Trauma/Haemorrhage Flashcards
Cardiac output
L/min Stroke volume (ml/beat) x heart rate (b/min)
Preload
Volume of venous return to left and right sides of heart
Determined by:
Venous capacitance
Volume status
Pressure difference between mean venous pressure and mean atrial pressure
Stroke volume
Preload
Myocardial contractility
Afterload
Afterload
Peripheral vascular resistance
Frank-Starling Law
Stroke volume of heart increases in response to increased volume of blood in ventricles.
Increased volume into ventricle stretches cardiac muscle, leading to increased force of contraction.
Types of shock
Hypovolaemic
Cardiogenic
Obstructive
Distributive (Neurogenic)
Hypovolaemic shock
Haemorrhage (traumatic, non-traumatic) Fluid loss (Gi losses, Burns, diuresis, pancreatitis)
Classes of haemorrhage
Class I
Class II
Class III
Class IV
Class I
Blood loss: <750ml (15%) BP: Normal HR: Normal RR: Normal GCS: Normal Urine: >30ml/hr
Class II
Blood loss: 750-1500 (15-30%) BP: normal (reduced pulse pressure) HR: 100-120 RR: 20-30 GCS: mild anxiety Urine: 20-30ml/hr
Class III
Blood loss: 1500-2000ml (30-40%) BP: Systolic <100 HR: 120-140 RR: 30-40 GCS: confused Urine: 20ml/hr
Class IV
Blood loss: >2000ml (>40%) BP: Systolic <70 HR: >140 RR: >40 GCS: reduced GCS Urine: Negligible
Cardiogenic shock
Impaired contractility (MI, Myocarditis, myocardial contusion) Dysrhythmia (tachycardias, bradycardias) Valvular diysfunction (severe aortic regurgitation/stenosis) Left ventricular outflow tract obstruction (HOCM, left atrial myxoma)
Obstructive shock
Massive PE Atrial thrombus/myxoma Emboli (e.g. air embolus) Cardiac tamponade Abdominal compartment syndrome Tension pneumothorax Dynamic hyperinflation (severe asthma) Caval compression (supine hypotension syndrome in pregnancy)
Distributive Shock
Sepsis Anaphylaxis Neurogenic shock Liver failure Adrenal insufficiency