Treatment of Erosion, Abrasion & Attrition Flashcards

1
Q

Tooth Wear
Ñ Tooth wear is the general term used for the surface loss of

A

dental hard tissues from causes other than developmental ones,
dental caries, and trauma.

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2
Q

Ñ Lambrechts et al. in 1989 estimated the normal vertical loss of
enamel resulting from natural wear is about – μm per year

A

65

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3
Q

Classification of tooth wear
(4)

A

Ñ Attrition
Ñ Abrasion
Ñ Abfraction
Ñ Erosion or corrosion

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4
Q

Attrition as defined in GPT 9 is the

A

mechanical wear resulting
from mastication or parafunction, limited to contacting surfaces
of the teeth

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5
Q

Attrition is related to the — process

A

aging

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6
Q

Attrition
Etiology is

A

multifactorial and is accelerated by extrinsic factors

such as coarse diet, chewing tobacco and snuff, abrasive dust,
parafunctional habits of clenching and bruxism, traumatic
occlusion in the partially edentulous dentition, anterior open
bite, and anterior teeth in edge-to-edge relationship or crossbite.

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7
Q

Consequences of loss of posterior support
Supraeruption was found in –% of unopposed teeth.

A

92

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8
Q

Consequences of loss of posterior support
Mean supraeruption measurement for unopposed teeth
was — mm

A

1.68

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9
Q

Consequences of loss of posterior support
Passive eruption:

A

After ten years of remaining
unopposed tooth, recession and root exposure occurred
without occlusal movement of the tooth as in case of
active eruption

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10
Q

Types of Supraeruption:
(3)

A

Relative wear
Periodontal growth
Active eruption

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11
Q

Relative wear
association with — and is
more prevalent in

A

increasing age
unopposed mandibular teeth

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12
Q

Periodontal growth

A

inverse association with attachment
loss. More prevalent in younger patients, in the maxilla, in
premolars, and in females

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13
Q

Active eruption

A

has an association with attachment loss.
Ó Active eruption increases, the presence of attachment
loss increases, and periodontal growth is reduced.
Ó Alters the C/R ratio, affecting the mechanical
characteristics of the tooth and its behavior under
functional loading

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14
Q

Abrasion as defined in GPT 9 as an

A

abnormal wearing away of
the tooth substance by some unusual or abnormal mechanical
process independent of mastication and occlusion.

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15
Q

Abrasion
Causative agent:

A

Foreign object or substance repeatedly
contacting the tooth surface

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16
Q

Abrasion
Site and pattern of the abrasive lesions:

A

Diagnostic as different
foreign bodies produce different patterns of abrasion

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17
Q

Abrasion
Example:

A

Overzealous horizontal tooth brushing with an
abrasive dentifrice produces a rounded or V-shaped ditch on the
facial aspects of teeth at the cemento-enamel junction

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18
Q

Clinical presentation of
Abrasion
Most commonly affected teeth:

A

Canines and premolar

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19
Q

Clinical presentation of
Abrasion
Causative agents:
(5)

A

1) Improper use of dental floss,
2) chewing tobacco;
3) Biting on hard objects such as
pens, pencils or pipe stems;
opening hair pins with teeth; and
biting fingernails.
4) Abrasion also can be produced
by the clasps of partial dentures.
5) Occupational abrasion:

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20
Q

5) Occupational abrasion:
(3)

A

a) Tailors – sever thread with their
teeth,
b) Shoemakers and upholsterers -
hold nails between their teeth,
c) Glassblowers and musicians-play
wind instruments

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21
Q

Abfraction is defined in GPT 9 as the

A

the pathologic loss of hard tooth
substance caused by biomechanical
loading forces; which is the result of
flexure and chemical fatigue
degradation of enamel and/or dentin
at some location distant from the
actual point of loading

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22
Q

Abfraction
Causative agent:

A

Stress

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23
Q

Abfraction
Stresses that lead to abfractions are transmitted by occlusal loading forces
such as (3)

A

occlusal interferences,
premature contacts, habits of bruxism
and clenching

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24
Q

Erosion as defined in GPT 9 as the

A

progressive loss of tooth substance
by chemical processes that do not involve bacterial action, producing
defects that are wedge-shaped depressions often in occlusal, facial
and cervical areas

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25
Erosion Causative agent:
Acids from external and internal sources
26
Erosion Extrinsic factor: (4)
1) Acidic foods such as citrus fruit, pickle, vinegar (acetic acid), sucking lemons, fruit juice and carbonated drinks, yogurt, herbal tea, spicy food. 2) Medicines such as effervescent and chewable vitamin C preparations46 and hydrochloric acid for achlorhydria, 3) Occupational exposure to acid fumes (sulfuric, hydrochloric, nitric, and tartaric acids), 4) Swimming in acidic gas-chlorinated pools.
27
Erosion Surface involved:
Labial surface of maxillary teeth affected
28
Erosion Appearance:
Scooped-out depressions
29
Erosion Intrinsic factor: (3)
1) Anorexia and bulimia nervosa or regurgitation of gastric contents because of abnormalities in the gastrointestinal tract, pregnancy morning sickness, and chronic alcoholism. 2) Certain drugs may cause nausea or vomiting such as estrogens, opiates, tetracycline, levodopa, aminophylline, digitalis, and disulfiram, 3) Other factors such as reduced salivary secretion and calcium and phosphorus levels
30
Erosion Surface involved:
Palatal surface of the maxillary teeth affected
31
Erosion Appearance:
Concave depression involving the entire surface.
32
Clinical presentation of Erosion Palatal surface and occlusal surface of posterior teeth involved due to --- Labial surface involved due to ---
GERD consumption of acidic food
33
Attrition- Abfraction:
Is the joint action of stress and friction when teeth are in tooth-to-tooth contact, as in bruxism or repetitive clenching.
34
Abrasion- Abfraction:
Is the loss of tooth substance caused by friction from an external material on an area in which stress concentration due to loading forces may cause tooth substance to break away
35
--- is an Occlusal Parafunctional Habit
Bruxism
36
Bruxism is an Occlusal Parafunctional Habit Ñ May be:
Sleep bruxism or Awake bruxism
37
bruxism It is defined as the
grinding of teeth during non functional movements of the masticatory system: it is a mandibular parafunction
38
Mechanical wear resulting from bruxism often results in
progressively greater wear towards the anterior teeth (with open bite as exception)
39
Bruxism Two etiological factors are: Ø Structural: (2) Ø Functional: (2)
a. Occlusal interferences b. Altered maxillo mandibular relationships a. Stress b. Children Brux
40
Clinical presentations of Bruxism Intraoral findings: (8)
Ñ Grooving of lateral borders of tongue Ñ Cheek biting Ñ Fractured porcelain restorations Ñ Cupping or cratering of occlusal surface Ñ Teeth grinding or clenching Ñ Teeth are worn down, flattened or chipped Ñ Increased tooth sensitivity
41
Clinical presentations of Bruxism Extraoral findings: (4)
Ñ Jaw pain or tightness in jaw muscles Ñ Earache Ñ Dull morning headache Ñ Chronic facial pain
42
Prevention rather treatment of Bruxism (3)
Ñ There is no specific treatment available at this time to stop bruxism, so that the focus has been to reduce the adverse effects of the habit. Ñ The use of inter occlusal appliances is the most common and accepted way to prevent wear of teeth and prosthodontic restorations Ñ When prosthetic intervention is indicated in a patient with bruxism, efforts should be made to reduce the effects of heavy occlusal loading on all the components that contribute to prosthetic structural integrity
43
Amelogenesis imperfecta:
Hereditary defect of dental enamel
44
Amelogenesis imperfecta: Characterized by
early loss of enamel with concomitant and more rapid attrition of tooth structure.
45
AI Classified into three basic types:
1) Hypoplastic: 2) Hypomaturation: 3) Hypocalcified:
46
1) Hypoplastic:
The enamel has only one eighth to one fourth of the normal thickness.
47
2) Hypomaturation:
The enamel has normal thickness and is softer than normal and tends to fracture from the dentin.
48
3) Hypocalcified:
The enamel normal thickness but is extremely friable and frequently lost soon after tooth eruption
49
Dentinogenesis imperfecta or hereditary opalescent dentin:
Is a dominant autosomal trait with a high degree of penetrance and the enamel tends to chip away from underlying dentin at an early stage and the teeth show gross attrition associated with obliterated pulp chambers and short roots.
50
DI This anomaly may or may not be associated with the
generalized skeletal disease osteogenesis imperfecta
51
DI Characterized by
an amber-colored translucency of the dentition
52
DI Ñ Type 1: (6)
* No Family History * Obliterated pulps frequent * Periapical radiolucency's less frequent * Defect not pitted * Mild or non existent anterior open bite * Always associated with osteogenesis imperfecta
53
DI Ñ Type 2: (1)
* All symptoms similar as type I only difference is that its is not associated with osteogenesis imperfecta
54
DI Ñ Type 3: (6)
* Positive family history * Pulps are large * Periapical radiolucency's frequent * Defect is pitted * Significant open bite * Not associated with osteogenesis imperfecta
55
How to diagnose and treat these cases? (7)
Ñ History taking Ñ Clinical examination Ñ Radiographs Ñ Diagnostic casts mounted in MIP and CR Ñ Clinical intraoral photographs Ñ TMJ examination Ñ Salivary gland examination
56
Management of Localized active tooth wear: 5 important factors to be considered are:
1) Pattern of tooth wear and the surfaces involved 2) Available inter occlusal space 3) Available restorative space for dental restorations proposed 4) The quantity and quality of available hard tissue and enamel respectively 5) Esthetic demands of the patient
57
Consequences of Wear (3)
1) Loss of VDO 2) Compromised Esthetics 3) Occlusal discrepancies
58
Management of Generalized tooth wear: The restorative management of patients presenting with generalized tooth wear (TW) will be considered according to the three categories described by Turner and Missirilian; * Category 1 – * Category 2 – * Category 3 –
Excessive wear with loss of vertical dimension of occlusion Excessive wear without loss of vertical dimension, but with space available Excessive wear without loss of vertical dimension, but with limited space.
59
Ñ Wear of the antagonist enamel surface was as follows:
Stained and glazed Zirconia > Stained Zirconia> Polished Zirconia
60
Ñ Wear of the antagonist enamel was even more with
layered feldspathic porcelain. Glazed or unglazed causes similar amount of wear
61
Ñ Care has to be taken to polish the porcelain surface well after chairside adjustments to minimize the
roughness in turn minimize wear of the opposing tooth.
62
Ñ The depth of enamel wear caused by monolithic zirconia and composite resin was significantly --- than that caused by glass ceramic and enamel
lower
63
Ñ Composite resin restorations caused --- wear of the antagonist enamel as compared to ceramics
less
64
Ñ Wear depth of --- is the closest to that of the enamel.
gold
65
Ñ The milled and not glazed Zirconia reinforced lithium disilicate shows --- wear depth compared with type III gold and human enamel.
small but significantly increased
66
Ñ Wear depth and volumetric loss for the --- did not statistically differ in comparison with the human enamel.
glaze-fired Zirconia reinforced lithium disilicate and for the other ceramics like heat pressed, CAD/CAM Feldspathic
67
Ñ The wear properties of the new zirconia-reinforced lithium silicate ceramic is improved by a
glaze firing cycle