Twin and Adoption Studies Flashcards

- twin studies - adoption studies - Brendgen et al - Heston

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1
Q

Strengths of twin studies

A
  • Useful as there is no other method to show genetic influence so clearly
  • High in validity - natural control over environmental effects
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2
Q

Weaknesses of twin studies

A
  • Lack generalisability - volunteer sampling
  • Validity issues - overemphasise role of genes (MZ twins more similar so are treated the same - greater environmental similarity)
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3
Q

Strength of adoption studies

A
  • Useful as useful way of separating genes from the environment
  • Allow trend in behaviour to be studied - use longitudinal method
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4
Q

Weaknesses of adoption studies

A
  • Overemphasise role of genes (children put in families similar to their own)
  • Lack generalisability (only certain families are allowed to adopt
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5
Q

Heston (1966) - Aim

A

To see how many adopted children of biological mothers with schizophrenia would go on to develop schizophrenia themselves

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6
Q

Heston (1966) - Method

A
  • Ppts asked a personal interview
  • Standardised - structured interview
  • General medical and environmental questionnaire which explored psychological dimensions
  • Gathered blindly by 2 independent psychiatrist
  • Final evaluation by 3rd researcher
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7
Q

Heston (1966) - Results

A
  • 5/47 developed schizophrenia (so 10% figure achieved)

- 0/50 of control group (adoption not a factor)

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8
Q

Heston (1966) - Conclusion

A

Results provided powerful evidence for the role of genes in schizophrenia
- No evidence for environmental factors

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9
Q

Heston (1966) - Strengths

A

+ High internal validity (inter-rater reliability)
+ High reliability (structured interview)
+ Application (if you know it can be inherited you can look for signs earlier)

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10
Q

Heston (1966) - Weakness

A
  • Low validity (less experimental subjects than control)

- Low validity (not scientifically credible - in 1966 ways of testing for schizophrenia weren’t as good)

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11
Q

Brendgen et al (2005) - Aims

A
  • see if social aggression could be caused by genes of the environment
  • see if social aggression shared the same cause as physical aggression
  • see if one type of aggression leads to another type
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12
Q

Brendgen et al (2005) - Sample

MZ & DZ

A

247 twin pairs from the Quebec Newborn Twin Study (QNTS)

  • 44 MZ males & 50 MZ females
  • 41 DZ males & 32 MZ females
  • 67 mixed set DZ twins
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13
Q

Brendgen et al (2005) - Method

A
  • Data gathered longitudinally at 5, 18, 30, 48, 60 months and again at 6 years
  • Two ratings from the teacher and from classmates
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14
Q

Brendgen et al (2005) - Teacher ratings

A
  • Rated social and physical aggression on a 3 point scale
  • e.g. “to what extent does the child spread nasty rumours about others” // “to what extent does the child get into fights”
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15
Q

Brendgen et al (2005) - Peer ratings

A
  • Research assistant made sure that each child could be recognised
  • Given a booklet with pictures of classmates
  • Asked to nominate 3 people for each description of a behaviour on each page
  • e.g. “tells others not to play with a child” // “hits,bites and kicks others”
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16
Q

Brendgen et al (2005) - Conclusion

A
  • strong genetic component to physical aggression but not social
  • children who were physically aggressive were more likely to be socially aggressive
  • As children grow they tend to be more socially aggressive because of social conventions on physical violence
17
Q

Brendgen et al (2005) - Strengths

A
  • High validity - natural control over environmental factors (teacher given time to get to know pupils)
  • High reliability - standardised procedure (questions and 3 point scale
  • Application - if we know aggressive behaviour is innate we can put more emphasis on positive role models
  • Ethics - parents fully informed
18
Q

Brendgen et al (2005) - Weaknesses

A
  • Low Generalisablility - enthnocentric and all 6 years old
  • ## Low validity - children asked to rate peer - could have chose children they disliked