Trauma Overview Flashcards
- What is shock?
Inadequate tissue perfusion
Inadequate amounts of O2 and glucose are delivered to cell
Impairing amount of waste products in body
- What is aerobic vs. anaerobic metabolism?
Aerobic metabolism is metabolism (production of energy) using oxygen
Anaerobic metabolism is metabolism in the absence of oxygen
Not sustainable as it causes decrease in ATP and increase in acid; induces sodium/potassium pumps to fail which brings sodium and water into the cell causing it swell and burst
- What are the 3 basic etiologies of shock?
1) Inadequate volume (hypovolemia): decreased blood volume = decrease in preload, which causes stroke volume and cardiac output to fall. Decr. in cardiac output drops systolic blood pressure (which is responsible for transporting the oxygenated blood throughout the systemic circulation). Decr. in systolic BP=inadequate tissue perfusion.
a. Hemorrhage=Loss of blood. Needs increase in blood volume to restore adequate perfusion. Losing whole blood= Needs administration of packed red blood cells and plasma to incr. oxygen-carrying capability of blood and improve pressure and perfusion.
b. Non-hemorrhage or hypovolemic=Loss of plasma/fluids. Resulting from excessive urination/diarrhea/burns/vomiting. Needs to have the fluid or water portion of his blood restored.
2) Inadequate pump function: if the pump fails, regardless of blood volume, delivery of oxygen and glucose to cells will decr.
Pump function failure results from injury to the heart that reduces its ability to generate contractions strong enough to push the blood forward throughout the body. Ex. heart attack. Can also be caused by mechanical obstruction of movement of blood. Pt requires improvement of pump function to eliminate shock state.
3) Inadequate vessel tone: tone is related to the size of the vessel and the resistance created within it and is referred to as systemic vascular resistance/peripheral vascular resistance. As vessel size decr., resistance incr. result is an increase in BP, which is governed by both CO and SVR. Incr. vessel size=reduced pressure and reduced preload and cardiac ouput, decr. in BP and diminished tissue perfusion.
- What are the 5 categories of shock? Pathophysiology of each? Examples of each?
- Hypovolemic shock=shock caused from a low blood volume. The most common form of shock. Can be due to blood loss or loss of some other body fluid. Basically any condition or injury that results in a reduction in fluid decreases blood content or fluid portion.
a. Most common=hemorrhage=loss of whole blood (ex. laceration, bleeding from gastrointestinal tract)
b. Non-hemorrhagic=hypovolemia associated w/ fluid loss from burns and dehydration - Distributive shock = associated with a decrease in intravascular volume caused by massive systemic vasodilation and an increase in capillary permeability. No fluid loss or blood loss but rather a relative reduction in volume because vessels incr. in size. Decr. in tissue perfusion.
- Cardiogenic shock = ineffective pump function of the heart. Adequate blood volume and vessel tone, but hypoperfusion because the heart cannot contract effectively.
a. Left ventricle fails to generate enough force to eject sufficient blood from the chamber into the system circulation, reduction in stroke volume, cardiac output, and systolic blood pressure; poor tissue perfusion - Obstructive shock = condition that obstructs forward blood flow. Volume is adequate, heart is not damaged, and vessels are of a normal size with adequate resistance. An obstruction is not allowing the blood to move forward. Large clot that will prevent adequate amount of blood from getting to lungs (pulmonary embolism) will prevent an adequate amount of blood from getting to left atrium and left ventricle, reducing preload and stroke volume, cardiac output, systolic BP, and tissue perfusion.
- Metabolic or respiratory shock: dysfunction in ability of oxygen to diffuse into blood, be carried by hemoglobin, off-load at the cell, or be used effectively by the cell for metabolism.
a. Ex. poison like cyanide, CO poisoning, etc.
- What is MODS? When does it occur? How might it be related to disseminated intravascular coagulation (DIC)?
MODS - Multiple organ dysfunction syndrome
Occurs - When multiple organs begin to fail throughout the body from extreme and prolonged hypoxia, altered metabolism, and evaluated carbon dioxide and acid levels.
Relation between MODS & DIC - MODS is not a single event but a continuous process. One such event that might occur is the production of microemboli that begin to block capillaries throughout the body, leading to lung failure, kidney failure, and multiple organ failures. Clotting factors are used in the formation of the microemboli in the blood. The body responds to the clots by releasing substances to attempt to break them up. Because the clotting factors were used up in the formation of the microemboli, the substances that are released to break down the clots are unopposed in the body and lead to widespread uncontrolled bleeding from any wound that was previously clotted, from intravenous catheter sites, from mucus membranes, and from the skin.
- Name three examples of obstructive shock, and explain the etiology of each.
Tension Pneumothorax - same as Pericardial tamponade → see below
Pulmonary embolism - A large clot that obstructs blood flow in the lungs will prevent an adequate amount of blood from getting to the left atrium and, subsequently, the left ventricle. This reduces the preload and decreases the stroke volume, cardiac output, systolic bp, and tissue perfusion.
Pericardial tamponade/Tension Pneumothorax - Prevent adequate ventricular filling and compress the heart. This reduces the preload, stroke volume, cardiac output, systolic BP, and tissue perfusion. Once the pressure is relieved, the heart regains its normal function. Although the pump function of the heart is disturbed, once the condition is reversed and the pressure on the heart is relieved, it functions normally.
- What are the s/s of the following types of shock: hemorrhagic hypovolemic vs. burn vs. anaphylactic vs. septic vs. neurogenic vs. cardiogenic?
Hemorrhagic hypovolemic: low blood pressure, cool/pale/clammy skin, increased HR
Burn: increased HR, cool/pale/clammy skin, (similar to hypovolemic)
Anaphylactic: altered mentation, low blood pressure, airway concerns, swelling, angioedema (lips, tongue, epiglottis, bronchioles swelling)
Septic: low blood pressure
Neurogenic: loss of consciousness, low blood pressure, lowered HR
Cardiogenic: rapid HR, low BP, pale/cool/clammy skin
- What are the 2 stages of shock? Signs/symptoms of each?
Compensatory: pale/cool/clammy skin, elevated HR and RR, slightly dilated pupils, narrow pulse pressure, anxious/restlessness, normal SBP
Decompensatory: BP dropping, altered mentation/unresponsive
IMPORTANT: the two s/s that indicate a shift from compensatory to decompensatory is altered mentation and decline in SBP
- What 2 body systems play the biggest roles in the body’s response to shock? What does each do?
The nervous and endocrine system
Nervous: Sympathetic nerve stimulation; vasoconstriction
Endocrine: Releases Hormones; norepinephrine and epinephrine stimulate alpha-1 receptors on arterioles that cause vasoconstriction and regulate blood pressure
- What are EMT treatments for shock?
- O2
- Stop any external bleeding
- Splint any fractures
- Supine position
- Blanket
6.Rapid Transport
- What is the formula for determining the lower limit of the SBP for a child less than 10 yoa?
Low SBP = 70 + 2(age in years)
- How does IV fluid replacement help hemorrhagic shock? What are its limitations?
IV fluid is a volume expander -> it takes the place of missing blood in vessels. The limitations are that it can’t carry oxygen to cells.
- How do geriatric patients differ in their response to shock?
Geriatric patients have a slower compensatory period, but a faster decompensatory period
- How do pediatric patients differ in their response to shock?
Pediatric patients have a faster everything (compensatory period, decompensatory period)
What is meant by “mechanism of injury” and “index of suspicion”?
Mechanism of injury - Is the force or forces that cause injury when applied to the human body.
Index of suspicion - is defined in medicine as the level of suspicion that a disease or condition is the underlying diagnosis based on the available findings in the patient.
What is the most significant factor in the transference of kinetic energy?
Rapid acceleration and deceleration
How many collisions are there in a typical MVC? What occurs with each one?
First, the vehicle is suddenly stopped and gets bent out of shape. This is called the vehicle collision.
Next, the patient comes to a quick stop on some part or parts of the inside of the vehicle, such as the steering wheel, causing injury to the chest. This is called the body collision.
Finally, there is the organ collision in which the patient’s internal organs, which are all suspended in their places by tissue, come to a quick stop, sometimes striking an inside surface of the body (e.g., the inner chest wall or the inner skull).
What are 2 pathways that might result from a frontal collision? What injuries are associated with each?
Abdomen: As abdomen strikes the dashboard or steering wheel, the liver, spleen, and hollow organs of the abdomen are compressed between the front and back abdominal walls and spine. The hollow organs are more easily displaced, leaving the solid liver and spleen to bear the brunt of the compression.
Chest: As the chest hosts the dashboard or the steering wheel, bones and soft tissues are both affected. The ribs and sternum can break, and the cartilage connecting the ribs to the sternum may separate. A torn intercostal artery can bleed 50 mL per minute into the chest cavity with no blood seen externally.
What injury patterns might you expect with rear and lateral impact MVCs? With rollover?
Rear:
Whiplash (injury to the neck and spine) (Hyperextension: head is pushed back all the way or Hyperflexion: Head is pushed forward, nearly touches your dashboard) which can shift the brain vertebrate and can potentially break the neck and spinal chord (if there is no headrest).
Coup-contrecoup injury (Anterior and posterior bruise on the brain. Happens when head hits headrest so fast; the brain pushes against occipital and then the frontal bone, causing the bruises)
Lateral:
Can be deadly because of the extreme impact on the side of the car (less protection). Spinal injury, broken bones, blunt-force trauma. Neck, head, chest, pelvis injuries
Rollover:
Many injuries, Both head on and lateral injury patterns, Multiple system injuries, Ejection (death 25x more likely), Crushing injuries, injuries to head, neck, spine, face, eye, arms from airbags
In a vehicle vs. pedestrian collision, what injury differences might you expect between child vs. adult?
Adults turn away from vehicle (side impact, thrown onto hood): leg, back, torso, shoulders, arms, abdomen, head, and neck injuries
Children turn toward vehicle (front impact and are so small they’re thrown forward and ran over) and tend to have Waddell’s Triad: femur, chest/abdomen head injuries
The extent of injury pedestrians experience depends on how fast the car is going, what part of the pedestrian’s body was hit, how far they were thrown, what surface they landed on, and the body part that first hit the ground
What is the proper position for a car seat?
Secured rear facing in the back seat
Reclined 45 degree angle
What injuries might you expect from seatbelts? From airbags?
Head, neck, spine, face, eye, arms
What are some limitations of airbags?
Airbags only work once.
The driver may still hit steering wheel after deflation.
Front airbags don’t protect from side impact.
- What are 3 questions you need to ask regarding any fall?
Distance, surface, and body part impacted first