurgitation
-what is the main problem?
- aortic valve is incompetent.
- results in volume overload of the L ventricle
acute aortic regurg
- size and compliance of LV?
- diastolic pressure?
- What is a serious complication?
aortic regurgitation is AN EMERGENCY
The size of the LV is normal and compliance is low
-Diastolic pressure increases quickly
-this could cause pulmonary edema and congestion
Chronic aortic regurgitation
- what happens to L ventricle size and compliance?
- What happens to L atrium and pulmonary vasculature?
chronically the LV will dilate and compliance will increase
L atrium and pulmonary vasculature will have LESS pressure because of compensatory actions of LV
aortic regurgitation
-sx presentation (4)
- dyspnea on exertion
- fatigue
- decreased exercise tolerance
- CP
-without surgery pt with angina will die in 4 years; pt with heart failure will die in 2
Aortic regurgitation
-Physical signs (4)
- Hyperdynamic pulses, head bobbing (Corrigan’s pulse in carotid) , water hammer pulse, “quincke’s pulse”, Duroziez murmur
- Widened pulse pressure =diastolic is less than 1/3 of systolic
- decrescendo diastolic murmur that is worsened with increased systemic pressure.
- Austin Flint murmur-diastolic rumble