valvular disorder part II Flashcards

1
Q

cardiac remodeling in mitral stenosis

A
  • reduced filling of LV = low LVEDP and LV volume

- RA and RA dilation impairs LV ejection

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2
Q

values for mild, moderate, server mitral stenosis

A

mild > 1.5 cm2
mod 1-1.5 cm2
severe > 1 cm2

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3
Q

Goal directed intraoperative management of mitral stenosis (LV preload, Rate, contractility, SVR, PVR)

A
LV preload: high 
rate: low, sinus
contractillity: even
SVR: even
PVR: low
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4
Q

why is it important to keep LV preload high in mitral stenosis

A

adequate preload is necessary for forward flow of VS existing LAP and danger of pulmonary edema

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5
Q

Things to avoid intraoperatively with mitral stenosis

A

hypoxia, hypercapnea, tachycardia

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6
Q

what should you do if an arrythmia develops in a pt with mitral stenosis

A

cardiovert if necessary

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7
Q

LV filling takes place during _____

A

diastole

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8
Q

PA monitoring for a patient with mitral stenosis?

A

PA cath with caution

increased risk of rupture of PA d/t thin walled pulmonary arteries

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9
Q

in patients with mitral stenosis seperation from bypass may reveal _____ how is treated

A

may reveal underlying RV and LV dysfunction treat with volume and inotropes

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10
Q

prophylactic treatment prior to maze procedure

A

amiodarone

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11
Q

causes of mitral regurgitation

A

mitral valve leaflet abnormalities, mitral annulus dilation, disproportionate LV elargement

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12
Q

etiology of acute MR

A
  • papillary muscle dysfunction or rupture due to ischemia
  • blunt chest trauma
  • infective pericarditis
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13
Q

etiology of chronic MR

A
  • mitral annulus dilation from ishcemic cardiomyopathy or AI
  • mitral leaflet disorders
  • disorders of subvalvular apparatus (chordae tendenae)
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14
Q

pathophysiology acute MR

A
  • marked left atrial overload
  • pulmonary congestion
  • sympathetic activation, tachycardia, incrased SVR, increased contractility, worsening ischemia, biventricular failure
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15
Q

pathophysiology of chronic MR

A
  • LAE, eccentric hypertrophy from relative increased volume preserves forward flow by increased SV
  • LAE frequently causes a-fib, loss of volume
  • worsening of disease causes pumonary congestion, increase PAP, RV failure eventually
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16
Q

remodeling in MR

A

acute: LVEDP rapidly increases and dilates left atrium to maintain stroke volume
Chronic: dilation occurs slowly, eccentric hypertrophy occurs. maintain forward flow by increaseing overall SV in the presence of a regurgitant percentage

17
Q

severity of regurg (mild, mod, severe)

A

mild 40%

18
Q

goal directed intraoperative management of MR

LV preload, rate, contracility, SVR, PVR

A
LV preload: high or low 
Rate: even or high
Contractility: even
SVR: low
PVR: low
19
Q

LV preload in MR

A

maintenance of preload frequently helps to maintain forward flow. however this depends on the patient. dilation of the LA in some folks also dialtes the annulus and actually increases regurg. must base decisions on patient response to a fluid challenge

20
Q

anesthetic managmenet

A
  • light premed
  • induction: peripheral vasodilation, good contractility, HR 90
  • PA
  • TEE
  • nitric oxide - pulm vasodilator
  • hyperventilation will decrease PA pressures
  • once new valve in place LV may have a hard time ejecting a full SV on separation from bypass
21
Q

problem with inadequate anesthesia for intuabation in MR

A

may cause sudden sympathetic outflow increase regur and cause pulm edema