Trauma in pregnancy and intimate partner violence Flashcards
Best initial treatment for the foetus
Resuscitation of the mother
Specific risks during pregnancy trauma
Amniotic fluid entering maternal intravascular space
Foetal risk from maternal pelvic fracture
Placental abruption due to lack of placental elasticity
Placental vasculature sensitivity to decrease in maternal intravascular volume
Increased blood components during pregnancy
Plasma volume
RBC volume
WCC
Fibrinogen and clotting factors
Arterial pH
PaO2
Gestation at which plasma volume plateaus
34 weeks
Decreased blood components during pregnancy
Haematocrit (physiological anaemia of pregnancy)
PT and APTT times
(but bleeding and clotting times unchanged)
Bicarbonate
PaCO2
Arterial pH range during pregnancy
vs non pregnant patients
7.40 - 7.45
(vs 7.35 - 7.45 in non pregnant pts)
Bicarbonate range during pregnancy
vs non pregnant patients
17 - 22
(vs 22 - 28 in non pregnant pts)
Changes to cardiac output during pregnancy
Increased due to increased plasma volume
Effect on cardiac output of laying supine in pregnancy
Reduced CO by up to 30% due to compression of vena cava
Changes to HR during pregnancy
Increases by up to 15 bpm over baseline by third trimester
Changes to ECG during pregnancy
Left axis deviation
TWI III and AVF
Ectopic beats
Changes to BP during pregnancy
Decreased in second trimester
Returns to near normal at term
Causes of reduced PaCO2 in pregnancy
Increased minute ventilation
Implication of upper limit of normal PaCO2 in pregnancy
Impending respiratory failure
Changes to GFR and renal blood flow during pregnancy
Increase
Changes to serum creatinine and urea during pregnancy
Decreased to around half of pre-pregnancy levels
Changes to MSK system during pregnancy
Widening of pubic symphysis and sacroiliac joints