Upper GI Surgery Flashcards

1
Q

where are the three areas of constriction of the oesophagus?

A

level of cricoid
posterior to left main bronchus and aortic arch
LOS

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2
Q

what is the oesophagus lined with?

A

non keratinising squamous epithelium

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3
Q

state some inflammatory causes of dysphagia?

A
tonsilitis 
oesophagitis 
GORD 
oral candidiasis 
aphthous ulcers
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4
Q

state some neurological/motility causes of dysphagia?

A
achalasia 
diffuse oesophageal spasm 
nutcracker oseophagus 
bulbar/pseudobulbar palsy 
systemic sclerosis 
MG
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5
Q

state some obstructive causes of dysphagia?

A
foreign body 
benign stricture - web (plummer vision), oesophagitis, trauma) 
malignant stricture 
pharyngeal pouch 
retrosternal goitre 
rolling hiatus hernia 
lung cancer 
mediastinal LNs 
thoracic AA
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6
Q

What surgery can be done for achalasia ?

A

Heller’s cardiomyotomy

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7
Q

what is the area of weakness in the inferior pharyngeal constrictor that causes pharyngeal pouch?

A

Killian dehiscence

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8
Q

what is the presentation of pharyngeal pouch?

A

Regurgitation, halitosis, gurgling sounds

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9
Q

what does diffuse oesophageal spasm look like on barium swallow ?

A

corkscrew oesophagus

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10
Q

what is the presentation of nutcracker oesophagus ?

A

Intermittent dysphagia ± chest pain

􏰀 ↑ contraction pressure ̄c normal peristalsis

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11
Q

is plummer vision syndrome pre malignant?

A

yes

20% risk of SCC

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12
Q

what causes plummer vision syndrome ?

A

iron deficiency anaemia

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13
Q

what oesophageal feature is seen in plummer vision syndrome?

A

pharyngeal web from hyperkeratinisation of upper 3rd of oesophagus

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14
Q

what are two syndromes caused by violent emesis ?

A

Boerhaave’s syndrome

Mallory Weiss syndrome

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15
Q

what is the difference between Boerhaave’s syndrome and Mallory Weiss syndrome ?

A

Boerhaave syndrome is a transmural perforation of the esophagus to be distinguished from Mallory-Weiss syndrome, a nontransmural esophageal tear also associated with vomiting.

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16
Q

what is the features of oesophageal rupture ?

A

Odonophagia
􏰀 Mediastinitis: tachypnoea, dyspnoea, fever, shock
􏰀 Surgical emphysema

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17
Q

what is the treatment of oesophageal rupture?

A

PPI, NGT, Abx

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18
Q

what is the commonest cause of oesophageal rupture?

A

iatrogenic - endoscopy, biopsy, dilation

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19
Q

state a secondary cause of achalasia ?

A

Chages disease

- caused by T cruzii parasite

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20
Q

what medication can increase risk of GORD?

A

anti-AChM, nitrates, CCB, TCAs

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21
Q

what surgical procedure can increase risk of GORD?

A

hellers myotomy

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22
Q

what are the epithelial changes seen in barretts oesophagus?

A

Metaplasia → dysplasia → adenocarcinoma

23
Q

what are extra oesophageal features of GORD?

A

Nocturnal asthma
􏰀 Chronic cough
􏰀 Laryngitis, sinusits

24
Q

what are DD for GORD?

A

oesophagitis
peptic ulcer disease
oesophageal cancer

25
what PPI should be used for GORD and for how long ?
Lansoprazole 30mg OD | 1-2months
26
what is Ranitidine and whats it used for?
H2RA | - used for GORD
27
what three criteria has to be met for nissen fundoplication for GORDD?
Severe symptoms 􏰁 Refractory to medical therapy 􏰁 Confirmed reflux (pH monitoring)
28
state some complications of fundoplication?
􏰁 Gas-bloat syn.: inability to belch / vomit 􏰁 | Dysphagia if wrap too tight
29
what is a sliding hiatus hernia associated with
GORD
30
is a sliding or paraoesophagheal hiatus hernia more common?
sliding (80%)
31
what hiatus hernia requires surgery and why?
paraoesophageal because it may cause strangulation and ischaemia
32
what makes duodenal ulcers worse and better?
worse before meals and at night | better by eating
33
what makes gastric ulcers worse and better?
worse on eating (which can result in weight loss) | relieved by antacids
34
state some risk factors for peptic ulcer disease ?
``` H pylori NSAIDs, Steroids Smoking EtOH Stress ```
35
what causes cushings ulcer?
gastric ulcer associated with elevated intracranial pressure.
36
what causes curling ulcers ?
complication from severe burns when reduced plasma volume leads to ischemia and cell necrosis
37
what do peptic ulcers look like?
punched out ulcers
38
are duodenal ulcers or gastric ulcers more common?
duodenal ulcers are more common
39
what tests can be done for peptic ulcers ?
FBC, urea C13 urea breath test CLO / urease test for H. pylori Gastrin levels if Zollinger-Ellison suspected
40
what is gastric acid stimulated by?
gastrin, histamine and ACh from vagus nerve increases acid production
41
state some features of dumping syndrome ?
Abdo distension, flushing, n/v, fainting, sweating 􏰁 Early: osmotic hypovolaemia 􏰁 Late: reactive hypoglycaemia
42
decreased activity of parietal cells will result in what deficiency ?
B 12
43
what is blind loop syndrome ?
associated with bacterial overgrowth in limb of intestine excluded from flow after a subtotal gastrectomy which causes malabsorption and diarrhoea
44
what is Mg trisilicate used for?
antacid
45
what is the management of upper GI bleeds ?
Beta blockers endoscopic banding TIPSS (Transjugular Intrahepatic PortoSystemic Shunt)
46
what is a DD of a perforated peptic ulcer?
pancreatitis acute cholecystitis AAA MI
47
what is Chailaditi’s sign?
when loop of large intestine (usually transverse colon) in between the diaphragm and the liver
48
what should be seen on a CXR with a ulcer perforation ?
Air under the diaphragm seen in 70%
49
state the presentation of gastric outlet obstruction?
Copious projectile, non-bilious vomiting a few hrs after meals. 􏰁 Contains stale food. 􏰁 Epigastric distension + succussion splash
50
what does a AXR show for gastric outlet obstruction?
Dilated gastric air bubble, air fluid level | 􏰁Collapsed distal bowel
51
what surgical interventions can be done for gastric outlet obstruction?
Endoscopic balloon dilatation 􏰁Pyloroplasty Stenting
52
what gender is most affected by hypertrophic pyloric stenosis ?
males
53
what is the presentation of hypertrophic pyloric stenosis ?
Projectile vomiting minutes after feeding 􏰀 RUQ mass: olive 􏰀 Visible peristalsis
54
what are the 5 criteria for bariatric surgery?
BMI ≥40 or ≥35 ̄c significant co-morbidities that could improve ̄c ↓ wt. 􏰁 Failure of non-surgical Mx to achieve and maintain clinically beneficial wt. loss for 6mo. 􏰁 Fit for surgery and anaesthesia 􏰁 Integrated program providing guidance on diet, physical activity, psychosocial concerns and lifelong medical monitoring 􏰁 Well-informed and motivated pt.