Txt Of Agd (d) Flashcards by Love Dog

Q

_____ is the lodgment and multiplication of a parasite in or
on the tissue of a host; does not invariably result in disease

A

infection

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Q

T/f

the inflammatory response initiated in gingival disease appears to be prerequisite for destruction of connective tissue attachment apical to CEJ

A

T

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Q

a microbial disease of the gingiva that most often occurs in an
impaired host

→necrotizing ulcerative gingivitis
→primary herpetic gingivostomatitis
→pericoronitis
→abscesses in the periodontium

A

necrotizing ulcerative gingivitis

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Q

What are the most severe inflammatory
periodontal disorders caused by plaque bacteria?

A

necrotizing ulcerative gingivitis (NUG), necrotizing periodontitis (NP), necrotizing stomatitis (NS)

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Q

recognized by Xenophon who stated that Greek soldiers were plagued with sore, ulcerated and foul-smelling mouths

A. 1st Event (4th B.C.)
B. 2nd Event ( 1778)
C. 3rd Event (1886)
D. 4th Event ( 1890s)
E. 5th Event (19th century)
F. 6th Event ( 1999 American Academy of Periodontics
Classification System)

A

A

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Q

John Hunter first delineated the clinical differences between NUG, scurvy, and chronic periodontitis

A. 1st Event (4th B.C.)
B. 2nd Event ( 1778)
C. 3rd Event (1886)
D. 4th Event ( 1890s)
E. 5th Event (19th century)
F. 6th Event ( 1999 American Academy of Periodontics
Classification System)

A

B

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Q

Hersch described that increased
salivation, enlarged lymph nodes, fever and malaise had been associated with
NUG

A. 1st Event (4th B.C.)
B. 2nd Event ( 1778)
C. 3rd Event (1886)
D. 4th Event ( 1890s)
E. 5th Event (19th century)
F. 6th Event ( 1999 American Academy of Periodontics
Classification System)

A

C

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Q

Plaut and Vincent described the disease and attributed its origin to fusiform bacilli
and spirochetes

A. 1st Event (4th B.C.)
B. 2nd Event ( 1778)
C. 3rd Event (1886)
D. 4th Event ( 1890s)
E. 5th Event (19th century)
F. 6th Event ( 1999 American Academy of Periodontics
Classification System)

A

D

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Q

NUG occurred as an epidemic

A. 1st Event (4th B.C.)
B. 2nd Event ( 1778)
C. 3rd Event (1886)
D. 4th Event ( 1890s)
E. 5th Event (19th century)
F. 6th Event ( 1999 American Academy of Periodontics
Classification System)

A

E

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Q

Acute Necrotizing Ulcerative Gingivitis (ANUG), now classified under Necrotizing
Periodontal Disease

A. 1st Event (4th B.C.)
B. 2nd Event ( 1778)
C. 3rd Event (1886)
D. 4th Event ( 1890s)
E. 5th Event (19th century)
F. 6th Event ( 1999 American Academy of Periodontics
Classification System)

A

F

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Q

ACCORDING TO PINDBORG

Erosion of only tip of interdental papilla

Stage?

A

1

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Q

ACCORDING TO PINDBORG

Lesion extending to marginal gingiva and causing
potentially a complete loss of papilla

Stage?

A

2

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Q

ACCORDING TO PINDBORG

Involving attached gingiva

Stage?

A

3

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Q

ACCORDING TO PINDBORG

Exposure of bone

Stage?

A

4

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Q

ACCORDING TO HORNING AND COHEN

Necrosis of tip of the interdental papilla

Stage?

A

1

93%

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Q

ACCORDING TO HORNING AND COHEN

Necrosis of entire papilla

Stage?

A

2

(19%)

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Q

ACCORDING TO HORNING AND COHEN

Necrosis extending to gingival margin

Stage?

A

3

21%

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Q

ACCORDING TO HORNING AND COHEN

Necrosis extending to attached gingiva

Stage?

A

4

1%

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Q

ACCORDING TO HORNING AND COHEN

Necrosis extending to buccal/labial mucosa

Stage?

A

5

6%

Q

ACCORDING TO HORNING AND COHEN

Necrosis perforating skin and cheek

Stage?

A

6

0%

Q

→ punched out appearance of papillae
→ gray pseudomembranous slough

A. necrotizing ulcerative gingivitis
B. primary herpetic gingivostomatitis
C. pericoronitis
D. abscesses in the periodontium

A

A

Q

pronounced foetor ex ore and metallic taste

A. necrotizing ulcerative gingivitis
B. primary herpetic gingivostomatitis
C. pericoronitis
D. abscesses in the periodontium

A

A

Q

→direct contact may cause lip and tongue ulcers
→pharyngeal involvement (Vincent’s angina)
→seasonal variations

A. necrotizing ulcerative gingivitis
B. primary herpetic gingivostomatitis
C. pericoronitis
D. abscesses in the periodontium

A

A

Q

Vincent’s angina

A. necrotizing ulcerative gingivitis
B. primary herpetic gingivostomatitis
C. pericoronitis
D. abscesses in the periodontium

A

A

BACTERIA in NUG

Prevotella Intermedia, Fusobacteria, Treponema, Selenomonas

the prevalent opinion is that it is produced by a complex of bacterial organisms but requires underlying tissue changes to facilitate the pathogenic activity of bacteria A. necrotizing ulcerative gingivitis B. primary herpetic gingivostomatitis C. pericoronitis D. abscesses in the periodontium

A

Plaut and Vincent in 1894 and 1896, respectively introduced the concept NUG is caused by specific bacteria–namely a _____ and a spirochetal organism

fusiform bacillus

Fusospirochetal organisms, P. intermedia, A. odontolyticus and various spirilla like Selenomonas species A. Constant Flora B. Variable Flora

A

heterogenous array of bacterial types A. Constant Flora B. Variable Flora

B

immunosuppression essential o NUG patients displayed depression in leukocyte chemotaxis and ____

phagocytosis

“fetor oris”

NECROTIZING ULCERATIVE GINGIVITI

T/f Littner MM in 1982 studied that bacterial smear shows a preponderance of streptococcal forms, which were identified as streptococcus viridans, and he reported it to be group A beta hemolytic streptococcus.

T

What visit → the pseudomembrane and debris are removed with a moist cotton pellet where the lesion is isolated with a cotton swab and local anesthetic may be used → ultrasonic scaling is done to remove the supragingival calculus

1st visit

What visit antibiotic – oral penicillin demonstrated in one study to show significant clinical improvement in three to six days → patient is also advised to avoid tobacco, alcohol and condiments

1st visit

What visit shrinkage of gingiva → expose calculus which is then gently removed

2nd visit

What visit discontinue hydrogen peroxide mouthwash but continue CHX mouthwash

3rd visit

What visit patient should now be scheduled for treatment of chronic disease

SUBSEQUENT VISITS

What visit appointments are scheduled for the treatment of the gingivitis, periodontal pockets, and peri-coronal flaps as well as for elimination of all forms of local irritation

SUBSEQUENT VISITS

What vist → the third molar flaps and other nidi of infection are eliminated → gingivoplasty and osseous surgery is done if the lost gingival architecture is not regained after nonsurgical therapy

SUBSEQUENT VISITS

occurs commonly in children and infants younger than 6 years of age, but is also seen in adolescents and adults → virus ascends through the sensory and autonomic nerves where it persists as latent A. necrotizing ulcerative gingivitis B. primary herpetic gingivostomatitis C. pericoronitis D. abscesses in the periodontium

B

primary infection is asymptomatic A. necrotizing ulcerative gingivitis B. primary herpetic gingivostomatitis C. pericoronitis D. abscesses in the periodontium

B

sites are sensitive to touch, thermal changes, foods such as condiments and fruit juices and action of coarse foods A. necrotizing ulcerative gingivitis B. primary herpetic gingivostomatitis C. pericoronitis D. abscesses in the periodontium

B

diffuse erythema and vesicles ANUG PHGS

PHGS

gingiva, buccal mucosa, lips ANUG PHGS

PHGS

7-10 days immunity seen ANUG PHGS

PHGS

inflammation of the gingiva in relation to the crown of an incompletely erupted tooth → occurs most commonly in mandibular third molar area

PERICORONITIS

clinically, it is seen as a markedly red, swollen, suppurating lesion that is exquisitely tender, with radiating pain to the ear, throat and floor of the mouth

PERICORONITIS