ulcers Flashcards

1
Q

this leads to distortion/damage to blood vessels, usually at a deep tissue level

A

Shear force

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2
Q

this is a risk factor for pressure injury

A

low diastolic BP - takes less pressure in order to impede circulation

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3
Q

patient should turn how often when lying down?

A

2 hours

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4
Q

patient should shift how often when sitting up

A

15 minutes

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5
Q

this stage of pressure injury is not yet an open wound, and has nonblanchable erythema & may be “boggy”

A

stage 1

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6
Q

This stage of pressure ulcer are superficial - partial thickness. include epidermis, sometimes part of dermis and includes blisters

A

stage II

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7
Q

these are deep ulcers, with extensive necrosis. they are full thickness with dermis and subcutaneous tissue involved - extend to BUT not through subctuaneous fascia

A

stage III

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8
Q

full thickness ulcer. deep ulceration, may have undermining

involves epidermis, dermis, subcutaneous tissue through fascia to muscle, tendon, joint capsule, and sometimes bone

A

stage IV

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9
Q

this pressure ulcer has high risk of osteomyelitis

A

stage IV

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10
Q

Can a stage IV ulcer reverse to a stage II ulcer?

A

NO no reversal omg

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11
Q

this scale is rated 0 to 7 based on observation of ulcer

A

sessing scale

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12
Q

this allows clinician to chart progress by subtracting score at reassessment from score initial evaluation

A

sessing scale

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13
Q

this scale ranges from score 8 to 34

A

PUSH - pressure ulcer scale for healing

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14
Q

this type of debridement method removes nectrotic tissue only, leaves uninvolved skin intact

A

selective

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15
Q

this debridement method goes from wet to dry

A

non-selective

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16
Q

this method of debridement uses semipermeable films, and absorbent dressings

A

autolytic debridement

17
Q

this type of debridement can use sharp of hydrotherapy

A

mechanical debridement

18
Q

what to diabetic ulcers stem from

A

peripheral neuropathy (damage to sensory nerves, autonomic nerves, motor nerves)

19
Q

this is the primary risk factor for development of diabetic foot ulcers

20
Q

these count for 50-70% of all non-traumatic amputations in the US

A

Diabetic foot ulcers

21
Q

this can lead to increased levels of intracellular glucose in nerves & disruptions in cellular metabolism with the neurons

A

hyperglycemia

22
Q

this is decreased perspiration and sebaceous secreations in the distal lower extremities and feet

A

autonomic neuropathy

23
Q

with this the skin is prone to dryness, cracks, callus formation and breakdown

A

autonomic neuropathy

24
Q

this affects intrinsic muscles of foot

A

motor neuropathy

25
atrophy will reduce what?
padding to pressure points
26
these are common foot deformities in DPN
plantarflexion contracture claw-toe deromity hallux varus forefoot varus or valgus
27
this is referred to as rocker bottom foot
charcot foot
28
this is a bone deterioration and pathologic fractures which leads to deformity
charcot foot
29
this will be involved in your circulatory assessment
peripheral pulses, capillary refill, skin color and temperature, ABI
30
what is an important thing to do during skin assessment?
indentify areas of callus as potential sites of breakdown, look for cracks and fissures
31
risk factors for ulcers
numbness, paresthesias. previous h/o ulcer, amputation, high scores on screen
32
these can be preventative tools for neuropathic ulcers
orthotics, proper footwear, daily inspection of shoes and feet, exercise
33
these are commonly located at plantar aspect of foot (midfoot, heel, metatarsal heads)
diabetic ulcers
34
these wound margins often show callus formation
diabetic ulcers
35
with total contact casting how often should cast be changes to avoid ulcer
at least ones a week - every 2 weeks at maximum
36
this can be injected under high pressure areas
liquid salicone
37
this may help replace fat padding that is displaces by bony deformatities
liquid silicone