Tissue Integrity: Burns Flashcards

1
Q

Shock

A

Widespread abnormal cellular metabolism that occurs when oxygenation and tissue perfusion needs are not met to the level necessary to maintain cell function

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2
Q

Cardiovascular manifestations of shock

A

decreased cardiac output, increased pulse rate, thread pulse, decreased blood pressure, narrowed pulse pressure, postural hypotension, low CVP, flat neck and hand veins in dependent positions, slow capillary refill, diminished peripheral pulses

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3
Q

Respiratory manifestations of shock

A

increased RR, shallow depth of respirations, increased PaCO2, decreased PaO2, cyanosis

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4
Q

Early neuromuscular manifestations of shock

A

anxiety, restlessness, increased thirst

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5
Q

Late neuromuscular manifestations of shock

A

decreased central nervous system activity (lethargy to coma), generalized muscle weakness, diminished or absent deep tendon reflexes, sluggish pupillary response to light

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6
Q

Kidney manifestations of shock

A

decreased urine output, increased specific gravity, sugar and acetone present in urine

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7
Q

Integumentary manifestations of shock

A

cool to cold, pale to mottled to cyanotic, moist, clammy, mouth dry; pastelike coating present

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8
Q

Gastrointestinal manifestations of shock

A

decreased motility, diminished or absent bowel sounds, nausea and vomiting, constipation

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9
Q

Hypovolemic shock

A

occurs when too little circulating blood volume causes a MAP decrease, resulting in inadequate total body oxygenation

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10
Q

Distributive shock

A

occurs when blood volume is not lost from the body but it is distributed to the interstitial tissues where it cannot circulate and deliver oxygen

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11
Q

Capillary leak syndrome

A

response of the capillaries to the presence of biologic mediator that change blood vessel integrity and allow fluid to shift from the blood vessels into the interstitial tissues where they cannot deliver oxygen or remove tissue waste products

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12
Q

Hypovolemic shock results in

A

a decreased MAP and loss of oxygen-carrying capacity from the loss of circulating RBCs; slows blood flow resulting in decreased tissue perfusion; decreases the ability for the blood to oxygenate the tissue it reaches

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13
Q

Continued MAP decrease in hypovolemic shock

A

some tissues function under anaerobic conditions but lactic acid levels and other harmful metabolites increase; causes electrolyte and acid-base imbalances; reversible if the cause of shock is corrected within 1-2 hrs after onset

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14
Q

Hypovolemic shock adaptive responses: initial stage

A

decrease in baseline MAP of 5-10 mm Hg; increased sympathetic stimulation: mild vasoconstriction, increased heart rate

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15
Q

Hypovolemic shock adaptive responses: nonprogressive stage

A

decrease in baseline MAP of 10-15 mm Hg; continued sympathetic stimulation: moderate vasoconstriction, increased heart rate, decreased pulse pressure; chemical compensation: renin, aldosterone, and antidiuretic hormone secretion result in increased vasoconstriction, decreased urine output, and stimulation of the thirst reflex; some anaerobic metabolism in nonvital organs: mild acidosis, mild hyperkalemia

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16
Q

Hypovolemic shock adaptive responses: progressive stage

A

decrease in baseline MAP of >20 mm Hg; anoxia of nonvital organs; hypoxia of vital organs; overall metabolism is anaerobic: moderate acidosis, moderate hyperkalemia, tissue ischemia

17
Q

Hypovolemic shock adaptive responses: refractory stage

A

severe tissue hypoxia with ischemia and necrosis; release of myocardial depressant factor from the pancreas; buildup of toxic metabolites; MODS; death

18
Q

Nursing Safety Priority

A

RN rather than LPN or UAP should assess the VS of pt who is at risk for or suspected of having hypovolemic shock

19
Q

Nursing Safety Priority

A

because changes in systolic BP are not always present in the initial stage of shock, use changes in pulse rate and quality as main indicators of shock presence or progression

20
Q

Hypovolemic shock: pH

A

Normal 7.35-7.45; will be decreased: insufficient tissue oxygenation causing anaerobic metabolism and acidosis

21
Q

Hypovolemic shock: PaO2

A

Normal 80-100; will be decreased: anaerobic metabolism

22
Q

Hypovolemic shock: PaCO2

A

Normal 35-45; will be increased: anaerobic metabolism

23
Q

Hypovolemic shock: lactic acid

A

normal 3-7 or 0.3-0.8; will be increased: anaerobic metabolism with buildup of metabolites

24
Q

Hypovolemic shock: hematocrit

A

normal 37-52: will be increased: fluid shift, dehydration; or decreased: hemorrhage

25
Q

Hypovolemic shock: hemoglobin

A

normal 12-18: will be increased: fluid shift, dehydration; or decreased: hemorrhage

26
Q

Hypovolemic shock: potassium

A

normal 3.5-5.0: will be increased: dehydration, acidosis

27
Q

Hypovolemic shock: best practice

A

ensure patent airway; start IV or maintain established IV; administer oxygen; elevate pts feet; head no higher than 30-degree angle; examine pt for overt bleeding; if overt bleeding present, apply direct pressure to site; administer drugs as prescribed; increase IV fluid rate of delivery; do not leave pt

28
Q

Hypovolemic shock: IV therapy

A

crystalloids (NS or LR); whole blood to replace large blood losses; PRBCs to replace moderate blood loss; plasma to restore osmotic pressure when Hgb and Hct are WNL

29
Q

Hypovolemic shock: drug therapy - vasoconstrictors

A

dopamine, norepinephrine: stimulate venous return by constricting blood vessels and decreasing venous pooling; increase cardiac output and MAP; improves perfusion and oxygenation

30
Q

Hypovolemic shock: drug therapy - inotropics

A

dobutamine: stimulates adrenergic receptors in the hear; improves heart muscle contractility

31
Q

Hypovolemic shock: drug therapy - myocardial perfusion

A

sodium nitroprusside: ensures heart is well perfused; dilates coronary blood vessels while minimally dilating systemic vessels

32
Q

Hypovolemic shock: monitoring pt

A

pulse, BP, pulse pressure, CVP, RR, skin and mucosal color, O2 sat, mental status, urine output