Trauma & Head Injury Flashcards
What is the GCS?
Eye
4) Spontaneous
3) Speech
2) Pain
1) None
Verbal
5) Oriented
4) Confused
3) Inappropriate
2) Incomprehensible
1) None
Motor
6) Follows Commands
5) Localizes Pain (pulls on source of pain)
4) Withdraws to pain (just pulls away, does not go after pain source)
3) Decorticate Flexion
2) Decerebrate Extension
1) No response
What are the GCS mild, mod, severe?
8 or less for 6+ hours is severe
9-12 is moderate
Mild is 13- 15
How is C-Spine cleared?
1) no cervical pain or tenderness
2) no paresthesia or neurological deficits
3) normal mental status
4) no distracting pain
5) >4 years of age
if you can’t meet above then
- cross-table lateral c-spine film C1 - T1
- open mouth odontoid view, thoracolumbar, A/P and lateral plain views required
Once patient is stable enough to tolerate a CT, you can evaluate c-spine then
How do you calculate CPP?
CPP = MAP - ICP
What is ideal CPP for patient with TBI?
- if head injury ideal CCP is unknown
- (normal CPP is 70 - 85)
- early studies suggest 70 - 80
- new evidence that if CPP> 70 then ARDS
- Cerebral ischemia if CPP < 50 - 60
- somewhere in middle CPP 60 - 70 reasonable
What MAP range allows cerebral autoregulation?
Cerebral autoregulation maintains CBF (cerebral blood flow) at constant rate with MAP between 60 - 150
How can you decrease ICP?
- no venous obstruction with draining blood from brain (check C-collar)
- elevate head 15 - 30 deg
- mannitol (decreases by osmotically shifting blood from brain to AVC, decreases CSF production)
- lasix
- barbiturate (reduces ICP 2/2 cerebral vasoconstriction and CMRO2)
- hyperventilation ETCO2 25 - 30 (has risk of cerebral ischemia). Used as last resort bc patients in first 24hrs after brain injury already have reduced cerebral blood flow. After 1-2 days the bicarb levels in CSF adjust to compensate for PaCo2 change so no longer effective.
How much does CMRO2 decrease per degree?
1 degree drops the CMRO2 by 7% (below 36)
What are normal values for
- Cardiac Index
- PCWP
- PA pressure
- Mixed venous oxygen sat
- Cardiac Index = 2.6 - 4.2
- PCWP = 2 - 15 mmHg
- PA pressure = 15-30 / 4- 12 mmHg
- Mixed venous oxygen sat = 65 - 75%
How do you treat fat embolism?
- 100% Oxygen
- treat hypotension
- correct hypovolemia
- replace blood and platelets
- c/w mechanical vent
- monitor, notify surgeon (may switch from IM nailing to external fixation of femur)
What is pathophysiology of ARDS
pulm manifestation to SIRS injury to cap / alveolar membrane b/l diffuse infilatrates severe dyspnea hypoxemia (2/2 intrapulm shunting) ARDS can cause fibrosis alveolitis and permanent lung scarring
What is the ARDS severity by the Berlin Definition?
PaO2 / FIO2
- 300 to 200 mild
- 200 to 100 mod
- under or equal to 100
During the case the BP drops to 95/60 and PIP increases mid 40s, O2 sat drops to 80s. What’s differential
- ETT to R mail stem
- expanding tension PNX
- cardiac tamponade
- fat emboli 2/2 fractured femur
- allergic reaction
- aspiration pneumonitis (increased PVR 2/2 hypoxic pulm vasoconstriction causing low BP)
Patient had TBI, on day 5 serum sodium is 129, high urine sodium. What’s the diagnosis?
High urine sodium and hyponatremia is consistent with cerebral salt wasting syndrome (CSWS) and SIADH
CSWS has hypovolemia, normal ADH, concentrates urine Na above 100
SIADH is euvolemic, high ADH, rare if urine Na is over 100
MACE scoring system
Need 2 or more
- IDDM
- ischemic heart dz
- CAD h/o heart failure
- h/o CVD
- CKD
- Supra-inguinal vascular, intraperitonal or intrathoracic surgery