THR stability and dislocation Flashcards
What is the incidence of THR dislocation in primary, revision hips?
- Primary THR 1-2%
- Revision 5-7%
- highest incidence found in >80 years for failed femoral neck orif converted to THR
What are the 4 important variables that determine stability of THR?
- Component design
- Component alignement
- Soft tissue tensioning
- Soft tissue function
How does component design affect stability of THR?
- Arc range before impingement -> dislocation
- factors include
-
Head neck ratio ( diameter of head/Diam of neck)
- Most important
- best to maximise
-
reduced by
- skirted femoral heads - early hip impingment-> dislocation
- acetabular hoods- decrease primary arc
- constrained acetabular liners- sig decrease in primary arc
-
Excursion distance
- distance head must travel to dislocate after primary impingement
- 1/2 diameter of femoral head
- larger heads have higher head/neck ratios and larger excursion distance
-
Leaverage range
- measure by excursion distance
-
Head neck ratio ( diameter of head/Diam of neck)
How does component alignement affect stability of THR?
-
Centre of rotation
- THR primary arc is smaller cf THR ( due to smaller prothetic head size)
- so THR arc range centred on antomical arc range
- if not centred -> unstabilty due to leavering
- neck length long enough so GT doesn’t impinge on acetabulum
-
Cup alignement
- avoid retroversion->post dislocation
- avoid anteversion-> ant dislocation
- Vertical cup- post sup dislocaiton
- horizontal cup- inferior dislocation
- Stem alignment
What are the ideal positions for the cup and stem alignement in THR?
- Acetabular cup
- 15-30o anteversion
- 35-45o coronal tilt
- Stem
- 10-15o anteversion
what is key in soft tissue tensioning which affect stability of THR?
-
Abductor complex ( Glut medius and medinimus)
- restoring tension of abdcutors is key to soft tissue tensioning by
- Head offset
-
Neck length
- if short- use long prosthetic neck
- skirt on long neck would decrease primary arc of motion and increase risk of dislocation
- if neck-shaft angle < than native hip then
- excessive increase in offset-> lateral hip pain and trochnateric bursitis
-
normal hip centre of rotation
- prevents blatant creation of unstable zone
-
Trochanteric deficiency escape
- occurs when GT pulls away from prox femur
- due to poor fixation of GT after revision THR/ trauma/osteolysis
- -> deficient abductor complex
- Increase risk of dislocation
- occurs when GT pulls away from prox femur
How is femoral offset decreased?
- weak abductor complex
- increased joint reaction force
- decreased lever arm
- Trendelenberg sign
- Gluteus medius lurch
- increased risk for dislocation
How is trochanteric deficiency/escape tx?
- Maximise femoral /head ratio
- resect GT fragment to prevent impingement/ Levering
- Contrainsed acetabular liner as last resort
How important is soft tissue function to THR stability
- Critical
- require coordinated vector forces ( muscle firing) to stablise THR
- both thry abductor complex and other vector forces that stabilise the hip
- 3 main factors controlling proper soft tissue function
-
CNS
- stroke/cerebellar dysfunction, dementia, parkinsons. MS, myeolpathy , delirum-> affect balance/coordination
-
PNS
- spinal stenosis ( glut medius L5) , peripheral neuropathy, radiculopathy, paralysis/paresis
-
Local soft tissue integrity
- trauma- soft tissue loss
- deconditioning
- age/poor health
- irradiation
- osteolysis
- collagen abnormalities
- myopthy
- malignancy
- infection
-
CNS
What is the epidemiology of hip dislocation?
- 2nd most common cause for revision surgery of THR
incidence 1-3% - 70% occur within first month
- 75-90% posterior
- Mechanism
- anterior= extension, ER
- posterior= Flexion, IR
- Risk factors
- prior hip surgery
- **Female **
- >70-80 years
-
Posterior surgical approach
- repairing capsule and reconstructing ER brings dislocation close to anterior approach
-
Malposition of components
- ideal cup- 10o anterversion, 40o coronal tilt
- Spastic neuromuscular disease ( parkinson’s)
- Inflammatory arthritis
- ETOH/ Drug abuse
- AVN
- Acute fracture
- decreased offset ( decreased tissue tension/stability)
- Decreased femoral head to neck ratio
What is the hx from the pt regarding dislocation?
- Activity that puts hip out- Hip flexion, adduction, IR
- shoe tying
- sitting in low seat or toilet
What is the tx of THR dislocation?
- Non operative
- Closed reduction and immobilisation
- 2/3rd of early dislocation tx with closed reduction adn immobilsation
- Operative
-
Revision THR
- 2 or more dislocation with evidence of
- polyethylene wear
-
malalignment
- vertical acetabular compenent may need revision
- also acetabular retroversion
- Hardware failure
-
Conversion to Hemiarthroplasty with larger femoral head
- for soft tissue deficiency or dysfunction
- CI if acetabular bone compromised
-
Resection arthroplasty
- when all options exhausted
- sig bone loss and soft tissue deficiency
- psychiatric pts
-
Revision THR
What are the aims in revision THR surgery?
- Realign components
- Head enlargement- optimise
-
Trochanteric osteotomy and advancement
- places abductor complex under tension with increases hip compression forces
- Conversion to a constrained acetabular compoenent
- when
- recurrent instability with a well postioned actetabular component due to soft tissue deficiency or dysfunciton
- must have compliant pt otherwise will fail by fx of cup or loosening from the pelvis
What does femoral subsidance have on the function of a THR?
- Femoral stem subsidence effectively decreases the neck length of the prosthesis resulting in a lax abductor complex
- -> increase in the joint reactive force
- This decrease in leg length can also lead to increased hip instability.
- Illustration A shows a free body diagram of the hip joint. The magnitude of the joint reaction force depends critically on the ratio of (d1:d2). As d2 decreases due to less offset, such as in this question, and body weight remains the same, the joint reaction forces increase.